| VCPI | Virtual Control Program Interface |
|---|---|
| VE | vaginal examination; Venezuelan encephalitis; venous emptying; venous extension; ventilation; ventil... |
| VEUD | virtual emergency and urgency department |
| VFID | virtual focus-isocenter-distance |
| VH | vaginal hysterectomy; venous hematocrit; ventricular hypertrophy; veterans hospital; viral hepatitis... |
| VE | Virtual Endoscopy |
|---|---|
| VE | Virtual Environment |
| VR | Virtual Reality |
| VRML | Virtual Reality Modeling Language |
| CESD | Cholesterol ester storage disease |
| virtual focus | The point from which divergent rays seem to proceed, or that at which they would meet if prolonged backward. (05 Mar 2000) |
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| virtual image | <microscopy> Such as seen in a mirror or through a magnifier. A virtual image has no real existence in space as does a real image from a lens. It does have a definite location, however, caused by the angles of divergence of the rays received by the eye. This can be shown by the common school experiment of placing a pin coincident with its mirror image behind a sheet of glass acting as a partial mirror. Its location can also be placed in design by extrapolating backwards to a focus. If a magnifier is used as it should be, with the object at its focus, the virtual image is at infinity. The same is true for a microscope focused for the relaxed eye. See: distance of virtual image. (05 Aug 1998) |
| distance of virtual image | <physics> When a simple lens is used as a magnifier for visual observation the eye becomes part of the optical system. A virtual image can be formulated by construction and its apparent distance will vary with the focus of the eye. This will vary among individuals. In a rather arbitrary but standardised assumption, the normal distance for close observation, or reading has been set at 10 inches (250 mm). The optics for the compound microscope have been designed to furnish parallel light from the eyepiece so that the eyes are relaxed for distant viewing. This makes the virtual image lie at infinity. Tests showed that the average observer accommodates somewhat, placing the virtual image rather variably, often about 20 - 25 feet. (05 Aug 1998) |
| image virtual | <microscopy> A virtual image has no real existence. It is the image seen when looking into a mirror. The field of view of the microscope is a good example of a virtual image. When the eye operates in conjunction with a lens to form an image on the retina, the visual sensation is as if the image existed in space. That its apparent location is very definite is proved when a pin can be made to coincide with the mirror (virtual) image of another pin that is seen by looking at a sheet of glass acting as a mirror. With a lens system a virtual image can be definitely located as by graphically tracing rays back to a focus. In a microscope, if the eye is relaxed as it should be, the virtual image will be at infinity. Measurements show that most observers place the aerial image at 20-25 feet, some as close as seven, because of partial accommodation. (11 Mar 1998) |
| brancher glycogen storage disease | Type of glycogen storage disease, due to deficiency of amylo-1,4-1,6-transglucosidase (brancher enzyme). Synonym: brancher deficiency glycogenosis, debrancher deficiency. (05 Mar 2000) |
| glycogen storage disease | <hepatology> A group of inherited metabolic disorders involving the enzymes responsible for the synthesis and degradation of glycogen. In some patients, prominent liver involvement is presented. In others, more generalised storage of glycogen occurs, sometimes with prominent cardiac involvement. Synonym: glycogenosis (12 Sep 2002) |
| glycogen storage disease type I | <disease> An autosomal recessive disease in which gene expression of glucose-6-phosphatase is absent, resulting in hypoglycaemia due to lack of glucose production. Accumulation of glycogen in liver and kidney leads to organomegaly, particularly massive hepatomegaly. Increased concentrations of lactic acid and hyperlipidemia appear in the plasma. Clinical gout often appears in early childhood. Inheritance: autosomal recessive. (12 Dec 1998) |
| glycogen storage disease type II | <disease> Glycogenosis due to alpha-1,4-glucosidase (acid maltase) deficiency. It affects muscle, heart, and other organs. (12 Dec 1998) |
| glycogen storage disease type III | <disease> An autosomal recessive metabolic disorder due to deficient expression of amylo-1,6-glucosidase (one part of the glycogen debranching enzyme system). The clinical course of the disease is similar to that of glycogen storage disease type I, but milder. Massive hepatomegaly, which is present in young children, diminishes and occasionally disappears with age. Levels of glycogen with short outer branches are elevated in muscle, liver, and erythrocytes. Six subgroups have been identified, with subgroups type IIIa and type IIIb being the most prevalent. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type IV | <disease> An autosomal recessive metabolic disorder due to a deficiency in expression of branching enzyme (alpha-1,4-glucan-6-alpha-glucosyltransferase), resulting in an accumulation of abnormal glycogen with long outer branches. Clinical features are muscle hypotonia and cirrhosis. Death from liver disease usually occurs before age 2. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type V | <disease> Glycogenosis due to muscle phosphorylase deficiency. Characterised by painful cramps following sustained exercise. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type VI | <disease> A hepatic glycogen storage disease in which there is an apparent deficiency of hepatic phosphorylase activity. However, studies have not been able to distinguish between phosphorylase deficiency and phosphorylase kinase deficiency in patients with hepatic glycogenosis. (12 Dec 1998) |
| glycogen storage disease type VII | <disease> An autosomal recessive muscle glycogen storage disease in which there is deficient expression of muscle phosphofructokinase activity, resulting in increased concentrations of glucose-6-phosphate and fructose-6-phosphate and low concentrations of fructose-1,6-diphosphate in muscle tissue. Glycogen storage in muscle is increased, perhaps due to activation of glycogen synthase by accumulated glucose-6-phosphate. It has been proposed that shunting of glucose-6-phosphate and fructose-6-phosphate into the pentose phosphate pathway may result in increased synthesis of purines and pyrimidines, causing hyperuricaemia and gout. Erythrocytes from patients may show decreased phosphofructokinase activity and 2,3-diphosphoglycerate deficiency. Exercise intolerance is present and severe congenital muscular dystrophy has been reported. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type VIII | <disease> An x-linked recessive hepatic glycogen storage disease resulting from lack of expression of phosphorylase-b-kinase activity. Symptoms are relatively mild; hepatomegaly, increased liver glycogen, and decreased leukocyte phosphorylase are present. Liver shrinkage occurs in response to glucagon. Inheritance: X-linked recessive (12 Dec 1998) |
| cholesterol ester storage disease | A rare benign adult form of inherited lysosomal lipid storage disease that is due to deficiency of acid lipase. It results in an accumulation of neutral lipids, particularly cholesterol esters, within cells (particularly leukocytes, fibroblasts, and liver cells). It is an allelic variant of wolman disease. (12 Dec 1998) |
| virtual storage | (computer science) memory created by using the hard disk to simulate additional random-access memory |
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제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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