| ¿µ¹® | protein | ÇÑ±Û | ´Ü¹éÁú |
|---|---|---|---|
| ¼³¸í | ź¼Ò, ¼ö¼Ò, »ê¼Ò, Áú¼Ò, ȲÀ» ÇÔÀ¯Çϰí ÀÖ´Â À¯±âÈÇÕ¹°·Î, ¸ðµç ¼¼Æ÷ÀÇ ¿øÇüÁúÀ» ÀÌ·ç°í ÀÖ´Â ±âº» ±¸¼º¹°ÁúÀÌ´Ù. ´Ü¹éÁúÀº ±× ´ÜÀ§ÀÎ ¾Æ¹Ì³ë»êµéÀÌ ÆéƼµå°áÇÕ¿¡ ÀÇÇØ °áÇյǾî ÀÖÀ¸¸ç, º¸Åë 20°³ÀÇ ¾Æ¹Ì³ë»êµéÀÌ ´Ù¸¥ ¼ø¼¿Í Á¶¼ºÀ» °¡Áö°í ¹è¿µÇ¾î, µ¶Æ¯ÇÑ ÇϳªÀÇ ´Ü¹éÁúÀ» Çü¼ºÇÏ°Ô µÈ´Ù. |
||
| ECG | Electro-Cardio-Graphy(-Gram); ½ÉÀüµµ = EKG 1. Conducting System Structu... |
|---|---|
| WPW Syndrome | Wolff-Parkinson-White Syndrome ? CIx 1. Drugs; AV Conduct... |
| UA | absorption unsharpness; ultra-audible; ultrasonic arteriography; umbilical artery; unauthorized abse... |
| UAP | unlicensed assistive personnel; unstable angina pectoris; urinary acid phosphatase; urinary alkaline... |
| UHD | unstable hemoglobin disease |
| UA | Unstable Angina |
|---|---|
| UAP | Unstable Angina Pectoris |
| UCAD | Unstable coronary artery disease |
| G protein | 5'-triphosphate-binding protein |
| G-protein | Guanine nucleotide-binding protein |
| angina, unstable | Precordial pain at rest, which may precede a myocardial infarction. (12 Dec 1998) |
|---|---|
| DNA sequence, unstable | DNA region comprised of a variable number of repetitive, contiguous trinucleotide sequences. Presence of these regions is associated with diseases such as fragile x syndrome and myotonia atrophica. (12 Dec 1998) |
| unstable angina | <cardiology> Angina which is new onset or prior existing angina which is increasing in severity, duration or frequency. (13 Nov 1997) |
| unstable bladder | Characterised by uninhibited detrusor contractions. (05 Mar 2000) |
| unstable cervical spine fractures | <radiology> Flexion: bilateral interfacetal dislocation, flexion teardrop fracture (usually C5 or C6) extension: extension teardrop fracture (usually C2 or C3), hangman's fracture, extension-dislocation, extension-fracture-dislocation, odontoid fracture vertical compression: Jefferson burst fracture see: cervical spine fractures (12 Dec 1998) |
| unstable colloid | A colloid that is not again soluble in water after having been dried at ordinary temperature. Synonym: unstable colloid. (05 Mar 2000) |
| unstable fracture | A fracture with an intrinsic tendency to slip out of place after reduction. (05 Mar 2000) |
| unstable haemoglobin haemolytic anaemia | A congenital haemolytic anaemia, due to autosomal inheritance of one of many unstable haemoglobins. The anaemia is of variable severity and characterised by the presence in vivo or in vitro of Heinz bodies. (05 Mar 2000) |
| unstable haemoglobins | A group of rare Hb's with amino acid substitutions (or amino acid deletions in three types) that alter the three-dimensional shape of the globin in a manner that renders the molecule unstable; they have an increased but variable tendency to auto-oxidation and Heinz body formation and are associated with congenital nonspherocytic haemolytic anaemia. The unstable b chain abnormalities include Hb's Genova, Gun Hill, Hammersmith, Koln, Philly, Sabine, Santa Ana, Sydney, Wien, and Zurich; unstable a chain abnormalities include Hb's Bibba, Sinai, and Torino. (05 Mar 2000) |
| unstable mutation | <molecular biology> A mutation that has a high likelihood of reverting to its original form. (13 Nov 1997) |
| unstable patient | The distinction of stability is made by a physician and based on a large number of variables: patient diagnosis, vital signs, physical findings, laboratory findings, subjective factors and patient prognosis. (27 Sep 1997) |
| acetoacetyl-acyl carrier protein synthase | <enzyme> E coli enzyme, that catalyses condensation of malonyl-acyl carrier protein plus acetyl-acyl carrier protein; not inhibited by cerulenin Registry number: EC 2.3.1.- Synonym: acetoacetyl-acp synthase (26 Jun 1999) |
| acid soluble spore protein | <molecular biology> A DNA binding protein in the spores of some bacteria, thought to stabilise the DNA in an A configuration, so protecting it from cleavage by enzymes or UV light. (18 Nov 1997) |
| acute-phase protein | <haematology> These plasma proteins (in addition to fibrinogen) increase 25% or more in response to inflammation and injury are under direct control of interleukin-6 (IL-6) (hepatocyte-stimulating factor). Other proteins which increase are ceruloplasmin, C3 and C4 which increase 50% or more; alpha-1 acid glycoprotein, alpha-1 antitrypsin, haptoglobin and fibrinogen (the major determinant of viscosity 1 ) which increase two- to fourfold; C-reactive protein (CRP) and serum amyloid A which increase several hundred-fold. Despite long-held clinical opinion to the contrary, available data indicate that neither ESR nor measurement of specific acute-phase reactants are useful in excluding underlying infection or inflammation regardless of the pretest probability. These proteins are secreted into the blood in increased or decreased quantities by hepatocytes in response to trauma, inflammation, or disease. They can serve as inhibitors or mediators of the inflammatory processes. Certain acute-phase proteins have been used to diagnose and follow the course of diseases or as tumour markers. See also: amyloid, c-reactive protein, erythrocyte sedimentation rate, viscosity. (25 Jun 1999) |
| acyl-(acyl-carrier-protein)-phospholipid acyltransferase | <enzyme> Catalyses the formation of phosphatidylethanolamine from acyl-acyl carrier protein and 2-acyl-sn-glycero-3-phosphoethanolamine Registry number: EC 2.3.1.40 Synonym: 2-acyl-gpe acyltransferase, 2-acylglycerophosphoethanolamine acyltransferase (26 Jun 1999) |
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