| 영문 | iron deficiency anemia | 한글 | 철결핍빈혈 |
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| 설명 | 적혈구의 기능은 산소를 운반하는데 있다. 적혈구 속에 산소와 결합을 하여 산소를 운반하는 혈색소라는 물질이 있다. 철은 이 혈색소의 중요한 부분을 이루는 것으로 철이 없으면 혈색소가 만들어질 수가 없다. 혈색소가 없으면 역시 적혈구도 만들어지지 않으므로 체내에 철이 부족하면 빈혈이 생긴다. 이 철결핍성 빈혈은 빈혈의 원인 중에서 가장 흔한 것이다(약 25%를 차지한다). 철저장량의 저하-결핍, 혈청철농도의 저하, 트란스페린량 상승, 트란스페린포화도의 저하, 혈색소농도 또는 헤마토크리트의 저하, 저색소성대적혈구를 특징으로 하는 빈혈로서, 생체 내에서 철이 장기에 걸쳐 결핍되며 그 때문에 혈색소 생산 감소에 의해 일어난다. 창자에서의 철흡수량 부족, 철의 수요 증대(유아기, 사춘기, 임신), 철소실과잉(출혈)에 의해 일어나며, 특히 사춘기에서 폐경기까지의 여성에게 많다. 증상으로서는 얼굴창백, 피로감, 피부창백, 손톱 변화(스푼 모양) 등을 나타낸다. 구강 영역에서는 혀의 접촉통, 발적, 건조감, 삼킴곤란을 수반하면 플러머-빈슨(Plummer-Vinson)증후군이라고 한다. 혈액 소견은 혈청철은 저하하며, 철결합능력의 상승, 저색소성 작은적혈구성을 나타낸다. |
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| MD | Doctor of Medicine [Lat. Medicinae Doctor]; magnesium deficiency; main duct; maintenance dose; major... |
|---|---|
| PAO | peak acid output; peripheral airway obstruction; plasma amine oxidase; polyamine oxidase; pulmonary ... |
| BS | Bachelor of Science; Bachelor of Surgery; Bacillus subtilis; Bartter syndrome; base strap; bedside; ... |
| BSA | benzenesulfonic acid; Biofeedback Society of America; bismuth-sulfite agar; bis-trimethylsilyl-aceta... |
| SST | sodium sulfite titration; somatostatin |
| ATD | 1-antitrypsin deficiency |
|---|---|
| AMD | Acid maltase deficiency |
| AIDS | Acquire Immune Deficiency Syndrome |
| AIDS | Acquired Immune Deficiency Disease Syndrome |
| alpha1ATD | Alpha-1-antitrypsin deficiency |
Nadsonieae
| calcium sulfite | Used as an intestinal antiseptic, and locally in the treatment of parasitic skin diseases. (05 Mar 2000) |
|---|---|
| sodium hydrogen sulfite | NaHSO3;acid sodium sulfite, used in gastric and intestinal fermentation, externally in the treatment of parasitic diseases, and as an antioxidant in certain injections (s. Metabisulfite). Synonym: sodium hydrogen sulfite, sodium pyrosulfite. (05 Mar 2000) |
| sodium sulfite | Na2SO3-7H2O;has been used for the relief of intestinal fermentation, and externally for aphthous stomatitis. (05 Mar 2000) |
| sulfite | A salt of sulfurous acid; elevated in cases of molybdenum cofactor deficiency. Sulfite dehydrogenase, an oxidoreductase catalyzing the reaction of sulfite with 2 ferricytochrome c and water to sulfate and 2-ferrocytochrome c. Sulfite oxidase, a liver oxidoreductase (haemoprotein) catalyzing the reaction of inorganic sulfite ion with O2 and water to produce sulfate ion and H2O2; a lower activity of this enzyme is observed in cases of molybdenum cofactor deficiency. Sulfite reductase, oxidoreductase catalyzing reduction of sulfite to H2S using some reduced acceptor. (05 Mar 2000) |
| sulfite oxidases | <enzyme> A molybdohemoprotein which catalyses the terminal reaction in the oxidative degradation of sulfur-containing amino acids with the formation of a sulfate. Cytochrome c, ferricyanide and molecular oxygen can act as acceptors. A deficiency is manifested by brain damage and mental deterioration. Utilises ferricytochrome c as acceptor. Uses molecular oxygen. Registry number: EC 1.8.3.1 (12 Dec 1998) |
| sulfite reductases | <enzyme> Hydrogen sulfide:(acceptor) oxidoreductases. Enzymes which reversibly catalyze the oxidation of hydrogen sulfide in the presence of various acceptors to sulfite and a reduced acceptor. Utilises NADP+ as the acceptor. Utilises oxidised ferredoxin as acceptor and EC 1.8.99.1 will utilise a variety of acceptors. Registry number: EC 1.8.- (12 Dec 1998) |
| exsiccated sodium sulfite | Anhydrous sodium sulfite, used as a preservative in pharmaceutical preparations. (05 Mar 2000) |
| abdominal muscle deficiency syndrome | <syndrome> Congenital absence (partial or complete) of abdominal muscles, in which the outline of the intestines is visible through the protruding abdominal wall; in males, genitourinary anomalies (urinary tract dilation and cryptorchidism) are also found; genetics unclear. (05 Mar 2000) |
| adult lactase deficiency | Onset of lactase deficiency, with resulting milk intolerance and malabsorption, in adulthood. Inherited forms may not be manifested until adulthood; any process that damages the intestinal lining cells can cause lactase deficiency in adults. (05 Mar 2000) |
| alpha-1 antitrypsin deficiency | <chest medicine> Deficiency of the protease inhibitor alpha-1 antitrypsin, leads primarily to degradation of elastin of the alveolar walls, as well as other structural proteins of a variety of tissues. The lack of this protein leads to damage of various organs, but mainly to the lung and liver. symptoms may become apparent at a very early age or in adulthood, manifesting either as shortness of breath or liver related symptoms (jaundice, fatigue, fluid in the abdomen, mental changes, or gastrointestinal bleeding). There are several options for treatment of the lung disease, including replacement of the missing protein. Treatment of the liver disease is a well-timed liver transplant (12 Dec 1998) |
| alpha-1-proteinase deficiency | Absence of a serum proteinase inhibitor that may cause nodular non-suppurative panniculitis. (05 Mar 2000) |
| alpha-antitrypsin deficiency | <enzyme> A specific enzyme (alpha 1 antitrypsinase) that when absent genetically can result in panacinar emphysema (lung disease) and liver disease. There is no specific treatment for this condition other than supportive care for the liver and lung complications. Medications such as alpha-1proteinase inhibitor is given regularly to these patients. Incidence: approximately 1 in 10,000. (02 Jan 1998) |
| anaemia, iron deficiency | Deficiency of iron results in anaemia because iron is necessary to make haemoglobin, the key molecule in red blood cells responsible for the transport of oxygen. In iron deficiency anaemia, the red cells are unusally small (microcytic) and pale (hypochromic). Characteristic features of iron deficiency anaemia in children include failure to thrive (grow) and increased infections. The treatment of iron deficiency anaemia, whether it be in children or adults, is with iron and iron-containing foods. Food sources of iron include meat, poultry, eggs, vegetables and cereals (especially those fortified with iron). According to the National Academy of Sciences, the Recommended Dietary Allowances of iron are 15 milligrams per day for women and 10 milligrams per day for men. Anaemia characterised by low or absent iron stores, low serum iron concentration, elevated free erythrocyte porphorin, low transferrin saturation, elevated transferrin, low serum ferritin, low haemoglobin concentration or haematocrit, and hypochromic microcytic red blood cells. Symptoms may include pallor, angular stomatitis and other oral lesions, gastrointestinal complaints, retinal haemorrhages and exudates, and thinning and brittleness of the nails. Among the causes of iron-deficiency anaemia are inadequate iron intake, impaired iron absorption, increased blood loss and increased requirements such as infancy, pregnancy, and lactation. (12 Dec 1998) |
| antibody deficiency disease | <syndrome> Any of a group of disorders associated with a defective antibody production due to defects in the B-type lymphocyte system or in T-type lymphocytes; chief manifestation is an increased susceptibility to infection by various microorganisms. See: agammaglobulinaemia, hypogammaglobulinaemia, immunodeficiency. Synonym: antibody deficiency disease. (05 Mar 2000) |
| antibody deficiency syndrome | <syndrome> Any of a group of disorders associated with a defective antibody production due to defects in the B-type lymphocyte system or in T-type lymphocytes; chief manifestation is an increased susceptibility to infection by various microorganisms. See: agammaglobulinaemia, hypogammaglobulinaemia, immunodeficiency. Synonym: antibody deficiency disease. (05 Mar 2000) |
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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