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| SMI | Self-Motivation Inventory; senior medical investigator; severe mental impairment; silent myocardial ... |
|---|---|
| SP | sacroposterior; sacrum to pubis; salivary progesterone; schizotypal personality; semi-private [room]... |
| ER | efficiency ratio; epigastric region; ejection rate; electroresection; emergency room; endoplasmic re... |
| RAR | rapidly adapting receptor; rat insulin receptor; retinoic acid receptor; right arm reclining; right ... |
| CR | calculation rate; calculus removed; calorie-restricted; cardiac rehabilitation; cardiac resuscitatio... |
| SMI | Silent myocardial ischemia |
|---|---|
| SIR | Silent Information Regulator |
| SP | Silent Period |
| SCI | Silent cerebral infarction |
| SI | Silent myocardial ischaemia |
| masticatory silent period | A pause in electromyographic patterns associated with tooth contacts during chewing and biting; a part of the complex feedback mechanism of mandibular control involving receptors in the periodontal ligament and muscles. (05 Mar 2000) |
|---|---|
| silent | Producing no detectable signs or symptoms, said of certain diseases or morbid processes. (05 Mar 2000) |
| silent allele | 1. <genetics> A gene which is inactive. Thus, an amorphic gene. 2. <cell biology> Something that lacks a discernible shape and thus can be describes as amorphous. (05 Feb 1998) |
| silent area | Any area of the cerebrum or cerebellum in which lesions cause no definite sensory or motor symptoms. (05 Mar 2000) |
| silent electrode | In unipolar electrocardiography, a remote electrode placed either upon a single limb or connected with the central terminal and paired with an exploring electrode; the indifferent electrode is supposed to contribute little or nothing to the resulting record. Synonym: dispersing electrode, silent electrode. (05 Mar 2000) |
| silent gap | The period during which Korotkoff sounds indicating true systolic pressure fade away and reappear at a lower pressure point; responsible for errors made in recording falsely low systolic blood pressure, especially in hypertensive patients, of up to 25 mm Hg, and avoided by pumping the cuff 30 mm Hg beyond palpable systolic pressure. Synonym: silent gap. (05 Mar 2000) |
| silent ischemia | Myocardial ischemia without accompanying signs or symptoms of angina pectoris; can be detected by EKG and other lab techniques. See: silent myocardial infarction. (05 Mar 2000) |
| silent mutant | A mutant that is not phenotypically manifest. Synonym: silent mutant. (05 Mar 2000) |
| silent mutation | Mutations that have no effect on phenotype because they do not affect the activity of the product of the gene, usually because of codon ambiguity. (18 Nov 1997) |
| silent myocardial infarction | Infarction that produces none of the characteristic symptoms and signs of myocardial infarction. (05 Mar 2000) |
| silent period | The time during which there is no electrical activity in a muscle following its rapid unloading, any pause in an otherwise continuous series of electrophysiologic events. (05 Mar 2000) |
| acetylcholine receptor antibodies | <neurology, investigation> A test used to measure the amount of antibodies to acetylcholine receptors on nerve endings. This is a diagnostic test for myasthenia gravis. A normal value is no antibodies in the bloodstream. Acetylcholine receptor (AChR) binding autoantibodies (i.e. Antibodies reactive with several epitopes other than the binding site for acetylcholine or alpha-bungarotoxin) are present in approximately 88% of patients with generalised myasthenia gravis, 70% of ocular myasthenia and in approximately 80% of myasthenia gravis in remission. Although serum concentrations of AChR binding autoantibodies do not in general correlate well with severity of weakness, there is typical decrease in concentration as weakness improves with immunosuppressive therapy. AChR blocking autoantibodies (i.e., antibodies reactive with the AChR binding site) are present in about 50% of patients with myasthenia gravis, 30% with ocular myasthenia gravis and 20% of myasthenia gravis in remission, AChR blocking autoantibodies are the only AChR autoantibodies present in about 1% of myasthenia gravis. AChR modulating autoantibodies (i.e., autoantibodies which cross-link AChRs and cause their removal from muscle membrane surfaces) are present in more than 90% of myasthenia gravis and occasionally are the only AchR autoantibodies detectable in mild, recent onset or ocular-restricted myasthenia gravis. Results for AChR modulating autoantibodies can be transiently false-positive due to curare-like drugs used during general anesthesia. AChR autoantibodies of one or more types are found in at least 80% of ocular myasthenia gravis. Although generally absent in neurological conditions other than myasthenia gravis(and consequently unlikely to cause confusion in neurodiagnosis), false-positive results for AChR autoantibodies occasionally occur in primary biliary cirrhosis, tardive dyskinesia, autoimmune thyroiditis, the elderly, amyotrophic lateral sclerosis patients treated with cobra venom and patients with thymoma in the absence of myasthenia gravis. Approximately 1% of patients with rheumatoid arthritis treated with D-penicillamine develop AChR autoantibodies and myasthenia gravis, both of which disappear when the drug is discontinued. Babies born to ~10% of myasthenia gravis mothers have a transient neonatal form of myasthenia gravis that responds well to anticholinesterase therapy and usually remits within 1 month as maternal IgG disappears. (29 Dec 1997) |
| amino acid receptor | <biochemistry> Ligand gated ion channels with specific receptors for amino acid transmitters. An extended protein superfamily that also includes subunits of the nicotinic acetylcholine receptor. (18 Nov 1997) |
| AMPA receptor | <cell biology> Glutamate operated ion channel. See: excitatory amino acid receptor channels. (05 Feb 1998) |
| ANP receptor | <molecular biology> Family of 3 receptors for atrial natriuretic peptide. ANP A and ANP B have intracellular guanylate cyclase and protein kinase like domains. ANP C, shares the extracellular ligand binding and transmembrane domains, but lacks the functional intracellular domains and is not thought to be involved in signal transduction. (18 Nov 1997) |
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