| ¿µ¹® | enzyme-linked immunoabsorbent assay | ÇÑ±Û | È¿¼Ò¸é¿ªÃøÁ¤¹ý |
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| ¼³¸í | È¿¼Ò°áÇո鿪ÈíÂøÁ¦ °ËÁ¤¹ýÀ¸·Î ¹ø¿ªµÇ°í ÀÖ´Ù. ÀÌ ¹ýÀº Ç׿ø(¶Ç´Â Ç×ü)¿¡ ¾ËÄ®¸® Æ÷½ºÆÄŸ¾ÆÁ¦ ¶Ç´Â Æä¸£¿Á½Ãµð¾ÆÁ¦ µîÀÇ »ê¼Ò¸¦ °áÇÕ½ÃÄÑ µÎ°í ±× »ê¼ÒȰ¼ºÀ» ÁöÇ¥·Î »ï¾Æ Ç׿øÇ×ü¹ÝÀÀÀÇ Á¤µµ¸¦ ¾È ´ÙÀ½ ¿©±â¿¡¼ Ç׿ø(¶Ç´Â Ç×ü)ÀÇ ¾çÀ» ±¸ÇÏ´Â °ÍÀÌ´Ù. ÀÌ ¹ýÀÇ ÀÌÁ¡À¸·Î¼ °í°¨µµ, Á¶ÀÛÀÇ °£´ÜÇÔ ¹× ¹æ»ç¼±¸é¿ªÃøÁ¤¹ýó·³ ¹æ»ç¼º¹°ÁúÀ» »ç¿ëÇÏÁö ¾Ê¾Æµµ µÈ´Ù´Â Á¡À» µé ¼ö ÀÖ´Ù. È£¸£¸óÀ̳ª ¸é¿ª±Û·ÎºÒ¸°ÀÇ Á¤·®¹ýÀ¸·Î¼ ÀÀ¿ë µÇ°í ÀÖÀ¸¸ç ÃøÁ¤¿ë ŰƮµµ ½ÃÆÇµÇ°í ÀÌÀÖ´Ù. |
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| ¿µ¹® | sex frigidity | ÇÑ±Û | ºÒ°¨Áõ |
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| ¼³¸í | ¼º¿åÀº ÀÖÀ¸³ª ¼º±³¿¡ µû¸¥ Äè°¨ÀÌ Àû°Å³ª ÀüÇô ´À³¢Áö ¸øÇÏ´Â »óÅÂ. ÀϹÝÀûÀ¸·Î ¿©¼º¿¡ ´ëÇÑ ¿ë¾îÀÌ´Ù. ³ÐÀº Àǹ̷δ ¼º°¨°¨ÅðÁõÀ» ÀǹÌÇϰí, 2´ë ¼º¿åÀÎ Á¢±Ù¿å(Á¤½ÅÀû-À°Ã¼ÀûÀ¸·Î À̼º¿¡ Á¢±ÙÇϰíÀÚ ÇÏ´Â ¼º¿å)°ú ¼º±³¿å(Á÷Á¢ÀûÀÎ ¼º¿å)ÀÇ ¾çÀÚ°¡ °¨ÅðÇϰí ÀÖ´Â °æ¿ì¸¦ ¸»Çϰí, Á¼Àº Àǹ̷δ ´Ü¼øÈ÷ ¼º±³¿¡ ¼ö¹ÝÇÏ¿©¾ß ÇÒ Äè°¨±Ø±â(³²ÀÚ´Â »çÁ¤±îÁö Æ÷ÇÔ)¸¦ ´À³¢Áö ¸øÇÏ´Â °æ¿ìÀÌ´Ù. ±×·¯³ª ¼º±³¿åÀÌ ¾ø¾îÁö´Â ³Ã°¨Áõ°úÀÇ ±¸º°Àº °ï¶õÇÑ °æ¿ì°¡ ¸¹´Ù. ¿øÀÎÀº ¼º±âÀÇ ¹ßÀ°ºÎÀü ¶Ç´Â ±âÇü À̿ܿ¡ ¿°Áõ µîÀ¸·Î ÀÎÇÑ ¼º±³½ÃÀÇ ÅëÁõÀ̶ó´Â ±âÁúÀû Àå¾Ö³ª ³»ºÐºñÁúȯ µîÀ¸·Î ÀÎÇÑ °ÍÀÌ 10% Á¤µµÀ̰í, ´Ù¸¥ ÇÑÆíÀ¸·Î´Â ¼º±³¿¡ ´ëÇÑ ºÒ¾È-°øÆ÷-Çø¿À-¼öÄ¡ µîÀÇ ¸¶À½Å¿ÀÎ °ÍÀÌ ¸¹´Ù. |
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| ¿µ¹® | sex | ÇÑ±Û | ¼º |
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| ¼³¸í | 1. ´ëºÎºÐÀÇ Á¾ÀÇ µ¿¹° ¹× ½Ä¹°¿¡¼ º¼ ¼ö ÀÖÀ¸¸ç, °³Ã¼°¡ ¸¸µç Á¢ÇÕÀÚÇü¿¡ ÀÇÇØ¼, ¶Ç´Â °³Ã¼ÀÇ ±âÁØ¿¡ µû¶ó¼ ³ª´©¾îÁö´Â ±âº»Àû Â÷ÀÌ. ³ÀÚ ¶Ç´Â ´ë¹è¿ìÀÚ´Â ¿©¼º¿¡ ÀÇÇØ¼ ¸¸µé¾îÁö°í, Á¤ÀÚ ¶Ç´Â ¼Ò¹è¿ìÀÚ´Â ³²¼º¿¡ ÀÇÇØ¼ ¸¸µé¾îÁö¸ç, ÀÌµé »óÀÌÇÑ »ý½Ä¼¼Æ÷ÀÇ °áÇÕÀº À¯¼º»ý½Ä¿¡ ÀÖ¾î¼ »õ·Î¿î °³Ã¼¸¦ »ý»êÇÏ´Â µ¥ ÀÚ¿¬ÀûÀÎ ÇʼöÁ¶°ÇÀÌ µÈ´Ù. 2. ³²¼º°ú ¿©¼º, ¼öÄÆ°ú ¾ÏÄÆÀÇ ±¸º°. ¶Ç´Â ³²¼ºÀ̳ª ¿©¼ºÀÇ À°Ã¼Àû Ư¡. |
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| ¿µ¹® | sex identification | ÇÑ±Û | ¼º°¨º° |
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| ¼³¸í | »ç¶÷ÀÇ ¼ºÀº Àû¾îµµ ÀÌÇÏÀÇ Àϰö °³ÀÇ Ç׸ñ¿¡ ÀÇÇÏ¿© °áÁ¤µÈ´Ù. Áï, ¨ç ¿°»öü¿¡ ÀÇÇÑ ¼º, ¨è »ý½Ä»ùÀÇ ¼º, ¨é ¼Ó»ý½Ä±â°ü ÇüÅ¿¡ ÀÇÇÑ ¼º, ¨ê ¹Ù±ù»ý½Ä±â°üÀÇ ÇüÅ¿¡ ÀÇÇÑ ¼º, ¨ë È£¸£¸óÀÛ¿ë¿¡ ÀÇÇÑ ¼º, ¨ì ¾çÀ°µÈ ¼º, ¨í ½É¸®ÇÐÀûÀÎ ¼º µîÀÌ´Ù. Åë»óÀÇ ¼ºº°ÆÇÁ¤Àº ¿°»öü¿¡ÀÇ ÇÑ ¼ºÀ» ÀǹÌÇϰí, ÀÌ¿¡´Â ÀÔ¾ÈÁ¡¸·¼¼Æ÷Ç¥º»¿¡ ÀÇÇÑ X¿°»öÁú, Y¿°»öÁú°Ë»öÀÌ ÀϹÝÀûÀÌ´Ù. |
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| ¿µ¹® | sex chromosome | ÇÑ±Û | ¼º¿°»öü |
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| ¼³¸í | ¾Ï¼öÀÇ ¼ºÀ» °áÁ¤ÇÏ´Â µ¥ Áß¿äÇÑ ±¸½ÇÀ» ÇÏ´Â ¿°»öü. À̰Ϳ¡ ´ëÇÏ¿© º¸ÅëÀÇ ¿°»öü¸¦ º¸Åë¿°»öü¶ó°í ÇÑ´Ù. ¾Ï¼öÀÇ ±¸º°ÀÌ ÀÖ´Â »ý¹°¿¡¼´Â ¾Ï¼ö¿¡ µû¶ó ´Ù¸¥ Çü°ú ¼ö¸¦ ³ªÅ¸³»´Â ¿°»öüÀ̸ç, º¸Åë¿°»öü¿¡ ºñÇØ ¿°»ö¼ºÀ̳ª Çൿ¿¡¼ Â÷À̰¡ ÀÖ´Ù. ƯÈ÷ µ¿¹°ÀÇ ¼º¿°»öü´Â ±×·± °æÇâÀÌ °ÇÏ´Ù. ÈÞÁö±â ¹× Çٺп Àü±â¿¡ ¶Ñ·ÇÇÑ ÀÌ»óÀÀÃàÀ» ³ªÅ¸³»¸ç °¨¼öºÐ¿ ¶§´Â ´Ù¸¥ ¿°»öüº¸´Ù ¸ÕÀú ¾Õ¼°Å³ª ²ø·Á°¡´Â ÇൿÀ» º¸¿©ÁØ´Ù. |
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| SCT | secretin; sex chromatin test; sexual compatibility test; sickle-cell trait; sperm cytotoxicity; spin... |
|---|---|
| SLHR | sex-linked hypophosphatemic rickets |
| MEN | Multiple Endocrine Neoplasia ; AD Trait 1. MEN Type I(= Wermer Syndro... |
| PBT | Paul-Bunnell test; phenacetin breath test; piebald trait; profile-based therapy |
| PTD | percutaneous transluminal dilatation; permanent total disability; personality trait disorder; preter... |
| SLD | Sex-linked dwarf |
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| SLRL | Sex-linked recessive lethal |
| N-linked | Asparagine linked |
| QTL | Aquantitative trait locus |
| QTL | Quantitative Trait Loci |
| sex-linked | <genetics> A genetic disorder where the genetic defect is linked to one of the chromosomes that determines sex (x or y). This is usually due to a gene on the unpaired portion of the X chromosome. Recessive X linked alleles are fully expressed in the heterogametic sex because they can have only one copy of the gene. Thus X linked mutant disorders are more common in human males than in females. An example is haemophilia, which is autosomal recessive and linked to the X chromosome, so only males (XY) actually manifest the disease. (06 Oct 1997) |
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| sex-linked character | An inherited character determined by a gene on a gonosome. See: gene. (05 Mar 2000) |
| sex-linked inheritance | The pattern of inheritance that may result from a mutant gene located on either the X or Y chromosome. (05 Mar 2000) |
| sex-linked locus | Any locus that in normal karyotypes is borne on a heterosome; commonly but incorrectly applied to an X-linked locus. (05 Mar 2000) |
| Bombay trait | A rare recessive trait at a locus that ordinarily manufactures H substance, the precursor from which the A and B phenotypes are elaborated; the mutant causes failure to produce H substance and no matter what the genotype at the ABO locus, the phenotype is O. The Bombay phenomenon is epistatic to the ABO locus. Origin: Bombay, India, where first reported (05 Mar 2000) |
| galtonian trait | A quantitative genetic trait due to contributions from many more of less equally important loci that resembles a continuous trait. (05 Mar 2000) |
| recessive trait | See: dominance of traits. (05 Mar 2000) |
| marker trait | A trait that may be of little importance in itself but which by association, linkage, or other means facilitates the detection, anticipation, or understanding of a disease or (for genetic diseases) the localization of the causative gene on the karyotype. (05 Mar 2000) |
| categorical trait | <genetics> A feature that can conveniently and effectively be analyzed by sorting into classes either because there is no satisfactory way of measuring it (as with blood groups) or because it falls into natural classes so that the variation among classes far exceeds that within classes (e.g., the phenotypic effects of many enzyme polymorphisms); existence of categories suggests but does not prove the operation of a major, simple, underlying cause. Synonym: qualitative trait. (05 Mar 2000) |
| penetrant trait | A trait that in the appropriate genotypes is phenotypically manifest; strictly, it is the trait that is penetrant, not the gene. See: penetrance. (05 Mar 2000) |
| mendelian trait | A categorical trait that segregates in accordance with a single-locus genetic system. (05 Mar 2000) |
| chromosomal trait | A trait dependent on a recurrent chromosomal aberration. (05 Mar 2000) |
| codominant trait | See: codominant. (05 Mar 2000) |
| sickle cell trait | <haematology> This condition occurs in people who have one of two possible genes (i.e., they are heterozygous forthe allele) that code for the defective haemoglobin responsible for sickle cell anaemia. The coditionis diagnosed by exposing an individual's red blood cells to a low oxygen environment, if the trait is present, the cells will turn to a sickle shape. People with this trait may suffer milder symptoms of sickle cell anaemia, or may have no symptoms. Some scientists believe the trait actually provides an evolutionary advantage in tropical environments because the slightly altered shape of the blood cells causes a person to be more resistant to malaria. (09 Oct 1997) |
| nonpenetrant trait | A genetic trait that is not phenotypically manifest because of non-genetic factors it therefore does not include recessivity, epistasis, hypostasis, or parastasis but does include environmental factors and pure random effects such as lyonization. (05 Mar 2000) |
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