| ¿µ¹® | rheumatoid arthritis | ÇÑ±Û | ·ù¸¶Æ¼½º°üÀý¿° |
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| ¿µ¹® | arthritis | ÇÑ±Û | °üÀý¿° |
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| ¿µ¹® | rheumatoid factor | ÇÑ±Û | ·ù¸¶Æ¼½º ÀÎÀÚ |
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| ¼³¸í | IgGÀÇ FcºÎÀ§¿¡ ÀÖ´Â Ç׿ø°áÁ¤ÀÎÀÚ¿¡ ´ëÇÑ Ç×ü·Î¼ ÀüÇüÀûÀÎ ¶Ç´Â È®½ÇÇÑ ·ù¸¶Æ¼½º°üÀý¿°(rheumatoid arthritis) ȯÀÚÀÇ 80%¿¡¼ ¹ß°ßµÈ´Ù. ·ù¸¶Æ¼½º ÀÎÀÚ´Â IgM, IgG, IgAÁß Çϳª°¡ µÉ ¼ö ÀÖÀ¸³ª ÁÖ·Î IgMÀÌ´Ù. ¼Ò¾Æ·ù¸¶Æ¼½º°üÀý¿°(juvenile rheumatoid arthritis: ¼Ò¾Æ±â¿¡ ¹ß»ýÇÏ´Â ·ù¸¶Æ¼½º°üÀý¿°)À» ºñ·ÔÇÑ, ´Ù¸¥ °áÇÕÁ¶Á÷º´À̳ª °¨¿°º´¿¡µµ ³ªÅ¸³¯ ¼ö ÀÖ´Ù |
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| ¿µ¹® | joint | ÇÑ±Û | °üÀý |
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| ¼³¸í | µÎ °³ÀÇ »À ¶Ç´Â ±× ÀÌ»óÀÇ »À°¡ ¼·Î À̾îÁ® ÀÖ´Â °÷. °üÀý¿¡¼´Â ÈçÈ÷ »À »çÀÌ¿¡ ¿òÁ÷ÀÏ ¼ö ÀÖ´Â °üÀý¿îµ¿ÀÌ ÀÖ°Ô µÇÁö¸¸ ÀüÇô ¿òÁ÷ÀÓÀÌ ¾ø´Â °üÀýµµ ÀÖ´Ù. °üÀýÀÇ Á¾·ù´Â ¸¶ÁÖ ´ëÇÏ´Â µÎ »À »çÀÌ¿¡ ¾î¶°ÇÑ Á¶Á÷ÀÌ µé¾î ÀÖ´À³Ä¿¡ µû¶ó¼ ³ª´µ¾îÁö¸ç ¿©±â¿¡´Â ¼¶À¯°üÀý, ¿¬°ñ°üÀý ¹× À±È°°üÀýÀÌ ÀÖ´Ù. »ÀÀÇ °üÀý¸éÀº °üÀý¿¬°ñ¿¡ µ¤À̰í, ÁÖÀ§´Â °üÀýÁָӴ϶ó ºÒ¸®´Â »À¸·¿¡ À̾îÁö´Â °áÇÕÁ¶Á÷¼ºÀÇ ¸·¿¡ ÀÇÇØ µÑ·¯½ÎÀδÙ. °üÀýÁÖ¸Ó´ÏÀÇ °¡Àå ¾ÈÂÊÃþ¿¡´Â À±È°¸·ÀÌ ÀÖ°í °üÀý°¿¡ Ȱ¾×À» ºÐºñÇÑ´Ù. °üÀý°¿¡´Â °üÀý¿øÆÇÀ̶ó´Â ¼¶À¯¿¬°ñ¼ºÀÇ °Ýº® ¶Ç´Â °üÀý¹Ý¿ùÀ̶ó´Â °üÀýÆ÷¿¡¼ ¿ïŸ¸®¸ð¾çÀ¸·Î µ¹ÃâµÇ´Â ºÎºÐÀÌ ÀÖ´Ù. °üÀý³¶ÀÇ ¿ÜºÎ¿¡´Â ¸¹Àº ¼¶À¯¼º Àδ밡 ºÎÂøµÇ¾î ÀÖ¾î °üÀýÀ» º¸°ÇÑ´Ù. °üÀý¿îµ¿ÀÇ ¼ºÁú, ¿îµ¿ÀÇ °¡´É ¹üÀ§´Â °üÀýÀ» ÀÌ·ç´Â °ñ´ÜÀÇ Çüųª °üÀý³¶-ÀδëÀÇ ºÎÂø¹æ¹ý¿¡ µû¶ó °¢°¢ ´Ù¸£´Ù. |
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| ¿µ¹® | joint capsule | ÇÑ±Û | °üÀýÁÖ¸Ó´Ï, °üÀý³¶ |
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| ¼³¸í | °üÀýÀÇ Á¾·ù Áß¿¡¼ À±È°°üÀýÀº µÎ »À »çÀ̰¡ ¾à°£ÀÇ °£°ÝÀ» µÎ°í ¶³¾îÁ® ÀÖÀ¸¸ç °üÀý¸é¿¡´Â ¾ãÀº À¯¸® ¿¬°ñÀÇ ÆÇÀÌ µ¤¿© ÀÖ´Â ÇüÅÂÀε¥, µÎ »À »çÀÌÀÇ °£°ÝÀ» °üÀý°À̶ó°í Çϸç ÀÌ °üÀý° ¼ÓÀº ¿¬°ñÀÌ µ¤¿© ÀÖ´Â °üÀý¸éÀ» Á¦¿ÜÇϰí´Â À±È°¸·¿¡ ÀÇÇØ µ¤¿© ÀÖ°í ÀÌ À±È°¸·Àº ´Ù½Ã ¹Û¿¡¼ Áú±ä ¼¶À¯·Î µÈ ¸·À¸·Î µ¤¿© ÀÖ´Ù. ÀÌ ¼¶À¯¸·À» °üÀýÁָӴ϶ó°í ÇÑ´Ù. |
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| ABCDES | abnormal alignment, bones-periarticular osteoporosis, cartilage-joint space loss, deformities, margi... |
|---|---|
| CHRONIC | chronic disease, rheumatoid arthritis, neoplasms, infections, cryoglobulinemia [conditions in which ... |
| JRA | Juvenile Rheumatoid Arthritis; ¿¬¼Ò±â ·ù¸¶ÅäÀÌµå °üÀý¿° = Juvenile Chronic Arthritis; ¿¬¼Ò±â ¸¸¼º °üÀý... |
| RA | 1) Refractory Anemia 2) Rheumatoid Arthritis ? Arthritis 3... |
| RA | radioactive; ragocyte; ragweed antigen; rapidly adapting [receptors]; reactive arthritis; reciprocal... |
| J.R.A. | Juvenile Rheumatoid Arthritis |
|---|---|
| MRA | Malignant rheumatoid arthritis |
| RANA | rheumatoid arthritis nuclear antigen |
| IgA RF | IgA rheumatoid factor |
| RF | IgA-rheumatoid factor |
self-care (ÀÚ°¡ Ä¡·á
acute arthritis
| rheumatoid arthritis: joint manifestations | <radiology> Early signs: fusiform periarticular soft tissue swelling (result of effusion), regional osteoporosis (disuse and local hyperaemia), widened joint space, marginal and central bone erosion (base of 4th proximal phalanx most common), change in ulnar styloid and distal radioulnar joint, atlantoaxial dislocation, giant synovial cysts late signs: flexion/extension contractures with ulnar subluxation/dislocation, destruction/fusion of joints, elevation of humeral heads (tear/atrophy of rotator cuff), resorption of distal clavicle, erosion of superior margins of posterior portions of 3-5th ribs, destruction/narrowing of disc spaces, destruction of zygapophyseal joints without osteophyte formation, resorption of spinous process, protrusio acetabuli (from osteoporosis) (12 Dec 1998) |
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| arthritis, juvenile rheumatoid | Rheumatoid arthritis of children occurring in three major subtypes defined by the symptoms present during the first six months following onset: systemic-onset (still's disease, juvenile-onset) polyarticular-onset, and pauciarticular-onset. Adult-onset cases of still's disease (still's disease, adult-onset) are also known. Only one subtype of juvenile rheumatoid arthritis (polyarticular-onset, rheumatoid factor-positive) clinically resembles adult rheumatoid arthritis and is considered its childhood equivalent. (12 Dec 1998) |
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| arthritis, rheumatoid | Autoimmune disease that is characterised by chronic inflammation of the joints and can cause inflammation of tissues in other areas of the body (such as the lungs, heart, and eyes). (12 Dec 1998) |
| rheumatoid arthritis | <rheumatology> Chronic inflammatory disease in which there is destruction of joints. Considered by some to be an autoimmune disorder in which immune complexes are formed in joints and excite an inflammatory response (complex mediated hypersensitivity). Cell-mediated (type IV) hypersensitivity also occurs and macrophages accumulate. This in turn leads to the destruction of the synovial lining (see pannus). (18 Nov 1997) |
| rheumatoid arthritis, systemic-onset juvenile | Also known as systemic-onset juvenile chronic arthritis. Still's disease presents with systemic (bodywide) illness including high intermittent fever, a salmon-coloured skin rash, swollen lymph glands, enlargement of the liver and spleen, and inflammation of the lungs (pleuritis) and around the heart (pericarditis). The arthritis may not be immediately apparent but it does appear and may persist after the systemic symptoms are gone. (12 Dec 1998) |
| systemic-onset juvenile rheumatoid arthritis | <rheumatology> A form of joint disease, arthritis, that presents with systemic upset. Clinical signs: high intermittent fever, a salmon-coloured skin rash, swollen lymph glands, enlargement of the liver and spleen, and inflammation of the lungs (pleuritis) and around the heart (pericarditis). The arthritis itself may not be immediately apparent but once apparent, it may persist after the systemic symptoms have resolved. Synonym: Still's disease. (03 Jul 1999) |
| juvenile rheumatoid arthritis | <pathology> Juvenile rheumatoid arthritis (JRA) is a form of rheumatoid arthritis in children that generally occurs prior to age 16. In contrast with the adult type, a fever is more pronounced. Cardiac involvement with pericarditis is more common. The arthritis favors one or more large joints and can interfere with normal bone growth. A positive rheumatoid factor is seen more uncommonly in this form of arthritis. Treatment is similar to the adult form of the disease. Up to 75% recover with treatment. Less than 10% are severely disabled by JRA. (27 Sep 1997) |
| juvenile rheumatoid arthritis, systemic-onset | Also known as systemic-onset juvenile chronic arthritis. Still's disease presents with systemic (bodywide) illness including high intermittent fever, a salmon-coloured skin rash, swollen lymph glands, enlargement of the liver and spleen, and inflammation of the lungs (pleuritis) and around the heart (pericarditis). The arthritis may not be immediately apparent but it does always surface and it may persists long after the systemic symptoms are gone. (12 Dec 1998) |
| amyloidosis: bone manifestations | <radiology> Joint pain without radiographic findings, osteoporosis, especially in axial skeleton, lytic lesions that destroy cortex and invade soft tissue, wrist, scaphoid and lunate lesions that may extend into the carpal tunnel, inducing the classic complaints of carpal tunnel syndrome amyloid arthropathy Differential diagnosis: pigmented villonodular synovitis, synovial chondromatosis, rheumatoid arthritis, TB (12 Dec 1998) |
| amyloidosis: gastrointestinal manifestations | <radiology> Oesophagus, loss of peristalsis, megaesophagus, stomach, small and rigid (simulate linitis plastica), effaced rugal pattern, diminished/absent peristalsis, may be localised to antrum, amyloidoma: well defined submucosal mass, small bowel, diffuse form (more common), diffuse, uniform thickening of valvulae conniventes, broadened flat undulated mucosal folds (mucosal atrophy), jejunalization of ileum, impaired motility, small bowel dilatation, localised form: multiple small deposits; associated with pseudoobstruction, colon, psudopolyps (12 Dec 1998) |
| crohn disease: extraintestinal manifestations | <radiology> Fatty liver, gallstones (28-34%), risk 3-5X higher than expected, secondary to malabsorption of bile salts in terminal ileum, correlation with length of diseased ileum and duration of disease, sclerosing cholangitis, bile duct carcinoma, amyloidosis, urolithiasis: oxalate/uric acid stones, migratory arthritis (5-20%), sacroilitis, ankylosing spondylitis, erythema nodosum, uveitis see: Crohn disease (12 Dec 1998) |
| sickle cell anaemia: bone manifestations | <radiology> 8-13% of blacks carry sickling factor, symptoms: chronic ulcers, pain crises, many infections, priapism X-ray findings: deossification due to marrow hyperplasia, decreased bone density in skull with widened diploe, H-shaped vertebrae or fish vertebrae, rib notching, thrombosis and infarction, avascular necrosis, especially femoral head, periosteal treatmentn (bone within bone), secondary osteomyelitis, Staph. Aureus greater than Salmonella, dactylitis = hand foot syndrome, growth effects, bone shortening secondary to diminished blood supply, death less than 40y (12 Dec 1998) |
| skin manifestations | Dermatologic disorders attendant upon non-dermatologic disease or injury. (12 Dec 1998) |
| skin manifestations of GI disease | <radiology> Pancreatic carcinoma . . . . . . . . Thrombophlebitis migrans, glucagonoma . . . . . . . . . Migratory necrolytic erythema, IBD . . . . . . . . . . . . . . . Pyoderma gangrenosum, sprue / coeliac disease. . . . Dermatitis herpetiformis, Whipple disease . . . . . . . Pigmentation, primary biliary cirrhosis . . . . Exanthemasma, hepatic cirrhosis . . . . . . . . Spider angiomata (12 Dec 1998) |
| neurologic manifestations | Neurologic disorders attendant upon non-neurologic disease or injury. (12 Dec 1998) |
| oral manifestations | Disorders of the mouth attendant upon non-oral disease or injury. (12 Dec 1998) |
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