| ¿µ¹® | gout | ÇÑ±Û | Åëdz |
|---|---|---|---|
| ¼³¸í | À¯ÀüÁ¤º¸¸¦ º¸°üÇÏ´Â ¹°ÁúÀÎ DNA¿Í RNA µîÀ» ÀÌ·ç´Â ÇÙ»êÀº ¿ä»êÀÇ ÇüÅ·Π¹è¼³ÀÌ µÈ´Ù. À̰ÍÀÇ ¹è¼³¿¡´Â ¿©·¯ °¡Áö È¿¼Ò°¡ ÀÛ¿ëÇÑ´Ù. ÀÌÁß¿¡¼ HGPRT (Hypoxanthine-Guanine Phosphoribosyl Transferase)¶ó´Â È¿¼Ò´Â ÀÌ¹Ì ¹è¼³ÀÌ µÇ·Á°í ¿ä»êÀ¸·Î º¯È¯ÇÏ´Â °úÁ¤ÀÇ ¹°ÁúÀ» ´Ù½Ã ÇÙ»êÀ¸·Î ¹Ù²Ù´Â ÀÛ¿ëÀ» ÇÏ´Â È¿¼ÒÀÌ´Ù. ÅëdzÀ̶õ ÀÌ HGPRT¶ó´Â È¿¼Ò Ȱ¼ºµµ°¡ °¨¼ÒµÇ¾î ¹ß»ýµÈ´Ù. ÀÌ È¿¼ÒÀÇ È°¼ºµµ°¡ °¨¼ÒµÇ¸é ÇÙ»êÀÇ ¹è¼³´Ü°è¿¡¼ Àç»ç¿ëÀ̶ó´Â ´Ü°è°¡ ¾ø¾îÁö¹Ç·Î °úµµÇÑ ¿ä»êÀÇ »ý¼ºÀÌ ÀÖ°Ô µÈ´Ù. Ç÷Áß¿¡ °úµµÇÑ ¿ä»êÀÌ Á¸ÀçÇϰí ÀÌ ¿ä»êÀº °áÁ¤À» ¸¸µé¾î¼ °üÀý ÁÖÀ§ ¹× ¿¬ºÎ Á¶Á÷¿¡ Ä§ÂøµÇ¾î °Ý½ÉÇÑ ¹ßÀÛ¼º °üÀýÅëÀ» ¾ß±âÇÏ´Â ±Þ¼º ¿°Áõ¼º °üÀý¿°À» ÀÏÀ¸Å°´Â Ư¡ÀûÀÎ º´ÀÌ´Ù. ¶Ç ÀÌ ¿ä»êÀÇ °áÁ¤µéÀÌ ÄáÆÏÀ̳ª ½ÉÀå¿¡ ÃàÀûµÇ¾î¼ ÄáÆÏ ¹× ½ÉÇ÷°ü°è¿¡ Àå¾Ö°¡ ¹ß»ýÇÒ ¼öµµ ÀÖ¾î ³»°úÀû Ä¡·á¸¦ ¿äÇÏ´Â º´ÀÌ´Ù. À¯Àü ¾ç½Ä»ó º¸Åë¿°»öü ¿ì¼ºÀ¸·Î À¯ÀüÇÏ´Â À¯Àüº´ÀÌ´Ù. º¸Åë¿°»öü ¿ì¼º À¯ÀüÀ» ÇÏÁö¸¸ ³²ÀÚ¿Í ¿©ÀÚ¿¡¼ Ç¥Çö·üÀÌ ´Þ¶ó¼ ³²ÀÚ°¡ 95%ÀÌ»óÀ» Â÷ÁöÇÑ´Ù. |
||
| ¿µ¹® | renal biopsy | ÇÑ±Û | ÄáÆÏ»ý°Ë |
|---|---|---|---|
| ¼³¸í | ÄáÆÏÀÇ º´º¯ÀÌ ÀÇ½ÉµÉ ¶§ È®ÁøÀ» À§ÇØ ÁÖ»ç¹Ù´Ã µîÀ» ÀÌ¿ëÇÏ¿© ÄáÆÏÁ¶Á÷À» ÀϺΠ¶¼¾î³»¼ Çö¹Ì°æÀ¸·Î °Ë°æÇÏ´Â °Í. |
||
| ¿µ¹® | renal hypertension | ÇÑ±Û | ÄáÆÏ¼º°íÇ÷¾Ð |
|---|---|---|---|
| ¼³¸í | ÄáÆÏ½ÇÁúÀÇ º´º¯À¸·Î ÀÎÇØ ¾ß±âµÈ °íÇ÷¾Ð. ÄáÆÏÀÇ ´ëÇ¥Àû ±â´ÉÀº ³ëÆó¹° ¹× ¼öºÐÀÇ ¹è¼³ÀÌ´Ù. ±×·±µ¥ ÀÌ·¯ÇÑ ÄáÆÏ±â´É¿¡ ÀÌ»óÀÌ »ý°åÀ» °æ¿ì ü³»¿¡ °úÀ×¼öºÐÀÇ ÃàÀûÀÌ ¹ß»ýÇÏ°Ô µÈ´Ù. À̿Ͱ°Àº °úÀ×¼öºÐÀÇ ÃàÀûÀº Ç÷°ü³» Á¤¼ö¾ÐÀ» »ó½Â½ÃÄÑ °íÇ÷¾ÐÀ» À¯¹ßÇÏ°Ô µÈ´Ù. Ä¡·á´Â ¿øÀÎ ÄáÆÏº´ÀÇ ±³Á¤À̸ç ÀÌÀ¯¸¦ ¸ð¸£´Â ¿ø¹ß°íÇ÷¾Ð°ú ´Þ¸® ÄáÆÏ¼º°íÇ÷¾ÐÀÇ °æ¿ì¿¡´Â ¿øÀÎ ÄáÆÏº´ÀÌ ±³Á¤µÇ¸é °íÇ÷¾Ðµµ »ç¶óÁö°Ô µÈ´Ù. |
||
| ¿µ¹® | renal cell carcinoma | ÇÑ±Û | ÄáÆÏ¼¼Æ÷¾ÏÁ¾ |
|---|---|---|---|
| ¼³¸í | ÄáÆÏ¿¡ »ý±ä ¿ø½ÃÄáÆÏÁ¶Á÷¿¡¼ ¹ß»ýÇÑ ¾Ï. ÁÖ·Î ¿ø½Ã¼¼´¢°üÁ¶Á÷¿¡¼ ¹ß»ýÇÑ´Ù. ´ëÇ¥ÀûÀÎ ¼¼Æ÷Á¶Á÷ÇüÀº ¿°»ö½Ã ¼¼Æ÷ÁúÀÌ ¸¼°Ô ºñ¾îº¸ÀÌ´Â ¸¼Àº¼¼Æ÷¾ÏÁ¾ÀÌ´Ù. Ä¡·á´Â ¼ö¼ú°ú Ç×¾ÏÈÇпä¹ýÀÌ¸ç ¾ÆÁÖ µå¹°Áö¸¸ ÀúÀý·Î ³´´Â °æ¿ìµµ ÀÖ´Â °ÍÀ¸·Î º¸°íµÇ¾î ÀÖ´Ù. |
||
| ¿µ¹® | renal transplantation | ÇÑ±Û | ÄáÆÏÀÌ½Ä |
|---|---|---|---|
| ¼³¸í | ÄáÆÏº´À» °¡Áö°í ÀÖÀ¸³ª Ä¡·á°¡ ºÒ°¡´ÉÇÑ ¸¸¼ºÄáÆÏ±â´É»ó½Ç µîÀÇ Áúº´À» °¡Áø ȯÀÚÀÇ ½ÅÀåÀ» ¶¼¾î³»°í ȯÀÚ¿Í Ç׿ø¼ºÀÌ À¯»çÇÑ »ç¶÷ÀÇ ÄáÆÏÀ» À̽ÄÇØÁÖ´Â °Í. ÀÌ ¶§ ¼·Î°£ÀÇ Ç׿ø¼ºÀÇ À¯»çÁ¡ÀÌ ¸¹¾Æ¾ß °ÅºÎ¹ÝÀÀÀÌ ÀϾÁö ¾Ê´Â´Ù. ±×¸®°í ÀÏ´Ü ÄáÆÏÀ̽ÄÀ» ¹ÞÀº »ç¶÷Àº ¿À·£±â°£ µ¿¾È ¸é¿ª¾ïÁ¦Á¦¸¦ Åõ¿©ÇÏ¿© °ÅºÎ¹ÝÀÀÀ» ÁÙ¿©¾ß ÇÑ´Ù. ´ë°³ ÀÌ½ÄµÈ ÄáÆÏÀº ¾ûµ¢»À¿À¸ñ¿¡ À§Ä¡ÇÏ°Ô µÈ´Ù. |
||
| ABC | absolute basophil count; absolute bone conduction; acalculous biliary colic; acid balance control; a... |
|---|---|
| PRAGMATIC | pregnancy, rheumatoid arthritis, acromegaly, glucose metabolism disorders, mechanical injury, amyloi... |
| CRD | carbohydrate-recognition domain; chronic renal disease; chronic respiratory disease; child restraint... |
| CRT | cadaveric renal transplant; cardiac resuscitation team; cathode-ray tube; certified; Certified Recor... |
| RVRA | renal vein rein activity; renal venous renin assay |
| ARI | 5--acute renal insufficiency |
|---|---|
| ARCD | Acquired renal cystic disease |
| ARF | Acute Renal Failure |
| ARN | Afferent renal nerves |
| BMRTC | Bone metastasising renal tumour of childhood |
| arthritis, gout | Joint inflammation caused by uric acid crystal deposits in the joint space An attack is usually extremely painful.The uric acid crystals are deposited in the joint fluid (synovial fluid) and joint lining (synovial lining). Intense joint inflammation occurs as white blood cells engulf the uric acid crystals, causing pain, heat, and redness of the joint tissues. The term gout commonly is used to refer to these painful arthritis attacks but gouty arthritis is only one manifeatation of gout. (12 Dec 1998) |
|---|---|
| calcium gout | <rheumatology> Chondrocalcinosis, chronic recurrent arthritis clinically similar to gout. (27 Sep 1997) |
| masked gout | latent gout |
| retrocedent gout | An obsolete term for the occurrence of severe gastric, cardiac, or cerebral symptoms during an attack of gout, especially when the joint and other symptoms suddenly subside at the same time. Saturnine gout, gout occurring in a person with lead poisoning. Synonym: lead gout. Secondary gout, gout resulting from increased serum uric acid levels as a result of an antecedent disease, such as a proliferative disease of the blood and bone marrow, lead poisoning, or prolonged chronic renal failure (on dialysis). Tophaceous gout, gout in which deposits of uric acid and urates occur as gouty tophi. (05 Mar 2000) |
| gout | <rheumatology> Recurrent acute arthritis of peripheral joints caused by the accumulation of monosodium urate crytals. Often presents as pain and swelling confined to one joint. The big toe joint is commonly affected. The arthritis occurs secondary to an inherited abnormality of purine metabolism, resulting in the deposition of uric acid crystals (sodium urate) within the joint space and articular cartilage. Usually due to overproduction of uric acid but may be a result of under excretion. The problems partly arise because neutrophils release lysosomal enzymes as a result of damage to the phagosome membrane by ingested crystals: colchicine acts to reduce the attack by inhibiting lysosome phagosome fusion. (18 Nov 1997) |
| gout arthritis | <rheumatology> Recurrent acute arthritis of peripheral joints caused by the accumulation of monosodium urate crytals. Often presents as pain and swelling confined to one joint. The big toe joint is commonly affected. The arthritis occurs secondary to an inherited abnormality of purine metabolism, resulting in the deposition of uric acid crystals (sodium urate) within the joint space and articular cartilage. Usually due to overproduction of uric acid but may be a result of under excretion. The problems partly arise because neutrophils release lysosomal enzymes as a result of damage to the phagosome membrane by ingested crystals: colchicine acts to reduce the attack by inhibiting lysosome phagosome fusion. (18 Nov 1997) |
| gout diet | A diet containing a minimal quantity of purine bases (meats); liver, kidney, and sweetbread especially are excluded and replaced by dairy products, fruits, and cereals; alcoholic beverages also are excluded. Synonym: purine-free diet. (05 Mar 2000) |
| gout suppressants | Agents that increase uric acid excretion by the kidney (uricosuric agents), decrease uric acid production (antihyperuricaemics), or alleviate the pain and inflammation of acute attacks of gout. (12 Dec 1998) |
| gout, tophaceous | A form of chronic gout. Nodular masses of uric acid crystals (tophi) are deposited in different soft tissue areas of the body. Even though tophi are most commonly found as hard nodules around the fingers, at the tips of the elbows, and around the big toe, tophi nodules can appear anywhere in the body. They have been reported in unexpected areas such as in the ears, vocal cords, or around the spinal cord! (12 Dec 1998) |
| primary gout | Acute episodes of crystal-induced synovitis due to abnormality of purine metabolism; lower than normal urinary excretion of urate leading to hyperuricaemia and acute episodes of joint inflammation. Synonym: primary gout. Interval gout, an asymptomatic phase between acute attacks of gout. Latent gout, hyperuricaemia without symptoms of gout. Often used synonymously with interval gout. Synonym: masked gout. (05 Mar 2000) |
| idiopathic gout | Acute episodes of crystal-induced synovitis due to abnormality of purine metabolism; lower than normal urinary excretion of urate leading to hyperuricaemia and acute episodes of joint inflammation. Synonym: primary gout. Interval gout, an asymptomatic phase between acute attacks of gout. Latent gout, hyperuricaemia without symptoms of gout. Often used synonymously with interval gout. Synonym: masked gout. (05 Mar 2000) |
| tophaceous gout | Nodular masses of uric acid crystals (tophi) are deposited in different soft tissue areas of the body. Even though tophi are most commonly found as hard nodules around the fingers, at the tips of the elbows, and around the big toe, tophi nodules can appear anywhere in the body. They have been reported in unexpected areas such as in the ears, vocal cords, or around the spinal cord! tophaceous gout is always a result of chronic gout. (12 Dec 1998) |
| lead gout | saturnine gout |
| acute renal failure | <nephrology> A sudden decline in renal function may be triggered by a number of acute disease processes. Examples include sepsis (infection), shock, trauma, kidney stones, kidney infection, drug toxicity (aspirin or lithium), poisons or toxins (drug abuse) or after injection with an iodinated contrast dye (adverse effect). Chronic renal failure represents a slow decline in kidney function over time. Chronic renal failure may be caused by a number of disorders which include long-standing hypertension, diabetes, congestive heart failure, lupus or sickle cell anaemia. Both forms of renal failure result in a life-threatening metabolic derangement. (27 Sep 1997) |
| aminoaciduria, renal | Impairment of renal tubular transport of amino acids. (12 Dec 1998) |
Á¦Ç°¸í |
ÆÇ¸Å»ç |
º¸ÇèÄÚµå | ¼ººÐ/ÇÔ·® | ±¸ºÐ/º¸Çè±Þ¿© |
|---|
Á¦Ç°¸í |
ÆÇ¸Å»ç |
º¸ÇèÄÚµå | ¼ººÐ/ÇÔ·® | ±¸ºÐ/º¸Çè±Þ¿© |
|---|