| ¿µ¹® | candidiasis | ÇÑ±Û | ĵð´ÙÁõ |
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| ¼³¸í | °õÆÎÀÌÀÇ ÇϳªÀÎ candida¿¡ ÀÇÇÑ °¨¿°. ´ë°³ ÇǺÎÀÇ °¨¿°ÀÌ °¡Àå ÈçÇϰí, ½Å»ý¾ÆÀÇ ÀÔÀ̳ª ¿©¼ºÀÇ Áú¿¡ °¨¿°À» ÀÏÀ¸Å°±âµµ ÇÑ´Ù. ±×¸®°í ¾ÆÁÖ µå¹°°Ô ĵð´Ù°¡ Àü½ÅÀû °¨¿°À» ÀÏÀ¸Å°±âµµ ÇÑ´Ù. À̰ÍÀ» ¸ð´Ò¸®¾ÆÁõ(moniliasis)À̶ó°íµµ ÇÑÀû ÀÖ´Ù. |
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| ¿µ¹® | renal biopsy | ÇÑ±Û | ÄáÆÏ»ý°Ë |
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| ¼³¸í | ÄáÆÏÀÇ º´º¯ÀÌ ÀÇ½ÉµÉ ¶§ È®ÁøÀ» À§ÇØ ÁÖ»ç¹Ù´Ã µîÀ» ÀÌ¿ëÇÏ¿© ÄáÆÏÁ¶Á÷À» ÀϺΠ¶¼¾î³»¼ Çö¹Ì°æÀ¸·Î °Ë°æÇÏ´Â °Í. |
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| ¿µ¹® | renal hypertension | ÇÑ±Û | ÄáÆÏ¼º°íÇ÷¾Ð |
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| ¼³¸í | ÄáÆÏ½ÇÁúÀÇ º´º¯À¸·Î ÀÎÇØ ¾ß±âµÈ °íÇ÷¾Ð. ÄáÆÏÀÇ ´ëÇ¥Àû ±â´ÉÀº ³ëÆó¹° ¹× ¼öºÐÀÇ ¹è¼³ÀÌ´Ù. ±×·±µ¥ ÀÌ·¯ÇÑ ÄáÆÏ±â´É¿¡ ÀÌ»óÀÌ »ý°åÀ» °æ¿ì ü³»¿¡ °úÀ×¼öºÐÀÇ ÃàÀûÀÌ ¹ß»ýÇÏ°Ô µÈ´Ù. À̿Ͱ°Àº °úÀ×¼öºÐÀÇ ÃàÀûÀº Ç÷°ü³» Á¤¼ö¾ÐÀ» »ó½Â½ÃÄÑ °íÇ÷¾ÐÀ» À¯¹ßÇÏ°Ô µÈ´Ù. Ä¡·á´Â ¿øÀÎ ÄáÆÏº´ÀÇ ±³Á¤À̸ç ÀÌÀ¯¸¦ ¸ð¸£´Â ¿ø¹ß°íÇ÷¾Ð°ú ´Þ¸® ÄáÆÏ¼º°íÇ÷¾ÐÀÇ °æ¿ì¿¡´Â ¿øÀÎ ÄáÆÏº´ÀÌ ±³Á¤µÇ¸é °íÇ÷¾Ðµµ »ç¶óÁö°Ô µÈ´Ù. |
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| ¿µ¹® | renal cell carcinoma | ÇÑ±Û | ÄáÆÏ¼¼Æ÷¾ÏÁ¾ |
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| ¼³¸í | ÄáÆÏ¿¡ »ý±ä ¿ø½ÃÄáÆÏÁ¶Á÷¿¡¼ ¹ß»ýÇÑ ¾Ï. ÁÖ·Î ¿ø½Ã¼¼´¢°üÁ¶Á÷¿¡¼ ¹ß»ýÇÑ´Ù. ´ëÇ¥ÀûÀÎ ¼¼Æ÷Á¶Á÷ÇüÀº ¿°»ö½Ã ¼¼Æ÷ÁúÀÌ ¸¼°Ô ºñ¾îº¸ÀÌ´Â ¸¼Àº¼¼Æ÷¾ÏÁ¾ÀÌ´Ù. Ä¡·á´Â ¼ö¼ú°ú Ç×¾ÏÈÇпä¹ýÀÌ¸ç ¾ÆÁÖ µå¹°Áö¸¸ ÀúÀý·Î ³´´Â °æ¿ìµµ ÀÖ´Â °ÍÀ¸·Î º¸°íµÇ¾î ÀÖ´Ù. |
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| ¿µ¹® | renal transplantation | ÇÑ±Û | ÄáÆÏÀÌ½Ä |
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| ¼³¸í | ÄáÆÏº´À» °¡Áö°í ÀÖÀ¸³ª Ä¡·á°¡ ºÒ°¡´ÉÇÑ ¸¸¼ºÄáÆÏ±â´É»ó½Ç µîÀÇ Áúº´À» °¡Áø ȯÀÚÀÇ ½ÅÀåÀ» ¶¼¾î³»°í ȯÀÚ¿Í Ç׿ø¼ºÀÌ À¯»çÇÑ »ç¶÷ÀÇ ÄáÆÏÀ» À̽ÄÇØÁÖ´Â °Í. ÀÌ ¶§ ¼·Î°£ÀÇ Ç׿ø¼ºÀÇ À¯»çÁ¡ÀÌ ¸¹¾Æ¾ß °ÅºÎ¹ÝÀÀÀÌ ÀϾÁö ¾Ê´Â´Ù. ±×¸®°í ÀÏ´Ü ÄáÆÏÀ̽ÄÀ» ¹ÞÀº »ç¶÷Àº ¿À·£±â°£ µ¿¾È ¸é¿ª¾ïÁ¦Á¦¸¦ Åõ¿©ÇÏ¿© °ÅºÎ¹ÝÀÀÀ» ÁÙ¿©¾ß ÇÑ´Ù. ´ë°³ ÀÌ½ÄµÈ ÄáÆÏÀº ¾ûµ¢»À¿À¸ñ¿¡ À§Ä¡ÇÏ°Ô µÈ´Ù. |
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| APECED | Autoimmune Poly-Endocrinopathy Candidiasis Ectodermal Dystrophy |
|---|---|
| MEDAC Syndrome | Multiple-Endocrine Deficiency Autoimmune-Candidiasis |
| CMC | carboxymethylcellulose; care management continuity; carpometacarpal; cell-mediated cytolysis or cyto... |
| CMCC | chronic mucocutaneous candidiasis |
| FCMC | familial chronic mucocutaneous candidiasis; family centered maternity care |
| APECED | Autoimmune polyendocrinopathy candidiasis ectodermal dystrophy |
|---|---|
| CMC | Chronic mucocutaneous candidiasis |
| OC | Oral candidiasis |
| OPC | Oropharyngeal candidiasis |
| VVC | Vulvovaginal candidiasis |
| candidiasis | <gastroenterology, microbiology, oncology> Infection with a fungus of the genus Candida. It is usually a superficial infection of the moist cutaneous areas of the body and is generally caused by Candida albicans, it most commonly involves the skin (dermatocandidiasis), oral mucous membranes (oral candidiasis), respiratory tract (bronchocandidiasis) and vagina (vaginal candidiasis or thrush). Rarely there is a systemic infection or endocarditis. Oral candidiasis: describes a fungal (yeast) infection of the oral cavity due to Candida. It is common in infants, diabetics or those on chemotherapy and is well recognised in patients with HIV infection and AIDS. Oesophageal candidiasis: Infection of the oesophagus by the yeast-like fungus Candidal albicans. Usually occurs in the immunocompromised individual (AIDS or following chemotherapy). Oral candidiasis is a predisposing factor but oesophageal involvement can occur without evidence of infection in the oral cavity. Symptoms include difficulty swallowing, pain on swallowing and oral lesions. Diagnosis is made using endoscopy. Treatment is with antifungal agents such as ketoconazole or fluconazole. Synonym: moniliasis, candidosis, oidiomycosis, blastodendriosis. (16 Dec 1997) |
|---|---|
| candidiasis, chronic mucocutaneous | A clinical syndrome characterised by development, usually in infancy or childhood, of a chronic, often widespread candidiasis of skin, nails, and mucous membranes. It may be secondary to one of the immunodeficiency syndromes, inherited as an autosomal recessive trait, or associated with defects in cell-mediated immunity, endocrine disorders, dental stomatitis, or malignancy. (12 Dec 1998) |
| candidiasis, cutaneous | Candidiasis of the skin manifested as eczema-like lesions of the interdigital spaces, perleche, or chronic paronychia. (12 Dec 1998) |
| candidiasis of oesophagus | <radiology> Findings: long oesophageal segments involved (more common in lower 1/2), 1-2 mm nodular filling defects with linear orientation (plaques), cobble stone: mucosal nodularity in early stage, shaggy, fuzzy, serrated contour (from pseudomembranes, erosions, ulcerations, intramural hemmorhage), narrowed lumen (spasm, pseudomembrane, oedema), intramural diverticulosis, sluggish/absent peristalsis Differential diagnosis: reflux oesophagitis, herpes oesophagitis, acute caustic ingestion, intramural pseudotics, squamous papillomatosis, glycogen acanthosis, Barrett oesophagus, superficial spreading carcinoma, epidermolysis bullosa, varices diagnostic sensitivity: endoscopy (97%), double contrast (88%), single contrast (55%) (12 Dec 1998) |
| candidiasis, oral | Infection of the mucous membranes of the mouth by a fungus of the genus candida. (12 Dec 1998) |
| candidiasis, vulvovaginal | Infection of the vulva and vagina with a fungus of the genus candida. (12 Dec 1998) |
| oesophageal candidiasis | <gastroenterology, microbiology, oncology> Infection with a fungus of the genus Candida. It is usually a superficial infection of the moist cutaneous areas of the body and is generally caused by Candida albicans, it most commonly involves the skin (dermatocandidiasis), oral mucous membranes (oral candidiasis), respiratory tract (bronchocandidiasis) and vagina (vaginal candidiasis or thrush). Rarely there is a systemic infection or endocarditis. Oral candidiasis: describes a fungal (yeast) infection of the oral cavity due to Candida. It is common in infants, diabetics or those on chemotherapy and is well recognised in patients with HIV infection and AIDS. Oesophageal candidiasis: Infection of the oesophagus by the yeast-like fungus Candidal albicans. Usually occurs in the immunocompromised individual (AIDS or following chemotherapy). Oral candidiasis is a predisposing factor but oesophageal involvement can occur without evidence of infection in the oral cavity. Symptoms include difficulty swallowing, pain on swallowing and oral lesions. Diagnosis is made using endoscopy. Treatment is with antifungal agents such as ketoconazole or fluconazole. Synonym: moniliasis, candidosis, oidiomycosis, blastodendriosis. (16 Dec 1997) |
| oral candidiasis | <gastroenterology, microbiology, oncology> Infection with a fungus of the genus Candida. It is usually a superficial infection of the moist cutaneous areas of the body and is generally caused by Candida albicans, it most commonly involves the skin (dermatocandidiasis), oral mucous membranes (oral candidiasis), respiratory tract (bronchocandidiasis) and vagina (vaginal candidiasis or thrush). Rarely there is a systemic infection or endocarditis. Oral candidiasis: describes a fungal (yeast) infection of the oral cavity due to Candida. It is common in infants, diabetics or those on chemotherapy and is well recognised in patients with HIV infection and AIDS. Oesophageal candidiasis: Infection of the oesophagus by the yeast-like fungus Candidal albicans. Usually occurs in the immunocompromised individual (AIDS or following chemotherapy). Oral candidiasis is a predisposing factor but oesophageal involvement can occur without evidence of infection in the oral cavity. Symptoms include difficulty swallowing, pain on swallowing and oral lesions. Diagnosis is made using endoscopy. Treatment is with antifungal agents such as ketoconazole or fluconazole. Synonym: moniliasis, candidosis, oidiomycosis, blastodendriosis. (16 Dec 1997) |
| acute renal failure | <nephrology> A sudden decline in renal function may be triggered by a number of acute disease processes. Examples include sepsis (infection), shock, trauma, kidney stones, kidney infection, drug toxicity (aspirin or lithium), poisons or toxins (drug abuse) or after injection with an iodinated contrast dye (adverse effect). Chronic renal failure represents a slow decline in kidney function over time. Chronic renal failure may be caused by a number of disorders which include long-standing hypertension, diabetes, congestive heart failure, lupus or sickle cell anaemia. Both forms of renal failure result in a life-threatening metabolic derangement. (27 Sep 1997) |
| aminoaciduria, renal | Impairment of renal tubular transport of amino acids. (12 Dec 1998) |
| back-pressure renal atrophy | <radiology> Caliectasis without obstruction, due to repeated episodes of obstruction, gradual loss of renal pyramids (12 Dec 1998) |
| base of renal pyramid | The outer broad part of a renal pyramid that lies next to the cortex. Synonym: basis pyramidis renis. (05 Mar 2000) |
| branchio-oto-renal syndrome | <syndrome> An autosomal dominant disorder manifested by various combinations of preauricular pits, branchial fistulae or cysts, lacrimal duct stenosis, hearing loss, structural defects of the outer, middle, or inner ear, and renal dysplasia. Associated defects include asthenic habitus, long narrow facies, constricted palate, deep overbite, and myopia. Hearing loss may be due to mondini type cochlear defect and stapes fixation. (12 Dec 1998) |
| capsular branches of renal artery | <anatomy, artery> Branches arising from the renal artery outside of the kidney that are distributed to the renal capsule. Synonym: rami capsulares arteriae renalis. (05 Mar 2000) |
| captopril renal scan | <radiology> In a kidney with a lesion in the afferent arteriole (e.g. Atherosclerotic plaque), reflex constriction of the efferent arteriole occurs through angiotensin system thus maintaining renal perfusion. ACE inhibition prevents constriction of efferent arteriole. Therefore, perfusion is decreased to a kidney with afferent lesions and the renal scan to looks WORSE. Bottom line: renal scans appear WORSE with captopril administration if there is a lesion in the afferent arteriole. See: renal artery stenosis (12 Dec 1998) |
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