¼±Åà - È­»ìǥŰ/¿£ÅÍŰ ´Ý±â - ESC

 
"receptors, tumour necrosis factor"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
¾Ë±â½¬¿î ÀÇÇпë¾îÇ®ÀÌÁý, ¼­¿ïÀÇ´ë ±³¼ö ÁöÁ¦±Ù, °í·ÁÀÇÇÐ ÃâÆÇ À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
¿µ¹® rheumatoid factor ÇÑ±Û ·ù¸¶Æ¼½º ÀÎÀÚ
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  IgGÀÇ FcºÎÀ§¿¡ Àִ Ç׿ø°áÁ¤ÀÎÀÚ¿¡ ´ëÇÑ Ç×ü·Î¼­ ÀüÇüÀûÀΠ¶Ç´Â È®½ÇÇÑ ·ù¸¶Æ¼½º°üÀý¿°(rheumatoid arthritis) È¯ÀÚÀÇ 80%¿¡¼­ ¹ß°ßµÈ´Ù. ·ù¸¶Æ¼½º ÀÎÀڴ IgM, IgG, IgAÁß Çϳª°¡ µÉ ¼ö ÀÖÀ¸³ª ÁַΠIgMÀÌ´Ù. ¼Ò¾Æ·ù¸¶Æ¼½º°üÀý¿°(juvenile rheumatoid arthritis: ¼Ò¾Æ±â¿¡ ¹ß»ýÇϴ ·ù¸¶Æ¼½º°üÀý¿°)À» ºñ·ÔÇÑ, ´Ù¸¥ °áÇÕÁ¶Á÷º´À̳ª °¨¿°º´¿¡µµ ³ªÅ¸³¯ ¼ö ÀÖ´Ù
¿µ¹® growth factor ÇÑ±Û ¼ºÀåÀÎÀÚ
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  ¼¼Æ÷ÀÇ ºÐÈ­ ¹× ¼ºÀå¿¡ °ü¿©Çϴ ´Ü¹éÁú. ¼ºÀåÀÎÀڴ Á¤»ó ¼¼Æ÷Áֱ⿡ ÇʼöÀûÀ̱⠶§¹®¿¡ µ¿¹°ÀÇ »ý¸í¿¡ Áß´ëÇÑ ¿ä¼Ò°¡ µÈ´Ù. ¹«¾ùº¸´Ùµµ ¼ºÀåÀÎÀڴ Å¾ÆÀÇ ¹ßÀ°À» Á¶Á¤Çϰí Á¶Á÷ÀÇ À¯Áö ¹× º¸¼ö¿¡ Áß´ëÇÑ ¿ªÇÒÀ» Çϸç, Ç÷±¸ÀÇ »ý¼ºÀ» ÀÚ±ØÇÑ´Ù. ¶ÇÇÑ ¾ÏÀÇ ÁøÇà°úÁ¤¿¡µµ °ü¿©ÇÑ´Ù.
¿µ¹® risk factor ÇÑ±Û À§ÇèÀÎÀÚ
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  ±¹Á¦¹æ»ç¼±¹æÈ£À§¿øÈ¸(ICRP)°¡ 1977³â ±Ç°í¿¡¼­ ¹æ»ç¼±¹æÈ£ÀÇ ¸ñÀûÀ¸·Î Ã¤ÅÃÇÑ Áö¼ö·Î, ´ÜÀ§¼±·®(1 Sv)´ç È®·üÀû ¿µÇâÀÇ ¹ß»ýÈ®·üÀ» ÃßÁ¤ÇÏ¿© ³ªÅ¸³½ °ÍÀÌ´Ù. »ý½Ä¼± ¼±·®¿¡ ´ëÇÑ À¯ÀüÀû¿µÇâÀÇ ¹ß»ý·ü(4¡¿10£­3/Sv)À̳ª Àû»ö°ñ¼ö¼±·®¿¡ ´ëÇÑ ¹éÇ÷º´ ¹ß»ý·ü(2¡¿10£­3/Sv)µî ¿Ü¿¡ »À, ÇãÆÄ, °©»ó»ù, Á¥»ù, ±âŸ Á¶Á÷ÀÇ À§ÇèÁö¼ö¸¦ ÃøÁ¤ÇÏ¿©, È®·üÀû ¿µÇâÀÇ Àü½Å¿¡ À־ Ä¡»çÀ§ÇèÁö¼öÀÇ Çհ踦 16.5¡¿10£­3/Sv·Î ÇÏ¿´´Ù. ±×ÈÄ ICRP´Â 1990³â ±Ç°í¿¡¼­ ´ë»óÀÌ µÇ´Â Á¶Á÷°ú Àå±â¸¦ Ãß°¡Çϰí, ¼öÄ¡ °³Á¤À» Çϸ鼭 ¸íεµ °¢¸ñÀûÈ®·üÁö¼ö¶ó°íÇÏ¿´´Ù. ÀÌ ±Ç°í¿¡ ÀÇÇϸé, Ä¡»çÀû È®·üÀû ¿µÇâÀÇ È®·üÁö¼öÀÇ ÇÕ°è´Â, ÀϹÝÀο¡ ÀÖ¾î 60.0¡¿10£­3/SvÀÌ´Ù.
¿µ¹® necrosis ÇÑ±Û ±«»ç
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  ¼¼Æ÷°¡ Á״ °ÍÀ» ¸»ÇÑ´Ù. ¿øÀÎÀº ¾î¶² ¿Ü»óÀÏ ¼öµµ ÀÖ°í, È¤Àº µ¿¸ÆÀÌ Á¼¾ÆÁ®¼­ Çǰ¡ ÅëÇÏÁö ¾Ê¾Æ Á×°Ô µÇ´Â °ÍÀÏ ¼öµµ ÀÖ´Ù. ¾î¶² °æ¿ìÀ̵ç, ±«»çµÈ Á¶Á÷À̠ü³»¿¡ ÀÖÀ¸¸é, °á±¹Àº ¿©·¯ ÇÕº´ÁõÀ» ºÒ·¯ ÀÏÀ¸Å°¹Ç·Î Á¦°ÅµÇ¾î¾ß ÇÑ´Ù.
  
  ±«Àú±«»ç: gangrenous necrosis
  
  ±«Àú±«»ç¶ó´Â ¿ë¾î´Â º¸Åë ±«»ç¶ó´Â °Í°ú Å©°Ô ´Ù¸¥ °ÍÀº ¾øÀ¸³ª ÀÓ»ó, Æ¯È÷ ¿Ü°ú¿¡¼­ ÈçÈ÷ ¾²°í Àִ ¸»ÀÌ´Ù. À̴ ÆÈ´Ù¸®, Æ¯È÷ ´Ù¸®¿¡¼­ ¸¹ÀÌ »ý±â´Âµ¥ Ç÷¾×°ø±ÞÀÌ ¼Ò½ÇµÇ°í ±× ÈÄ¿¡ ¼¼±Õ°¨¿°À» ¹Þ¾Æ¼­ »ý±â´Â º´ÅÍÀÌ´Ù. ¸¸¾à Ç÷·ù°ø±ÞÀÇ Â÷´Ü¿¡ ÀÇÇÑ Á¶Á÷ÀÇ Á×À½ÀÌ ÁÖº´º¯À̰í ÀÌÂ÷Àû ¼¼±ÕÀÇ °¨¿°ÀÌ Àû°í, ÀÖ´õ¶óµµ ¾ÆÁÖ ¹Ì¾àÇØ¼­ º´ÅͰ¡ ¸¶¸£°í ¾×ü¼ººÐÀÌ ÀûÀ» °æ¿ì¿¡ À̰ÍÀ» °Ç¼º±«Àú(dry gangrene)À̶ó°í Çϰí, ¼¼±ÕÀÇ °¨¿°ÀÌ ¾ÆÁÖ ¸¹¾Æ¼­ Á×Àº Á¶Á÷ÀÌ ºÐÇØ°¡ µÇ¾î¼­ ¾×ü¼ººÐÀÌ ±«ÀúÀÇ Á¶Á÷¿¡ ¸¹ÀÌ À־ º¸±â¿¡ ½ÀÇØ º¸À̸é À̰ÍÀ» ½À¼º±«Àú(wet gangrene)À̶ó°í ÇÑ´Ù.
¿µ¹® fibrinoid necrosis ÇÑ±Û ¼¶À¯¼Ò¼º±«»ç
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  ÃʱâÀÇ ·ù¸¶Æ¼½º È¤Àº ¾Ç¼ºÁ¾¾ç µî¿¡¼­ º¼ ¼ö Àִ Ç÷°ü-°áÇÕÁ¶Á÷ÀǠƯÀ¯ÇÑ º¯È­·Î Ç÷°üº®À̳ª ÁÖÀ§ÀÇ °áÇÕÁ¶Á÷ÀÌ ¼¶À¯¼Ò¸ð¾çÀ¸·Î ±ÕÁúÇÑ ¹°Áú·Î µÇ°í, È£»ê¼ºÀ¸·Î ¿°»öµÈ´Ù. À̰ÍÀº ¼¶À¯¼Ò¿Í °ÅÀÇ ¶È°°Àº ¿°»ö¼ºÀ» ³ªÅ¸³»±â ¶§¹®¿¡ ¿°Áõ¿¡¼­ Ç÷°üÀ¸·ÎºÎÅÍ ¼¶À¯¼Ò ¶Ç´Â ¼¶À¯¼Ò¸ð¾çÀÇ ¹°ÁúÀÌ »ïÃâÇÏ¿© °áÇÕÁ¶Á÷ ¼¶À¯°£¿¡ Ä§ÂøÇÏ¿© ±¸Á¶°¡ ºÒ¸í·áÇÑ °ÍÀ¸·Î »ý°¢µÇ¾úÀ¸³ª, ÃÖ±Ù Á¶Á÷È­ÇÐ, ÀüÀÚÇö¹Ì°æ µî¿¡ ÀÇÇÏ¿© °áÇÕ¼¶À¯ÀÚü¿¡µµ º¯È­°¡ Àִ °ÍÀ¸·Î ¸é¿ª±Û·ÎºÒ¸°ÀÇ Ä§Âøµµ Áõ¸íµÇ°í ÀÖ´Ù.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • angiosclerotic necrosis
    Ç÷°ü°æÈ­±«»ç
  • anterior segment necrosis
    ¾È±¸¾ÕºÎºÐ±«»ç
  • arteriolar necrosis
    ¼¼µ¿¸Æ±«»ç
  • aseptic necrosis
    ¹«±Õ±«»ç
  • avascular necrosis
    ¹«Ç÷°ü±«»ç, ¹«Ç÷¼º±«»ç
  • acute tubular necrosis
    ±Þ¼º¿ä¼¼°ü±«»ç
  • bacillary necrosis
    ¸·´ë±Õ±«»ç
  • coagulation necrosis
    ÀÀ°í±«»ç
  • colliquative necrosis
    ¾×È­±«»ç
  • cortical necrosis
    °ÑÁú±«»ç, ÇÇÁú±«»ç
  • cystic medial necrosis
    ³¶¼ºÁß°£¸·±«»ç
  • caseous necrosis
    Ä¡Á»ç
  • central hemorrhagic necrosis
    Áß½ÉÃâÇ÷±«»ç
  • central necrosis
    Á߽ɱ«»ç
  • centrilobular necrosis
    Á߽ɼҿ±±«»ç
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • avascular necrosis of the femoral head
    ³Ò´Ù¸®»À¸Ó¸®¹«Ç÷°ü±«»ç, ´ëÅð°ñµÎ¹«Ç÷°ü±«»ç
  • necrosis
    񮯇
  • acute tubular necrosis
    ±Þ¼º¿ä¼¼°ü±«»ç, ±Þ¼º¼¼´¢°ü±«»ç
  • aseptic necrosis
    ¹«±Õ±«»ç
  • avascular necrosis
    ¹«Ç÷°ü±«»ç
  • caseous necrosis
    Ä¡Á»ç, °Ç¶ô±«»ç
  • coagulation necrosis
    ÀÀ°í±«»ç
  • cystic medial necrosis
    ³¶¼ºÁßÃþ±«»ç
  • fibrinoid necrosis
    ¼¶À¯¼Ò¼º±«»ç
  • gangrenous necrosis
    ±«Àú±«»ç
  • massive hepatic necrosis
    ±¤¹üÀ§°£±«»ç
  • postpartum pituitary necrosis
    »êÈijúÇϼöü±«»ç
  • radiation necrosis
    ¹æ»ç¼±±«»ç
  • renal papillary necrosis
    ÄáÆÏÀ¯µÎ±«»ç
  • subacute hepatic necrosis
    ¾Æ±Þ¼º°£±«»ç
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • tumor necrosis factor
    Á¾¾ç±«»çÀÎÀÚ
  • angiosclerotic necrosis
    Ç÷°ü°æÈ­±«»ç
  • anterior segment necrosis
    ¾È±¸¾ÕºÎºÐ±«»ç
  • arteriolar necrosis
    ¼¼µ¿¸Æ±«»ç
  • aseptic necrosis
    ¹«±Õ±«»ç
  • avascular necrosis
    ¹«Ç÷°ü±«»ç
  • bacillary necrosis
    °£±Õ¼º±«»ç
  • caseous necrosis
    Ä¡Á»ç, °Ç¶ô±«»ç
  • central hemorrhagic necrosis
    Áß½ÉÃâÇ÷±«»ç
  • centrilobular necrosis
    Á߽ɼҿ±±«»ç
  • coagulation necrosis
    ÀÀ°í±«»ç
  • colliquative necrosis
    ¾×È­±«»ç
  • cortical necrosis
    °ÑÁú±«»ç
  • cystic medial necrosis
    ³¶¼ºÁßÃþ±«»ç
  • fibrinoid necrosis
    ¼¶À¯¼Ò¼º±«»ç
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • TNF => tumor necrosis factor
    Á¾¾ç±«»çÀÎÀÚ
  • F factor (fertility factor)
    FÀÎÀÚ, ¼öÅÂÀÎÀÚ
  • GABA A receptors
    GABA A ¼ö¿ëü(áôé»ô÷)
  • GABA receptors
    GABA ¼ö¿ëü(áôé»ô÷)
  • GABAB receptors
    GABA B ¼ö¿ëü(áôé»ô÷)
  • H1 receptors
    H1¼ö¿ëü(â¥é»ô÷)
  • HT receptors
    5-ÇÏÀ̵å·Ï½ÃÆ®¸³Å¸¹Î¼ö¿ëü
  • HT2 receptors, action of hallucinogens
    5-ÇÏÀ̵å·Ï½ÃÆ®¸³Å¸¹Î2¼ö¿ëü
  • Hair follicle receptors
    ¸ð³¶¼ö¿ëü(Ù¾Ò¥â¥é»ô÷)
  • Hair receptors
    ¸ð¼ö¿ëü(Ù¾â¥é»ô÷)
  • Heat receptors
    ¿­¼ö¿ëü(æðâ¥é»ô÷)
  • Histamine receptors
    È÷½ºÅ¸¹Î¼ö¿ëü
  • Immunoglobulin adhesion receptors
    ¸é¿ª±Û·ÎºÒ¸° ºÎÂø(ݾó·)¼ö¿ëü
  • Irritant receptors, ventilatory responses , ventilatory responses
    Àڱؼö¿ëü(í©Ð½áôé»ô÷), È£Èí¹ÝÀÀ(û¼ýåÚãëë)
  • J receptors, ventilatory responses mediated
    J ¼ö¿ëü(áôé»ô÷), È£Èí¹ÝÀÀ(û¼ýåÚãëë)
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • tumor =tumour
    Á¾¾ç(ðþåË), Á¾Ã¢(ðþóì)
  • atrial receptors
    (½É)¹æ¼ö¿ëü(¡­áôé»ô÷)
  • atrial receptors
    ½É¹æ¼ö¿ëü(¡­áôé»ô÷).
  • down-regulation (of receptors)
    (¼ö¿ëü)ÇÏÇâÁ¶Àý.
  • down-regulation (of receptors)
    ¼ö¿ëü(áôé»ô÷)ÇÏÇâÁ¶Àý(ù»ú¾ðàï½).
  • presynaptic receptors
    ½Å°æÁ¢ÇÕÀü ¼ö¿ëü
  • receptors
  • tumor necrosis factor
    Á¾¾ç±«»çÀÎÀÚ
  • tumor necrosis factor
    Á¾¾ç±«»çÀÎÀÚ
  • tumor necrosis factor (TNF)
    Á¾¾ç ±«»ç ÀÎÀÚ
  • col factor(»ýÈ­) cÀÌ ÀÎÀÚ colicin factor
    Äݸ®½ÅÀÎÀÚ.
  • lupus erythematosus factor = LE factor
    È«¹Ý¼º ·çǪ½ºÀÎÀÚ(ûõÚèàõ¡­ì×í­)
  • time, dose and fractionation factor, TDF factor
    ½Ã°£¼±·®ºÐÇÒÀÎÀÚ
  • acute retinal necrosis
    ±Þ¼º¸Á¸·±«»ç
  • acute tubular necrosis
    ±Þ¼º ¼¼´¢°ü±«»ç (¡­á¬èñηÎÕÞÝ).
´ëÇѱâ»ýÃæÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • Duffy factor
    ´õÇÇÀÎÀÚ
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • tumor necrosis factor
    Á¾¾ç ±«»çÀÎÀÚ(ðþåËÎÕÞÝì×í­)
  • acetate-replacing factor
    ¾Æ¼¼Æ®»ê(ß«)ġȯ ÀÎÀÚ(öÇüµì×í­)
  • achromotrichia factor
    ¸ð¹ß»ö¼Ò°áÇÌÀÎÀÚ (Ù¾Û¥ßäáÈÌÀù¹ì×í­)
  • angiogenic factor
    Ç÷°üÇü¼ºÀÎÀÚ(úìηû¡à÷ì×í­)
  • animal protein factor
    µ¿¹°´Ü¹éÁúÀÎÀÚ(ÔÑÚªÓ±ÛÜòõì×í­)
  • anti-egg-white injury factor
    Ç׳­¹é ¼Õ»óÀÎÀÚ(ù÷ÑëÛÜáßß¿ì×í­)
  • anti-fatty-liver-factor
    Ç×Áö¹æ°£ÀÎÀÚ(ù÷ò·Û¸ÊÜì×í­)
  • anti-gray-hair factor
    Ç×¹é¸ð¹ß ÀÎÀÚ(ù÷ÛÜÙ¾Û¥ì×í­)
  • antiacrodynia factor
    Ç×Áö´Üµ¿Åë ÀÎÀÚ(ù÷ò¶Ó®ÔÙ÷Ôì×í­)
  • antianemia factor
    Ç׺óÇ÷ÀÎÀÚ(ù÷Þ¸úìì×í­)
  • antiberiberi factor
    Ç×°¢±âÀÎÀÚ(ù÷ÊÅѨì×í­)
  • antiblack-tongue factor
    Ç×Èæ¼³ÀÎÀÚ(ù÷ýÙàßì×í­)
  • antihemophilic factor
    Ç×Ç÷¿ìº´ÀÎÀÚ(ù÷úìéÒÜ»ì×í­)
  • antihemophilic factor B
    Ç×Ç÷¿ìº´ÀÎÀÚ (ù÷úìéÒÜ»ì×í­) B
  • antihemophilic factor C
    Ç×Ç÷¿ìº´ÀÎÀÚ (ù÷úìéÒÜ»ì×í­) C
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • attenuation factor
    °¨¾à¿ä¼Ò, °¨¼è¿äÀÎ
  • conversion factor
    º¯È¯ÀÎÀÚ
  • factor
    ÀÎÀÚ
  • predisposing factor
    ¼ÒÀÎ, Áúº´¼ÒÁú
  • Q factor
    Å¥ÀÎÀÚ
  • rheumatoid factor
    ·ù¸¶Æ¼½º¾çÀÎÀÚ
  • risk factor
    À§Çè¿äÀÎ
  • turbo factor
    Åͺ¸ÀÎÀÚ
  • avascular necrosis
    ÇãÇ÷±«»ç
  • caseous necrosis
    °Ç¶ô±«»ç, Ä¡Á»ç
  • ischemic necrosis
    ÇãÇ÷¼º±«»ç
  • medial necrosis
    ÁßÃþ±«»ç, µ¿¸Æ±ÙÃþ±«»ç
  • myocardial necrosis
    ½É±Ù±«»ç
  • necrosis
    񮯇
  • pressure necrosis
    ¾Ð¹Ú±«»ç
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
CF calcaneal fibular [ligament]; calcium leucovorin; calf blood flow; calibration factor; cancer-free; ...
EF ectopic focus; edema factor; ejection fraction; elastic fibril; electric field; elongation factor; e...
PF pair feeding; peak flow; perfusion fluid; pericardial fluid; periosteal fibroblast; peritoneal fluid...
AChRs Acetylcholine Receptors
RA radioactive; ragocyte; ragweed antigen; rapidly adapting [receptors]; reactive arthritis; reciprocal...
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
TNF alpha Anti-tumour necrosis factor alpha
rTNF Recombinant human tumour necrosis factor
rhTNF Recombinant human tumour necrosis factor
rhTNF-alpha Recombinant human tumour necrosis factor-alpha
TNF alpha Tumour Necrosis Factor alpha
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • tumor necrosis factor
    Á¾¾ç ±«»ç ÀÎÀÚ
    TNF. Á¾¾ç¿¡ °ü°èÇϰí ÀÖ´Â ¸é¿ª°èÀÇ ¼¼Æ÷ÀÎ Á¾¾ç ħÀ±¼º ¸²ÇÁ±¸
  • IGF-I : insulin like growth factor-IÀÇ ¾àÀÚ. ¼ºÀåÆÇÀÎ epiphyseal plate¿¡ ¿¬°ñ »ý¼ºÀ» Áõ°¡½ÃÄÑ »ÀÀÇ ¼ºÀåÀ» ÃËÁøÇÑ´Ù.

    IGF-II : insulin like growth factor-IIÀÇ ¾àÀÚ. ¸¹Àº Àå±â¿Í Á¶Á÷¿¡ ÀÛ¿ëÇÏ¿© ´Ü¹é ÇÕ¼º°ú DNA, RNAÀÇ ÇÕ¼ºÀ» Áõ°¡½ÃÄÑ ¼¼Æ÷ÀÇ ¼ö¿Í ¾çÀ» Áõ°¡

    Àν¶¸°¾ç ¼ºÀå ÀÎÀÚ °áÇÕ ´Ü¹éÁú
    insulinelike growth factor binding
  • single-factor versus multiple-factor analysis
    ´ÜÀÏ ¿ä¼Ò ºÐ¼® ´ë º¹ÇÕ ¿ä¼Ò ºÐ¼®
  • angiosclerotic necrosis
    Ç÷°ü °æÈ­¼º ±«»ç
  • avaseular necrosis
    ÇãÇ÷ ±«»ç
    µ¿ÀǾî=ischemic necrosis.
  • caseous necrosis
    °Ç¶ô ±«»ç, °Ç¶ô¼º ±«»ç
    µ¿ÀǾî=cheesy necrosis. °áÇÙ °¨¿°ÀÇ À°¾ÆÁ¾ Áß¾Ó¿¡¼­ º¼ ¼ö ÀÖ´Â ¶Ç ´Ù¸¥ Ư¡ÀûÀÎ ÀÀ°í ±«»çÀÇ ÀÏÁ¾. Á¶Á÷ÀÌ À¯¿¬ÇÏ°í °ÇÁ¶ÇÏ¿© cottage cheese¿Í Èí»çÇÑ ±«»ç. ´ëºÎºÐ °áÇÙ ¹× ¸Åµ¶¿¡¼­ º¼ ¼ö ÀÖ´Ù. °áÇÙ °¨¿° º´¼ÒÀÎ À°¾ÆÁ¾ÀÇ Áß¾Ó¿¡¼­ º¼ ¼ö ÀÖ´Â ¶Ç ´Ù¸¥ Ư¡ÀûÀÎ ÀÀ°í ±«»çÀÇ ÀÏÁ¾ÀÌ´Ù. °Ç¶ôÀ̶ó´Â ¿ë¾î´Â ±«»ç ºÎÀ§ÀÇ À°¾ÈÀûÀÎ ¸ð¾ç
  • central hemorrhagic necrosis
    ÃâÇ÷¼º Á᫐ Á¤¾× ±«»ç
  • central necrosis
    Á᫐ ±«»ç
  • colliquative necrosis
    ¾×È­, ±«»ç
  • contraction band necrosis
    ¼öÃà´ë ±«»ç
  • diffuse cortical necrosis
    ¹Ì¸¸¼º ÇÇÁú ±«»ç
  • electrical necrosis
    Àü±â¼º ±«»ç
  • embolic necrosis
    »öÀü¼º ±«»ç
  • fat necrosis
    Áö¹æ ±«»ç
  • liquefaction necrosis
    ¾×È­ ±«»ç
    °­ÇÑ °¡¼öºÐÇØ È¿¼ÒÀÇ ÀÛ¿ëÀ¸·Î ¹ß»ýÇÏ´Â ±«»ç.
CancerWEB ¿µ¿µ ÀÇÇлçÀü ¸ÂÃã °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
receptors, tumour necrosis factor Cell surface receptors that bind tumour necrosis factor and trigger changes which influence the behaviour of cells. The two recognised tumour necrosis factor receptors are designated alpha and beta receptors. Both receptors bind both alpha and beta tumour necrosis factors with high affinity, and both are members of the nerve growth factor receptor family.
(12 Dec 1998)
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
p60 tumour necrosis factor receptor-associated kinase <enzyme> Interacts with and causes phosphorylation of the cytoplasmic domain of the tnf receptor
Registry number: EC 2.7.10.-
Synonym: p60 tnf receptor-associated kinase, p60-trak
(26 Jun 1999)
tumour necrosis factor <cytokine> Originally described as a tumour inhibiting factor in the blood of animals exposed to bacterial lipopolysaccharide or Bacille Calmette-Guerin.
Preferentially kills tumour cells in vivo and in vitro, causes necrosis of certain transplanted tumours in mice and inhibits experimental metastases. Human Tumour Necrosis factor alpha is a protein of 157 amino acids and has a wide range of pro inflammatory actions. Usually considered a cytokine.
Synonym: cachectin.
Acronym: TNF
(13 Nov 1997)
tumour necrosis factor-beta <cytokine> A cytolytic factor that is produced by CD4 and CD8 T-cells after their exposure to an antigen.
(05 Mar 2000)
fat necrosis tumour <surgery> Destruction of fat cells in the breast due to trauma or injury that can cause a hard noncancerous lump.
(09 Oct 1997)
tumor necrosis factor <cytokine> Originally described as a tumour inhibiting factor in the blood of animals exposed to bacterial lipopolysaccharide or Bacille Calmette-Guerin.
Preferentially kills tumour cells in vivo and in vitro, causes necrosis of certain transplanted tumours in mice and inhibits experimental metastases. Human Tumour Necrosis factor alpha is a protein of 157 amino acids and has a wide range of pro inflammatory actions. Usually considered a cytokine.
Synonym: cachectin.
Acronym: TNF
(13 Nov 1997)
receptors, atrial natriuretic factor Cell surface proteins that bind atrial natriuretic factor with high affinity and trigger intracellular changes influencing the behaviour of cells.
(12 Dec 1998)
receptors, colony-stimulating factor Cell surface receptors for colony-stimulating factors, local mediators, and hormones that regulate the survival, proliferation, and differentiation of haemopoietic cells.
(12 Dec 1998)
receptors, epidermal growth factor-urogastrone Glycoproteins of about 170 kD that have protein kinase activity and span the plasma membranes of growing cells, including tumours. They are activated by the binding of epidermal growth factor-urogastrone which then initiates DNA and protein synthesis. They are not found on mitotically quiescent cells except in the stomach where they control the synthesis and release of digestive enzymes and gastric acid. Transforming growth factor alpha also binds to and activates these receptors.
(12 Dec 1998)
receptors, fibroblast growth factor Specific molecular sites or structures on cell membranes that react with fibroblast growth factors (both the basic and acidic forms), their analogs, or their antagonists to elicit or to inhibit the specific response of the cell to these factors. These receptors frequently possess tyrosine kinase activity.
(12 Dec 1998)
receptors, granulocyte-colony-stimulating factor Receptors that bind and internalise granulocyte-colony-stimulating factor. Their mw is believed to be 150 kD. These receptors are found mainly on a subset of myelomonocytic cells.
(12 Dec 1998)
receptors, granulocyte-macrophage colony-stimulating factor Receptors that bind and internalise the granulocyte-macrophage stimulating factor. Their mw is believed to be 84 kD. The most mature myelomonocytic cells, specifically human neutrophils, macrophages, and eosinophils, express the highest number of affinity receptors for this growth factor.
(12 Dec 1998)
receptors, growth factor Cell surface receptors that bind growth or trophic factors with high affinity, triggering intracellular responses which influence the growth, differentiation, or survival of cells.
(12 Dec 1998)
receptors, insulin-like-growth factor I Specific proteins on or in cells to which insulin-like growth factor I (somatomedin c) binds and thereby modifies the function of the cells. These receptors contain transmembrane and cytosolic domains, bind igf-I preferentially, and have high-affinity sites for igf-II. The alpha-subunit has a mw of 130 kD and the beta subunit possesses tyrosine kinase activity.
(12 Dec 1998)
receptors, insulin-like-growth-factor II Specific proteins on or in cells to which insulin-like growth factor II and mannose-6-phosphate bind and thereby modify the function of the cells. These receptors have a mw of 250 kD and possess no tyrosine kinase activity.
(12 Dec 1998)
receptors, macrophage colony-stimulating factor Glycoproteins of mw 165 kD which are encoded by the c-fms proto-oncogene. The binding of csf-1 to its receptors activates an intrinsic tyrosine kinase activity resulting in autophosphorylation of the receptors on tyrosine, rapid receptor down-regulation, and phosphorylation of as yet unidentified physiologic substrates that initiate a mitogenic response.
(12 Dec 1998)
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