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| ER | efficiency ratio; epigastric region; ejection rate; electroresection; emergency room; endoplasmic re... |
|---|---|
| RAR | rapidly adapting receptor; rat insulin receptor; retinoic acid receptor; right arm reclining; right ... |
| CR | calculation rate; calculus removed; calorie-restricted; cardiac rehabilitation; cardiac resuscitatio... |
| CRL | cell repository line; Certified Record Librarian; complement receptor location; complement receptor ... |
| DR | degeneration reaction; delivery room; deoxyribose; diabetic retinopathy; diagnostic radiology; digit... |
| CRLR | Calcitonin Receptor-Like Receptor |
|---|---|
| EGF-receptor | Epidermal Growth Factor receptor |
| IRR | Insulin receptor- related receptor |
| alpha 2MR/LRP | alpha (2)-macroglobulin receptor/low density lipoprotein receptor-related protein |
| ORL1 | opioid receptor like receptor |
| amblyopia | <ophthalmology> The impairment of vision without detectable organic lesion of the eye. (18 Nov 1997) |
|---|---|
| amblyopia ex anopsia | The suppression of the central vision in one eye when the images from the two eyes are so different that they cannot be fused into one. This may be due to: 1) faulty image formation (sensory amblyopia); 2) a large difference in refraction between the two eyes (anisometropic amblyopia); or 3) the two eyes pointing in different directions (strabismic amblyopia). Most suppression amblyopia can be reversed if appropriately treated before age 6 years. Synonym: amblyopia ex anopsia. (05 Mar 2000) |
| anisometropic amblyopia | A suppression of central vision due to an unequal refractive error (anisometropia) of at least two diopters. This induces a sufficient difference in image size (aniseikonia) that the two images cannot be fused. In order to avoid confusion, the blurrier image is suppressed. Synonym: refractive amblyopia. (05 Mar 2000) |
| refractive amblyopia | A suppression of central vision due to an unequal refractive error (anisometropia) of at least two diopters. This induces a sufficient difference in image size (aniseikonia) that the two images cannot be fused. In order to avoid confusion, the blurrier image is suppressed. Synonym: refractive amblyopia. (05 Mar 2000) |
| sensory amblyopia | A suppression of central vision in one eye due to faulty image formation; for example, by a corneal scar, a cataract, or a droopy eyelid. Synonym: deprivation amblyopia. (05 Mar 2000) |
| hysterical amblyopia | An apparent loss of visual acuity or visual field with no substantiating physical signs; often due to a natural concern about visual loss combined with suggestibility and a fear of the worst; best treated with reassurance. (05 Mar 2000) |
| strabismic amblyopia | A suppression of central vision due to the two eyes pointing in different directions. The two scenes cannot be fused into a single image, so, to avoid confusion, one of the images is suppressed. (05 Mar 2000) |
| nocturnal amblyopia | Night blindness or difficulty in seeing at night. Symptom of vitamin A deficiency. (27 Sep 1997) |
| nutritional amblyopia | Amblyopia resulting from lack of vitamin B-complex constituents. (05 Mar 2000) |
| suppression amblyopia | The suppression of the central vision in one eye when the images from the two eyes are so different that they cannot be fused into one. This may be due to: 1) faulty image formation (sensory amblyopia); 2) a large difference in refraction between the two eyes (anisometropic amblyopia); or 3) the two eyes pointing in different directions (strabismic amblyopia). Most suppression amblyopia can be reversed if appropriately treated before age 6 years. Synonym: amblyopia ex anopsia. (05 Mar 2000) |
| deprivation amblyopia | A suppression of central vision in one eye due to faulty image formation; for example, by a corneal scar, a cataract, or a droopy eyelid. Synonym: deprivation amblyopia. (05 Mar 2000) |
| toxic amblyopia | Blindness due to optic neuritis caused by methyl alcohol, lead, arsenic, quinine, or other poisons. (05 Mar 2000) |
| acetylcholine receptor antibodies | <neurology, investigation> A test used to measure the amount of antibodies to acetylcholine receptors on nerve endings. This is a diagnostic test for myasthenia gravis. A normal value is no antibodies in the bloodstream. Acetylcholine receptor (AChR) binding autoantibodies (i.e. Antibodies reactive with several epitopes other than the binding site for acetylcholine or alpha-bungarotoxin) are present in approximately 88% of patients with generalised myasthenia gravis, 70% of ocular myasthenia and in approximately 80% of myasthenia gravis in remission. Although serum concentrations of AChR binding autoantibodies do not in general correlate well with severity of weakness, there is typical decrease in concentration as weakness improves with immunosuppressive therapy. AChR blocking autoantibodies (i.e., antibodies reactive with the AChR binding site) are present in about 50% of patients with myasthenia gravis, 30% with ocular myasthenia gravis and 20% of myasthenia gravis in remission, AChR blocking autoantibodies are the only AChR autoantibodies present in about 1% of myasthenia gravis. AChR modulating autoantibodies (i.e., autoantibodies which cross-link AChRs and cause their removal from muscle membrane surfaces) are present in more than 90% of myasthenia gravis and occasionally are the only AchR autoantibodies detectable in mild, recent onset or ocular-restricted myasthenia gravis. Results for AChR modulating autoantibodies can be transiently false-positive due to curare-like drugs used during general anesthesia. AChR autoantibodies of one or more types are found in at least 80% of ocular myasthenia gravis. Although generally absent in neurological conditions other than myasthenia gravis(and consequently unlikely to cause confusion in neurodiagnosis), false-positive results for AChR autoantibodies occasionally occur in primary biliary cirrhosis, tardive dyskinesia, autoimmune thyroiditis, the elderly, amyotrophic lateral sclerosis patients treated with cobra venom and patients with thymoma in the absence of myasthenia gravis. Approximately 1% of patients with rheumatoid arthritis treated with D-penicillamine develop AChR autoantibodies and myasthenia gravis, both of which disappear when the drug is discontinued. Babies born to ~10% of myasthenia gravis mothers have a transient neonatal form of myasthenia gravis that responds well to anticholinesterase therapy and usually remits within 1 month as maternal IgG disappears. (29 Dec 1997) |
| amino acid receptor | <biochemistry> Ligand gated ion channels with specific receptors for amino acid transmitters. An extended protein superfamily that also includes subunits of the nicotinic acetylcholine receptor. (18 Nov 1997) |
| AMPA receptor | <cell biology> Glutamate operated ion channel. See: excitatory amino acid receptor channels. (05 Feb 1998) |
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