| ¿µ¹® | non-Hodgkin lymphoma | ÇÑ±Û | ºñÈ£ÁöŲ ¸²ÇÁÁ¾ |
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| ¼³¸í | ¸²ÇÁÁ¾Àº ¸²ÇÁÀý¿¡ »ý±ä ¾Ç¼º Á¾¾çÀ¸·Î ¹éÇ÷º´°ú´Â ´Ù¸£´Ù. ¹éÇ÷º´Àº °ñ¼ö¿¡¼ »ý±ä ¸²ÇÁ±¸¼º ¾Ç¼ºÁ¾¾çÀÌÁö¸¸, ¸²ÇÁÁ¾Àº ¸²ÇÁÁ¶Á÷¿¡¼ »ý±ä ¸²ÇÁ±¸¼º ¾Ç¼ºÁ¾¾çÀÌ´Ù. Å©°Ô ¸²ÇÁÁ¾Àº 2°¡Áö·Î ³ª´ ¼ö ÀÖ´Â µ¥, È£ÁöŲº´(Hodgkin's lymphoma)°ú ºñÈ£ÁöŲ¸²ÇÁÁ¾(non-Hodgkin's lymphoma)À¸·Î ³ª´ ¼ö ÀÖ´Ù. ¶ÇÇÑ ºñÈ£ÁöŲ¸²ÇÁÁ¾Àº ¿©·¯ °¡ÁöÀÇ ¼¼Æ÷ÇüÅ¿¡ µû¶ó ³ª´©°í ÀÖ´Ù. ÀÌ º´Àº ´ÜÁö ¸²ÇÁÀý»Ó¸¸ ¾Æ´Ï¶ó Áö¶ó, °ñ¼ö, ÀÌ¿Ü ´Ù¸¥ ±â°üÀ» ħ¹üÇÔÀ¸·Î½á ¾Ç¼ºÀ̸ç, »ý¸í¿¡ Å« À§ÇùÀ» ÁØ´Ù. ¾Ç¼º¸²ÇÁÁ¾ÀÇ ÀÏÁ¾. ÀüÀÌÀÇ ÇüŰ¡ ´Ù¾çÇÏ¿©, ÁÖÀ§ ¸²ÇÁÀýÀ» °Ç³Ê¶Ù¾î ´Ù¸¥ ¸²ÇÁÀý·Î ÀüÀÌÇϱ⵵ ÇÑ´Ù. ¶ÇÇÑ ¼¼Æ÷ÀÇ ÇüÅ¿¡ µû¶ó ¿©·¯ °¡Áö Á¾·ù°¡ ÀÖÀ¸¸ç ´ë°³ Ä¡·á´Â ¹æ»ç¼±Ä¡·á¿Í ÈÇпä¹ýÀ» º¹ÇÕÇÏ¿© »ç¿ëÇÑ´Ù. |
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| ¿µ¹® | acidosis | ÇÑ±Û | »êÁõ, ¾Æ½Ãµµ½Ã½º |
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| ¼³¸í | ¸ö¼ÓÀÇ Ã¼¾×ÀÌ »ê¼ºÈ µÇ·Á´Â °æÇâ. ¸ö¼Ó¿¡ »êÀÌ ÃàÀûµÇ°Å³ª ¾ËÄ®¸®ÀÇ °¨¼Ò°¡ »ý±æ ¶§¿¡ ÀϾÙ. |
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| ¿µ¹® | severe acute respiratory syndrome(SARS) | ÇÑ±Û | »ç½º |
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| ¼³¸í | Áß±¹ ±¤µ¿ Áö¿ª¿¡¼ °¡Àå ¸ÕÀú ¹ß»ýÇÑ Àü¿°¼º È£Èí±â ÁúȯÀ¸·Î ¼¼°èº¸°Ç±â±¸(WHO)¿¡¼ ¡®ÁßÁõ±Þ¼ºÈ£ÈíÁõÈıº(SARS)'À¸·Î ¸í¸íÇß´Ù. ¼·¾¾ 38µµ ÀÌ»óÀÇ °í¿°ú ±âħ, È£Èí°ï¶õ, Àú»ê¼ÒÁõ, X¼±»óÀÇ Æó·ÅÁõ»ó Áß Çϳª ÀÌ»óÀÇ Áõ»óÀÌ ³ªÅ¸³ª¸ç, µÎÅë, ±ÙÀ°Åë, ½Ä¿åºÎÁø, ÇǷΰ¨, ¹ßÁø, ¼³»ç¸¦ µ¿¹ÝÇÒ ¼ö ÀÖ´Ù. Ãʱâ Áõ»óÀº °¨±â¿Í ºñ½ÁÇÏÁö¸¸ Æó·ÅÀ¸·Î ¹ßÀüÇϸé Ä¡¸íÀûÀÏ ¼ö ÀÖ´Ù. ÇöÀç ¹àÇôÁø °¨¿°°æ·Î´Â ȯÀÚ°¡ Àçä±â³ª ±âħÇÒ ¶§ ³»»Õ´Â ħ¹æ¿ïÀ̰í, À̰ÍÀÌ ´Ù¸¥ »ç¶÷ÀÇ È£Èí±â·Î µé¾î°¥ ¶§ Àü¿°µÈ´Ù. ħ¹æ¿ïÀÌ Àü´ÞµÇ´Â °Å¸®´Â º¸Åë 1m·Î º¸°í ÀÖ´Ù. °ø±â¸¦ ÅëÇØ Àü¿°ÀÌ °¡´ÉÇÏ´Ù´Â ÁÖÀåÀÌ Á¦±âµÆÁö¸¸ ¾ÆÁ÷ È®ÀεÇÁö ¾Ê¾Ò´Ù. ¿øÀαÕÀº º¯Á¾ Äڷγª¹ÙÀÌ·¯½º·Î ¹àÇôÁ³´Ù. |
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| ¿µ¹® | severe acute respiratory syndrome(SARS) | ÇÑ±Û | ÁßÁõ±Þ¼ºÈ£ÈíÁõÈıº |
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| ¼³¸í | Áß±¹ ±¤µ¿ Áö¿ª¿¡¼ °¡Àå ¸ÕÀú ¹ß»ýÇÑ Àü¿°¼ºÈ£Èí±âº´À¸·Î ¼¼°èº¸°Ç±â±¸(WHO)¿¡¼ ¡®ÁßÁõ±Þ¼ºÈ£ÈíÁõÈıº(SARS)'À¸·Î ¸í¸íÇß´Ù. ¼·¾¾ 38µµ ÀÌ»óÀÇ °í¿°ú ±âħ, È£Èí°ï¶õ, Àú»ê¼ÒÁõ, X¼±»óÀÇ Æó·ÅÁõ»ó Áß Çϳª ÀÌ»óÀÇ Áõ»óÀÌ ³ªÅ¸³ª¸ç, µÎÅë, ±ÙÀ°Åë, ½Ä¿åºÎÁø, ÇǷΰ¨, ¹ßÁø, ¼³»ç¸¦ µ¿¹ÝÇÒ ¼ö ÀÖ´Ù. Ãʱâ Áõ»óÀº °¨±â¿Í ºñ½ÁÇÏÁö¸¸ Æó·ÅÀ¸·Î ¹ßÀüÇϸé Ä¡¸íÀûÀÏ ¼ö ÀÖ´Ù. ÇöÀç ¹àÇôÁø °¨¿°°æ·Î´Â ȯÀÚ°¡ Àçä±â³ª ±âħÇÒ ¶§ ³»»Õ´Â ħ¹æ¿ïÀ̰í, À̰ÍÀÌ ´Ù¸¥ »ç¶÷ÀÇ È£Èí±â·Î µé¾î°¥ ¶§ Àü¿°µÈ´Ù. ħ¹æ¿ïÀÌ Àü´ÞµÇ´Â °Å¸®´Â º¸Åë 1m·Î º¸°í ÀÖ´Ù. °ø±â¸¦ ÅëÇØ Àü¿°ÀÌ °¡´ÉÇÏ´Ù´Â ÁÖÀåÀÌ Á¦±âµÆÁö¸¸ ¾ÆÁ÷ È®ÀεÇÁö ¾Ê¾Ò´Ù. ¿øÀαÕÀº º¯Á¾ Äڷγª¹ÙÀÌ·¯½º·Î ¹àÇôÁ³´Ù. |
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| ¿µ¹® | respiratory distress syndrome(RDS) | ÇÑ±Û | È£Èí°ï¶õÁõÈıº |
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| ¼³¸í | ÆóÆ÷¿Í Æó¸ð¼¼Ç÷°ü »çÀÌ¿¡ ºÎÁ¾À¸·Î ÀÎÇÑ È®»ê´É °¨¼Ò·Î È£Èí°ï¶õ°ú û»öÁõÀ» º¸ÀÌ´Â »óÅ·Π°¨¿°, ¼ö¼ú, ¿Ü»ó µî ¸ðµç Á¾·ùÀÇ ½ºÆ®·¹½º»óȲ¿¡¼ ¹ß»ýÇÒ ¼ö ÀÖ´Ù. Ä¡·á´Â ¼±Çà ¿äÀÎÀÇ ±³Á¤°ú ÀûÀýÇÑ Ç÷¾×³» »ê¼Ò³óµµ À¯ÁöÀÌ´Ù. |
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| ARD | absolute reaction of degeneration; acute radiation disease; acute respiratory disease; adult respira... |
|---|---|
| RC | an electronic circuit containing a resistor and capacitor in series; radiocarpal; reaction center; r... |
| ECG | Electro-Cardio-Graphy(-Gram); ½ÉÀüµµ = EKG 1. Conducting System Structu... |
| NANB | non-A, non-B [hepatitis] |
| NANBH | non-A, non-B hepatitis |
| CMA | Chronic metabolic acidosis |
|---|---|
| DRTA | Distal renal tubular acidosis |
| MA | Metabolic acidosis |
| MELAS | Mitochondrial Encephalomyopathy with Lactic Acidosis and Stroke-like episodes |
| MELAS | Mitochondrial Encephalomyopathy, Lactic Acidosis, and Stroke-like episodes |
| non-A, non-B, non-C hepatitis | Hepatitis caused by viral organisms other than hepatitis viruses A, B or C. (05 Mar 2000) |
|---|---|
| respiratory acidosis | <biochemistry> A metabolic derangement of acid-base balance where the blood pH is abnormally low. Causes include haemorrhagic shock, cardiogenic shock, severe dehydration, sepsis, toxic ingestion (for example isopropyl alcohol, methanol), alcoholic ketoacidosis, lactic acidosis, renal failure and diabetic ketoacidosis. Respiratory acidosis will occur if the lungs are not ventilating properly resulting in an excess of carbon dioxide in the body. (25 Jun 1999) |
| compensated respiratory acidosis | Retention of bicarbonate by the renal tubules to minimise the effect on the pH of the blood of retention of carbon dioxide by the lungs, such as occurs with hypoventilation. (05 Mar 2000) |
| hepatitis non-A, non-B | <virology> A virus somewhat similar in size to Hepatitis A but has no antigenic cross reaction with either A or B. Many of the cases are in fact hepatitis C and this was the old term for hepatitis C. (20 Sep 2002) |
| non-A, non-B hepatitis | Hepatitis caused by two or more infectious agents not detectable by methods that reveal the presence of hepatitis viruses A and B; one cause, now called type C hepatitis has been identified; may follow blood transfusion and is often seen in chronic renal dialysis patients. Synonym: NANB hepatitis. (05 Mar 2000) |
| non-A, non-B hepatitis virus | Term used to group any of a number of viruses, other than A or B, which cause hepatitis in humans. (05 Mar 2000) |
| acidosis | <biochemistry> A metabolic condition, characterised by an increase in hydrogen ion concentration, that occurs when the body is no longer able to buffer free hydrogen ions in the blood, resluting from either the accumulation of acid or depletion of the alkaline reserve (bicarbonate) in the blood and body tissues. This usually causes the pH of the blood to drop (and become more acidic). Compare: alkalosis. (10 May 1997) |
| carbon dioxide acidosis | <biochemistry> A metabolic derangement of acid-base balance where the blood pH is abnormally low. Causes include haemorrhagic shock, cardiogenic shock, severe dehydration, sepsis, toxic ingestion (for example isopropyl alcohol, methanol), alcoholic ketoacidosis, lactic acidosis, renal failure and diabetic ketoacidosis. Respiratory acidosis will occur if the lungs are not ventilating properly resulting in an excess of carbon dioxide in the body. (25 Jun 1999) |
| renal tubular acidosis | <nephrology> A rare sometimes familial disorder of the renal tubule characterised by the inability to excrete urine of normal acidity. This leads to a hyperchloraemic acidosis which is often associated with one or more secondary complications such as hypercalcinuria with nephrolithiasis and nephrocalcinosis, rickets, or osteomalacia and severe potassium depletion. (25 Jun 1999) |
| metabolic acidosis | <biochemistry> A metabolic derangement of acid-base balance where the blood pH is abnormally low. Causes include haemorrhagic shock, cardiogenic shock, severe dehydration, sepsis, toxic ingestion (for example isopropyl alcohol, methanol), alcoholic ketoacidosis, lactic acidosis, renal failure and diabetic ketoacidosis. Respiratory acidosis will occur if the lungs are not ventilating properly. (27 Jun 1999) |
| compensated acidosis | An acidosis in which the pH of body fluids is normal; compensation is achieved by respiratory or renal mechanisms. (05 Mar 2000) |
| primary renal tubular acidosis | A metabolic defect in the mechanism of urinary acidification that may be either the transient type, with onset in infancy, or the persistent type, with onset in childhood or adult years; both types are familial. (05 Mar 2000) |
| secondary renal tubular acidosis | Renal tubular acidosis that may occur as a complication of hypercalcaemic states, hyperglobulinaemic disorders, and in some other chronic renal conditions; a regular component of De Toni-Fanconi syndrome. (05 Mar 2000) |
| hyperchloraemic acidosis | <nephrology> A rare sometimes familial disorder of the renal tubule characterised by the inability to excrete urine of normal acidity. This leads to a hyperchloraemic acidosis which is often associated with one or more secondary complications such as hypercalcinuria with nephrolithiasis and nephrocalcinosis, rickets, or osteomalacia and severe potassium depletion. (25 Jun 1999) |
| starvation acidosis | Ketoacidosis resulting from lack of food intake, leading to fat catabolism to provide energy, releasing acidic ketone bodies. (05 Mar 2000) |
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