| ¿µ¹® | multiple sclerosis | ÇÑ±Û | ´Ù¹ß°æÈÁõ |
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| ¼³¸í | ½Å°æÃà»èÀ» µÑ·¯½Î°í ÀÖ´Â ¸»ÀÌÁý(myelin sheath)ÀÇ ÆÄ±«·Î ÀÎÇÑ º´Àû»óŸ¦ ¸»ÇÔ. ÆÄ±«µÈ ¸»ÀÌÁýÀº ÈäÅ͸¦ ³²±â°Ô µÇ¾î ½Å°æÃà»èÀ» ÅëÇÑ ½Å°æÀü´ÞÀÌ Á¦´ë·Î µÇÁö ¾Ê¾Æ ¿îµ¿, °¨°¢, ÀÚÀ²½Å°æ ¸ðµÎÀÇ ½Å°æÀü´ÞÀå¾Ö°¡ ³ªÅ¸³´Ù. ÀÌ º´ÅÍ´Â ¾îµð¼³ª ³ªÅ¸³¯ ¼ö ÀÖ¾î¼ ±× Àå¾Ö°¡ ³ªÅ¸³ª´Â ºÎÀ§¿¡ µû¶ó ¼·Î ´Ù¸¥ Áõ»óÀ» È£¼ÒÇÑ´Ù. |
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| ¿µ¹® | multiple myeloma | ÇÑ±Û | ´Ù¹ß°ñ¼öÁ¾ |
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| ¼³¸í | ´Ù¹ß¼º ¿ø¹ß¼º°ñÁ¾¾ç. ¸Ó¸®»À-°¥ºñ»À-º¹Àå»À-ôÃß»À-°ñ¹Ý µî¿¡ Àß ³ªÅ¸³ª°í, ¹°··¹°··ÇÑ Á¾±«¸¦ Çü¼ºÇϸç, »ÀÀÇ Èí¼ö°¡ ÀϾ°í, 40~60¼¼ ³²ÀÚ¿¡°Ô ¸¹ÀÌ ¹ß»ýÇÑ´Ù. °ñ¼öÁ¾ Á¾¾ç¼¼Æ÷´Â ÇüÁú¼¼Æ÷¿¡¼ À¯·¡ÇÑ °ÍÀÌ¾î¼ ÇüÁú¼¼Æ÷Á¾À̶ó°íµµ ÇÑ´Ù. °ú°Å¿¡´Â ÇüÁú¼¼Æ÷¼º°ñ¼öÁ¾ À̿ܿ¡´Â ´Ù¸¥ °ñ¼öÁ¶Ç÷¿ä¼Ò¿¡¼ »ý±â´Â °ñ¼öÁ¾À̶ó°í »ý°¢ÇßÁö¸¸ ÇöÀç´Â ºÎÁ¤µÇ°í ÀÖ´Ù. ÇüÁú¼¼Æ÷´Â ¿ø·¡ ¸é¿ª±Û·ÎºÒ¸°À» »ý»êÇÏ´Â ¼¼Æ÷À̸ç, ±×°ÍÀÌ Á¾¾çÈÇÑ ´Ù¹ß °ñ¼öÁ¾ ȯÀÚ¿¡¼µµ ´ëºÎºÐ Ç÷û ¼Ó¿¡ ¸é¿ª ±Û·ÎºÒ¸°ÀÌ Áõ°¡µÈ °ÍÀ» º¼ ¼ö ÀÖ´Ù. Áõ°¡ÇÑ ±Û·ÎºÒ¸°Àº IgG³ª IgAÀÎ °æ¿ì°¡ ¸¹Áö¸¸ ´Ù¸¥ Çüµµ ÀÖ´Ù. °ñ¼öÁ¾ ȯÀÚ ¾à 50%´Â ¿ÀÁÜ¿¡¼ º¥½ºÁÔ½º´Ü¹éÁúÀÌ °ËÃâµÇ´Âµ¥, ÀÌ ´Ü¹éÁúÀÇ ÃàÀû¿¡ ÀÇÇØ ¿ä¼¼°üÀÌ ÆÄ±«µÇ°í, ÄáÆÏ°æÈ°¡ ÀϾÙ. °ñ¼öÁ¾ ȯÀÚ¿¡¼´Â Ç÷û´Ü¹é ÀÌ»óÀ¸·Î °¡²û ¾Æ¹Ð·ÎÀ̵åÁõÀÌ ³ªÅ¸³´Ù. »À X¼± ¼Ò°ßÀ¸·Î¼´Â µµ·Á³½ º´ÅÍ, °ñÀ¶ÇØ»ó, º´Àû°ñÀýÀÌ °üÂûµÈ´Ù. |
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| ¿µ¹® | multiple personality | ÇÑ±Û | ´ÙÀμº ÀÎ°Ý |
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| ¼³¸í | ÇØ¸®¼º Á¤½ÅÀå¾ÖÀÇ Çϳª·Î ³ªÅ¸³´Ù. ÇÑ »ç¶÷ÀÌ ¿©·¯ »ç¶÷ÀÇ ¼º°ÝÀ» ¼ÒÀ¯Çϰí ÀÖ´Â °ÍÀ¸·Î ¸¶Ä¡ ¡°Áöų¹Ú»ç¿Í ÇÏÀÌµå ¾¾¡±¿Í °°Àº °æ¿ìÀÌ´Ù. ¾Æ¸¶, ÇöÀç ÀÚ½ÅÀÇ Ã³Áö¿¡¼ ¹þ¾î³ª°í ½ÍÀº ¹«ÀǽÄÀûÀÎ ¿å¸Á¿¡¼ ºñ·ÔµÇ´Â °ÍÀ¸·Î ¿©°ÜÁø´Ù. |
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| ¿µ¹® | congenital syphilis | ÇÑ±Û | ¼±Ãµ¸Åµ¶ |
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| ¼³¸í | ÀӺΰ¡ ¸Åµ¶¿¡ °¨¿°µÇ¾î ÀÖÀ¸¸é ÀӽŠÈı⿡ ¸Åµ¶±ÕÀÌ Å¹ÝÀ» ÅëÇØ Ç÷Ç༺À¸·Î žƿ¡ °¨¿°(¼öÁ÷°¨¿°)µÈ °ÍÀ» ¸»ÇÏ´Ù. ´ëºÎºÐÀº À¯»ê, »ç»êÀÌ µÇÁö¸¸ Ãâ»ýÇϸé Á¦2±â ÀÌÈÄÀÇ ¹ßÁøÀ» º¸ÀδÙ. ¹ßÇö½Ã±â¿¡ µû¶ó¼ ¨ç žƸŵ¶, ¨è À¯¾Æ¸Åµ¶, ¨é ¸¸¹ß¼º ¼±Ãµ¸Åµ¶À¸·Î ºÐ·ùµÈ´Ù. ¨ç¿¡¼´Â »À¿¬°ñ¿°, °£-Áö¶ó ºñ´ë¿Í ¸Åµ¶¼º õÆ÷â, ¨è¿¡¼´Â ÆÄ·Î°¡¼º¸¶ºñ¿Í ¸Åµ¶¼º ÄÚ¿°, ¨é¿¡¼´Â ÇãÄ£½¼ ¼¼Â¡ÈÄ(ÇãÄ£½¼ Ä¡¾Æ, ¼Ó±Í¼º ³Ã», ½ÇÁú¼º °¢¸·¿°)¿¡ µû¶ó Ư¡ÀÌ ÀÖ´Ù. ±âŸ ¼öµÎÁõ, Áö´É¹ßÀ° ºÒ·® µîÀ» ÀÚÁÖ º¼ ¼ö ÀÖ´Ù. ¸Åµ¶ Ç÷û¹ÝÀÀÀº ´ëºÎºÐÀÇ °æ¿ì ¾ç¼ºÀ¸·Î ³ª¿Â´Ù. ¸Å¿ì µå¹°°Ô °£¼¼Æ÷³»¿¡¼ ¸Åµ¶±ÕÀ» ¹«¼öÈ÷ º¼ ¼ö ÀÖ´Ù. °£¼¼Æ÷ ÁÖº¯ÀÇ ¼¶À¯È¿Í ÇÔ²² ºÒ±ÔÄ¢ÇÑ ÈäÅÍ(hepar lobatum)¸¦ ¸¸µé ¼ö ÀÖ´Ù. |
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| ¿µ¹® | congenital rubella syndrome | ÇÑ±Û | ¼±ÃµÇ³ÁøÁõÈıº |
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| ¼³¸í | ÀӽűⰣ Áß¿¡ »ê¸ð°¡ dzÁø¿¡ °É¸®¸é ÀÌ Ç³Áø ¹ÙÀÌ·¯½º´Â ŹÝÀ» ÅëÇØ¼ žƿ¡°Ô Àü´ÞµÇ¾î¼ žÆÀÇ Ç³Áø°¨¿°À» ÀÏÀ¸Å²´Ù. ÀӽŠù 3°³¿ù µ¿¾È, ƯÈ÷ ÀӽŠù´Þ¿¡ žư¡ dzÁøÀÇ °¨¿°À» ¹ÞÀ¸¸é, ½Å»ý¾Æ¿¡¼ ¼±Ãµ±âÇü, Áï ´«¿¡¼ ÃÐÁ¡À» Á¤È®È÷ ¸ÂÃß¾îÁÖ´Â ·»ÁîÀÇ ¿ªÇÒÀ» ÇÏ´Â ¼öÁ¤Ã¼ÀÇ È¥Å¹(¹é³»Àå), ½ÉÀå±âÇü, ±Í¸Ó°Å¸® ¹× ½ÉÇÑ Áö´É¹Ú¾àÀ» µ¿¹ÝÇÏ´Â ¼ÒµÎÁõ µîÀÌ ¹ß»ýÇÏ´Â ¼ö°¡ ¸¹´Ù. |
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| ECG | Electro-Cardio-Graphy(-Gram); ½ÉÀüµµ = EKG 1. Conducting System Structu... |
|---|---|
| CDH | 1) Chronic Daily Headache = CTH = ... |
| CDH | ceramide dihexoside; congenital diaphragmatic hernia; congenital dislocation of hip; congenital dysp... |
| AP | accessory pathway; accounts payable; acid phosphatase; acinar parenchyma; action potential; active p... |
| APC | acetylsalicylic acid, phenacetin, and caffeine; activated protein C; adenoidal-pharyngeal-conjunctiv... |
| MLP | Multiple lymphomatous polyposis |
|---|---|
| MCA | Multiple congenital anomalies |
| MCA/MR | multiple congenital anomalies mental retardation |
| MCA | multiple congenital anomaly |
| APC | Adenomatous Polyposis Coli |
| multiple intestinal polyposis | Begins usually in late childhood; polyps increase in numbers, causing symptoms of chronic colitis, and carcinoma of the colon almost invariably develops in untreated cases; autosomal dominant inheritance. In the Gardner syndrome there are extracolonic changes (desmoid tumours, etc.). Synonym: polyposis coli. Hamartomatous polyposis of the small or large intestine, Peutz-Jeghers syndrome with melanin spots on the lips, less common, miscellaneous, rare, and doubtful occurrences. Synonym: familial intestinal polyposis. (05 Mar 2000) |
|---|---|
| adenomatous polyposis coli | An autosomal dominant polyposis syndrome in which the colon contains few to thousands of adenomatous polyps, often occurring by age 15 to 25. (12 Dec 1998) |
| polyposis | Presence of several polyps. Origin: polyp + G. -osis, condition (05 Mar 2000) |
| polyposis coli | Hereditary disorder (Mendelian dominant) characterised by the development of hundreds of adenomatous polyps in the large intestine, which show a tendency to progress to malignancy. The APC gene has also been implicated in a chromosome 5 gastric and pancreatic cancer. (18 Nov 1997) |
| polyposis syndromes | <radiology> Inher. Malig. Type familial polyposis coli dom and adenoma Gardner syndrome dom and Turcot syndrome rec CNS Peutz-Jeghers syndrome dom (+) hamartoma Cowden syndrome dom ? juvenile polyposis coli (?) - juvenile Cronkhite-Canada syndrome (12 Dec 1998) |
| juvenile polyposis coli | <radiology> Benign polyposis, inheritance uncertain, inflammatory or retention polyps: round, smooth, soft, mucin-filled, non-neoplastic, onset less than 10 yrs, polyps can prolapse through anus, associated with diarrhoea, protein loss see: polyposis syndromes, Cronkhite-Canada syndrome (12 Dec 1998) |
| familial adenomatous polyposis | <gastroenterology> Genetic disease with numerous precancerous polyps in the colon and rectum. Also called familial polyposis. (12 Dec 1998) |
| familial intestinal polyposis | Begins usually in late childhood; polyps increase in numbers, causing symptoms of chronic colitis, and carcinoma of the colon almost invariably develops in untreated cases; autosomal dominant inheritance. In the Gardner syndrome there are extracolonic changes (desmoid tumours, etc.). Synonym: polyposis coli. Hamartomatous polyposis of the small or large intestine, Peutz-Jeghers syndrome with melanin spots on the lips, less common, miscellaneous, rare, and doubtful occurrences. Synonym: familial intestinal polyposis. (05 Mar 2000) |
| familial polyposis | An inherited condition in which several hundred polyps develop in the colon and rectum. (12 Dec 1998) |
| familial polyposis coli | <gastroenterology, oncology> A inherited, disorder where there are multiple adenomatous polyps (up to several thousand) in the colon. Malignant degeneration of the polyps (to colon carcinoma) occurs in virtually 100% by age 40. Inheritance: autosomal dominant. (27 Sep 1997) |
| filiform polyposis | <radiology> Benign, non-specific sequela of diffuse, severe mucosal inflammation, UC, Crohn's, XR: thin, straight filling defects, resembles stalks of polyps without heads (12 Dec 1998) |
| abortion, multiple | Couples who have had 2 or more miscarriages (spontaneous abortions) have about a 5% chance that one member of the couple is carrying a chromsome translocation responsible for the miscarriages. (12 Dec 1998) |
| advanced multiple-beam equalization radiography | A variant of scanning equalization radiography using several X-ray beams. (05 Mar 2000) |
| amyloidosis of multiple myeloma | Foci of amyloidosis in mesenchymal tissues of some persons with multiple myeloma; no direct relation between amyloid and Bence Jones protein is conclusively known. (05 Mar 2000) |
| cancer, multiple myeloma | A bone marrow cancer involving a type of white blood cell called a plasma (or myeloma) cell. The tumour cells can form a single collection (a plasmacytoma) or many tumours (multiple myeloma). Plasma cells are part of the immune system and make antibodies. Because patients have an excess of identical plasma cells, they have too much of one type of antibody. As myeloma cells increase in number, they damage and weaken the bones, causing pain and often fractures. When bones are damaged, calcium is released into the blood leading to hypercalcaemia (excess calcium in the blood) and that causes loss of appetite, nausea, thirst, fatigue, muscle weakness, restlessness, and confusion. Myeloma cells prevent the bone marrow from forming normal plasma cells and other white blood cells important to the immune system so patients may not be able to fight infections. The cancer cells can also prevent the growth of new red blood cells, causing anaemia. Excess antibody proteins and calcium may prevent the kidneys from filtering and cleaning the blood properly Cancer, non-Hodgkin's lymphoma: A lymphoma is a cancer that develops in the lymphatic system. The most common symptom of non-Hodgkin's lymphomas is a painless swelling in the lymph nodes in the neck, underarm, or groin. Non-Hodgkin's lymphomas are diagnosed with a biopsy of an enlarged lymph node. Follow-up examinations are important after lymphoma treatment. Most relapses occur in the first 2 years after therapy. (12 Dec 1998) |
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