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"multiple congenital polyposis"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
¾Ë±â½¬¿î ÀÇÇпë¾îÇ®ÀÌÁý, ¼­¿ïÀÇ´ë ±³¼ö ÁöÁ¦±Ù, °í·ÁÀÇÇÐ ÃâÆÇ À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
¿µ¹® multiple sclerosis ÇÑ±Û ´Ù¹ß°æÈ­Áõ
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  ½Å°æÃà»èÀ» µÑ·¯½Î°í Àִ ¸»ÀÌÁý(myelin sheath)ÀÇ ÆÄ±«·Î ÀÎÇÑ º´Àû»óŸ¦ ¸»ÇÔ. ÆÄ±«µÈ ¸»ÀÌÁýÀº ÈäÅ͸¦ ³²±â°Ô µÇ¾î ½Å°æÃà»èÀ» ÅëÇÑ ½Å°æÀü´ÞÀÌ Á¦´ë·Î µÇÁö ¾Ê¾Æ ¿îµ¿, °¨°¢, ÀÚÀ²½Å°æ ¸ðµÎÀÇ ½Å°æÀü´ÞÀå¾Ö°¡ ³ªÅ¸³­´Ù. ÀÌ º´Åʹ ¾îµð¼­³ª ³ªÅ¸³¯ ¼ö À־ ±× Àå¾Ö°¡ ³ªÅ¸³ª´Â ºÎÀ§¿¡ µû¶ó ¼­·Î ´Ù¸¥ Áõ»óÀ» È£¼ÒÇÑ´Ù.
¿µ¹® multiple myeloma ÇÑ±Û ´Ù¹ß°ñ¼öÁ¾
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  ´Ù¹ß¼º ¿ø¹ß¼º°ñÁ¾¾ç. ¸Ó¸®»À-°¥ºñ»À-º¹Àå»À-ôÃß»À-°ñ¹Ý µî¿¡ Àß ³ªÅ¸³ª°í, ¹°··¹°··ÇÑ Á¾±«¸¦ Çü¼ºÇϸç, »ÀÀÇ Èí¼ö°¡ ÀϾ°í, 40~60¼¼ ³²ÀÚ¿¡°Ô ¸¹ÀÌ ¹ß»ýÇÑ´Ù. °ñ¼öÁ¾ Á¾¾ç¼¼Æ÷´Â ÇüÁú¼¼Æ÷¿¡¼­ À¯·¡ÇÑ °ÍÀ̾ ÇüÁú¼¼Æ÷Á¾À̶ó°íµµ ÇÑ´Ù. °ú°Å¿¡´Â ÇüÁú¼¼Æ÷¼º°ñ¼öÁ¾ À̿ܿ¡´Â ´Ù¸¥ °ñ¼öÁ¶Ç÷¿ä¼Ò¿¡¼­ »ý±â´Â °ñ¼öÁ¾À̶ó°í »ý°¢ÇßÁö¸¸ ÇöÀç´Â ºÎÁ¤µÇ°í ÀÖ´Ù. ÇüÁú¼¼Æ÷´Â ¿ø·¡ ¸é¿ª±Û·ÎºÒ¸°À» »ý»êÇϴ ¼¼Æ÷À̸ç, ±×°ÍÀÌ Á¾¾çÈ­ÇÑ ´Ù¹ß °ñ¼öÁ¾ È¯ÀÚ¿¡¼­µµ ´ëºÎºÐ Ç÷û ¼Ó¿¡ ¸é¿ª ±Û·ÎºÒ¸°ÀÌ Áõ°¡µÈ °ÍÀ» º¼ ¼ö ÀÖ´Ù. Áõ°¡ÇÑ ±Û·ÎºÒ¸°Àº IgG³ª IgAÀΠ°æ¿ì°¡ ¸¹Áö¸¸ ´Ù¸¥ Çüµµ ÀÖ´Ù. °ñ¼öÁ¾ È¯ÀÚ ¾à 50%´Â ¿ÀÁÜ¿¡¼­ º¥½ºÁÔ½º´Ü¹éÁúÀÌ °ËÃâµÇ´Âµ¥, ÀÌ ´Ü¹éÁúÀÇ ÃàÀû¿¡ ÀÇÇØ ¿ä¼¼°üÀÌ ÆÄ±«µÇ°í, ÄáÆÏ°æÈ­°¡ ÀϾ´Ù. °ñ¼öÁ¾ È¯ÀÚ¿¡¼­´Â Ç÷û´Ü¹é ÀÌ»óÀ¸·Î °¡²û ¾Æ¹Ð·ÎÀ̵åÁõÀÌ ³ªÅ¸³­´Ù. »À X¼± ¼Ò°ßÀ¸·Î¼­´Â µµ·Á³½ º´ÅÍ, °ñÀ¶ÇØ»ó, º´Àû°ñÀýÀÌ °üÂûµÈ´Ù.
¿µ¹® multiple personality ÇÑ±Û ´ÙÀμº ÀΰÝ
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  Çظ®¼º Á¤½ÅÀå¾ÖÀÇ Çϳª·Î ³ªÅ¸³­´Ù. ÇÑ »ç¶÷ÀÌ ¿©·¯ »ç¶÷ÀÇ ¼º°ÝÀ» ¼ÒÀ¯Çϰí Àִ °ÍÀ¸·Î ¸¶Ä¡ ¡°Áöų¹Ú»ç¿Í ÇÏÀ̵堾¾¡±¿Í °°Àº °æ¿ìÀÌ´Ù. ¾Æ¸¶, ÇöÀç ÀÚ½ÅÀǠóÁö¿¡¼­ ¹þ¾î³ª°í ½ÍÀº ¹«ÀǽÄÀûÀΠ¿å¸Á¿¡¼­ ºñ·ÔµÇ´Â °ÍÀ¸·Î ¿©°ÜÁø´Ù.
¿µ¹® congenital syphilis ÇÑ±Û ¼±Ãµ¸Åµ¶
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  ÀӺΰ¡ ¸Åµ¶¿¡ °¨¿°µÇ¾î ÀÖÀ¸¸é ÀӽŠÈı⿡ ¸Åµ¶±ÕÀ̠ŹÝÀ» ÅëÇØ Ç÷Ç༺À¸·Î Å¾ƿ¡ °¨¿°(¼öÁ÷°¨¿°)µÈ °ÍÀ» ¸»ÇÏ´Ù. ´ëºÎºÐÀº À¯»ê, »ç»êÀÌ µÇÁö¸¸ Ãâ»ýÇϸé Á¦2±â ÀÌÈÄÀÇ ¹ßÁøÀ» º¸ÀδÙ. ¹ßÇö½Ã±â¿¡ µû¶ó¼­ ¨ç Å¾Ƹŵ¶, ¨è À¯¾Æ¸Åµ¶, ¨é ¸¸¹ß¼º ¼±Ãµ¸Åµ¶À¸·Î ºÐ·ùµÈ´Ù. ¨ç¿¡¼­´Â »À¿¬°ñ¿°, °£-Áö¶ó ºñ´ë¿Í ¸Åµ¶¼º ÃµÆ÷â, ¨è¿¡¼­´Â ÆÄ·Î°¡¼º¸¶ºñ¿Í ¸Åµ¶¼º ÄÚ¿°, ¨é¿¡¼­´Â ÇãÄ£½¼ ¼¼Â¡ÈÄ(ÇãÄ£½¼ Ä¡¾Æ, ¼Ó±Í¼º ³­Ã», ½ÇÁú¼º °¢¸·¿°)¿¡ µû¶ó Æ¯Â¡ÀÌ ÀÖ´Ù. ±âŸ ¼öµÎÁõ, Áö´É¹ßÀ° ºÒ·® µîÀ» ÀÚÁÖ º¼ ¼ö ÀÖ´Ù. ¸Åµ¶ Ç÷û¹ÝÀÀÀº ´ëºÎºÐÀÇ °æ¿ì ¾ç¼ºÀ¸·Î ³ª¿Â´Ù. ¸Å¿ì µå¹°°Ô °£¼¼Æ÷³»¿¡¼­ ¸Åµ¶±ÕÀ» ¹«¼öÈ÷ º¼ ¼ö ÀÖ´Ù. °£¼¼Æ÷ ÁÖº¯ÀÇ ¼¶À¯È­¿Í ÇÔ²² ºÒ±ÔÄ¢ÇÑ ÈäÅÍ(hepar lobatum)¸¦ ¸¸µé ¼ö ÀÖ´Ù.
¿µ¹® congenital rubella syndrome ÇÑ±Û ¼±ÃµÇ³ÁøÁõÈıº
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  ÀӽűⰣ Áß¿¡ »ê¸ð°¡ Ç³Áø¿¡ °É¸®¸é À̠dzÁø ¹ÙÀÌ·¯½º´Â Å¹ÝÀ» ÅëÇØ¼­ Å¾ƿ¡°Ô Àü´ÞµÇ¾î¼­ Å¾ÆÀǠdzÁø°¨¿°À» ÀÏÀ¸Å²´Ù. ÀӽŠù 3°³¿ù µ¿¾È, Æ¯È÷ ÀӽŠù´Þ¿¡ Å¾ư¡ Ç³ÁøÀÇ °¨¿°À» ¹ÞÀ¸¸é, ½Å»ý¾Æ¿¡¼­ ¼±Ãµ±âÇü, Áï ´«¿¡¼­ ÃÐÁ¡À» Á¤È®È÷ ¸ÂÃß¾îÁִ ·»ÁîÀÇ ¿ªÇÒÀ» Çϴ ¼öÁ¤Ã¼ÀǠȥŹ(¹é³»Àå), ½ÉÀå±âÇü, ±Í¸Ó°Å¸® ¹× ½ÉÇÑ Áö´É¹Ú¾àÀ» µ¿¹ÝÇϴ ¼ÒµÎÁõ µîÀÌ ¹ß»ýÇϴ ¼ö°¡ ¸¹´Ù.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • multiple intestinal polyposis
    ´Ù¹ßÀåÆú¸³Áõ
  • familial adenomatous polyposis
    °¡Á·¼º»ùÁ¾Æú¸³Áõ
  • familial polyposis
    °¡Á·¼ºÆú¸³Áõ
  • intestinal polyposis
    âÀÚÆú¸³Áõ, ÀåÆú¸³Áõ
  • juvenile coli polyposis
    ¼Ò¾ÆÀß·ÏâÀÚÆú¸³Áõ, ¼Ò¾Æ´ëÀåÆú¸³Áõ
  • polyposis
    Æú¸³Áõ
  • polyposis coli
    Àß·ÏâÀÚÆú¸³Áõ, ´ëÀåÆú¸³Áõ
  • congenital
    ¼±Ãµ-
  • congenital adrenal hyperplasia
    ¼±ÃµºÎ½Å°ú´ÙÇü¼º, ¼±ÃµÄáÆÏÀ§»ù°ú´ÙÇü¼º
  • congenital amputation
    ¼±Ãµ¼ºÀý´Ü
  • congenital aural fistula
    ¼±Ãµ±Ó¹ÙÄû¾Õ»û±æ, ¼±ÃµÀÌÀüºÎ´©°ø
  • congenital bullous icthyosiform erythroderma
    ¼±Ãµ¹°Áýºñ´ÃÁõ¸ð¾çÈ«»öÇǺÎ(Áõ), ¼±Ãµ¼öÆ÷ºñ´ÃÁõ¸ð¾çÈ«»öÇǺÎ(Áõ)
  • congenital cataract
    ¼±Ãµ¹é³»Àå
  • congenital constriction band
    ¼±ÃµÇùÂø¶ì
  • congenital contractural arachnodactyly
    ¼±Ãµ±¸Ãà°Å¹Ì°¡¶ôÁõ
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • congenital cataract
    ¼±Ãµ¹é³»Àå
  • congenital heart disease
    ¼±Ãµ½ÉÀ庴
  • congenital adrenal hyperplasia
    ¼±ÃµºÎ½Å°ú´ÙÇü¼º, ¼±ÃµÄáÆÏÀ§»ù°ú´ÙÇü¼º
  • congenital megacolon
    ¼±Ãµ°Å´ëūâÀÚÁõ, ¼±Ãµ°Å´ë´ëÀåÁõ, ¼±Ãµ°Å´ë°áÀåÁõ
  • polyposis coli
    (¢¡ familial adenomatous polyposis) °¡Á·¼ºÅ«Ã¢ÀÚÆú¸³Áõ, °¡Á·¼º´ëÀåÆú¸³Áõ
  • polyposis
    Æú¸³Áõ, »ì¹ö¼¸Áõ
  • familial adenomatous polyposis
    °¡Á·¼ºÅ«Ã¢ÀÚÆú¸³Áõ, °¡Á·¼º´ëÀåÆú¸³Áõ
  • juvenile coli polyposis
    ¼Ò¾ÆÃ¢ÀÚÆú¸³Áõ
  • multiple birth
    ´Ù»ê, ´ÙÅÂÃâ»ê
  • plural multiple birth
    ´Ù»ê, ´ÙÅÂÃâ»ê
  • multiple
    ´Ù¹ß-, ¿©·¯-, ¹µ-, ´Ù-
  • multiple myeloma
    ´Ù¹ß°ñ¼öÁ¾
  • multiple endocrine neoplasia
    º¹ÇÕ³»ºÐºñ»ù½Å»ý¹°
  • multiple sclerosis
    ´Ù¹ß°æÈ­Áõ
  • multiple causation theory
    Áúº´¹ß»ý´Ù¿äÀμ³
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial adenomatous polyposis
    °¡Á·»ùÁ¾Æú¸³Áõ
  • intestinal polyposis
    ÀåÆú¸³Áõ, âÀÚ»ì¹ö¼¸Áõ
  • polyposis
    Æú¸³Áõ, »ì¹ö¼¸Áõ
  • acyanotic congenital cardiopathy
    ºñû»ö¼±Ãµ½ÉÀ庴Áõ
  • congenital contractural arachnodactyly
    ¼±Ãµ±¸Ãà°Å¹Ì°¡¶ôÁõ
  • congenital oculomotor apraxia
    ¼±ÃµÈ´º¸±â¸øÇÔÁõ
  • congenital
    ¼±Ãµ-
  • congenital cataract
    ¼±Ãµ¹é³»Àå
  • congenital defect
    ¼±Ãµ°áÇÔ, ¼±Ãµ°á¼Õ(Áõ)
  • congenital megacolon
    ¼±ÃµÅ«°áÀåÁõ
  • congenital syphilis
    ¼±Ãµ¸Åµ¶
  • congenital torticollis
    ¼±Ãµ±â¿î¸ñ
  • congenital alveolar dysplasia
    ¼±ÃµÆóÆ÷Çü¼ºÀÌ»ó, ¼±ÃµÇãÆÄ²Ê¸®Çü¼ºÀÌ»ó
  • congenital aural fistula
    (¢¡congenital preauricular fistula) ¼±Ãµ±Ó¹ÙÄû¾Õ»û±æ, ¼±ÃµÀÌÀüºÎ´©°ø
  • congenital bullous icthyosiform erythroderma
    ¼±Ãµ¹°Áýºñ´ÃÇǺÎÁõ¸ð¾çÈ«»öÇǺÎÁõ
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • adenomatous polyposis coli
    ¼±Á¾¼º °áÀåÆú¸³Áõ(¡­Ì¿íó¡­ñø)
  • gastric polyposis
    À§Æú¸³Áõ.
  • Cowdens syndrome = multiple hamartoma syndrome
    ´Ù¹ß¼º °ú¿ÀÁ¾ ÁõÈıº
  • MOTSA (multiple overlapping thin-slab acquisition)
    ´ÙÁß Áߺ¹ ¼¼ÆíÆÇ ȹµæ
  • infection, multiple
    ´ÙÁß°¨¿°, º¹¼ö±Õ°¨¿°
  • infectious multiple gangrene of skin
    Àü¿°¼º ÇǺΠ´Ù¹ß¼º ±«Àú
  • personality disorder, multiple
    ´ÙÁß(Òýñì) ÀΰÝÀå¾Ö
  • personality, multiple
    ´ÙÁßÀΰÝ.
  • plural birth =multiple b.
    ´Ùźи¸(Òý÷à ÝÂØ´).
  • Gunthers disease => congenital erythropoietic porphyria
    ¼±Ãµ¼º ÀûÇ÷±¸ Á¶Ç÷¼º Æ÷¸£ÇǸ° Áõ
  • Hemolytic icterus, congenital
    ¿ëÇ÷¼ºÈ²´Þ(éÁúìàõüÜÓ¸)
  • Lebers congenital amaurosis
    ·¹º£¸£¼±ÃµÈæ¾Ï½Ã
  • acyanotic congenital cardiopathy
    ºñû»ö¼º ¼±Ãµ½É(Àå)º´Áõ(Þªôìßäàõà»ô¸ãýíôÜ»ñø).
  • anorchia congenital
    ¼±Ãµ¼º ¹«°íȯÁõ.
  • fusiform congenital cataract
    ¹æÃ߸ð¾ç¼±Ãµ¹é³»Àå, ¹æÃß»ó¼±Ãµ¹é³»Àå
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • multiple congenital polyposis
    ´Ù¹ß¼º ¼±Ãµ¼º(¡­à»ô¸àõ) Æú¸³Áõ(¡­ñø)
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • adenomatous polyposis coli
    ¼±Á¾¼º °áÀåÆú¸³Áõ(¡­Ì¿íó¡­ñø)
  • familial adenomatous polyposis
    °¡Á·¼º¼±Á¾¼º¿ëÁ¾Áõ.
  • familial adenomatous polyposis
    °¡Á·¼º ¼±Á¾¼º Æú¸³Áõ
  • familial polyposis
    °¡Á·¼º Æú¸³Áõ.
  • familial polyposis
    °¡Á·¼º Æú¸³Áõ
  • gastric polyposis
    À§Æú¸³Áõ.
  • gastrointestinal polyposis
    À§Àå¿ëÁ¾Áõ
  • intestinal polyposis
    ÀåÆú¸³Áõ.
  • juvenile polyposis syndrome
    ¿¬¼Ò¼º Æú¸³Áõ ÁõÈıº(¡­ ñøý¦ÏØ)
  • polyposis
    Æú¸³Áõ, ¿ëÁ¾Áõ.
  • polyposis
    Æú¸³Áõ(¡­ñø)
  • polyposis
    ¿ëÁ¾Áõ
  • polyposis adenomatosa ³ª
    ¼±Á¾¼º(àÍðþàõ) Æú¸³Áõ
  • polyposis coli <³ª>
    °áÀå(°áÀå)Æú¸³Áõ.
  • polyposis coli ³ª
    °áÀå(Ì¿íó)Æú¸³Áõ
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 7 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • Multiple placenta
    ¹µÅ¹Ý
    [¿¾ ¿ë¾î] ´Ù¹ß¼ºÅ¹Ý
  • Multiple deformity
    º¹ÇÕ±âÇü
    [¿¾ ¿ë¾î] ´Ù¹ß¼º±âÇü
  • Multiple morphologic defect
    º¹ÇÕÇüŰáÇÔ
    [¿¾ ¿ë¾î] ´Ù¹ß¼ºÇüÅÂÇÐÀû°áÇÔ
  • Congenital defect
    ¼±Ãµ°áÇÔ
    [¿¾ ¿ë¾î] ¼±Ãµ¼º°áÇÔ
  • Congenital glaucoma
    ¼±Ãµ³ì³»Àå
    [¿¾ ¿ë¾î] ¼±Ãµ¼º³ì³»Àå
  • Congenital metabolic defect
    ¼±Ãµ´ë»ç°áÇÔ
    [¿¾ ¿ë¾î] ¼±Ãµ¼º´ë»ç¼º°áÇÔ
  • Congenital cataract
    ¼±Ãµ¹é³»Àå
    [¿¾ ¿ë¾î] ¼±Ãµ¼º¹é³»Àå
´ëÇѱâ»ýÃæÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • multiple budding
    ´Ù¼öÃâ¾Æ
  • multiple fission
    ´ÙºÐ¿­
  • congenital infection
    ¼±Ãµ°¨¿°
  • congenital malaria
    ¼±Ãµ¸»¶ó¸®¾Æ
  • congenital toxoplasmosis
    ¼±ÃµÅå¼ÒÆ÷ÀÚÃæÁõ
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 11 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • congenital goiter
    "¼±Ãµ¼º °©»ó¼±Á¾(à»ô¸àõË£ßÒàÍðþ), (ÔÒ) =familial goiter"
  • congenital hyperammonemia
    ¼±Ãµ¼º(à»ô¸àõ) °ú(Φ)¾Ï¸ð´Ï¾ÆÇ÷Áõ(úìñø)
  • congenital parahemophilia
    ¼±Ãµ¼º ÃøÇ÷¿ìº´(à»ô¸àõö°úìéÒÜ»)
  • congenital porphyria
    ¼±Ãµ¼º(à»ô¸àõ) Æ÷¸£ÇǸ°Áõ(ñø)
  • multiple alleles
    º¹´ë¸³À¯ÀüÀÚ(ÜÜÓߨ¡ë¶îîí­)
  • multiple binding
    ´ÙÁß°áÇÕ(ÒýñëÌ¿ùê)
  • multiple codon recognition
    ´Ù(Òý)ÄÚµ· ÀÎÁö(ìãò±)
  • multiple displacement mechanism
    ´Ù(Òý)´ëü(ÓÛôð) ±âÀü(Ѧï®)
  • multiple factor hypothesis
    ´ÙÀÎÀÚ¼³(Òýì×í­àã)
  • multiple gene
    ´ÙÀ¯ÀüÀÚ(Òýë¶îîí­)
  • multiple inhibition analysis
    ´ÙÁß(Òýñì)ÀúÇØ ºÐ¼®(îÁúªÝÂà°)
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • multiple lymphomatous polyposis
    ´Ù¹ß¼º¸²ÇÁÁ¾¼º¿ëÁ¾Áõ
  • congenital
    ¼±Ãµ¼ºÀÇ
  • familial polyposis
    °¡Á·¼ºÆú¸³Áõ
  • polyposis
    Æú¸³Áõ
  • MOTSA [=multiple overlapping thin-slab acquisition]
    ´ÙÁßÁߺ¹¼¼ÆíÆÇȹµæ
  • multiple
    ´Ù¹ß¼º
  • multiple cranial nerve palsy
    ´Ù¹ß¼º³ú½Å°æ¸¶ºñ
  • multiple echo
    ´ÙÁß¿¡ÄÚ
  • multiple epiphyseal dysplasia
    ´Ù¹ß¼º°ñ´ÜÀÌÇü¼ºÁõ
  • multiple excitaiton
    ´ÙÁß¿©±â
  • multiple exostoses
    ´Ù¹ß¼º¿Ü°ñÁõ
  • multiple fibroma
    ´Ù¹ß¼º¼¶À¯Á¾
  • multiple myeloma
    ´Ù¹ß¼º°ñ¼öÁ¾
  • multiple overlapping thin slab acquisition [=MOTSA]
    ´ÙÁßÁߺ¹¼¼ÆíÆÇȹµæ
  • multiple polyp
    ´Ù¹ß¼ºÆú¸³
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
ECG Electro-Cardio-Graphy(-Gram); ½ÉÀüµµ
   = EKG
  1. Conducting System Structu...
CDH   1) Chronic Daily Headache
    = CTH
    = ...
CDH ceramide dihexoside; congenital diaphragmatic hernia; congenital dislocation of hip; congenital dysp...
AP accessory pathway; accounts payable; acid phosphatase; acinar parenchyma; action potential; active p...
APC acetylsalicylic acid, phenacetin, and caffeine; activated protein C; adenoidal-pharyngeal-conjunctiv...
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
MLP Multiple lymphomatous polyposis
MCA Multiple congenital anomalies
MCA/MR multiple congenital anomalies mental retardation
MCA multiple congenital anomaly
APC Adenomatous Polyposis Coli
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  • polyposis coli
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multiple intestinal polyposis Begins usually in late childhood; polyps increase in numbers, causing symptoms of chronic colitis, and carcinoma of the colon almost invariably develops in untreated cases; autosomal dominant inheritance. In the Gardner syndrome there are extracolonic changes (desmoid tumours, etc.).
Synonym: polyposis coli.
Hamartomatous polyposis of the small or large intestine, Peutz-Jeghers syndrome with melanin spots on the lips, less common, miscellaneous, rare, and doubtful occurrences.
Synonym: familial intestinal polyposis.
(05 Mar 2000)
adenomatous polyposis coli An autosomal dominant polyposis syndrome in which the colon contains few to thousands of adenomatous polyps, often occurring by age 15 to 25.
(12 Dec 1998)
polyposis Presence of several polyps.
Origin: polyp + G. -osis, condition
(05 Mar 2000)
polyposis coli Hereditary disorder (Mendelian dominant) characterised by the development of hundreds of adenomatous polyps in the large intestine, which show a tendency to progress to malignancy. The APC gene has also been implicated in a chromosome 5 gastric and pancreatic cancer.
(18 Nov 1997)
polyposis syndromes <radiology> Inher. Malig. Type familial polyposis coli dom and adenoma Gardner syndrome dom and Turcot syndrome rec CNS Peutz-Jeghers syndrome dom (+) hamartoma Cowden syndrome dom ? juvenile polyposis coli (?) - juvenile Cronkhite-Canada syndrome
(12 Dec 1998)
juvenile polyposis coli <radiology> Benign polyposis, inheritance uncertain, inflammatory or retention polyps: round, smooth, soft, mucin-filled, non-neoplastic, onset less than 10 yrs, polyps can prolapse through anus, associated with diarrhoea, protein loss see: polyposis syndromes, Cronkhite-Canada syndrome
(12 Dec 1998)
familial adenomatous polyposis <gastroenterology> Genetic disease with numerous precancerous polyps in the colon and rectum. Also called familial polyposis.
(12 Dec 1998)
familial intestinal polyposis Begins usually in late childhood; polyps increase in numbers, causing symptoms of chronic colitis, and carcinoma of the colon almost invariably develops in untreated cases; autosomal dominant inheritance. In the Gardner syndrome there are extracolonic changes (desmoid tumours, etc.).
Synonym: polyposis coli.
Hamartomatous polyposis of the small or large intestine, Peutz-Jeghers syndrome with melanin spots on the lips, less common, miscellaneous, rare, and doubtful occurrences.
Synonym: familial intestinal polyposis.
(05 Mar 2000)
familial polyposis An inherited condition in which several hundred polyps develop in the colon and rectum.
(12 Dec 1998)
familial polyposis coli <gastroenterology, oncology> A inherited, disorder where there are multiple adenomatous polyps (up to several thousand) in the colon. Malignant degeneration of the polyps (to colon carcinoma) occurs in virtually 100% by age 40.
Inheritance: autosomal dominant.
(27 Sep 1997)
filiform polyposis <radiology> Benign, non-specific sequela of diffuse, severe mucosal inflammation, UC, Crohn's, XR: thin, straight filling defects, resembles stalks of polyps without heads
(12 Dec 1998)
abortion, multiple Couples who have had 2 or more miscarriages (spontaneous abortions) have about a 5% chance that one member of the couple is carrying a chromsome translocation responsible for the miscarriages.
(12 Dec 1998)
advanced multiple-beam equalization radiography A variant of scanning equalization radiography using several X-ray beams.
(05 Mar 2000)
amyloidosis of multiple myeloma Foci of amyloidosis in mesenchymal tissues of some persons with multiple myeloma; no direct relation between amyloid and Bence Jones protein is conclusively known.
(05 Mar 2000)
cancer, multiple myeloma A bone marrow cancer involving a type of white blood cell called a plasma (or myeloma) cell. The tumour cells can form a single collection (a plasmacytoma) or many tumours (multiple myeloma). Plasma cells are part of the immune system and make antibodies. Because patients have an excess of identical plasma cells, they have too much of one type of antibody. As myeloma cells increase in number, they damage and weaken the bones, causing pain and often fractures. When bones are damaged, calcium is released into the blood leading to hypercalcaemia (excess calcium in the blood) and that causes loss of appetite, nausea, thirst, fatigue, muscle weakness, restlessness, and confusion. Myeloma cells prevent the bone marrow from forming normal plasma cells and other white blood cells important to the immune system so patients may not be able to fight infections. The cancer cells can also prevent the growth of new red blood cells, causing anaemia. Excess antibody proteins and calcium may prevent the kidneys from filtering and cleaning the blood properly Cancer, non-Hodgkin's lymphoma: A lymphoma is a cancer that develops in the lymphatic system. The most common symptom of non-Hodgkin's lymphomas is a painless swelling in the lymph nodes in the neck, underarm, or groin. Non-Hodgkin's lymphomas are diagnosed with a biopsy of an enlarged lymph node. Follow-up examinations are important after lymphoma treatment. Most relapses occur in the first 2 years after therapy.
(12 Dec 1998)
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