| ¿µ¹® | kidney stones | ÇÑ±Û | ÄáÆÏµ¹, ÄáÆÏ°á¼® |
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| ¿µ¹® | kidney | ÇÑ±Û | ÄáÆÏ, ½ÅÀå |
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| ¿µ¹® | infectious disease | ÇÑ±Û | °¨¿°º´ |
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| ¿µ¹® | hypertensive heart disease | ÇÑ±Û | °íÇ÷¾Ð½ÉÀ庴 |
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| ¿µ¹® | pelvic inflammatory disease | ÇÑ±Û | °ñ¹Ý¿°Áúȯ |
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| MCDK | multicystic dysplastic kidney |
|---|---|
| MCKD | multicystic kidney disease |
| CD | cadaver donor; canine distemper; canine dose; carbohydrate dehydratase; carbon dioxide; cardiac dise... |
| HD | Haab-Dimmer [syndrome]; Hajna-Damon [broth]; Hansen disease; hearing distance; heart disease; helix ... |
| MD | Doctor of Medicine [Lat. Medicinae Doctor]; magnesium deficiency; main duct; maintenance dose; major... |
| MCDK | Multicystic dysplastic kidney |
|---|---|
| DNS | Dysplastic Naevus Syndrome |
| DMN | Dysplastic melanocytic nevi |
| DN | Dysplastic naevi |
| ACDK | Acquired cystic disease of the kidney |
| multicystic dysplastic kidney disease | <radiology> most severe form of renal dysplasia, most common cause of palpaple abdominal mass in infants, unilateral (almost always; if bilateral, death occurs in utero), ureter absent or atretic, not associated with other cysts or with periportal fibrosis, Potter type II (12 Dec 1998) |
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| congenital dysplastic angiectasia | <syndrome> A congenital malformation syndrome characterised by the triad of asymmetric limb hypertrophy, haemangiomata, and nevi. Asymmetric limb hypertrophy is enlargement of one limb and not the corresponding limb on the other side, the enlarged limb being 3 times more likely to be a leg than an arm in ktw; and the limb enlargement is of bone as well as soft tissue. The haemangiomas, abnormal nests of blood vessels that proliferate inappropriately and excessively, cover a remarkable range from small innocuous capillary haemangiomas ( strawberry marks ) to huge cavernous haemangiomas. The nevi are pigmented moles on the skin; in ktw there are often also dark linear streaks on the skin, streaks due to too much pigment. There can be other abnormalities but the triad is the consistent clinical centrepiece of the disease. most persons with ktw have an enlarged leg and do relatively well without treatment or, for example, with only compression from an elastic stocking. Skin ulcers and other skin problems can occur over the swollen leg. Usually, the treatment is conservative. Surgery is almost never needed. The only possible exceptions are the very rare situations in which the leg reaches gigantic proportions or secondary clotting difficulties arise (due to trapping and destruction of blood platelets in a huge haemangioma). Then, amputation may become necessary. The cause of ktw syndrome is unknown. (12 Dec 1998) |
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| congenital dysplastic angiomatosis | Autosomal dominant angiomatosis in which there is dysplasia of the underlying tissues, sometimes with overgrowth of bone (Klippel-Trenaunay-Weber syndrome), or encephalotrigeminal angiomatosis (Sturge-Weber syndrome) in which there is an angioma in the distribution of one or more branches of the trigeminal nerve, with vascular anomalies and calcification of the cerebral cortex. (05 Mar 2000) |
| dysplastic | Pertaining to or marked by dysplasia. (05 Mar 2000) |
| dysplastic nevi | Atypical moles; moles whose appearance is different from that of common moles. Dysplastic nevi are generally larger than ordinary moles and have irregular borders. Their colour often is not uniform; they usually are flat, but parts may be raised above the skin surface. (12 Dec 1998) |
| dysplastic nevus | <syndrome> Clinically atypical nevi (usually exceeding 5 mm in diameter and having variable pigmentation and ill defined borders) with an increased risk for development of non-familial cutaneous malignant melanoma. Biopsies show melanocytic dysplasia. Nevi are clinically and histologically identical to the precursor lesions for melanoma in the b-k mole syndrome. (12 Dec 1998) |
| dysplastic nevus syndrome | <syndrome> Clinically atypical nevi (usually exceeding 5 mm in diameter and having variable pigmentation and ill defined borders) with an increased risk for development of non-familial cutaneous malignant melanoma. Biopsies show melanocytic dysplasia. Nevi are clinically and histologically identical to the precursor lesions for melanoma in the b-k mole syndrome. (12 Dec 1998) |
| medullary cystic kidney disease | <radiology> Mode of inheritance uncertain, usually affects young adults (early 20s), associated with red or blond hair (!!), causes salt wasting, polyuria, azotaemia, affects both kidneys, with progressive disease patients need dialysis or transplant (12 Dec 1998) |
| polycystic kidney disease | <disease> A rare inherited condition in which the kidney are composed of multiple cysts. Kidney cysts are associated with an increased incidence of cerebral aneurysm. Symptoms usually appear later (if they do at all) and include blood in the urine, flank pain, excessive urination at night and abdominal pain. Individuals may also have elevated blood pressure. Chronic (end-stage renal disease) renal failure is the most common result in the 5th to 6th decades of life. Incidence: 1 in 5,000. (02 Jan 1998) |
| pulpy kidney disease | An enterotoxaemia of sheep caused by the bacterium Clostridium perfringens type D and characterised by sudden death preceded in some cases by excitement, incoordination, and convulsions; also occurs in goats and rarely in cattle. (05 Mar 2000) |
| diabetic kidney disease | Kidney disease and resultant kidney function impairment due to the long standing effects of diabetes on the microvasculature (glomerulus) of the kidney. Features include increased urine protein and declining kidney function. Severe diabetic nephropathy can lead to kidney failure and end-stage renal disease. (27 Sep 1997) |
| disease, polycystic kidney | Genetic (inherited) disorders characterised by the development of innumerable cysts in the kidneys filled with fluid that replace much of the mass of the kidneys and reduce kidney function leading to kidney failure. (12 Dec 1998) |
| kidney disease | <disease> Any one of several chronic conditions that are caused by damage to the cells of the kidney. People who have had diabetes for a long time may have kidney damage. (09 Oct 1997) |
| acute kidney failure | <nephrology> A sudden decline in renal function may be triggered by a number of acute disease processes. Examples include sepsis (infection), shock, trauma, kidney stones, kidney infection, drug toxicity (aspirin or lithium), poisons or toxins (drug abuse) or after injection with an iodinated contrast dye (adverse effect). Chronic renal failure represents a slow decline in kidney function over time. Chronic renal failure may be caused by a number of disorders which include long-standing hypertension, diabetes, congestive heart failure, lupus or sickle cell anaemia. Both forms of renal failure result in a life-threatening metabolic derangement. (27 Sep 1997) |
| acute kidney transplant rejection | <radiology> Findings on ultrasound: globular enlargment of the kidney, swelling and hypoechogenicity of the medullary pyramids, indistinct cortico-medullary junction, foci in the renal cortex (12 Dec 1998) |
| african green monkey kidney cell | <cell culture> Cells taken from the kidneys of the African green monkey Cercopithecus aethiops sabaeus and used to grow certain viruses like poliovirus. (05 Feb 1998) |
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