| ¿µ¹® | hemolytic anemia | ÇÑ±Û | ¿ëÇ÷ºóÇ÷ |
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| ¼³¸í | ¿ëÇ÷ºóÇ÷À̶õ ÀûÇ÷±¸ÀÇ °úµµÇÑ ÆÄ±«¿¡ ÀÇÇÑ ºóÇ÷ÀÌ´Ù. ¿ø·¡ 120ÀÏ Á¤µµÀÇ ¼ö¸íÀ» °¡Áö´Â ÀûÇ÷±¸ÀÇ ¼ö¸íÀÌ Âª¾ÆÁö´Â °ÍÀÌ´Ù. ¿©±â¿¡´Â ¿©·¯ °¡Áö ¿øÀÎÀÌ ÀÖÀ» ¼ö°¡ Àִµ¥ ´ëÇ¥ÀûÀÎ ¿øÀÎÀ¸·Î´Â ÀûÇ÷±¸¿¡ ´ëÇÑ Ç×ü°¡ »ý±â´Â °Í(¹ßÀÛ¼º¾ß°£Ç÷»ö¼Ò´¢Áõ)°ú ÀûÇ÷±¸ÀÚüÀÇ ÀÌ»ó(À¯Àü¼ºµÕ±ÙÀûÇ÷±¸Áõ), ±×¸®°í ´Ù¸¥ Áúº´¿¡ ÀÇÇØ¼ 2Â÷ÀûÀ¸·Î »ý±â´Â °ÍÀÌ ÀÖ´Ù. |
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| ¿µ¹® | hemolytic disease of newborn | ÇÑ±Û | ½Å»ý¾Æ¿ëÇ÷º´ |
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| ¼³¸í | ½Å»ý¾Æ¿¡¼ ÀûÇ÷±¸°¡ ºñÁ¤»óÀûÀ¸·Î ¸¹ÀÌ ÆÄ±«µÇ´Â º´À¸·Î žÆÀû¸ð±¸Áõ(erythroblastosis fetalis)¿Í °°Àº ¶æÀ¸·Î ¾²ÀδÙ. À̰ÍÀº ¾î¸Ó´Ï¿¡°Ô¼ »ý»êµÈ ½Å»ý¾Æ³ª žÆÀÇ ÀûÇ÷±¸¿¡ ´ëÇÑ Ç×ü°¡ ŹÝÀ» °Ç³Ê¿Í¼ žÆÀÇ ÀûÇ÷±¸¿Í °áÇÕÇÏ¿©¼ »ý±â´Â ¿ëÇ÷¼ººóÇ÷À» À̸£´Â ¸». Áï ½Å»ý¾Æ³ª žÆÀÇ ÀûÇ÷±¸ÀÇ Ç×ü°¡ ¾î¸Ó´ÏÀÇ ¸ö¿¡¼ »ý»êÀÌ µÇ°í À̰ÍÀÌ Å¹ÝÀ» ÅëÇØ¼ žƿ¡°Ô ³Ñ¾î°¡¼ žÆÀÇ ÀûÇ÷±¸¿Í °áÇÕÀ» Çϰí ÀÌ Ç×ü¿Í °áÇÕÇÑ ÀûÇ÷±¸´Â ÆÄ±«°¡ µÇ¾î¼ ºóÇ÷ÀÌ »ý±ä °ÍÀ» žÆÀû¸ð±¸ÁõÀ̶ó°í ÇÑ´Ù. À̰ÍÀº Rh Àû¸ð±¸Áõ(Rh erythroblastosis)¿Í ABO Àû¸ð±¸Áõ(ABO erythroblastosis)·Î ³ª´ ¼ö°¡ ÀÖ´Ù. |
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| ¿µ¹® | anemia | ÇÑ±Û | ºóÇ÷ |
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| ¼³¸í | Ç÷¾×ÀÌ ¼øÈ¯ÇÏ´Â ¸ñÀû Áß¿¡¼ °¡Àå Áß¿äÇÑ °ÍÀº ¿©·¯ °¡Áö ¿µ¾ç¼Ò¸¦ ¸»ÃÊÀÇ Àå±â·Î º¸±ÞÇÏ°í ¸»ÃÊÀÇ Àå±â¿¡¼ ³ª¿À´Â ¿©·¯ ³ëÆó¹°À» ÄáÆÏÀ̳ª Æó·Î º¸³» ¹è¼³¹°À» ó¸®ÇÏ´Â µ¥ ÀÖ´Ù. ±× Áß¿¡¼ »ê¼ÒÀÇ ¿î¹ÝÀº °¡Àå Áß¿äÇѵ¥ ¹Ù·Î ÀÌ »ê¼ÒÀÇ ¿î¹ÝÀ» ´ã´çÇÏ´Â °ÍÀÌ ÀûÇ÷±¸ÀÌ´Ù. ÀûÇ÷±¸¿¡´Â Ç÷»ö¼Ò¶ó´Â ¹°ÁúÀÌ ÀÖ¾î À̰ÍÀÌ »ê¼Ò¿Í °áÇÕÇÏ¿© »ê¼Ò¸¦ ¸»ÃÊÀÇ Àå±â·Î ¿î¹ÝÇÒ ¼ö°¡ ÀÖ´Ù. ºóÇ÷À̶õ ´ÜÀ§ºÎÇÇÀÇ Ç÷¾×¼Ó¿¡ ÀûÇ÷±¸ÀÇ ¾çÀÌ ÀûÀº °æ¿ì¸¦ ¸»ÇÑ´Ù. ÀûÇ÷±¸ÀÇ ¾çÀ» ³ªÅ¸³»´Â °ÍÀ¸·Î´Â 3°¡Áö ¹æ¹ýÀÌ ÀÖ´Ù. ÀûÇ÷±¸ÀÇ ¼ýÀÚ¸¦ Á÷Á¢ Ç¥ÇöÇÏ´Â ¹æ¹ý°ú, Ç÷»ö¼ÒÀÇ ¾çÀ» Á¤·®ÇÏ¿© ±× ¾çÀ» Ç¥½ÃÇÏ´Â ¹æ¹ý°ú, Ç÷¾×¼Ó¿¡¼ ÀûÇ÷±¸°¡ Â÷ÁöÇÏ´Â ¾ç(ÀûÇ÷±¸µîÀûÀ²)À» ³ªÅ¸³»´Â ¹æ¹ýÀÌ ±×°ÍÀÌ´Ù. ´ë°³ ºóÇ÷À̶ó ÇÔÀº ³²¼º¿¡¼ Ç÷»ö¼Ò < 14g/dl, Ç÷»ö¼Ò < 42%, ÀûÇ÷±¸ÀÇ ¼ö < 4,000,000/mm3ÀÏ °æ¿ìÀ̰í, ¿©¼º¿¡¼± Ç÷»ö¼Ò < 12g/dl, Ç÷»ö¼Ò < 36%, ÀûÇ÷±¸ÀÇ ¼ö < 3,300,000/mm3ÀÏ °æ¿ì¸¦ ÁöĪÇÑ´Ù. |
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| ¿µ¹® | pernicious anemia | ÇÑ±Û | ¾Ç¼ººóÇ÷ |
|---|---|---|---|
| ¼³¸í | ¾Ç¼º(»ý¸íÀ» À§ÇùÇϸç, Ä¡·á¿¡ ÀúÇ×ÇÏ´Â °æ¿ì¿¡ ´ë°³ ¾Ç¼ºÀ̶ó ºÎ¸§. ¿¹¸¦ µé¾î ÁøÇàµÈ ¾ÏÀÇ °æ¿ì)À̶ó À̸§ºÙ¾î ÀÖÁö¸¸, ½ÇÁ¦ÀûÀ¸·Î´Â ¾Ç¼ºÀÌ ¾Æ´Ï´Ù. ºóÇ÷ÀÇ ÀÏÁ¾ÀÌ´Ù. Á¤»óÀûÀ¸·Î ÀûÇ÷±¸´Â ¹ß´Þ°ú ¼º¼÷°úÁ¤¿¡¼ ºñŸ¹Î B12°¡ ÇʼöÀûÀÌ´Ù. ÀÌ ºñŸ¹Î B12ÀÇ Ç÷Áß³óµµ°¨¼Ò¿¡ ÀÇÇØ ÀûÇ÷±¸»ý¼º¿¡ ÁöÀåÀ» °¡Á®¿À°Ô µÇ°í, Ç÷¾×³»¿¡ Ư¡ÀûÀÎ °Å´ëÀû¸ð±¸(megaloblast)ÀÇ Çü¼ºÀÌ ³ªÅ¸³ª´Â Áúº´À» ¸»ÇÑ´Ù. |
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| ¿µ¹® | aplastic anemia | ÇÑ±Û | Àç»ýºÒ·®ºóÇ÷ |
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| ¼³¸í | Àç»ýºÒ·®¼º ºóÇ÷À̶õ °ñ¼öÀÇ ÀÌ»óÀ¸·Î »ý±â´Â ºóÇ÷ÀÌ´Ù. °ñ¼ö¶õ »À¼Ó¿¡ Á¸ÀçÇÏ´Â °ÍÀ¸·Î Ç÷±¸¸¦ »ý¼ºÇÏ´Â ¼¼Æ÷µé°ú ¹Ì¼º¼÷ÇÑ Ç÷±¸µé·Î ÀÌ·ç¾îÁ® ÀÖ´Ù. À̰͵éÀÌ ¿©·¯ °¡Áö ¿øÀο¡ ÀÇÇØ¼ ÆÄ±«µÇ¾úÀ» ¶§ »ý±â´Â ºóÇ÷À» Àç»ýºÒ·®ºóÇ÷À̶ó°í ÇÑ´Ù. ±×·¯¹Ç·Î ÀûÇ÷±¸¸¸ÀÇ °¨¼Ò°¡ ¾Æ´Ï¶ó ¸ðµç Ç÷±¸ ¼¼Æ÷ÀÇ °¨¼Ò¸¦ º¼ ¼ö ÀÖ´Ù. Ä¡·á·Î´Â Ç÷±¸ »ý¼ºÀ» ÀÚ±ØÇϴ ȣ¸£¸óÀ» Åõ¿©ÇÏ´Â °ÍÀÌ ÀÖÁö¸¸ À̰ÍÀ¸·Î´Â ÆÄ±«µÈ Ç÷±¸¸¦ »ý¼ºÇÏ´Â ¼¼Æ÷ÀÇ Àç»ýÀÌ ÀϾÁö ¸øÇϹǷΠ¿ÏÀüÇÑ Ä¡·á¶ó°í´Â º¼ ¼ö°¡ ¾ø´Ù. ¿ÏÀüÇÑ Ä¡·á·Î´Â ³²ÀÇ °ñ¼ö¸¦ äÃëÇØ¼ À̰Ϳ¡¼ºÎÅÍ Ç÷±¸¸¦ »ý¼ºÇÏ´Â ¼¼Æ÷¸¦ ºÐ¸®, ȯÀÚ¿¡°Ô À̽ÄÇÏ´Â °ñ¼öÀ̽ÄÀÌ ÀÖ´Ù. |
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| AHA | acetohydroxamic acid; acquired hemolytic anemia; acute hemolytic anemia; American Heart Association;... |
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| MAHA | microangiopathic hemolytic anemia |
| MHA | major histocompatibility antigen; May-Hegglin anomaly; Mental Health Association; methemalbumin; mic... |
| MHD | maintenance hemodialysis; mean hemolytic dose; mental health department; minimum hemolytic dilution;... |
| MAHA | Micro-Angiopathic Hemolytic Anemia; PB»ó Helmet Cell ThrombocytopeniaÁß MAHAÀ¯¹ß &nbs... |
| MAHA | Microangiopathic hemolytic anemia |
|---|---|
| BHS | Beta-hemolytic streptococci |
| CH 50 | Complement hemolytic activity |
| DHTR | Delayed hemolytic transfusion reaction |
| GABHS | Group A beta hemolytic streptococcal |
| anemia | <haematology> Too few red blood cells in the bloodstream, resulting in insufficient oxygen to tissues and organs. Origin: Gr. Haima = blood (16 Dec 1997) |
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| microangiopathic haemolytic anaemia | <haematology> Consequence of disseminated intravascular coagulation (DIC): fragments of red blood cells, damaged by being forced through a fibrin meshwork, are found in the circulation. Origin: Gr. Haima = blood (18 Nov 1997) |
| hemolytic anaemia | <disease, haematology> Anaemia resulting from reduced red cell survival time and haemolysis, either due to an intrinsic defect in the erythrocyte (hereditary spherocytosis or ellipsocytosis, enzyme defects, haemoglobinopathy) or an extrinsic damaging agent. For example autoantibody (autoimmune haemolytic anaemia), iso antibody, parasitic invasion of the cells (malaria), bacterial or chemical haemolysins, mechanical damage to erythrocytes. Origin: Gr. Haima = blood (18 Nov 1997) |
| microangiopathic hemolytic anemia |
Hemolysis due to narrowing or obstruction of small blood vessels usually due to inflammation, causing fragmentation and distortion in the shape of red blood cells (Dirckx, 1997).
Ãâó: www.med.uwo.ca/ecosystemhealth/education/glossary....
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