¼±Åà - È­»ìǥŰ/¿£ÅÍŰ ´Ý±â - ESC

 
"mesangial nephritis"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
¾Ë±â½¬¿î ÀÇÇпë¾îÇ®ÀÌÁý, ¼­¿ïÀÇ´ë ±³¼ö ÁöÁ¦±Ù, °í·ÁÀÇÇÐ ÃâÆÇ À¯»ç °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
¿µ¹® nephritis(interstitial) ÇÑ±Û ÄáÆÏ¿°, ½ÅÀå¿°(°£Áú¼º)
¼³¸í   
  ÄáÆÏ»çÀÌÁú(renal interstitium: ÄáÆÏÀÇ ¼¼´¢°ü »çÀÌÀÇ °áÇÕÁ¶Á÷)ÀÇ ¿°Áõ. ±Þ¼ºÀ¸·Î ÀϾ´Â °£ÁúÄáÆÏ¿°Àº ´ë°³ ¸¹Àº ¾àÀ» º¹¿ëÇÑ ÈÄ¿¡ ¹ß»ýÇϴ µ¥, ¿­, ÇǺÎÀÇ º¯»ö, Ç÷¾×³»ÀǠȣ»ê±¸Áõ ±×¸®°í ¼Òº¯ÀÇ °¨¼Ò¿Í ÄáÆÏ±â´ÉÀÇ °¨¼Ò¸¦ Æ¯Â¡À¸·Î ÇÑ´Ù. Ä¡·á´Â º¹¿ëÁßÀΠ¾àÀ» ²÷°í, ºÎ½Å°ÑÁúÈ£¸£¸óÁ¦¸¦ Åõ¿©ÇÑ´Ù.
  
  ÄáÆÏÀº °¢Á¾ ³ëÆó¹°, ÀüÇØÁú, ¼öºÐ µîÀ» Æ÷ÇÔÇÑ ¿ä¸¦ »ý»êÇÏ¿© ¹èÃâÇϴ µ¿½Ã¿¡ ¼ö¼ÒÀÌ¿Â, ³ªÆ®·ý, Ä®·ý, Àλê À̿ ³óµµ µîÀ» Á¶ÀýÇϸ砳»ºÐºñ¿Í ¿ÜºÐºñ ±â´É¿¡ °ü¿©Çϱ⵵ ÇÑ´Ù.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • mesangial cell
    Ç÷°ü»çÀ̼¼Æ÷, ¸Þ»êÁö¿ò¼¼Æ÷
  • mesangial matrix
    Ç÷°ü»çÀ̹ÙÅÁÁú, ¸Þ»êÁö¿ò±âÁú
  • mesangial proliferative glomerulonephritis
    Ç÷°ü»çÀÌÁúÁõ½ÄÅ丮ÄáÆÏ¿°, Ç÷°ü°£¼¼Æ÷Áõ½Ä»ç±¸Ã¼½Å¿°
  • arteriosclerotic nephritis
    µ¿¸Æ°æÈ­ÄáÆÏ¿°, µ¿¸Æ°æÈ­½ÅÀå¿°
  • acute nephritis
    ±Þ¼ºÄáÆÏ¿°, ±Þ¼º½ÅÀå¿°
  • analgesic nephritis
    ÁøÅëÁ¦ÄáÆÏ¿°, ÁøÅëÁ¦½ÅÀå¿°
  • glomerular nephritis
    Å丮ÄáÆÏ¿°, »ç±¸Ã¼½Å(Àå)¿°
  • glomerulotubular nephritis
    Å丮¿ä¼¼°üÄáÆÏ¿°, »ç±¸Ã¼¿ä¼¼°ü½ÅÀå¿°
  • hemorrhagic nephritis
    ÃâÇ÷ÄáÆÏ¿°, ÃâÇ÷½ÅÀå¿°
  • Henoch-Schonlein purpura nephritis
    Çì³ëÈå-¼é¶óÀÎÀÚ»ö¹ÝÄáÆÏ¿°, Çì³ëÈå-¼é¶óÀÎÀÚ»ö¹Ý½ÅÀå¿°
  • hereditary tubulointerstitial nephritis
    À¯Àü¿ä¼¼°ü»çÀÌÁúÄáÆÏ¿°, À¯Àü¿ä¼¼°ü°£Áú½ÅÀå¿°
  • interstitial nephritis
    »çÀÌÁúÄáÆÏ¿°, °£Áú½ÅÀå¿°
  • lupus nephritis
    ·çǪ½ºÄáÆÏ¿°, ·çǪ½º½ÅÀå¿°
  • nephritis
    ÄáÆÏ¿°, ½ÅÀå¿°
  • nephrotoxic nephritis
    ÄáÆÏµ¶¼ºÄáÆÏ¿°, ½Åµ¶¼º½ÅÀå¿°
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 7 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • mesangial proliferative glomerulonephritis
    Ç÷°ü»çÀÌÁúÁõ½ÄÅ丮ÄáÆÏ¿°, Ç÷°ü°£¼¼Æ÷Áõ½Ä»ç±¸Ã¼½Å¿°
  • nephritis
    ÄáÆÏ¿°, ½ÅÀå¿°
  • acute nephritis
    ±Þ¼ºÄáÆÏ¿°
  • glomerular nephritis
    Å丮ÄáÆÏ¿°, »ç±¸Ã¼½ÅÀå¿°
  • Henoch-Schoenlein purpura nephritis
    Çì³ëÈ彨¶óÀÎÀÚ»ö¹ÝÄáÆÏ¿°, Çì³ëÈ彨¶óÀÎÀÚ»ö¹Ý½ÅÀå¿°
  • interstitial nephritis
    »çÀÌÁúÄáÆÏ¿°, °£Áú½ÅÀå¿°
  • lupus nephritis
    ·çÇÁ½ºÄáÆÏ¿°, ·çÇÁ½º½ÅÀå¿°
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • mesangial cell
    Ç÷°ü»çÀ̼¼Æ÷
  • mesangial matrix
    Ç÷°ü»çÀ̹ÙÅÁÁú
  • analgesic nephritis
    ÁøÅëÁ¦ÄáÆÏ¿°
  • arteriosclerotic nephritis
    µ¿¸Æ°æÈ­ÄáÆÏ¿°
  • glomerular nephritis
    Å丮ÄáÆÏ¿°, »ç±¸Ã¼ÄáÆÏ¿°
  • glomerulotubular nephritis
    Å丮¿ä¼¼°üÄáÆÏ¿°, »ç±¸Ã¼¿ä¼¼°üÄáÆÏ¿°
  • hemorrhagic nephritis
    ÃâÇ÷ÄáÆÏ¿°
  • hereditary tubulointerstitial nephritis
    À¯Àü´¢¼¼°ü»çÀÌÁúÄáÆÏ¿°
  • interstitial nephritis
    »çÀÌÁúÄáÆÏ¿°
  • nephritis
    ÄáÆÏ¿°, ½ÅÀå¿°
  • nephrotoxic nephritis
    ÄáÆÏµ¶¼ºÄáÆÏ¿°
  • parenchymatous nephritis
    ½ÇÁúÄáÆÏ¿°
  • potassium-losing nephritis
    Ä®·ý»ó½ÇÄáÆÏ¿°
  • radiation nephritis
    ¹æ»ç¼±ÄáÆÏ¿°
  • saturnine nephritis
    ³³µ¶¼ºÄáÆÏ¿°
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 14 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • acute nephritis
    ±Þ¼º ½Å¿°(Ðáàõãìæú).
  • anti-glomerular,basement menbrane nephritis
    Ç׻籸ü±âÀú¸·½Å¿°(ù÷Þêϳô÷Ðñî¼Ø¯ãìæú)
  • glomerular nephritis
    »ç±¸Ã¼¼º ½Å¿°.
  • glomerulocapsular nephritis
    »ç±¸Ã¼Çdz¶¼º ½Å¿°(ÞêϹô÷ù«Ò¥àõãì
  • glomerulotubular nephritis
    »ç±¸Ã¼¼¼´¢°ü½Å¿°(¡­á¬èñηãìæú).
  • hemorrhagic nephritis
    ÃâÇ÷(¼º)½Å¿°(¡­ãìæú).
  • hereditary nephritis
    À¯Àü¼º ½Å¿°.
  • hereditary tubulointerstitial nephritis
    À¯Àü¼º¼¼´¢°ü°£Áú½Å¿°
  • heymans nephritis
    ÇÏÀ̸¸ ½Å¿°, Heyman ½Å¿°
  • potassium losing nephritis
    Ä®·ý»ó½Ç¼º ½Å¿°.
  • potassium losing nephritis
    Ä®·ý»ó½Ç¼º ½Å¿°(¡­ßÃã÷àõ ãìæú)
  • radiation nephritis
    ¹æ»ç¼±½Å¿°(Û¯ÞÒàÊãìæú).
  • radiation nephritis
    ¹æ»ç¼±½Å¿°(Û¯ÞÒàÊãìæú)
  • radiation nephritis
    ¹æ»ç¼±½Å¿°
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • mesangial cell
    Ç÷°ü»çÀ̼¼Æ÷, Ç÷°ü°£¼¼Æ÷(úìηÊàá¬øà).
  • mesangial cell
    Ç÷°ü»çÀ̼¼Æ÷
  • mesangial matrix
    Ç÷°ü»çÀ̱âÁú, Ç÷°ü°£±âÁú (¡­Ñ¨òõ).
  • mesangial region
    ¸Þ»êÁöÀ½¿µ¿ª.
  • acute nephritis
    ±Þ¼º ½Å¿°(Ðáàõãìæú).
  • anti-glomerular,basement menbrane nephritis
    Ç׻籸ü±âÀú¸·½Å¿°(ù÷Þêϳô÷Ðñî¼Ø¯ãìæú)
  • arteriosclerotic nephritis
    µ¿¸Æ°æÈ­¼º ½Å¿°(¡­ãìæú).
  • azotemic nephritis
    (°í)Áú¼ÒÇ÷¼º ½Å¿°.
  • cheesy nephritis<³ª> n. caseoaa
    °Ç¶ô¼º ½Å¿°(ËëÕ¬àõãìæú).
  • crush nephritis
    Á¸ê½Å¿°, ºÐ¼â¼º ½Å¿°.
  • exudative nephritis
    »ïÃ⼺ ½ÅÀå¿°.
  • fibrous nephritis
    ¼¶À¯¼º ½Å¿°(¡­ãìæú).
  • focal bacterial nephritis
    ±Þ¼º ±¹¼Ò¼º ¼¼±Õ¼º ½Å¿°
  • focal nephritis
    ÃÊÁ¡¼º ½Å¿°.
  • glomerular nephritis
    »ç±¸Ã¼¼º ½Å¿°.
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • Mesangial cell
    Ç÷°ü»çÀ̼¼Æ÷
    [¿¾ ¿ë¾î] Ç÷°ü°£¼¼Æ÷
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 3 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • nephritis
    ½ÅÀå¿°
  • radiation nephritis
    ¹æ»ç¼±½Å¿°
  • salt-losing nephritis
    ¿°·ù¼Ò½Ç¼º½Å¿°
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
MPGN Mesangial(Membrano-) Proliferative Glomerulo-Nephritis
DMH diffuse mesangial hypercellularity
DMP diffuse mesangial proliferation; dimercaprol; dimethylphthalate
DMS delayed match-to-sample; delayed microembolism syndrome; demarcation membrane system; department of ...
MesPGN mesangial proliferative glomerulonephritis
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
DMS Diffuse mesangial sclerosis
HMC Human mesangial cell
MC Mesangial cell
MesPGN Mesangial proliferative glomerulonephritis
GMC glomerular mesangial cell
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 9 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • mesangial matrix
    Ç÷°ü »çÀÌ ±âÁú, Ç÷°ü°£ ±âÁú
  • acute nephritis
    ±Þ¼º ½Å¿°
    ±Þ¼ºÀ¸·Î ¹ß»ýÇÏ´Â ½Å¿°ÀÇ ÃÑĪÀÌ´Ù. ±Þ¼º
  • anti-glomerular, basement menbrane nephritis
    Ç׻籸ü ±âÀú¸· ½Å¿°
  • cheesy nephritis
    °Ç¶ô¼º ½Å¿°
  • Lancereaux's nephritis
    ¶û¼¼·Î ½Å¿°
    ·ù¸ÓƼ½º¼º °£Áú¼º ½Å¿°.
  • lupus nephritis
    ³¶Ã¢¼º ½ÅÀå¿°
  • nephritis
    ½Å¿°, ½ÅÀå¿°
    1. ½ÅÀåÀÇ ¿°Áõ. glomerulus, tubule, interstitial renal tissue¿¡¼­ ¹ß»ýÇÏ°Ô µÇ´Â ¸ð¿©¼­ »ý±â°Å³ª Èð¾îÁ®¼­ »ý±â´Â, Áõ½ÄÇϰųª ÆÄ±«µÇ´Â °úÁ¤. 2. ºÎÁ¾, Ç÷´¢ ¹× ´Ü¹é´¢¸¦ ÁÖÁõÀ¸·Î ÇÏ´Â Áúȯ. ½Å¿°, »ç±¸Ã¼½Å¿°À̶ó°íµµ ÇÑ´Ù. ÁÖ·Î ¿¬¼â ±¸±ÕÀÇ °¨¿°ÀÌ Ã¼³»ÀÇ ¾îµò°¡¿¡ ÀÖÀ¸¸é ±× µ¶¼Ò¿¡ ´ëÇÑ ¾Ë·¹¸£±â Çö»óÀ¸·Î ¹ßÁõÇÑ´Ù. ½ÅÀå ÀüüÀÇ ¿°ÁõÀÎ ¹ü¹ß¼º ½ÅÀå¿°°ú ÀϺο¡ ÀϾ´Â ±¹ÇѼº ½ÅÀå¿°ÀÌ ÀÖ°í, ¹ü¹ß¼º ½ÅÀå¿°¿¡´Â ±Þ¼º°ú ¸¸¼ºÀÌ ÀÖ´Ù. ¡¼±Þ¼º ½ÅÀå¿°¡½ »ó±âµµ °¨¿°Áõ, ¼ºÈ«¿­, ¼¼±Õ¼º ³»¸·¿°, ¼¼±Õ¼º Æó·Å, È­³ó Áúȯ µî¿¡ ¼Ó¹ßÇϰí, ÀþÀº »ç¶÷¿¡°Ô ¸¹À¸¸ç, ƯÈ÷ ±Þ¼º Æíµµ¿°¿¡ ¼Ó¹ßÇÏ´Â ÀÏÀÌ ¸¹´Ù. ºÎÁ¾Àº ¿ì¼± À§ÂÊ ´«²¨Ç®¿¡ ³ªÅ¸³ª¸ç, ƯÈ÷ ¾ÆÄ§¿¡ ÀϾÀ» ¶§ ´«¿¡ ¶ì°Ô ³ªÅ¸³­´Ù. ½ÉÇÏ°Ô µÇ¸é Àü½Å¿¡ ÆÄ±ÞµÇ³ª, ºÎÁ¾ÀÇ Á¤µµ´Â ¹Ýµå½Ã º´ÀÇ °æÁß°ú´Â ÀÏÄ¡ÇÏÁö ¾Ê´Â´Ù. ¿ÀÁÜÀº ´Ü¹éÀÌ ¾ç¼ºÀ̰í, ÀûÇ÷±¸, ¿øÁÖ´¢µµ ÀÖ´Ù. ÇÑÆí, À°¾ÈÀûÀ¸·Î Ç÷´¢¸¦ »ý°¢ÇÏ°Ô Çϱ⵵ ÇÏÁö¸¸ °Ë°æÇϸé Ç×»ó ÀûÇ÷±¸°¡ ³ªÅ¸³­´Ù. ³×ÇÁ·ÎÁ¦
  • nephritis
    ½Å¿°, ½ÅÀå¿°
    »ç±¸Ã¼, ¼¼´¢°ü, ½Å °£Áú Á¶Á÷À» ħ½ÀÇÏ´Â ¼Ò»ó ¶Ç´Â ¹Ì¸¸¼º Áõ½Ä¼º ¶Ç´Â ÆÄ±«¼º °úÁ¤.
  • scarlatinal nephritis
    ¼ºÈ«¿­¼º ½Å¿°, ¼ºÈ«¿­ ½Å¿°
    ¹ßº´ ÈÄ 3ÁÖ ³»¿¡ ¹ß»ýÇϸç, Ç÷´¢, ºÎÁ¾, °íÇ÷¾Ð µîÀÌ ³ªÅ¸³­´Ù.
CancerWEB ¿µ¿µ ÀÇÇлçÀü ¸ÂÃã °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
mesangial nephritis Glomerulonephritis with an increase in glomerular mesangial cells or matrix, or mesangial deposits.
(05 Mar 2000)
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
mesangial Referring to the mesangium.
(05 Mar 2000)
mesangial cell <pathology> Cells found within the glomerular lobules of mammalian kidney, where they serve as structural supports, may regulate blood flow, are phagocytic and may act as accessory cells, presenting antigen in immune responses.
(18 Nov 1997)
mesangial proliferative glomerulonephritis <nephrology, pathology> Inflammation of the kidney glomerulus (blood filtering portion of the kidney) due to the abnormal deposition of IgM antibody in the mesangium layer of the glomerular capillary. A form of glomerulonephritis that appears to be caused by an abnormal immune response. This disorder generally manifests as nephrotic syndrome.
Symptoms include swelling, dark urine, weight gain, hypertension, anorexia and bloody urine.
Acronym: MPGN
(05 Jan 1998)
diffuse mesangial proliferation <nephrology, pathology> Inflammation of the kidney glomerulus (blood filtering portion of the kidney) due to the abnormal deposition of IgM antibody in the mesangium layer of the glomerular capillary. A form of glomerulonephritis that appears to be caused by an abnormal immune response. This disorder generally manifests as nephrotic syndrome.
Symptoms include swelling, dark urine, weight gain, hypertension, anorexia and bloody urine.
Acronym: MPGN
(05 Jan 1998)
acute interstitial nephritis <nephrology> A relatively uncommon condition which is characterised by inflammation of the renal tubules, glomeruli and surrounding tissue. Interstitial nephritis is usually be temporary and often associated with the use of a particular medication. Chronic and progressive forms do exist.
Drugs that have been associated with interstitial nephritis include penicillins, ampicillin, sulpha drugs, non-steroidal anti-inflammatory agents, furosemide and thiazide diuretics. May result in mild kidney dysfunction or acute renal failure. Treatment may be with corticosteroids.
Incidence: 1 in 25,000.
(29 Dec 1997)
acute nephritis <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood).
Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis).
(27 Sep 1997)
analgesic nephritis Chronic interstitial nephritis with renal papillary necrosis, occurring in patients with a long history of excessive consumption of analgesics, especially those containing phenacetin.
Synonym: analgesic nephropathy.
(05 Mar 2000)
anti-basement membrane nephritis Glomerulonephritis produced by autologous or heterologous antibodies to the glomerular capillary basement membranes, the latter known as anti-kidney serum nephritis.
(05 Mar 2000)
anti-kidney serum nephritis Experimental glomerulonephritis produced by injection of antiserum to kidney.
(05 Mar 2000)
Masugi's nephritis Glomerulonephritis produced by injecting into rats a rabbit antiserum prepared against rat kidney tissue suspensions.
(05 Mar 2000)
glomerular nephritis <nephrology> A variety of nephritis characterised by inflammation of the capillary loops in the glomeruli of the kidney. It occurs in acute, subacute and chronic forms and may be secondary to haemolytic streptococcal infection. Evidence also supports possible immune or autoimmune mechanisms.
(18 Nov 1997)
chronic nephritis Glomerulonephritis that presents with persisting proteinuria, chronic renal failure, and hypertension, of insidious onset or as a late sequel of acute glomerulonephritis; the kidneys are symmetrically contracted and granular, with scarring and loss of glomeruli and the presence of tubular atrophy and interstitial fibrosis.
Synonym: chronic nephritis.
(05 Mar 2000)
haemorrhagic nephritis Acute glomerulonephritis accompanied by haematuria.
(05 Mar 2000)
salt-losing nephritis A rare disorder resulting from renal tubular damage of a variety of aetiologies; mimics adrenocortical insufficiency in that abnormal renal loss of sodium chloride occurs, accompanied by hyponatraemia, azotemia, acidosis, dehydration, and vascular collapse.
Synonym: salt-losing syndrome, Thorn's syndrome.
(05 Mar 2000)
hereditary nephritis <pathology> An inherited disorder involving damage to the kidneys, haematuria and hearing loss. In some individuals vision may also be affected. This genetic disease is uncommon.
Symptoms include loss of hearing, abnormal colour to urine, swelling, cough and decline in vision.
Inheritance: sex-linked autosomal dominant.
Incidence: 1 in 50,000.
(27 Sep 1997)
ÇÑ¿µ/¿µÇÑ »çÀü À¯»ç °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • nephritis
    (º´¸®)½Å¿°;BTIGHT'W DISEASE
ÀÌ ¾Æ·¡ ºÎÅÍ´Â °á°ú°¡ ¾ø½À´Ï´Ù.
KMLE ¾àǰ/ÀǾàǰ ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • Á¦Ç°¸í
    ¼ººÐ/ÇÔ·®
    ±¸ºÐ/º¸Çè±Þ¿©
KMLE ¾àǰ/ÀǾàǰ À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • Á¦Ç°¸í
    ¼ººÐ/ÇÔ·®
    ±¸ºÐ/º¸Çè±Þ¿©
¾Ë±â½¬¿î ÀÇÇпë¾îÇ®ÀÌÁý, ¼­¿ïÀÇ´ë ±³¼ö ÁöÁ¦±Ù, °í·ÁÀÇÇÐ ÃâÆÇ ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
´ëÇѽŰæ¿Ü°úÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ÇÑÀÚ
´ëÇѽŰæ¿Ü°úÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ÇÑÀÚ
´ëÇѱâ»ýÃæÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
´ëÇѱâ»ýÃæÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
KI ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
KMLE ÀÇÇоà¾î »çÀü ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
ÀÇÇÐ³í¹® ¾àÀÚ(Pubmed/Entrez) °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
Çѱ¹Ç¥ÁØÁúº´»çÀκзù ¾àÀÚ ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ÄÚµå
    ¿µ¹®
    ÇѱÛ
Çѱ¹Ç¥ÁØÁúº´»çÀκзù ¾àÀÚ À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ÄÚµå
    ¿µ¹®
    ÇѱÛ
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
MeSH(Medical Subject Headings) ¸ÂÃã °Ë»ö (http://www.nlm.nih.gov) °á°ú : 0 ÆäÀÌÁö: 1
MeSH(Medical Subject Headings) À¯»ç °Ë»ö (http://www.nlm.nih.gov) °á°ú : 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - Merriam-Webster's ÀÇÇлçÀü ¸ÂÃã °Ë»ö (https://www.merriam-webster.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - Merriam-Webster's ÀÇÇлçÀü À¯»ç °Ë»ö (https://www.merriam-webster.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - A.D.A.M. Medical Encyclopedia ¸ÂÃã °Ë»ö (http://www.nlm.nih.gov) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - A.D.A.M. Medical Encyclopedia À¯»ç °Ë»ö (http://www.nlm.nih.gov) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - MedlinePlus Health Topics ¸ÂÃã °Ë»ö (http://www.nlm.nih.gov) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - MedlinePlus Health Topics À¯»ç °Ë»ö (http://www.nlm.nih.gov) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - µå·¯±×ÀÎÆ÷ ¾àÇÐ Á¤º¸ ¸ÂÃã °Ë»ö (http://www.druginfo.co.kr) °á°ú: 0 ÆäÀÌÁö: 1
Á¦Ç°¸í
ÆÇ¸Å»ç
º¸ÇèÄÚµå ¼ººÐ/ÇÔ·®
±¸ºÐ/º¸Çè±Þ¿©
¿ÜºÎ ¸µÅ© - µå·¯±×ÀÎÆ÷ ¾àÇÐ Á¤º¸ À¯»ç °Ë»ö (http://www.druginfo.co.kr) °á°ú: 0 ÆäÀÌÁö: 1
Á¦Ç°¸í
ÆÇ¸Å»ç
º¸ÇèÄÚµå ¼ººÐ/ÇÔ·®
±¸ºÐ/º¸Çè±Þ¿©
¿ÜºÎ ¸µÅ© - WebMD.com Drug Reference ¸ÂÃã °Ë»ö (http://www.webmd.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - WebMD.com Drug Reference À¯»ç °Ë»ö (http://www.webmd.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - Drug.com Drugs by Medical Condition ¸ÂÃã °Ë»ö (http://www.drugs.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - Drug.com Drugs by Medical Condition À¯»ç °Ë»ö (http://www.drugs.com) °á°ú: 0 ÆäÀÌÁö: 1
KMLE À¥ ¿ë¾î ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
KMLE À¥ ¿ë¾î À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
ÇÑ¿µ/¿µÇÑ »çÀü ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
WordNet ÀÏ¹Ý ¿µ¿µ »çÀü °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - American Heritage Dictionary ¿µ¿µ»çÀü ¸ÂÃã °Ë»ö (https://www.ahdictionary.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - American Heritage Dictionary ¿µ¿µ»çÀü À¯»ç °Ë»ö (https://www.ahdictionary.com) °á°ú: 0 ÆäÀÌÁö: 1
ÅëÇÕ°Ë»ö ¿Ï·á