| ¿µ¹® | hemoglobin | ÇÑ±Û | Ç÷»ö¼Ò |
|---|---|---|---|
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| ¿µ¹® | fetal monitoring | ÇÑ±Û | žư¨½Ã |
|---|---|---|---|
| ¼³¸í | ÀӽŠÁßÀ̳ª ºÐ¸¸ ÁßÀÇ Å¾ÆÀÇ »óŸ¦ ¾Ë¾Æº¸´Â °ÍÀ» žư¨½Ã¶ó°í ÇÑ´Ù. ¿©±â¿¡´Â ¿©·¯ °¡Áö ¹æ¹ýÀÌ Àִµ¥ Å©°Ô ºÐ¸¸Àü žư¨½Ã¿Í ºÐ¸¸Áß Å¾ư¨½Ã·Î Å©°Ô ³ª´«´Ù. 1. ºÐ¸¸Àü žư¨½Ã´Â ºÐ¸¸Çϱâ ÀÌÀüÀÇ Å¾ÆÀÇ »óŸ¦ °Ë»çÇÏ´Â ¹æ¹ýÀ¸·Î ´ÙÀ½°ú °°Àº ¿©·¯ °¡Áö ¹æ¹ýÀÌ ÀÖ´Ù. 1)¾ç¼öõÀÚ-ÁÖ»ç±â¸¦ ÀÌ¿ëÇØ¼ »ê¸ðÀÇ ¹è¸¦ ÅëÇØ¼ ¾ç¼ö¸¦ ¾ò¾î¼ ºÐ¼®ÇÏ¿© žÆÀÇ »óŸ¦ ¾Ë¾Æº¸´Â ¹æ¹ý. ÀӽŠ15ÁÖ À̻󿡼 ½Ç½ÃÇÒ ¼ö°¡ ÀÖ´Ù. 2)ÅÈÁÙõÀÚ-ÃÊÀ½ÆÄ°Ë»ç¸¦ ½Ç½ÃÇÏ¿© ¿µ»óÀ» º¸¸é¼ ÅÈÁÙ¼Ó¿¡ ¹Ù´ÃÀ» ³Ö¾î¼ ±×°÷ÀÇ Ç÷°üÀ» ã¾Æ Ç÷¾×À» äÃëÇÏ¿© °Ë»çÇÏ´Â ¹æ¹ý. 3)½ºÆ®·¹½º°Ë»ç-žƵµ Àڱüӿ¡¼ ¿îµ¿À» ÇÑ´Ù. ±×¸®°í Á¤»óÀûÀ¸·Î žư¡ Àڱüӿ¡¼ ¿îµ¿À» ÇÒ °æ¿ì¿¡´Â žÆÀÇ ½ÉÀå ¹Úµ¿ÀÌ »¡¶óÁö°Ô µÈ´Ù. žÆÀÇ ¿îµ¿°ú žÆÀÇ ½ÉÀå¹Úµ¿À» µ¿½Ã¿¡ °¨½ÃÇÏ¿© ¿îµ¿½Ã¿¡ žÆÀÇ ½ÉÀå¹Úµ¿ÀÌ »¡¶óÁö´Â °¡¸¦ ¾Ë¾Æº¸´Â °ÍÀÌ´Ù. 4)žÆÃ»°¢Àڱذ˻ç-30ÁÖ ÀÌ»óÀÌ µÈ žƴ ¼Ò¸®ÀÇ Àڱؿ¡ ´ëÇØ¼ ¹ÝÀÀÀ» ÇÑ´Ù. Áï ¼Ò¸®¸¦ µé·ÁÁÖ¾úÀ» °æ¿ì¿¡ žư¡ ¹ÝÀÀÀ» ÇØ¼ ½ÉÀåÀÇ ¹Úµ¿¼ö°¡ Áõ°¡¸¦ ÇÑ´Ù. 5)¼öÃེƮ·¹½º°Ë»ç-Á¤»óÀûÀΠžƴ ÀÚ±ÃÀÌ ¼öÃàÇϸé ÀÌ¿¡ ¹ÝÀÀÀ» ÇØ¼ ½É¹Ú¼ö°¡ º¯ÈÇÑ´Ù. À̰ÍÀ» ÀÌ¿ëÇÏ¿© žÆÀÇ »óŸ¦ °Ë»çÇÏ´Â ¹æ¹ýÀÌ´Ù. 2. ºÐ¸¸Áß Å¾ư¨½Ã ºÐ¸¸µµÁß¿¡ žÆÀÇ °Ç°»óŸ¦ °¨½ÃÇÏ´Â °ÍÀÌ´Ù. ´ëü·Î ºÐ¸¸µµÁß¿¡ žư¡ Àú»ê¼ÒÁõÀ̳ª ¿©·¯ °¡Áö Ãæ°ÝÀ» ¹Þ´Â °æ¿ì°¡ ¸¹À¸¹Ç·Î ºÐ¸¸µµÁßÀÇ Å¾ư¨½Ã´Â Áß¿äÇÑ Àǹ̸¦ °¡Áø´Ù. 1)ÀüÀڽĞư¨½Ã-Àü±âÀû ÀåÄ¡¸¦ ÀÌ¿ëÇÏ¿© žÆÀÇ »óŸ¦ ¾Ë¾Æº¸´Â °ÍÀ¸·Î °¨½ÃÀÇ ´ë»óÀº ÀÚ±ÃÀÇ ¼öÃà°ú žÆÀÇ ½ÉÀå¹Úµ¿¼öÀÌ´Ù. 2)žƸӸ®µ¤°³ Ç¥º»Ã¤Ãë Ç÷¾×-ºÐ¸¸µµÁß¿¡ žÆÀÇ µÎÇÇÀÇ Ç÷°ü¿¡¼ Ç÷¾×À» äÃëÇÏ¿© °Ë»ç¸¦ ÅëÇØ¼ žÆÀÇ »óŸ¦ ¾Æ´Â ¹æ¹ýÀÌ´Ù. ÁÖ·Î °Ë»ç¸¦ ÇàÇÏ´Â Á¾¸ñÀº Ç÷¾×ÀÇ pHÀÌ´Ù. |
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| ¿µ¹® | fetal assessment | ÇÑ±Û | žƻçÁ¤ |
|---|---|---|---|
| ¼³¸í | ÀӽŠÁßÀ̳ª ºÐ¸¸ ÁßÀÇ Å¾ÆÀÇ »óŸ¦ ¾Ë¾Æº¸´Â °ÍÀ» žư¨½Ã¶ó°í ÇÑ´Ù. ¿©±â¿¡´Â ¿©·¯ °¡Áö ¹æ¹ýÀÌ Àִµ¥ Å©°Ô ºÐ¸¸Àü žư¨½Ã¿Í ºÐ¸¸Áß Å¾ư¨½Ã·Î Å©°Ô ³ª´«´Ù. 1. ºÐ¸¸Àüžư¨½Ã: ºÐ¸¸Çϱâ ÀÌÀüÀÇ Å¾ÆÀÇ »óŸ¦ °Ë»çÇÏ´Â ¹æ¹ýÀ¸·Î ´ÙÀ½°ú °°Àº ¿©·¯ °¡Áö ¹æ¹ýÀÌ ÀÖ´Ù. ¨ç ¾ç¼öõÀÚ: ÁÖ»ç±â¸¦ ÀÌ¿ëÇØ¼ »ê¸ðÀÇ ¹è¸¦ ÅëÇØ¼ ¾ç¼ö¸¦ ¾ò¾î¼ ºÐ¼®ÇÏ¿© žÆÀÇ »óŸ¦ ¾Ë¾Æº¸´Â ¹æ¹ý. ÀӽŠ15ÁÖ À̻󿡼 ½Ç½ÃÇÒ ¼ö ÀÖ´Ù. ¨è ÅÈÁÙõÀÚ: ÃÊÀ½ÆÄ°Ë»ç¸¦ ½Ç½ÃÇÏ¿© ¿µ»óÀ» º¸¸é¼ ÅÈÁÙ ¼Ó¿¡ ¹Ù´ÃÀ» ³Ö¾î¼ ±× °÷ÀÇ Ç÷°üÀ» ã¾Æ Ç÷¾×À» äÃëÇÏ¿© °Ë»çÇÏ´Â ¹æ¹ý. ¾ÆÁÖ À§ÇèÇÒ °Í °°Áö¸¸ »ó´çÈ÷ ¾ÈÀüÇÑ °ÍÀ¸·Î µÇ¾î ÀÖ´Ù. ´Ù¸¥ °Ë»ç¿¡ ºñÇØ¼ °Ë»ç¿¡ ¸¹Àº ±â¼úÀÌ ÇÊ¿äÇÏÁö¸¸ ¾ÆÁÖ ¸¹Àº Á¤º¸¸¦ Á¦°øÇØ ÁØ´Ù. ¨é ºñ½ºÆ®·¹½º°Ë»ç(nonstress test) : žƵµ Àڱà ¼Ó¿¡¼ ¿îµ¿À» ÇÑ´Ù. ±×¸®°í Á¤»óÀûÀ¸·Î žư¡ Àڱà ¼Ó¿¡¼ ¿îµ¿À» ÇÒ °æ¿ì¿¡´Â žÆÀÇ ½ÉÀå ¹Úµ¿ÀÌ »¡¶óÁö°Ô µÈ´Ù. žÆÀÇ ¿îµ¿°ú žÆÀÇ ½ÉÀå¹Úµ¿À» µ¿½Ã¿¡ °¨½ÃÇÏ¿© ¿îµ¿½Ã¿¡ žÆÀÇ ½ÉÀå¹Úµ¿ÀÌ »¡¶óÁö´Â °¡¸¦ ¾Ë¾Æº¸´Â °ÍÀÌ´Ù. |
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| ¿µ¹® | fetal alcohol syndrome | ÇÑ±Û | žƾËÄÚ¿ÃÁõÈıº |
|---|---|---|---|
| ¼³¸í | ÀӽűⰣ Áß ¸¸¼ºÀûÀ¸·Î ¾ËÄÚ¿ÃÀ» ¼·ÃëÇÑ ¿©ÀÚ¿¡°Ô¼ ÅÂ¾î³ ¿µ¾Æ¿¡°Ô ³ªÅ¸³ª´Â ÇüŹ߻ýÀÇ ÀÌ»óÀ» ³ªÅ¸³»´Â ÁõÈıºÀ¸·Î¼ À§ÅλÀ¹ßÀ°ºÎÀü, ¾Õ¸Ó¸®¿Í ¾Æ·¡ÅÎÀÇ µ¹Ãâ, ªÀº°Ë¿, ÀÛÀº¾È±¸Áõ, ´«±¸¼®ÁÖ¸§, ½ÉÇÑ ¼ºÀåÁö¿¬, Á¤½ÅÁöü µîÀ» ³ªÅ¸³½´Ù. |
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| HPFH | hereditary persistence of fetal hemoglobin |
|---|---|
| HCG, hCG | Human Chorionic Gonadotropin; »ç¶÷À¶¸ð¼º¼º¼±ÀÚ±ØÈ£¸£¸ó 1. Placental Glycoprotein Hormone &nbs... |
| HPAFT | hereditary persistence of alfa-fetoprotein |
| HbF | fetal hemoglobin, hemoglobin F |
| N-P | need-persistence |
| HPFH | Hereditary Persistence of Fetal Hemoglobin |
|---|---|
| DCLHb | Diaspirin Cross-Linked Hemoglobin |
| GHb | Glycosylated Hemoglobin |
| HbA1C | Hemoglobin A1C |
| Hb A2 | Hemoglobin A2 |
| hereditary persistence of foetal haemoglobin | <haematology> Hereditary persistence of foetal haemoglobin is a genetic condition where adult types of haemoglobin fail to develop and the types of haemoglobin the individual had as a foetus remains present well past the point when they would normally have stopped being produced. (09 Oct 1997) |
|---|---|
| hemoglobin | <cell biology, haematology> Four subunit globular oxygen carrying protein of the erythrocytes of vertebrates and some invertebrates. It is a conjugated protein containing four haem groups and globin. There are two alpha and two beta chains (very similar to myoglobin) in adult humans, the haem moiety (an iron containing substituted porphyrin) is firmly held in a nonpolar crevice in each peptide chain. There are four globin polypeptide chains, designated alpha, beta, gamma, delta in the adult. Each is composed of several hundred amino acids. (08 Mar 2000) |
| fetal | Of or pertaining to a foetus, pertaining to in utero development after the embryonic period. (18 Nov 1997) |
| persistence | 1. The tendency of a cell to continue moving in one direction: an internal bias on the random walk behaviour that cells exhibit in isotropic environments. 2. Of viruses that persist in a cell population, animal, plant or population for long periods often in a nonreplicating form, by such strategies as integration into host DNA, immunological suppression or mutation into forms with slow replication. (18 Nov 1997) |
| Albright's hereditary osteodystrophy | An inherited form of hyperparathyroidism associated with ectopic calcification and ossification and skeletal defects, notably the small fourth metacarpals, but intelligence is normal. There are dominant, recessive and X-linked forms. See: pseudohypoparathyroidism. Synonym: Albright's syndrome. (05 Mar 2000) |
| angioedema, hereditary | A genetic form of angioedema. (Angioedema is also referred to as Quinke's disease.) Persons with it are born lacking an inhibitor protein (called C1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of C1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioneurotic oedema. (12 Dec 1998) |
| angioneurotic oedema, hereditary | A genetic form of angioedema. (Angioedema is also referred to as Quinke's disease.) Persons with it are born lacking an inhibitor protein (called C1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of C1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioedema. (12 Dec 1998) |
| canine hereditary blindness | An autosomal dominant condition seen in dogs of the collie and several other breeds. (05 Mar 2000) |
| colourectal neoplasms, hereditary nonpolyposis | A syndrome characterised by autosomal dominant inheritance, a low mean age (41 years) for occurrence of colon cancer, and a marked increase in the proportion of tumours in the proximal colon. (12 Dec 1998) |
| corneal dystrophies, hereditary | Bilateral hereditary disorders of the cornea, usually autosomal dominant, which may be present at birth but more frequently develop during adolescence and progress slowly throughout life. Central macular dystrophy is transmitted as an autosomal recessive defect. (12 Dec 1998) |
| hereditary | <genetics> Transferred via genes from parent to child. (16 Dec 1997) |
| hereditary amyloidosis | <neurology> A disorder in which various peripheral nerves are infiltrated with amyloid and their functions disturbed, an abnormal prealbumin is also formed and is present in the blood; characteristically, it begins during mid-life and is found largely in persons of Portuguese descent. Other rare clinical types occur. Inheritance: autosomal dominant. Synonym: familial amyloidosis, hereditary amyloidosis. (05 Mar 2000) |
| hereditary angioedema | A genetic form of angioedema. (angioedema is also referred to as quinke's disease.) persons with it are born lacking an inhibitor protein (called c1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of c1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioneurotic oedema. (12 Dec 1998) |
| hereditary angioneurotic oedema | A genetic form of angioedema. (angioedema is also referred to as quinke's disease.) persons with it are born lacking an inhibitor protein (called c1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of c1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioedema. (12 Dec 1998) |
| hereditary angio oedema | <biochemistry> Condition in which there seems to be uncontrolled production of C2 kinin because of a deficiency in C1 inhibitor levels. (18 Nov 1997) |
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