| ¿µ¹® | hemolytic disease of newborn | ÇÑ±Û | ½Å»ý¾Æ¿ëÇ÷º´ |
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| ¼³¸í | ½Å»ý¾Æ¿¡¼ ÀûÇ÷±¸°¡ ºñÁ¤»óÀûÀ¸·Î ¸¹ÀÌ ÆÄ±«µÇ´Â º´À¸·Î žÆÀû¸ð±¸Áõ(erythroblastosis fetalis)¿Í °°Àº ¶æÀ¸·Î ¾²ÀδÙ. À̰ÍÀº ¾î¸Ó´Ï¿¡°Ô¼ »ý»êµÈ ½Å»ý¾Æ³ª žÆÀÇ ÀûÇ÷±¸¿¡ ´ëÇÑ Ç×ü°¡ ŹÝÀ» °Ç³Ê¿Í¼ žÆÀÇ ÀûÇ÷±¸¿Í °áÇÕÇÏ¿©¼ »ý±â´Â ¿ëÇ÷¼ººóÇ÷À» À̸£´Â ¸». Áï ½Å»ý¾Æ³ª žÆÀÇ ÀûÇ÷±¸ÀÇ Ç×ü°¡ ¾î¸Ó´ÏÀÇ ¸ö¿¡¼ »ý»êÀÌ µÇ°í À̰ÍÀÌ Å¹ÝÀ» ÅëÇØ¼ žƿ¡°Ô ³Ñ¾î°¡¼ žÆÀÇ ÀûÇ÷±¸¿Í °áÇÕÀ» Çϰí ÀÌ Ç×ü¿Í °áÇÕÇÑ ÀûÇ÷±¸´Â ÆÄ±«°¡ µÇ¾î¼ ºóÇ÷ÀÌ »ý±ä °ÍÀ» žÆÀû¸ð±¸ÁõÀ̶ó°í ÇÑ´Ù. À̰ÍÀº Rh Àû¸ð±¸Áõ(Rh erythroblastosis)¿Í ABO Àû¸ð±¸Áõ(ABO erythroblastosis)·Î ³ª´ ¼ö°¡ ÀÖ´Ù. |
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| ¿µ¹® | hemolytic anemia | ÇÑ±Û | ¿ëÇ÷ºóÇ÷ |
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| ¼³¸í | ¿ëÇ÷ºóÇ÷À̶õ ÀûÇ÷±¸ÀÇ °úµµÇÑ ÆÄ±«¿¡ ÀÇÇÑ ºóÇ÷ÀÌ´Ù. ¿ø·¡ 120ÀÏ Á¤µµÀÇ ¼ö¸íÀ» °¡Áö´Â ÀûÇ÷±¸ÀÇ ¼ö¸íÀÌ Âª¾ÆÁö´Â °ÍÀÌ´Ù. ¿©±â¿¡´Â ¿©·¯ °¡Áö ¿øÀÎÀÌ ÀÖÀ» ¼ö°¡ Àִµ¥ ´ëÇ¥ÀûÀÎ ¿øÀÎÀ¸·Î´Â ÀûÇ÷±¸¿¡ ´ëÇÑ Ç×ü°¡ »ý±â´Â °Í(¹ßÀÛ¼º¾ß°£Ç÷»ö¼Ò´¢Áõ)°ú ÀûÇ÷±¸ÀÚüÀÇ ÀÌ»ó(À¯Àü¼ºµÕ±ÙÀûÇ÷±¸Áõ), ±×¸®°í ´Ù¸¥ Áúº´¿¡ ÀÇÇØ¼ 2Â÷ÀûÀ¸·Î »ý±â´Â °ÍÀÌ ÀÖ´Ù. |
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| ¿µ¹® | infection | ÇÑ±Û | °¨¿° |
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| ¼³¸í | º´¿ø¹Ì»ý¹°ÀÌ »ç¶÷À̳ª µ¿¹° ¶Ç´Â ½Ä¹°ÀÇ Á¶Á÷. ü¾×-Ç¥¸é¿¡ Á¤ÂøÇÏ¿© Áõ½ÄÇÏ´Â »óÅÂ. ÀÌ °æ¿ì µ¿¹° ¶Ç´Â ÀÎü¿¡ ¿©·¯ °¡Áö Áõ»ó, Áï Áúº´À» ÀÏÀ¸Å°´Â °æ¿ì¿Í ÀÏÀ¸Å°Áö ¾Ê´Â °æ¿ì°¡ ÀÖ´Ù. ¿¹¸¦ µé¸é, ÀϺ»³ú¿°¹ÙÀÌ·¯½º°¡ ÀÎü¿¡ ħÀÔÇÏ¿© ü³»¿¡ Áõ½ÄÇÏ¸é ¾î¶² »ç¶÷¿¡°Ô´Â °í¿-µÎÅë-ÀǽÄÀå¾Ö-°æ·Ã µîÀÇ Áõ»óÀÌ ÀϾ ¹ßº´À» ¾ËÁö¸¸, ´ë´Ù¼öÀÇ »ç¶÷Àº ü³»¿¡¼ ¹ÙÀÌ·¯½º°¡ Áõ½ÄÇÏ´õ¶óµµ Áõ¼¼ÀÇ Á¤µµ°¡ ³·°í ¹ß¿À̳ª ±× ¹ÛÀÇ Áõ¼¼µµ ¾ø¾î °¨¿°À» ¸ð¸¥´Ù. ÀÌ¿Í °°ÀÌ º´¿ø¹Ì»ý¹°Àº ÀÎü¿¡ °¨¿°µÇ´õ¶óµµ ¹ßº´ÇÏ´Â °æ¿ì¿Í ÇÏÁö ¾Ê´Â °æ¿ì°¡ ÀÖ´Ù. ÀüÀÚ¸¦ Áõ»ó°¨¿°, ÈÄÀÚ¸¦ ¹«Áõ»ó°¨¿°À̶ó ÇÑ´Ù. °¨¿°ÀÇ ±Ù¿øÀÌ µÇ´Â ȯÀÚ-º¸±ÕÀÚ-°¨¿°µ¿¹°-¸Å°³µ¿¹°-º´¿øÃ¼¸¦ Æ÷ÇÔÇÑ ¹è¼³¹° ¹× ±×¿¡ ÀÇÇØ °¨¿°µÈ °ÍÀ» °¨¿°¿øÀ̶ó Çϰí, ÀÌ·¯ÇÑ °¨¿°¿ø¿¡¼ Á÷Á¢ ¶Ç´Â °£Á¢À¸·Î »ýü¿¡ º´¿øÃ¼°¡ ħÀÔÇÏ´Â °æ·Î¸¦ °¨¿°°æ·Î¶ó ÇÑ´Ù. °¨¿°°æ·Î¿¡´Â °ø±â°¨¿°-Á¢Ã˰¨¿°-°æ±¸°¨¿°-°æÇǰ¨¿° µîÀÌ ÀÖ´Ù. ¶ÇÇÑ °¨¿°ÁõÀº Àü¿°¼º°ú ºñÀü¿°¼ºÀÇ µÎ °¡Áö·Î ³ª´ ¼ö ÀÖ´Ù. ÀüÀÚ´Â Áúº´ÀÇ °æ°ú Áß¿¡(¶§·Î´Â Àẹ±â³ª ȸº¹±â¿¡) °¨¿°ÇÑ »ýüÀÇ ºÐºñ¹° ¶Ç´Â ¹è¼³¹°°ú ÇÔ²² º´¿øÃ¼°¡ ³ª¿Í¼ Á¢ÃË ¶Ç´Â ¸Å°³¿¡ ÀÇÇØ ´Ù¸¥ °³Ã¼¸¦ °¨¿°½ÃŰ´Â °æ¿ì¸¦ ¸»ÇÑ´Ù. ¸¶¸¶-µðÇÁÅ׸®¾Æ-¼ºÈ«¿-Æä½ºÆ®-ÄÝ·¹¶ó-ÀÌÁú µîÀÌ ÀÌ¿¡ ¼ÓÇÑ´Ù. ÈÄÀÚ´Â º´¿øÃ¼°¡ °¨¿°ÇÑ »ýü¿¡¼ ¹è¼³µÇÁö ¾Ê°Å³ª ¹è¼³µÇ´õ¶óµµ ´Ù¸¥ °³Ã¼¿¡´Â °¨¿°À» ÀÏÀ¸Å°Áö ¾Ê´Â °ÍÀ¸·Î ¿©±â¿¡´Â ÆÄ»ódz-¸»¶ó¸®¾Æ-¹ßÁøÆ¼Çª½º-»êÈÄ¿ µîÀÌ ÀÖ´Ù. |
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| ¿µ¹® | droplet infection | ÇÑ±Û | ºñ¸»°¨¿°, ÀÛÀº¹æ¿ï°¨¿° |
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| ¼³¸í | º¸±ÕÀÚ³ª Áõ»óÀÌ Àִ ȯÀÚ È¤Àº ÀÌ¹Ì °¨¿°µÇ¾î ÀÖ´Â »ç¶÷ÀÇ È£Èí¿¡¼ ³ª¿Â Á÷°æ 10¸¶ÀÌÅ©·Ð ¶Ç´Â ±× ÀÌÇÏÀÇ ¾×üÀÔÀÚ¿¡ ºÎÀ¯Çϰí ÀÖ´Â º´¿øÃ¼ÀÇ ÈíÀÔ¿¡ ÀÇÇÑ È£Èí±â°¨¿°À» À̸¥´Ù. ÀÎÇ÷翣ÀÚ³ª Æíµµ¿°°ú °°ÀÌ È¯ÀÚ°¡ ±âħÀ» Çϰųª ´ëÈ µµÁß¿¡ ÀÚÀßÇÑ ºñ¸»°ú ÇÔ²² º´¿ø±ÕÀÌ °ø±â¿Í ÇÔ²² º´¿ø±ÕÀÌ ¹æÃâµÇ¾î °ø±â¿Í ÇÔ²² È£Èí±â·Î ÈíÀÔµÊÀ¸·Î½á °¨¿°µÇ´Â °ÍÀ» ¸»ÇÑ´Ù. °áÇÙ-À¯Ç༺°¨±â-¹éÀÏÇØ-µðÇÁÅ׸®¾Æ-Æó·Å µîÀÌ ÀÌ¿¡ ÀÇÇÏ¿© ÀüÆÄµÈ´Ù. |
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| ¿µ¹® | wound infection | ÇÑ±Û | »ó󰨿° |
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| ¼³¸í | Àý¼Õ µîÀÇ ±â°èÀû »óÇØ, ÀÎÀ§Àû ºÎ»ó ¶Ç´Â Ÿ±ÕÀÇ Ä§ÀÔ¿¡ ÀÇÇØ »óó³ Á¶Á÷¿¡¼ ħÀÔÇÏ¿© °¨¿°½ÃŰ´Â °Í. |
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| AHA | acetohydroxamic acid; acquired hemolytic anemia; acute hemolytic anemia; American Heart Association;... |
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| MHD | maintenance hemodialysis; mean hemolytic dose; mental health department; minimum hemolytic dilution;... |
| GRABS | group A beta-hemolytic streptococcal pharyngitis |
| EOGBS | early onset group B streptococcal [infection] |
| MAHA | Micro-Angiopathic Hemolytic Anemia; PB»ó Helmet Cell ThrombocytopeniaÁß MAHAÀ¯¹ß &nbs... |
| GABHS | Group A beta hemolytic streptococcal |
|---|---|
| BHS | Beta-hemolytic streptococci |
| CH 50 | Complement hemolytic activity |
| DHTR | Delayed hemolytic transfusion reaction |
| GBS | Group B beta hemolytic streptococci |
| hemolytic anaemia | <disease, haematology> Anaemia resulting from reduced red cell survival time and haemolysis, either due to an intrinsic defect in the erythrocyte (hereditary spherocytosis or ellipsocytosis, enzyme defects, haemoglobinopathy) or an extrinsic damaging agent. For example autoantibody (autoimmune haemolytic anaemia), iso antibody, parasitic invasion of the cells (malaria), bacterial or chemical haemolysins, mechanical damage to erythrocytes. Origin: Gr. Haima = blood (18 Nov 1997) |
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| acute post-streptococcal glomerulonephritis | <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood). Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis). (27 Sep 1997) |
| group A streptococcal necrotizing fasciitis | A complication of infection with GAS (group A streptococci) in which the bacteria attacks and destroys muscle tissue. According to the CDC, 5-10% of people with severe GAS infection develop necrotizing fasciitis. Though the infection can be treated with antibiotics, the fatality rate is close to 30%. This complication often develops as a wound infection after surgery or injury. (05 Mar 2000) |
| streptococcal | Relating to or caused by any organism of the genus Streptococcus. (05 Mar 2000) |
| streptococcal fibrinolysin | <enzyme> Plasminogen activator released by Streptococcus pyogenes. Occurs in two forms, A and B. (18 Nov 1997) |
| streptococcal infections | Infections with bacteria of the genus streptococcus. (12 Dec 1998) |
| streptococcal lymphadenitis | A contagious bacterial disease of pigs caused by a group E streptococcus and characterised by the formation of abscesses in the cervical and/or cephalic lymph nodes. (05 Mar 2000) |
| streptococcal nuclease | <enzyme> From streptococcus haemolyticus; degrades RNA and DNA producing oligonucleotides terminating in 5'-phosphate Registry number: EC 3.1.30.- (26 Jun 1999) |
| streptococcal pneumonia | Pneumonia due to Streptococcus pyogenes. (05 Mar 2000) |
| streptococcal toxins | Group of haemolytic exotoxins released by Streptococci. _ haemolysin: 26-39 Kd (four types), forms ring like structures in membranes (see Streptolysin O). Lipid target unclear. _ haemolysin: a hot cold haemolysin with sphingomyelinase C activity. _ haemolysin: complex of two proteins (29 and 26 kD) that act synergistically, rabbit erythrocytes particularly sensitive. _ toxin: heat stable peptide (5 kD) with high proportion of hydrophobic amino acids. Seems to act in a detergent like manner (c.f. Subtilysin), but may form hydrophilic transmembrane pores by cooperative interaction with other _ toxin molecules. Leucocidin (Panton Valentine leucocidin): two components f (fast migration on CM cellulose column: 32 kD) and s (slow: 38 kD). Mode of action contentious. See: Streptococcus, streptolysins O and S, erythrogenic toxin. (18 Nov 1997) |
| agonal infection | An acute infection, commonly pneumonic or septic, occurring toward the end of any disease and often the cause of death. Synonym: agonal infection. (05 Mar 2000) |
| airborne infection | A mechanism of transmission of an infectious agent by particles, dust, or droplet nuclei suspended in the air. (05 Mar 2000) |
| apical infection | Implantation of microorganisms at the apex of a tooth, usually the result of the migration of microorganisms from the pulp canal through the apical foramen. (05 Mar 2000) |
| arbovirus infection | <virology> A type of viral infection that is transmitted by mosquitoes in late spring to early autumn. One manifestation is encephalitis (central nervous system infection). (27 Sep 1997) |
| ascariasis infection | <microbiology> Infection by the nematode Ascaris lumbricoides and is characterised by an early pulmonary phase related to larval migration and a later, prolonged intestinal phase. Adult worms are 15-40 cm in length and maintain themselves in the lumen of the small intestine. Infection occurs after ingesting eggs contained in contaminated food or more commonly, by transmission to the mouth by the hands after contact with contaminated soil. Treatment is with mebendazole or pyrantel pamoate. (27 Sep 1997) |
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