| ¿µ¹® | receptor | ÇÑ±Û | ¼ö¿ëü |
|---|---|---|---|
| ¼³¸í | ¼¼Æ÷Áú³» ¶Ç´Â ¼¼Æ÷Ç¥¸é¿¡ Á¸ÀçÇÏ´Â ºÐÀÚ±¸Á¶·Î¼ ƯÀ̹°Áú°ú ¼±ÅÃÀûÀ¸·Î °áÇÕÇÏ¸ç °áÇÕ¿¡ ÀÇÇØ ƯÀÌÇÑ »ý¸®Àû ÀÛ¿ëÀ» ³ªÅ¸³½´Ù. ÆéƼµåÈ£¸£¸ó, ½Å°æÀü´Þ¹°Áú, Ç׿ø, º¸Ã¼, ¸é¿ª±Û·ÎºÒ¸°¿¡ ´ëÇÑ ¼¼Æ÷Ç¥¸é ¼ö¿ëü¿Í ½ºÅ×·ÎÀ̵忡 ´ëÇÑ ¼¼Æ÷Áú³» ¼ö¿ëü°¡ ÀÖ´Ù. |
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| ¿µ¹® | basal layer of skin | ÇÑ±Û | ±âÀúÃþ |
|---|---|---|---|
| ¼³¸í | Ç¥ÇÇÀÇ °¡Àå ¾Æ·¡ ºÎºÐÀÌ¸ç ¼¼Æ÷ÀÇ ¸ð¾çÀÌ ºñ±³Àû Á÷»ç°¢Çü¿¡ °¡±õ°í ÀÏÁ¤ÇÑ ¹è¿À» ÇÑ´Ù. |
||
| ¿µ¹® | skin tag | ÇÑ±Û | ÁãÁ¥, ÇǺο¬¼º¼¶À¯Á¾ |
|---|---|---|---|
| ¼³¸í | ÁÖ·Î ¸ñ, °Üµå¶ûÀÌ, ´Ù¸®¿¡ »ý±â´Â »ì»ö ¶Ç´Â °ú»ö¼Ò Ä§Âø¼ºÀÇ ÀÛÀº ¸ñÀ» °¡Áø ¼¶À¯¼º µ¹Ãâ¹°·Î ´ë°³ ¹«Áõ»óÀÌ´Ù. |
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| ¿µ¹® | skin | ÇÑ±Û | ÇǺΠ|
|---|---|---|---|
| ¼³¸í | ½Åü¸¦ ½Î°í ÀÖ´Â ¹Ù±ùÃþÀ¸·Î¼ Ç¥ÇÇ, ÁøÇÇ, ÇÇÇÏÁ¶Á÷À¸·Î ±¸¼ºµÇ¸ç, ¿ÜºÎ ȯ°æÀ¸·ÎºÎÅÍ ½Åü¸¦ º¸È£Çϰí ü¿ÂÀ» Á¶ÀýÇÏ´Â ±â´ÉÀ» °¡Áü. 1.Ç¥ÇÇ: ÇǺÎÀÇ °¡Àå ¹Ù±ùÃþÀ¸·Î¼ ¿Ü¹è¿±¿¡¼ ±â¿øÇÏ¸ç °¢ÁúÀ̶ó´Â ±¸Á¶ ´Ü¹éÁúÀ» »ý»êÇÏ´Â ±â´ÉÀ» °¡Áü. 4°¡ÁöÀÇ ¼¼Æ÷°¡ Àִµ¥, °¢Áú¼¼Æ÷, ¸á¶ó´Ñ¼¼Æ÷, Langerhans ¼¼Æ÷, ºÎÁ¤Çü ¼¼Æ÷, Merkel¼¼Æ÷°¡ ÀÖ´Ù. Ç¥ÇÇ¿¡´Â ÀÌ 4°¡Áö ¼¼Æ÷¿Ü¿¡ ´ÙÀ½°ú °°Àº Ç¥ÇÇ ºÎ¼Ó±â°¡ ÀÖ´Ù. ¿¡Å©¸° ¶¡»ù(eccrine sweat gland): Á¡¸· ÇǺΠ°æ°è ºÎÀ§ µî Ư¼öÇÑ ºÎÀ§¸¦ Á¦¿ÜÇÑ ¸ðµç ÇǺο¡¼ ¹ß°ßµÇ¸ç ¶¡À» ºÐºñÇÏ´Â »ùÀ¸·Î¼ ½Åü ¿ Á¶Àý ü°èÀÇ Áß¿äÇÑ ¿ä¼Ò. ¾ÆÆ÷Å©¸°»ù(apocrine gland): °Üµå¶ûÀÌ, Á¥²ÉÆÇ, Ç×¹® »ý½Ä±â ºÎÀ§, ¿ÜÀ̵µ, ´«²¨Ç® µî¿¡¼¸¸ ¹ß°ßµÇ¸ç ºÐºñ¹°ÀÇ ±â´ÉÀº Àß ¾Ë·ÁÁ® ÀÖÁö ¾ÊÀ¸³ª µ¿¹°¿¡¼´Â ¹æ¾î ¹× ¼ºÀû ±â´ÉÀÌ ÀÖÀ½. ÅÐÁÖ¸Ó´Ï(hair follicle): ¼Õ¹Ù´Ú, ¹ß¹Ù´Ú µî Ư¼ö ºÎÀ§¸¦ Á¦¿ÜÇÑ ½Åü Àü ºÎÀ§¿¡ ºÐÆ÷Çϸç Àΰ£¿¡¼ Áß¿äÇÑ »ý¸®Àû ±â´ÉÀº ¾øÀ¸³ª ¼ºÀû ¸Å·ÂÀ» Áö´Ï´Â ½Åü Àå½ÄÀÇ ±â´ÉÀ» °¡Áü. ±â¸§»ù(sebaceous gland): ¼Õ¹Ù´Ú°ú ¹ß¹Ù´ÚÀ» Á¦¿ÜÇÑ Àü ÇǺο¡ ºÐÆ÷ÇÏ¸ç ºÐºñ¹°À» ±â¸§(sebum)¶ó ºÎ¸§. ³²¼º È£¸£¸óÀÇ Àڱؿ¡ ÀÇÇØ ¼ºÀåÇÏ¸ç »ý¸®Àû ±â´ÉÀº Àß ¾Ë·ÁÁ® ÀÖÁö ¾ÊÀ½. ¼Õ¹ßÅé(nail) 2. ÁøÇÇ: Ç¥ÇÇ ¾Æ·¡¿¡ Á¸ÀçÇÏ´Â Á߹迱 ±â¿øÀÇ Á¶Á÷À¸·Î¼ Ç÷°ü, ¸²ÇÁ°ü, ½Å°æ, ±ÙÀ°, Ç¥ÇÇ ºÎ¼Ó±â µîÀ» Æ÷ÇÔÇϰí ÀÖÀ½. Ç¥ÇÇ ¹Ù·Î ¹ØÀÇ ºÎÀ§¸¦ À¯µÎ ÁøÇǶó ÇÏ°í ¾Æ·¡ÃþÀ» ¸Á»ó ÁøÇǶó ÇÑ´Ù. Á¶Á÷ ¼¶À¯¿¡´Â ¾Æ±³Áú ¼¶À¯, ±×¹° ¼¶À¯, ź·Â ¼¶À¯ÀÇ ¼¼ Á¾·ù°¡ ÀÖ´Ù. 3. ÇÇÇÏ Á¶Á÷: ÁøÇÇ ¾Æ·¡¿¡ Á¸ÀçÇϸç Á߹迱 ±â¿øÀ¸·Î¼ º¸¿Â ÀÛ¿ë, Ãæ°Ý Èí¼ö ÀÛ¿ë, ¿µ¾ç ÀúÀå ÀÛ¿ëÀÇ ±â´ÉÀÌ ÀÖ´Ù. |
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| ¿µ¹® | skin dose | ÇÑ±Û | ÇǺμ±·® |
|---|---|---|---|
| ¼³¸í | 1. ÇǺÎÇ¥¸é¿¡¼ÀÇ ¹æ»ç¼±ÀÇ °øÁß¼±·®À¸·Î ¿©±â¿¡´Â 1Â÷¹æ»ç¼±°ú ÈĹæ»ê¶õÀÌ Æ÷ÇԵȴÙ. 2. ÇǺο¡ Èí¼öµÇ´Â ¼±·®. |
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| GPHN | giant pigmented hairy nevus |
|---|---|
| HC | hair cell; hairy cell; handicapped; head circumference; head compression; health care; healthy contr... |
| HCL | hairy-cell leukemia; human cultured lymphoblasts |
| HL | hairline; hairy leukoplakia; half life; hearing level; hearing loss; heparin lock; histiocytic lymph... |
| OHL | oral hairy leukoplakia |
| HCL | Hairy Cell Leukaemia |
|---|---|
| HC | Hairy Cells |
| HL | Hairy leucoplakia |
| OHL | Oral "hairy" leukoplakia |
| Su(Hw) | Suppressor of Hairy wing |
Nd:YAG
| giant hairy nevus | <dermatology> These large pigmented (often hairy) congenital nevi are important because of their increased risk (10 to 15%) of conversion into malignant melanoma. A biopsy can confirm if cells have turned malignant. Any change in a pre-existing nevus should prompt a physician evaluation. (17 Dec 1997) |
|---|---|
| hairy | A pair rule gene of Drosophila. (18 Nov 1997) |
| hairy cell leukaemia | <haematology, oncology> A rare chronic disorder characterised by proliferation of hairy cells in reticuloendothelial organs and blood. Origin: Gr. Haima = blood (13 Nov 1997) |
| hairy cells | Medium sized leukocytes that have features of reticuloendothelial cell's and multiple cytoplasmic projections (hairs) on the cell surface, but which may be a variety of B lymphocyte; they are found in hairy cell leukaemia. (05 Mar 2000) |
| hairy heart | Acute pericarditis with fibrinous exudate. See: bread-and-butter pericardium. Synonym: hairy heart, pericarditis villosa, shaggy pericardium. (05 Mar 2000) |
| hairy leukoplakia | <dermatology, gastroenterology> A white lesion appearing on the tongue, occasionally on the buccal mucosa, of patients with AIDS, the lesion appears raised, with a corrugated or hairy surface due to keratin projections. (13 Nov 1997) |
| hairy mole | A mole covered with an abundant growth of hair. Synonym: hairy mole. (05 Mar 2000) |
| hairy root culture | A relatively new type of plant culture, which consists of highly branched roots of a plant, covered with a mass of tiny root hairs. These cultures do not require hormones or vitamins to grow, so they can grow on simple media of salts and sugars. Just as importantly, they produce secondary metabolites at levels similar to those made in the original plant, so they can be used as replacement plants for making such compounds as food flavours and fragrances. (14 Nov 1997) |
| hairy shaker disease | Congenital disorder of lambs caused by a virus closely related to or identical with certain strains of bovine viral diarrhoea virus. (12 Dec 1998) |
| hairy tongue | A tongue with abnormal elongation of the filiform papillae, resulting in a thickened furry appearance. Synonym: glossotrichia, trichoglossia. (05 Mar 2000) |
| oral hairy leukoplakia | A whitish lesion that appears on the side of the tongue and inside cheeks. The lesion appears raised, with a ribbed or hairy surface. Oral hairy leukoplakia occurs mainly in people with declining immunity and may be caused by Epstein-Barr virus infection. (11 Jan 1998) |
| tongue, hairy | A benign condition of the tongue characterised by hypertrophy of the filiform papillae that give the dorsum of the tongue a furry appearance. The colour of the elongated papillae varies from yellowish white to brown or black, depending upon staining by substances such as tobacco, food, or drugs. (12 Dec 1998) |
| leukaemia, hairy cell | A neoplastic disease of the lymphoreticular cells which is considered to be a rare type of chronic leukaemia; it is characterised by an insidious onset, splenomegaly, anaemia, granulocytopenia, thrombocytopenia, little or no lymphadenopathy, and the presence of "hairy" or "flagellated" cells in the blood and bone marrow. (12 Dec 1998) |
| leukoplakia, hairy | Epithelial hyperplasia of the oral mucosa associated with epstein-barr virus and found almost exclusively in persons with HIV infection. The lesion consists of a white patch that is often corrugated or hairy. (12 Dec 1998) |
| acetylcholine receptor antibodies | <neurology, investigation> A test used to measure the amount of antibodies to acetylcholine receptors on nerve endings. This is a diagnostic test for myasthenia gravis. A normal value is no antibodies in the bloodstream. Acetylcholine receptor (AChR) binding autoantibodies (i.e. Antibodies reactive with several epitopes other than the binding site for acetylcholine or alpha-bungarotoxin) are present in approximately 88% of patients with generalised myasthenia gravis, 70% of ocular myasthenia and in approximately 80% of myasthenia gravis in remission. Although serum concentrations of AChR binding autoantibodies do not in general correlate well with severity of weakness, there is typical decrease in concentration as weakness improves with immunosuppressive therapy. AChR blocking autoantibodies (i.e., antibodies reactive with the AChR binding site) are present in about 50% of patients with myasthenia gravis, 30% with ocular myasthenia gravis and 20% of myasthenia gravis in remission, AChR blocking autoantibodies are the only AChR autoantibodies present in about 1% of myasthenia gravis. AChR modulating autoantibodies (i.e., autoantibodies which cross-link AChRs and cause their removal from muscle membrane surfaces) are present in more than 90% of myasthenia gravis and occasionally are the only AchR autoantibodies detectable in mild, recent onset or ocular-restricted myasthenia gravis. Results for AChR modulating autoantibodies can be transiently false-positive due to curare-like drugs used during general anesthesia. AChR autoantibodies of one or more types are found in at least 80% of ocular myasthenia gravis. Although generally absent in neurological conditions other than myasthenia gravis(and consequently unlikely to cause confusion in neurodiagnosis), false-positive results for AChR autoantibodies occasionally occur in primary biliary cirrhosis, tardive dyskinesia, autoimmune thyroiditis, the elderly, amyotrophic lateral sclerosis patients treated with cobra venom and patients with thymoma in the absence of myasthenia gravis. Approximately 1% of patients with rheumatoid arthritis treated with D-penicillamine develop AChR autoantibodies and myasthenia gravis, both of which disappear when the drug is discontinued. Babies born to ~10% of myasthenia gravis mothers have a transient neonatal form of myasthenia gravis that responds well to anticholinesterase therapy and usually remits within 1 month as maternal IgG disappears. (29 Dec 1997) |
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