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  • ¿µ¹®
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  • Wilms¡¯ tumor
    ºô¸§½ºÁ¾¾ç
  • caretaker genes
    °ü¸®À¯ÀüÀÚ
  • homologous genes
    »óµ¿À¯ÀüÀÚ
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    Àª¸§ÁîÁ¾¾ç
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    Àª¸§ÁîÁ¾¾ç
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  • ¿µ¹®
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  • Wilms tumor
    Àª¸§Á¾¾ç
  • hox genes
    Ȥ½º À¯ÀüÀÚ, Hox À¯ÀüÀÚ
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  • ¿µ¹®
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  • tumor =tumour
    Á¾¾ç(ðþåË), Á¾Ã¢(ðþóì)
  • kidney,wilms tumor
    Àª¸§½º Á¾¾ç, Wilm Á¾¾ç(ðþåË)
  • nephroblastoma =wilms tumor
    ½Å¸ð¼¼Æ÷Á¾ Àª¸§ÁîÁ¾¾ç(¡­ðþåË)
  • tumor,wilms
    Àª¸§Á¾¾ç(¡­ðþåË)
  • wilms tumor
    Àª¸§Á¾¾ç
  • wilms tumor
    ºô¸§½ºÁ¾¾ç
  • c2 genes
    C2 À¯ÀüÀÚ (¡­ë¶îîí­)
  • cancer suppressor genes
    ¾Ï¾ïÁ¦À¯ÀüÀÚ(äßåäð¤ë¶îîí­)
  • ced genes
    ced À¯ÀüÀÚ(¡­ë¶îîí­)
  • complementary genes
    »óº¸¼ºÀ¯ÀüÀÚ(ßÀÜÍàõë¶îîí­).
  • cooperating genes
    Çùµ¿À¯ÀüÀÚ
  • fcc genes
    FCC À¯ÀüÀÚ
  • hox genes
    Ȥ½º À¯ÀüÀÚ, Hox À¯ÀüÀÚ
  • multiple genes
    ´ÙÀ¯ÀüÀÚ.
  • multiple genes
    º¹¼öÀ¯ÀüÀÚ.
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    C À¯ÀüÀÚ(ë¶îîí­)
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    »ì¸² À¯ÀüÀÚ(ë¶îîí­)
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  • joining genes
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  • luxury genes
    ƯȰÀ¯ÀüÀÚ(÷åüÀë¶îîí­)
  • reiterated genes
    ¹Ýº¹ À¯ÀüÀÚ(ÚãÜÖë¶îîí­)
  • syn genes
    ½Å À¯ÀüÀÚ(ë¶îîí­)
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    µ¿¿°»öü À¯ÀüÀÚ(ÔÒæøßäô÷ë¶îîí­)
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    ÀÌÀ¯ÀüÀÚ(ì£ë¶îîí­)- ÀÏ(ìé)Æú¸®ÆéŸÀÌµå »ç½½
  • ur genes
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  • variable genes
    °¡º¯ºÎÀ§(ʦܨݻêÈ) À¯ÀüÀÚ (ë¶îîí­)
  • V genes
    V À¯ÀüÀÚ (ë¶îîí­)
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KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
WAGR syndrome   Wilms's Tumor
  Aniridia
  Genital Anomalies
  Me...
AWTA aniridia-Wilms tumor association
NWTS National Wilms' Tumor Study
WAGR Wilms tumor, aniridia, genitourinary abnormalities, and mental retardation
WT wall thickness; water temperature; wavelet transform; wild type [strain]; Wilms tumor; wisdom teeth;...
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
WT1 Wilms tumour
NWTS National Wilms Tumor Study
WT-1 Wilms' Tumor-1
ISG IFN stimulated genes
mdr Multidrug resistance genes
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
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  • Wilms tumor
    ºô¸§½º Á¾¾ç, Àª¸§ Á¾¾ç, Àª¸§Áî Á¾¾ç
    1. ¼Ò¾ÆÀÇ º¹ºÎ¿¡ ¹ß»ýÇÏ´Â ¾Ç¼º Á¾¾ç. µ¶ÀÏ ¿Ü°úÀÇ»ç M. ºô¸§½º
CancerWEB ¿µ¿µ ÀÇÇлçÀü ¸ÂÃã °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
genes, wilms' tumour Tumour suppressor genes located in the 11p13 region on the short arm of human chromosome 11. The absence of these genes is associated with the formation of wilms' tumour.
(12 Dec 1998)
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
wilms tumour <radiology> Nephroblastoma, 3rd most common kiddie tumour (most common abdominal tumour), arises from embryonal renal tissue (nephroblastomatosis), large, only 10% are calcified, 4-10% bilateral, metastasis may lead to lungs, para-aortic nodes associated with: aniridia, Beckwith-Wiedemann syndrome, hemihypertrophy, Drash syndrome Cf: neuroblastoma (more likely calcified)
(12 Dec 1998)
Wilms' tumour A malignant renal tumour of young children, composed of small spindle cells and various other types of tissue, including tubules and, in some cases, structures resembling foetal glomeruli, and striated muscle and cartilage. Often inherited as an autosomal dominant trait .
Synonym: adenomyosarcoma, embryoma of the kidney, nephroblastoma.
(05 Mar 2000)
Wilms, Max <person> German surgeon, 1867-1918.
See: Wilms' tumour.
(05 Mar 2000)
genes, suppressor, tumour Genes that inhibit expression of the tumourigenic phenotype. They are normally involved in holding cellular growth in check. When tumour suppressor genes are inactivated or lost, a barrier to normal proliferation is removed and deregulated growth is possible.
(12 Dec 1998)
breast cancer susceptibility genes Inherited factors that predispose to breast cancer. Put otherwise, these genes make one more susceptible to the disease and so increase the risk of developing breast cancer. Two of these genes, BRCA1 and BRCA2, have been identified (and prominently publicised). Several other genes (those for the Li-Fraumeni syndrome, Cowden disease, Muir-Torre syndrome, and ataxia-telangiectasia) are also known to predispose to breast cancer. However, since all of these known breast cancer susceptibility genes together do not account for more than a minor fraction (1/5th at most) of breast cancer that clusters in families, it is clear that more breast cancer genes remain to be discovered.
(12 Dec 1998)
cancer, breast, susceptibility genes Inherited factors that predispose to breast cancer. Put otherwise, these genes make one more susceptible to the disease and so increase the risk of developing breast cancer. Two of these genes, BRCA1 and BRCA2, have been identified (and prominently publicised). Several other genes (those for the Li-Fraumeni syndrome, Cowden disease, Muir-Torre syndrome, and ataxia-telangiectasia) are also known to predispose to breast cancer. However, since all of these known breast cancer susceptibility genes together do not account for more than a minor fraction (1/5th at most) of breast cancer that clusters in families, it is clear that more breast cancer genes remain to be discovered.
(12 Dec 1998)
genes Located in the nucleus of the cell, genes contain hereditary information that is transferred from cell to cell.
(09 Oct 1997)
genes, abl Retrovirus-associated DNA sequences (abl) originally isolated from the abelson murine leukaemia virus (ab-mulv). The proto-oncogene abl (c-abl) codes for a protein that is a member of the tyrosine kinase family. The human c-abl gene is located at 9q34.1 on the long arm of chromosome 9. It is activated by translocation to bcr on chromosome 22 in chronic myelogenous leukaemia.
(12 Dec 1998)
genes, apc Tumour suppressor genes located in the 5q21 region on the long arm of chromosome 5. The mutation of these genes is associated with familial adenomatous polyposis (apc stands for adenomatous polyposis coli) and gardner's syndrome, as well as some sporadic colourectal cancers.
(12 Dec 1998)
genes, arac Regulatory genes which encode a cyclic AMP receptor protein required for l-arabinose utilization in e. Coli. It is an example of positive control or regulation of gene expression in the bacterial operon.
(12 Dec 1998)
genes, archaeal The genetic material of archaea.
(12 Dec 1998)
genes, bacterial The genetic material of bacteria.
(12 Dec 1998)
genes, bcl-1 The B-cell leukaemia/lymphoma-1 genes, associated with various neoplasms when overexpressed. Overexpression results from the t(11;14) translocation, which is characteristic of mantle zone-derived B-cell lymphomas. The human c-bcl-1 gene is located at 11q13 on the long arm of chromosome 18.
(12 Dec 1998)
genes, bcl-2 The B-cell leukaemia/lymphoma-2 genes, responsible for blocking apoptosis in normal cells, and associated with follicular lymphoma when overexpressed. Overexpression results from the t(14;18) translocation. The human c-bcl-2 gene is located at 18q24 on the long arm of chromosome 18.
(12 Dec 1998)
genes, BRCA1 Tumour suppressor genes located on human chromosome 17q12-21. The mutation of these genes is associated with the formation of familial breast and ovarian cancer.
(12 Dec 1998)
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