| ¿µ¹® | side effects of drugs | ÇÑ±Û | ¾à¹°ÀÇ ºÎÀÛ¿ë |
|---|---|---|---|
| ¼³¸í | ¾àÀ» »ç¿ëÇÒ ¶§ ±â´ëÇÏ´Â È¿°ú ¿Ü¿¡ ´Ù¸¥ Àå±â¿¡¼ ³ªÅ¸³ª´Â ¿øÄ¡ ¾Ê´Â È¿°ú¸¦ ÃÑĪÇÏ´Â ¸»ÀÌ´Ù. ¿¹¸¦ µé¾î Ç÷¾ÐÀ» ³·Ãß±â À§ÇØ Ç×°íÇ÷¾ÐÁ¦¸¦ »ç¿ë ½Ã, Ç÷¾ÐÀÌ ¶³¾îÁö´Â µ¿½Ã¿¡ ±â°üÁöÀÇ ¼öÃàÀÌ ÀϾ´Â Çö»ó. |
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| ¿µ¹® | alcohol | ÇÑ±Û | ¾ËÄÚ¿Ã |
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| ¼³¸í | ´ë°³ ¿¡Åº¿Ã(ethanol, ethyl alcohol)À» ÁöĪÇÏ´Â ¸»ÀÌ´Ù. ¹°°ú °°ÀÌ ¸¼Àº »öÀÌÁö¸¸ ƯÀÌÇÑ ³¿»õ°¡ ÀÖ°í ²ú´Â Á¡ÀÌ ³·Àº ¾×üÀÌ´Ù. ³óµµ°¡ ³ôÀº °ÍÀº ÀÇ·á¿ë ¶Ç´Â ¼Òµ¶Á¦·Î ¾²ÀÌ°í ³·Àº ³óµµ·Î µÈ °ÍÀº À½·á(¼ú)·Îµµ ÀÌ¿ëµÈ´Ù. |
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| ¿µ¹® | ethyl alcohol | ÇÑ±Û | ¿¡Æ¿¾ËÄÚ¿Ã |
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| ¼³¸í | Áö¹æÁ· Æ÷ȾËÄÚ¿ÃÀÇ Çϳª. °¢Á¾ ¾ËÄÚ¿Ã À½·á ¼Ó¿¡ ÇÔÀ¯µÇ¾î ÀÖ¾î ÁÖÁ¤À̶ó°íµµ Çϰí, ¶Ç ¿¡Åº¿ÃÀ̶ó°íµµ ÇÑ´Ù. º¸Åë ¾ËÄÚ¿ÃÀ̶ó°í Çϸé ÀÌ ¿¡Æ¿¾ËÄÚ¿ÃÀ» °¡¸®Å²´Ù. ¼úÀÇ ¼ººÐÀ¸·Î¼ ¿¹ÀüºÎÅÍ ¾Ë·ÁÁ® ÀÖ¾úÀ¸³ª, ¼úÀÌ ÃëÇÏ´Â ¿øÀÎÀÌ ¿¡Åº¿Ã¿¡ ÀÖ´Ù´Â °ÍÀ» ¾È °ÍÀº 15¼¼±â ÀÌÈÄÀÇ ÀÏÀÌ´Ù. Á¶¼ºÀº óÀ½¿¡ ¶óºÎ¾ÆÁö¿¡³ª N.T. ¼Ò½´¸£ µî¿¡ ÀÇÇØ ÃøÁ¤µÇ°í, °ÔÀÌ·ò»èÀ̳ª J.B. µÚ¸¶ µî¿¡ ÀÇÇØ¼ È®Á¤µÇ¾ú´Ù. ¾ËÄÚ¿ÃÀ̶ó´Â À̸§Àº ¿ø·¡ ´«½ç¿¡ Ä¥ÇÏ´Â Èæ»ö ¾È·áäÔÖù¸¦ °¡¸®Å°´Â ¾Æ¶óºñ¾Æ¾î¿´´Âµ¥. À̰ÍÀÇ ¹Ì¼ÒºÐ¸»À» ¸¸µå´Âµ¥ ½ÂȹýÀ» »ç¿ëÇÑ µ¥¼ ¼úÀ» Áõ·ùÇÏ¿© °¡¿¬¼º ¿¢½º¸¦ ¸¸µå´Â °ÍÀ¸·Î ÀüÈï®ûùÇϰí, À̰ÍÀÌ ´Ù½Ã Áõ·ù¹°À» °¡¸®Å°´Â ¸»ÀÌ µÇ¾ú´Ù. ¾ËÄÚ¿ÃÀ̶ó´Â ¸»ÀÌ ¿¡Åº¿ÃÀ» °¡¸®Å°°Ô µÇ°í, ´Ù½Ã ¾ËÄÚ¿Ã Àü¹ÝÀ» °¡¸®Å°°Ô µÈ °ÍÀº 19¼¼±â ÀÌÈÄÀÇ ÀÏÀÌ´Ù. ¿¡Åº¿ÃÀ» º¹¿ëÇÏ¸é ´ë³úÀÇ Á¦¾î±â´ÉÀÌ ¾ïÁ¦µÇ¾î ÈïºÐ»óŰ¡ µÇ°í, ÀÌ¾î¼ ÁßÃ߽ŰæÀÌ ¾ïÁ¦µÈ´Ù. ¹«»öÅõ¸íÇÑ Èֹ߼º ¾×ü. ƯÀ¯ÇÑ ³¿»õ¿Í ¸ÀÀ» °¡Áö¸ç, ÀÎü¿¡ Èí¼öµÇ¸é ÈïºÐÀ̳ª ¸¶Ãë ÀÛ¿ëÀ» ÀÏÀ¸Å²´Ù. ÈÇоàǰÀÇ ÇÕ¼º ¿ø·á, ¿ëÁ¦, ¿¬·á, ¾ËÄڿüº À½·á µûÀ§·Î ¾´´Ù. ¾ËÄÚ¿ÃÀº °£¼¼Æ÷¿¡ Á¸ÀçÇÏ´Â ¾ËÄÚ¿ÃÅ»¼ö¼ÒÈ¿¼Ò(alcohol dehydrogenase)°¡ ´ë»çÇÑ´Ù. ±Þ¼º Áßµ¶ Áõ»óÀº Ç÷Áß ¾ËÄÚ¿Ã ³óµµ¿Í ºñ·ÊÇÑ´Ù. Ç÷Áß ¾ËÄÚ¿Ã ³óµµ°¡ ³ô¾ÆÁú¼ö·Ï ÁßÃ߽Űæ°è´Â ¾ïÁ¦µÇ¾î Ç÷Áß ³óµµ°¡ 3.5~4.5mg/mLÀ̸é È¥¼ö¿¡ ºüÁ® »ç¸ÁÇÒ ¼öµµ ÀÖ´Ù. |
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| ¿µ¹® | fetal alcohol syndrome | ÇÑ±Û | žƾËÄÚ¿ÃÁõÈıº |
|---|---|---|---|
| ¼³¸í | ÀӽűⰣ Áß ¸¸¼ºÀûÀ¸·Î ¾ËÄÚ¿ÃÀ» ¼·ÃëÇÑ ¿©ÀÚ¿¡°Ô¼ ÅÂ¾î³ ¿µ¾Æ¿¡°Ô ³ªÅ¸³ª´Â ÇüŹ߻ýÀÇ ÀÌ»óÀ» ³ªÅ¸³»´Â ÁõÈıºÀ¸·Î¼ À§ÅλÀ¹ßÀ°ºÎÀü, ¾Õ¸Ó¸®¿Í ¾Æ·¡ÅÎÀÇ µ¹Ãâ, ªÀº°Ë¿, ÀÛÀº¾È±¸Áõ, ´«±¸¼®ÁÖ¸§, ½ÉÇÑ ¼ºÀåÁö¿¬, Á¤½ÅÁöü µîÀ» ³ªÅ¸³½´Ù. |
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| AA/AD | alcohol abuse/alcohol dependence |
|---|---|
| ECG | Electro-Cardio-Graphy(-Gram); ½ÉÀüµµ = EKG 1. Conducting System Structu... |
| HCG, hCG | Human Chorionic Gonadotropin; »ç¶÷À¶¸ð¼º¼º¼±ÀÚ±ØÈ£¸£¸ó 1. Placental Glycoprotein Hormone &nbs... |
| BCM | B-cell maturation; birth control medication; blood-clotting mechanism effects; body cell mass; body ... |
| BEAR | biological effects of atomic radiation |
| FAE | Fetal Alcohol Effects |
|---|---|
| FHR | Foetal heart rate |
| IUGR | Intrauterine foetal growth retardation |
| AE | Adverse effects |
| E | Effects |
acute angle
| foetal alcohol effects | A softer diagnosis than foetal alcohol syndrome (FAS). The diagnosis of possible FAE is considered when: 1. The person has some signs of FAS; 2. The person does not meet all of the necessary criteria for FAS; and 3. There is a history of alcohol exposure before birth. (12 Dec 1998) |
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| baby, foetal alcohol syndrome | <syndrome> Alcohol is capable of causing birth defects. FAS (foetal alcohol syndrome) always involves brain damage. And impaired growth. FAS also always involves head and face abnormalities. No amount of alcohol has been proven safe during pregnancy. Women who are or may become pregnant are advised to avoid alcohol. (12 Dec 1998) |
|---|---|
| syndrome, foetal alcohol | The sum total of a person's problems caused by maternal alcohol intake during pregancy. (12 Dec 1998) |
| foetal alcohol syndrome | <syndrome> The most common cause of defective cerebral development in industrialised nations, affecting 1 in 700 live births in the United States. This clinical syndrome results from the direct toxic effects of alcohol on the developing foetus. Growth retardation, mental retardation, small brain and heart valve lesions are common. Infants can be identified by close set eyes, small head, small nasal bridge, reduction in the vermilion border of the upper lip, eye folds and small teeth. (27 Sep 1997) |
| cumulative effects | Effects on the environment resulting from actions that are individually minor but that add up to a greater total effect as they take place over a period of time. (05 Dec 1998) |
| prenatal exposure delayed effects | Delayed effects on offspring of maternal or foetal prenatal exposure to drugs, radiation and other physical agents, manipulation, nutrition, stress, etc. (12 Dec 1998) |
| side effects | Problems that occur when treatment affects healthy cells. Common side effects of cancer treatment are fatigue, nausea, vomiting, decreased blood cell counts, hair loss, and mouth sores. (12 Dec 1998) |
| diamagnetic effects | <radiobiology> Application of a magnetic field to a plasma will tend to create circulating current within the plasma that will reduce the strength of the magnetic field. (09 Oct 1997) |
| experimenter effects | The influence of the experimenter's behaviour, personality traits, or expectancies on the results of that person's own research. See: double blind study. (05 Mar 2000) |
| baseline foetal heart rate | <paediatrics> The average heart rate for a particular foetus during the diastolic phase of uterine contractions. (05 Mar 2000) |
| baseline variability of foetal heart rate | The beat-to-beat changes in foetal heart rate as recorded on a graph. (05 Mar 2000) |
| rate, foetal mortality | The ratio of foetal deaths divided by the sum of the births (the live births + the foetal deaths) in that year. In the united states, the foetal mortality rate plummeted from 19.2 per 1,000 births in 1950 to 9.2 per 1,000 births in 1980. (12 Dec 1998) |
| marked foetal bradycardia | A foetal heart rate less than 100 beats per minute. (05 Mar 2000) |
| version, foetal | The manual conversion of or changing of the polarity of the foetus with reference to the mother. (12 Dec 1998) |
| maternal-foetal exchange | Exchange of substances between the maternal blood and the foetal blood through the placental barrier. It excludes microbial or viral transmission. (12 Dec 1998) |
| persistent foetal circulation syndrome | <syndrome> A syndrome of persistent pulmonary hypertension in the newborn infant, without demonstrable cardiac disease. It is characterised by cyanosis and acidosis, severe pulmonary vasoconstriction, hypertrophy of pulmonary arterial muscle, and elevated pulmonary vascular resistance, with resultant right-to-left shunting of blood through a patent ductus arteriosus and at times a patent foramen ovale. (12 Dec 1998) |
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