¼±Åà - È­»ìǥŰ/¿£ÅÍŰ ´Ý±â - ESC

 
"familial scleroderma"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
¾Ë±â½¬¿î ÀÇÇпë¾îÇ®ÀÌÁý, ¼­¿ïÀÇ´ë ±³¼ö ÁöÁ¦±Ù, °í·ÁÀÇÇÐ ÃâÆÇ À¯»ç °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
¿µ¹® scleroderma ÇÑ±Û °øÇÇÁõ
¼³¸í   
  ¸¸¼ºÀûÀ¸·Î °è¼Ó ÁøÇàµÇ¾î ÇǺΰ¡ µÎ²¨¿ö Áö°í µüµüÇØÁö´Â º´À¸·Î, ¼Ò¾Æ¿Í ¾î¸¥¿¡°Ô¼­ ¸ðµÎ ¹ß»ýÇÒ ¼ö ÀÖ°í, Æ¯Â¡ÀûÀ¸·Î ¸ñ°ú µîÀÇ À­ºÎºÐ¿¡¼­ ½ÃÀ۵ǾƷ¡Âʰú ¾ÕÂÊÀ¸·Î ÆÛÁö´Â º´ÀÌ´Ù. À̰ÍÀº ÀÏÁ¾ÀÇ ÀÚ°¡¸é¿ªÁúȯ(ÀÚ½ÅÀÇ ¼¼Æ÷¸¦ À̹°Áú·Î ÀÎÁöÇÏ¿© ¸é¿ª¹ÝÀÀÀ» ÀÏÀ¸Å°´Â Áúȯ)À¸·Î »ý°¢µÇ¸ç, ÇǺοܿ¡ ¶ÇÇÑ ³»ºÎ±â°ü(¿¹¸¦ µé¾î ÄáÆÏ, ÇãÆÄ, ½Äµµ µî)¿¡µµ ¿ª½Ã °°Àº ¹ÝÀÀÀÌ ÀϾ´Ù. Ä¡·á´Â Æ¯ÀÌÇÑ °ÍÀº ¾ø°í, ¸é¿ª¾ïÁ¦Á¦ÀÇ Ä¡·á¿Í ¹°¸®Ä¡·á µîÀ» ÅëÇÑ °í½ÄÀûÄ¡·á¿¡ ±×Ä£´Ù.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • circumscribed scleroderma
    ±¹ÇÑÇǺΰæÈ­Áõ, ±¹¼ÒÇǺΰæÈ­Áõ
  • diffuse scleroderma
    ±¤¹üÀ§ÇǺΰæÈ­Áõ
  • guttate scleroderma
    ¹æ¿ïÇǺΰæÈ­Áõ
  • linear scleroderma
    ¼±ÇǺΰæÈ­Áõ
  • localized scleroderma
    ±¹ÇÑÇǺΰæÈ­(Áõ)
  • scleroderma
    ÇǺΰæÈ­Áõ, ÇǺα»À½Áõ
  • scleroderma acrosclerosis
    ¸»´ÜÇǺΰæÈ­Áõ
  • scleroderma diabeticorum
    ´ç´¢ÇǺΰæÈ­Áõ
  • scleroderma en bande
    ¶ì¸ð¾çÇǺΰæÈ­Áõ
  • systemic scleroderma
    Àü½ÅÇǺΰæÈ­Áõ
  • acholuric familial jaundice
    ¹«´ãÁó´¢°¡Á·¼ºÈ²´Þ
  • familial
    °¡Á·(¼º)-
  • familial adenomatous polyposis
    °¡Á·¼º»ùÁ¾Æú¸³Áõ
  • familial aminoglycoside ototoxicity
    °¡Á·¼º¾Æ¹Ì³ë±Û¸®Äڽõå±Íµ¶¼º
  • familial amyloid neuropathy
    °¡Á·¼º¾Æ¹Ð·ÎÀ̵å½Å°æº´(Áõ)
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 2 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial adenomatous polyposis
    °¡Á·¼ºÅ«Ã¢ÀÚÆú¸³Áõ, °¡Á·¼º´ëÀåÆú¸³Áõ
  • scleroderma
    ÇǺΰæÈ­Áõ, ÇǺα»À½Áõ
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • circumscribed scleroderma
    (¢¡localized scleroderma) ±¹ÇÑÇǺΰæÈ­Áõ
  • diffuse scleroderma
    ±¤¹üÀ§ÇǺΰæÈ­Áõ
  • guttate scleroderma
    ¹æ¿ïÇǺΰæÈ­Áõ
  • linear scleroderma
    ¼±»óÇǺΰæÈ­Áõ
  • localized scleroderma
    ±¹ÇÑÇǺΰæÈ­Áõ
  • scleroderma
    ÇǺΰæÈ­Áõ, ÇǺα»À½Áõ
  • scleroderma acrosclerosis
    ¸»´ÜÇǺΰæÈ­Áõ
  • scleroderma diabeticorum
    ´ç´¢º´ÇǺΰæÈ­Áõ
  • scleroderma en bande
    ¶ì¸ð¾çÇǺΰæÈ­Áõ
  • systemic scleroderma
    Àü½ÅÇǺΰæÈ­Áõ
  • acholuric familial jaundice
    (¢¡hereditary spherocytosis) À¯ÀüµÕ±ÙÀûÇ÷±¸Áõ, À¯Àü±¸ÇüÀûÇ÷±¸
  • familial cardiomyopathy
    °¡Á·½ÉÀå±ÙÀ°º´Áõ
  • familial dyskeratotic comedo
    °¡Á·°¢È­ÀÌ»ó¸éÆ÷
  • familial dysautonomia
    °¡Á·ÀÚÀ²½Å°æ±â´ÉÀÌ»ó
  • endogenous familial hypertriglyceridemia
    ³»Àΰ¡Á·°íÁß¼ºÁö¹æÇ÷Áõ
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 12 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • guttate scleroderma
    ¹°¹æ¿ï °æÇÇÁõ
  • Familial hypercholesterolemia
    °¡Á·¼º(Ê«ðéàõ) °í(ÍÔ)ÄÝ·¹½ºÅ×·ÑÇ÷Áõ(úìñø)
  • Hailey-Hailey disease = familial benign chronic pemphigus
    ÇìÀϸ®-ÇìÀϸ®º´
  • amaurotic familial idiocy =Tay-Sachs disease
    °¡Á·¼ºÈæ¾Ï½Ã¹éÄ¡(Ê«ðéàõýÙÒ®î¡ÛÜöÁ).
  • amaurotic familial idiocy =Tay-Sachs disease
    °¡Á·¼º Èæ³»Àå ¹éÄ¡(Ê«ðéàõýÙÒ®î¡ÛÜöÁ).
  • hearing loss, familial pregressive
    °¡Á· ÁøÇà(¼º) ³­Ã»
  • hyperbilirubinemia,asymptomatric familial
    ¹«ÁõÈÄ °¡Á·¼º(Ùíñøý¦ Ê«ðéàõ)
  • hypercholesterolemia,familial
    °¡Á·¼º(Ê«ðéàõ)
  • idiopathic familial fibromatosis
    Ư¹ß¼º °¡Á·¼º ¼¶À¯Á¾Áõ.
  • idiopathic familial fibromatosis
    Ư¹ß¼º °¡Á·¼º ¼¶À¯Á¾Áõ
  • infantile amaurotic familial idiocy
    ¿µ¾Æ¼º Èæ³»À强 °¡Á·¼º ¹éÄ¡.
  • infantile amaurotic familial idiocy
    ¿µ¾Æ¼º Èæ³»À强 °¡Á·¼º ¹éÄ¡.
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial scleroderma
    °¡Á·¼º ÇǺΠ°æÈ­Áõ
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • circumscribed scleroderma
    ±¹ÇѼº) °æÇÇÁõ
  • diffuse scleroderma
    ±¤¹üÀ§ ÇǺΠ°æÈ­Áõ
  • guttate scleroderma
    ¹°¹æ¿ï °æÇÇÁõ
  • linear scleroderma
    ¼±»ó °æÇÇÁõ
  • localized scleroderma
    ±¹ÇѼº°æÇÇÁõ
  • scleroderma
    ÇǺΰæÈ­Áõ
  • scleroderma
    ÇǺΰæÈ­Áõ,°æÇÇÁõ(Ìãù«ñø)
  • scleroderma
    ÇǺΰæÈ­Áõ(ù«Ý±Ìãûùñø), °æÇÇÁõ(Ìãù«ñø)
  • scleroderma acrosclerosis
    ¸»´Ü°æÇÇ(Áõ)
  • scleroderma diabeticorum
    ´ç´¢º´ °æÇÇ(Áõ)
  • scleroderma en bande
    ¶ì¸ð¾ç °æÇÇ(Áõ)
  • systemic scleroderma
    Àü½Å¼º °æÇÇÁõ(¡­Ìã ù«ñø)
  • systemic sclerosis [=scleroderma]
    Àü½Å¼º°æÈ­Áõ[= °æÇÇÁõ]
  • amaurotic familial idiocy =Tay-Sachs disease
    °¡Á·¼º Èæ³»Àå ¹éÄ¡(Ê«ðéàõýÙÒ®î¡ÛÜöÁ).
  • amaurotic familial idiocy =Tay-Sachs disease
    °¡Á·¼ºÈæ¾Ï½Ã¹éÄ¡(Ê«ðéàõýÙÒ®î¡ÛÜöÁ).
´ëÇѱâ»ýÃæÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial aggregation
    °¡Á·ÁýÀû¼º
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 4 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial high-density lipoprotein deficiency °¡Á·¼º °í¹Ðµµ ÁöÁú´Ü¹éÁú °áÇÌ (Ê«ðéàõÍÔÚËÓøò·òõ
    Ó±ÛÜòõÌÀù¹)
  • familial hypercholesterolemia
    °¡Á·¼º(Ê«ðéàõ) °ú(Φ)ÄÝ·¹½ºÅ×·ÑÇ÷Áõ(úìñø)
  • familial lysosomal lipase deficiency
    °¡Á·¼º(Ê«ðéàõ) ¶óÀÌ¼Ò¼Ø ¶óÀÌÆäÀ̽º °áÇÌ(ÌÀù¹)
  • familial methemoglobinemia
    °¡Á·¼º(Ê«ðéàõ) ¸ÞÅ׸ð±Û·ÎºóÇ÷Áõ(úìñø)
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 3 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • scleroderma
    ÇǺΰæÈ­Áõ, °æÇÇÁõ
  • familial
    °¡Á·¼ºÀÇ
  • familial polyposis
    °¡Á·¼ºÆú¸³Áõ
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
FHH Familial Hypocalciuric Hypercalcemia
  = Familial Benign Hypercalcemia
FAD familial Alzheimer dementia; familial autonomic dysfunction; fetal activity-acceleration determinati...
FAP familial adenomatous polyposis; familial amyloid polyneuropathy; fatty acid polyunsaturated; fatty a...
DS dead air space; dead space; deep sedative; deep sleep; defined substrate; dehydroepiandrosterone sul...
LS lateral suspensor; left sacrum; left septum; left side; legally separated; leiomyosarcoma; length of...
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
LS Localized scleroderma
PSS Progressive Systemic Scleroderma
SRC Scleroderma renal crisis
SSc; scleroderma Systemic Sclerosis
SSc Systemic scleroderma
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • circumscribed scleroderma
    ±¹ÇѼº °øÇÇÁõ
  • progressive scleroderma
    ÁøÇ༺ °øÇÇÁõ
  • scleroderma circumscripta
    ±¹ÇѼº °øÇÇÁõ
  • scleroderma in plaque
    ġų» °øÇÇÁõ
  • amaurotic familial idiocy
    °¡Á·¼º Èæ³»Àå ¹éÄ¡
    µ¿ÀǾî=Tay-Sach's disease.
  • asymptomatric familial hyperbilirubinemia
    ¹«ÁõÈÄ °¡Á·¼º °úºô¸®·çºóÇ÷Áõ
  • benign familial pemphigus
    ¾ç¼º °¡Á·¼º õÆ÷â
    µå¹°°Ô ¹ß»ýÇÏ´Â, À¯Àü¼ºÀ̸ç Áö¼ÓÀûÀ¸·Î Àç¹ßÀ» ¹Ýº¹ÇÏ´Â ¼ÒÆ÷¼º ¹× ÀÛÀº ¼öÆ÷¼º ÇǺο°À¸·Î, ¾×¿Í, ¼­ÇýºÎ ¹× ¸ñ ºÎÀ§¸¦ °¡Àå Àß Ä§¹üÇÏÁö¸¸, ¶§·Î´Â ±¤¹üÇÑ ºÎÀ§¸¦ ħ¹üÇÑ´Ù. º´º¯Àº ´Ù¹ß¼ºÀ̸ç, ¼öÁÖÀÏ ³»Áö ¼ö°³¿ù ÈÄ¿¡ ¼èÅðÇÑ´Ù. ÀÌ ÁúȯÀº »ó¿°»öü ¿ì¼º À¯ÀüÀ» ÇÑ´Ù.
  • familial adenomatous polyposis
    °¡Á·¼º ¼±Á¾¼º ¿ëÁ¾Áõ
  • familial amyloid polyneuropathy
    °¡Á·¼º ¾Æ¹Ð·ÎÀÌµå ´Ù¹ß ½Å°æº´Áõ
  • familial benign chronic pemphigus
    °¡Á·¼º ¾ç¼º ¸¸¼º õÆ÷â
  • familial cold urticaria
    °¡Á·¼º Çѳà µÎµå·¯±â
  • familial cutaneous collagenosis
    °¡Á·¼º ÇǺΠ±³¿øÁõ
  • familial dysbetalipoproteinemia
    °¡Á·¼º ÀÌ»ó º£Å¸ ¸®Æ÷ ÇÁ·ÎÅ×ÀÎ Ç÷Áõ
  • familial fibrous dysplasia
    °¡Á·¼º ¼¶À¯ ÀÌÇü¼º
  • familial genuine malfomation of root
    °¡Á·¼º ¼±Ãµ¼º Ä¡±Ù ±âÇü
    µ¿ÀǾî=dentinal dys
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
progressive familial scleroderma A syndrome characterised by calcinosis cutis, Raynaud's phenomenon, sclerodactyly, and telangiectasia; usually due to scleroderma; autosomal dominant form of progressive systemic sclerosis.
(05 Mar 2000)
scleroderma <dermatology> Hardening of skin.
(04 Mar 1998)
scleroderma, circumscribed A chronic, localised hardening and thickening of the skin. Lesions may be categorised as morphea (guttate, profunda, pansclerotic) or linear (with or without melorheostosis or hemiatrophy). It is twice as common in women as in men. The condition is characterised by skin ischemia, lymphocytic infiltrates, swollen collagen bundles, and thickening of the dermis with reduction of subcutaneous fat.
(12 Dec 1998)
scleroderma, systemic A chronic, progressive dermatosis characterised by boardlike hardening and immobility of the affected skin, with visceral involvement, especially of lungs, oesophagus, kidneys and heart. It may be accompanied by calcinosis, raynaud's phenomenon, and telangiectasis (crest syndrome). It includes acrosclerosis and sclerodactyly.
(12 Dec 1998)
oesophagus: scleroderma <radiology> Females (80%), 35-55 years of age, decreased LES pressure, decreased peristalsis, smooth muscle atrophy with or without loose fibrosis, dermatomyositis may include involvement of upper 1/3 (striated), dysphagia to solids more than liquids (steakhouse syndrome), wide-open LES or HH, with or without basilar pulmonary fibrosis, other GI sites associated with CREST syndrome
(12 Dec 1998)
localised scleroderma A skin lesion that is characterised by the presence of localised, indurated, slightly depressed areas of thickened dermal tissue that may be white or yellow in colour and surrounded by a pink or purplish halo.
See: in cutaneus scleroderma.
(27 Sep 1997)
benign familial chorea A rare, nonprogressive movement disorder characterised by chorea and athetosis appearing in early childhood, most commonly manifested as gait ataxia and upper limb coordination. Intellect is unaffected. Probably autosomal-dominance inheritance with incomplete penetrance.
(05 Mar 2000)
benign familial chronic pemphigus Recurrent eruption of vesicles and bullae that become scaling and crusted lesions with vesicular borders, predominantly of the neck, groin, and axillary regions; autosomal dominant inheritance, presenting in late adolescence or early adult life.
Synonym: Hailey-Hailey disease.
(05 Mar 2000)
benign familial icterus Mild jaundice due to increased amounts of unconjugated bilirubin in the plasma without evidence of liver damage, biliary obstruction, or haemolysis; thought to be due to an inborn error of metabolism in which the excretion of bilirubin by the liver is defective, ascribed to decreased conjugation of bilirubin as a glucuronide or impaired uptake of hepatic bilirubin.
Synonym: benign familial icterus, constitutional hepatic dysfunction, Gilbert's disease, Gilbert's syndrome, Hebra's disease.
(05 Mar 2000)
cancer, breast, familial A number of factors have been identified that increase the risk of breast cancer. One of the strongest of these risk factors is the history of breast cancer in a relative. About 15-20% of women with breast cancer have such a family history of the disease, clearly reflecting the participation of inherited (genetic) components in the development of some breast cancers. Dominant breast cancer suceptibility genes, including BRCA1 and BRCA2, appear responsible for about 5% of all breast cancer.
(12 Dec 1998)
paralysis, familial periodic An autosomal dominant trait marked by recurring attacks of rapidly progressive flaccid paralysis. There are three types: I, associated with a fall in serum potassium levels (hypokalaemic periodic paralysis); II, associated with a rise therein (hyperkalaemic periodic paralysis, called also adynamia episodica hereditaria); and III, with normal levels (normokalaemic periodic paralysis).
(12 Dec 1998)
pemphigus, benign familial Rare hereditary disease characterised by recurrent eruptions of vesicles and bullae mainly on the neck, axillae, and groin. It exhibits autosomal dominant inheritance and is unrelated to pemphigus vulgaris though it closely resembles that disease.
(12 Dec 1998)
chronic familial icterus <haematology> A hereditary disorder that leads to a chronic haemolytic anaemia due to an abnormality in the red blood cell membrane.
This disorder is caused by a defective gene. Red cells are resistant to stress and rupture easily. Infants may appear jaundiced and pale. Fatigue, weakness and shortness of breath are other symptoms that may be seen in older patients. The spleen may also be enlarged.
Treatment includes splenectomy (removal of the spleen). After this is accomplished the life-span of the red blood cells returns to normal.
(27 Sep 1997)
chronic familial jaundice <haematology> A hereditary disorder that leads to a chronic haemolytic anaemia due to an abnormality in the red blood cell membrane.
This disorder is caused by a defective gene. Red cells are resistant to stress and rupture easily. Infants may appear jaundiced and pale. Fatigue, weakness and shortness of breath are other symptoms that may be seen in older patients. The spleen may also be enlarged.
Treatment includes splenectomy (removal of the spleen). After this is accomplished the life-span of the red blood cells returns to normal.
(27 Sep 1997)
chronic familial polyneuritis Inflammation of nerves related to infiltration by amyloid.
(05 Mar 2000)
ÇÑ¿µ/¿µÇÑ »çÀü À¯»ç °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial
    °¡Á·ÀÇ;°¡Á·¼ºÀÇ;°¡Á· ƯÀ¯ÀÇ
ÀÌ ¾Æ·¡ ºÎÅÍ´Â °á°ú°¡ ¾ø½À´Ï´Ù.
KMLE ¾àǰ/ÀǾàǰ ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • Á¦Ç°¸í
    ¼ººÐ/ÇÔ·®
    ±¸ºÐ/º¸Çè±Þ¿©
KMLE ¾àǰ/ÀǾàǰ À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • Á¦Ç°¸í
    ¼ººÐ/ÇÔ·®
    ±¸ºÐ/º¸Çè±Þ¿©
¾Ë±â½¬¿î ÀÇÇпë¾îÇ®ÀÌÁý, ¼­¿ïÀÇ´ë ±³¼ö ÁöÁ¦±Ù, °í·ÁÀÇÇÐ ÃâÆÇ ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
´ëÇѽŰæ¿Ü°úÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ÇÑÀÚ
´ëÇѽŰæ¿Ü°úÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ÇÑÀÚ
´ëÇѱâ»ýÃæÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
KI ÀÇÇпë¾î »çÀü °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
KMLE ÀÇÇоà¾î »çÀü ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
ÀÇÇÐ³í¹® ¾àÀÚ(Pubmed/Entrez) °Ë»ö ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
Çѱ¹Ç¥ÁØÁúº´»çÀκзù ¾àÀÚ ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ÄÚµå
    ¿µ¹®
    ÇѱÛ
Çѱ¹Ç¥ÁØÁúº´»çÀκзù ¾àÀÚ À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ÄÚµå
    ¿µ¹®
    ÇѱÛ
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
CancerWEB ¿µ¿µ ÀÇÇлçÀü ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
MeSH(Medical Subject Headings) ¸ÂÃã °Ë»ö (http://www.nlm.nih.gov) °á°ú : 0 ÆäÀÌÁö: 1
MeSH(Medical Subject Headings) À¯»ç °Ë»ö (http://www.nlm.nih.gov) °á°ú : 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - Merriam-Webster's ÀÇÇлçÀü ¸ÂÃã °Ë»ö (https://www.merriam-webster.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - Merriam-Webster's ÀÇÇлçÀü À¯»ç °Ë»ö (https://www.merriam-webster.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - A.D.A.M. Medical Encyclopedia ¸ÂÃã °Ë»ö (http://www.nlm.nih.gov) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - A.D.A.M. Medical Encyclopedia À¯»ç °Ë»ö (http://www.nlm.nih.gov) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - MedlinePlus Health Topics ¸ÂÃã °Ë»ö (http://www.nlm.nih.gov) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - MedlinePlus Health Topics À¯»ç °Ë»ö (http://www.nlm.nih.gov) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - µå·¯±×ÀÎÆ÷ ¾àÇÐ Á¤º¸ ¸ÂÃã °Ë»ö (http://www.druginfo.co.kr) °á°ú: 0 ÆäÀÌÁö: 1
Á¦Ç°¸í
ÆÇ¸Å»ç
º¸ÇèÄÚµå ¼ººÐ/ÇÔ·®
±¸ºÐ/º¸Çè±Þ¿©
¿ÜºÎ ¸µÅ© - µå·¯±×ÀÎÆ÷ ¾àÇÐ Á¤º¸ À¯»ç °Ë»ö (http://www.druginfo.co.kr) °á°ú: 0 ÆäÀÌÁö: 1
Á¦Ç°¸í
ÆÇ¸Å»ç
º¸ÇèÄÚµå ¼ººÐ/ÇÔ·®
±¸ºÐ/º¸Çè±Þ¿©
¿ÜºÎ ¸µÅ© - WebMD.com Drug Reference ¸ÂÃã °Ë»ö (http://www.webmd.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - WebMD.com Drug Reference À¯»ç °Ë»ö (http://www.webmd.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - Drug.com Drugs by Medical Condition ¸ÂÃã °Ë»ö (http://www.drugs.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - Drug.com Drugs by Medical Condition À¯»ç °Ë»ö (http://www.drugs.com) °á°ú: 0 ÆäÀÌÁö: 1
KMLE À¥ ¿ë¾î ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
KMLE À¥ ¿ë¾î À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
ÇÑ¿µ/¿µÇÑ »çÀü ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
WordNet ÀÏ¹Ý ¿µ¿µ »çÀü °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - American Heritage Dictionary ¿µ¿µ»çÀü ¸ÂÃã °Ë»ö (https://www.ahdictionary.com) °á°ú: 0 ÆäÀÌÁö: 1
¿ÜºÎ ¸µÅ© - American Heritage Dictionary ¿µ¿µ»çÀü À¯»ç °Ë»ö (https://www.ahdictionary.com) °á°ú: 0 ÆäÀÌÁö: 1
ÅëÇÕ°Ë»ö ¿Ï·á