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"familial amyloid neuropathy"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
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¿µ¹® diabetic neuropathy ÇÑ±Û ´ç´¢º´½Å°æº´Áõ
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  • ¿µ¹®
    ÇѱÛ
  • familial amyloid neuropathy
    °¡Á·¼º¾Æ¹Ð·ÎÀ̵å½Å°æº´(Áõ)
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • amyloid angiopathy
    ¾Æ¹Ð·ÎÀ̵åÇ÷°üº´(Áõ)
  • amyloid kidney
    ¾Æ¹Ð·ÎÀ̵åÄáÆÏ, ¾Æ¹Ð·ÎÀ̵å½ÅÀå
  • amyloid nephrosis
    ¾Æ¹Ð·ÎÀ̵åÄáÆÏÁõ, ¾Æ¹Ð·ÎÀ̵å½ÅÀåÁõ
  • amyloid plaque
    ¾Æ¹Ð·ÎÀ̵åÆÇ
  • acholuric familial jaundice
    ¹«´ãÁó´¢°¡Á·¼ºÈ²´Þ
  • familial
    °¡Á·(¼º)-
  • familial adenomatous polyposis
    °¡Á·¼º»ùÁ¾Æú¸³Áõ
  • familial aminoglycoside ototoxicity
    °¡Á·¼º¾Æ¹Ì³ë±Û¸®Äڽõå±Íµ¶¼º
  • familial annulare erythema
    °¡Á·¼ºÀ±»óÈ«¹Ý, °¡Á·¼º°í¸®È«¹Ý
  • familial cardiomyopathy
    °¡Á·¼º½ÉÀå±ÙÀ°º´(Áõ), °¡Á·¼º½É±Ùº´(Áõ)
  • familial dysautonomia
    °¡Á·¼ºÀÚÀ²½Å°æÀÌ»ó(Áõ)
  • familial goiter
    °¡Á·¼º°©»ó»ùÁ¾
  • familial hemophagocytic lymphohistiocytosis
    °¡Á·¼ºÀûÇ÷±¸Æ÷½Ä¼º¸²ÇÁÁ¶Á÷±¸Áõ½ÄÁõ
  • familial paroxysmal polyserositis
    °¡Á·¼º¹ßÀÛ´Ù¹ßÀ帷¿°
  • familial polyposis
    °¡Á·¼ºÆú¸³Áõ
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 11 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial adenomatous polyposis
    °¡Á·¼ºÅ«Ã¢ÀÚÆú¸³Áõ, °¡Á·¼º´ëÀåÆú¸³Áõ
  • neuropathy
    ½Å°æº´Áõ
  • acute ischemic brachial neuropathy
    ±Þ¼ºÇãÇ÷À§ÆÈ½Å°æº´Áõ
  • autonomic neuropathy
    ÀÚÀ²½Å°æº´Áõ
  • diabetic neuropathy
    ´ç´¢º´½Å°æº´Áõ
  • entrapment neuropathy
    ÁËÀӽŰ溴Áõ, Æ÷Âø½Å°æº´Áõ
  • hereditary motor sensory neuropathy
    À¯Àü¿îµ¿°¨°¢½Å°æº´Áõ
  • ischemic optic neuropathy
    ÇãÇ÷½Ã°¢½Å°æº´Áõ
  • paraneoplastic neuropathy
    (¢¡paraneoplastic polyneuropathy) ½Å»ý¹°µþ¸²½Å°æº´Áõ
  • peripheral neuropathy
    ¸»ÃʽŰ溴Áõ
  • polyneuropathy neuropathy
    ½Å»ý¹°µþ¸²¿©·¯½Å°æº´Áõ, ½Å»ý¹°µþ¸²´Ù¹ß½Å°æº´Áõ
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • amyloid kidney
    ¾Æ¹Ð·ÎÀ̵åÄáÆÏ, ¾Æ¹Ð·ÎÀ̵å½ÅÀå
  • amyloid nephrosis
    ¾Æ¹Ð·ÎÀ̵åÄáÆÏÁõ
  • amyloid plaque
    ¾Æ¹Ð·ÎÀ̵åÆÇ
  • amyloid angiopathy
    ¾Æ¹Ð·ÎÀ̵åÇ÷°üº´Áõ
  • autonomic neuropathy
    ÀÚÀ²½Å°æº´Áõ
  • compression neuropathy
    ¾Ð¹Ú½Å°æº´Áõ
  • diabetic neuropathy
    ´ç´¢º´½Å°æº´Áõ
  • entrapment neuropathy
    µ£½Å°æº´Áõ, Æ÷Âø½Å°æº´Áõ, ÁËÀӽŰ溴Áõ
  • genetically determined neuropathy
    À¯Àü½Å°æº´Áõ
  • hereditary motor sensory neuropathy
    À¯Àü¿îµ¿°¨°¢½Å°æº´Áõ
  • hypertrophic interstitial neuropathy
    ºñ´ë»çÀÌÁú½Å°æº´Áõ
  • idiopathic neuropathy
    Ư¹ß½Å°æº´Áõ
  • ischemic optic neuropathy
    ÇãÇ÷½Ã°¢½Å°æº´Áõ
  • lead neuropathy
    ³³Áßµ¶½Å°æº´Áõ
  • neuropathy
    ½Å°æº´Áõ
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • anterior ischemic optic neuropathy
    Àü¹æÇãÇ÷¼º ½Ã½Å°æº´Áõ(îñÛ°úÈúìàõãÊãêÌèÜ»ñø).
  • anterior ischemic optic neuropathy
    ¾ÕÇãÇ÷½Ã½Å°æº´Áõ
  • genetically determined neuropathy
    À¯Àü¼º ½Å°æº´Áõ.
  • hereditary motor and sensory neuropathy
    À¯Àü¼º¿îµ¿ °¨°¢½Å°æº´Áõ
  • hypertrophic interstitial neuropathy
    ºñÈļº °£Áú¼º ½Å°æº´Áõ.
  • idiopathic neuropathy
    Ư¹ß¼º ½Å°æº´Áõ.
  • posterior ischemic optic neuropathy
    ÈÄÇãÇ÷½Ã½Å°æº´Áõ
  • amyloid
    ¾Æ¹Ð·ÎÀ̵å.
  • amyloid
    ¾Æ¹Ð·ÎÀ̵å
  • amyloid angiopathy
    À¯ÀüºÐÇ÷°üº´Áõ(ëºîþÝÏ úìηܻñø)
  • amyloid bodies<³ª> corpora amylacea
    ¾Æ¹Ð·ÎÀ̵å¼Òü(¡­á³ô÷).
  • amyloid degeneration
    ¾Æ¹Ð·ÎÀ̵庯¼º(¡­Ü¨àõ).
  • amyloid disease
    ¾Æ¹Ð·ÎÀ̵庴(¡­Ü»).
  • amyloid kidney
    ¾Æ¹Ð·ÎÀÌµå ½Å
  • amyloid nephrosis
    ¾Æ¹Ð·ÎÀ̵å½ÅÁõ(¡­ãìñø).
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial amyloid elastosis
    °¡Á·¼º À¯ÀüºÐ ź·Â¼¶À¯Áõ
  • familial amyloid polyneuropathy
    °¡Á·¼º ¾Æ¹Ð·ÎÀÌµå ´Ù¹ß½Å°æº´Áõ
  • amyloid
    ¾Æ¹Ð·ÎÀ̵å.
  • amyloid
    ¾Æ¹Ð·ÎÀ̵å
  • amyloid angiopathy
    À¯ÀüºÐÇ÷°üº´Áõ(ëºîþÝÏ úìηܻñø)
  • amyloid bodies<³ª> corpora amylacea
    ¾Æ¹Ð·ÎÀ̵å¼Òü(¡­á³ô÷).
  • amyloid degeneration
    ¾Æ¹Ð·ÎÀ̵庯¼º(¡­Ü¨àõ).
  • amyloid disease
    ¾Æ¹Ð·ÎÀ̵庴(¡­Ü»).
  • amyloid kidney
    ¾Æ¹Ð·ÎÀÌµå ½Å
  • amyloid nephrosis
    ¾Æ¹Ð·ÎÀ̵å½ÅÁõ(¡­ãìñø).
  • amyloid precurssor protein
    ¾Æ¹Ð·ÎÀ̵å Àü±¸ ´Ü¹éÁú
  • amyloid tumor
    ¾Æ¹Ð·ÎÀ̵åÁ¾¾ç.
  • laryngeal amyloid
    Èĵξƹ̷ÎÀ̵å
  • papular amyloid elastosis
    ±¸Áø¼º À¯ÀüºÐ ź·Â ¼¶À¯Áõ
  • anterior ischemic optic neuropathy
    Àü¹æÇãÇ÷¼º ½Ã½Å°æº´Áõ(îñÛ°úÈúìàõãÊãêÌèÜ»ñø).
´ëÇѱâ»ýÃæÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial aggregation
    °¡Á·ÁýÀû¼º
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • amyloid
    ¾Æ¹Ð·ÎÀ̵å
  • familial high-density lipoprotein deficiency °¡Á·¼º °í¹Ðµµ ÁöÁú´Ü¹éÁú °áÇÌ (Ê«ðéàõÍÔÚËÓøò·òõ
    Ó±ÛÜòõÌÀù¹)
  • familial hypercholesterolemia
    °¡Á·¼º(Ê«ðéàõ) °ú(Φ)ÄÝ·¹½ºÅ×·ÑÇ÷Áõ(úìñø)
  • familial lysosomal lipase deficiency
    °¡Á·¼º(Ê«ðéàõ) ¶óÀÌ¼Ò¼Ø ¶óÀÌÆäÀ̽º °áÇÌ(ÌÀù¹)
  • familial methemoglobinemia
    °¡Á·¼º(Ê«ðéàõ) ¸ÞÅ׸ð±Û·ÎºóÇ÷Áõ(úìñø)
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 4 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • amyloid infiltration
    ¾Æ¹Ð·ÎÀ̵åħÀ±
  • neuropathy
    ½Å°æº´Áõ
  • familial
    °¡Á·¼ºÀÇ
  • familial polyposis
    °¡Á·¼ºÆú¸³Áõ
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
FAP familial adenomatous polyposis; familial amyloid polyneuropathy; fatty acid polyunsaturated; fatty a...
APP acute phase protein; alum-precipitated pyridine; aminopyrazolopyrimidine; amyloid peptide precursor;...
FAC Familial Amyloid Cardiomyopathy
FAP Familial Amyloid Polyneuropathy
FHH Familial Hypocalciuric Hypercalcemia
  = Familial Benign Hypercalcemia
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
FAP Familial Amyloid Polyneuropathy
AMAN Acute Motor Axonal Neuropathy
AION Anterior Ischemic Optic Neuropathy
AN Autonomic neuropathy
CAN Cardiac autonomic neuropathy
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • familial amyloid polyneuropathy
    °¡Á·¼º ¾Æ¹Ð·ÎÀÌµå ´Ù¹ß ½Å°æº´Áõ
  • amyloid degeneration
    ¾Æ¹Ð·ÎÀÌµå º¯¼º
  • amyloid infiltration
    ¾Æ¹Ð·ÎÀ̵å ħÀ±
  • serum amyloid protein A
    Ç÷û ¾Æ¹Ð·ÎÀ̵å ÇÁ·Îƾ A
  • diabetic peripheral neuropathy
    ´ç´¢º´¼º ½Å°æº´Áõ
    ÀÓ»óÀûÀ¸·Î ¿øÀ§ ½Å°æÀ» ´ëμºÀ¸·Î ħ¹üÇϸç ÁÖ·Î °¨°¢¼º ´Ù¹ß ½Å°æº´ÁõÀÇ ÇüŸ¦ ÃëÇÑ´Ù. º´¸®ÇÐÀûÀ¸·Î´Â ÁÖ·Î Ãà»èÀÇ º´º¯À̳ª ºÎÀ§¿¡ µû¶ó Å»¼öÃÊÀÇ Çö»óÀ» º¸À̱⵵ ÇÑ´Ù. ½Å°æ¼¶À¯ ³»¸·ÀÇ ¼¼µ¿¸Æ º®Àº ÃÊÀھ缺 ºñÈĸ¦ º¸ÀÌ¸ç ±âÀú¸·ÀÇ ºñÈĵµ µ¿¹ÝµÇ¾î ÀÖ´Ù. ÀÌ·¯ÇÑ ¸»ÃʽŰ溴ÁõÀÇ ¿øÀÎÀº ºÐ¸íÄ¡ ¾ÊÀ¸³ª Ç÷°ü º¯È­¿¡ ÀÇÇÑ ÇãÇ÷¼º ¼Õ»ó ¶Ç´Â ¼Ò¸£ºñÅç »ó½Â¿¡ ÀÇÇÑ »ýÈ­ÇÐÀû ¼Õ»ó µîÀÌ Áß¿äÇÑ ¿ªÇÒÀ» ÇÑ´Ù°í ÃßÁ¤µÇ°í ÀÖ´Ù.
  • lebers hereditary optic neuropathy
    ·¹¹ö¾¾ ¼±Ãµ¼º ½Ã½Å°æº´Áõ
  • neuropathy
    ½Å°æ Àå¾Ö, ½Å°æº´Áõ, ½Å°æº´
    ¸»ÃʽŰæÀÇ ±â´ÉÀå¾Ö³ª º´¸®ÇÐÀû º¯È­. ¸»ÃʽŰæ°èÀÇ ±â´ÉÀû Àå¾Ö ¹× ¶Ç´Â º´Àû º¯È­¸¦ ³ªÅ¸³»´Â ÀÏ¹Ý ¿ë¾î. ±× ¿øÀÎÀº ¾Ë·ÁÁø °æ¿ì
  • optic neuropathy
    ½Ã½Å°æº´Áõ
  • posterior ischemic optic neuropathy
    ÈÄÇãÇ÷ ½Ã½Å°æº´Áõ
  • retrobulbar neuropathy
    ±¸ÈÄ ½Ã½Å°æº´Áõ
  • subacute myelo-optic neuropathy
    ¾Æ±Þ¼º ô¼ö ½Ã½Å°æº´Áõ
  • amaurotic familial idiocy
    °¡Á·¼º Èæ³»Àå ¹éÄ¡
    µ¿ÀǾî=Tay-Sach's disease.
  • asymptomatric familial hyperbilirubinemia
    ¹«ÁõÈÄ °¡Á·¼º °úºô¸®·çºóÇ÷Áõ
  • benign familial pemphigus
    ¾ç¼º °¡Á·¼º õÆ÷â
    µå¹°°Ô ¹ß»ýÇÏ´Â, À¯Àü¼ºÀ̸ç Áö¼ÓÀûÀ¸·Î Àç¹ßÀ» ¹Ýº¹ÇÏ´Â ¼ÒÆ÷¼º ¹× ÀÛÀº ¼öÆ÷¼º ÇǺο°À¸·Î, ¾×¿Í, ¼­ÇýºÎ ¹× ¸ñ ºÎÀ§¸¦ °¡Àå Àß Ä§¹üÇÏÁö¸¸, ¶§·Î´Â ±¤¹üÇÑ ºÎÀ§¸¦ ħ¹üÇÑ´Ù. º´º¯Àº ´Ù¹ß¼ºÀ̸ç, ¼öÁÖÀÏ ³»Áö ¼ö°³¿ù ÈÄ¿¡ ¼èÅðÇÑ´Ù. ÀÌ ÁúȯÀº »ó¿°»öü ¿ì¼º À¯ÀüÀ» ÇÑ´Ù.
  • familial adenomatous polyposis
    °¡Á·¼º ¼±Á¾¼º ¿ëÁ¾Áõ
CancerWEB ¿µ¿µ ÀÇÇлçÀü ¸ÂÃã °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
familial amyloid neuropathy <neurology> A disorder in which various peripheral nerves are infiltrated with amyloid and their functions disturbed, an abnormal prealbumin is also formed and is present in the blood; characteristically, it begins during mid-life and is found largely in persons of Portuguese descent. Other rare clinical types occur.
Inheritance: autosomal dominant.
Synonym: familial amyloidosis, hereditary amyloidosis.
(05 Mar 2000)
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
amyloid Glycoprotein deposited extracellularly in tissues in amyloidosis. The glycoprotein may either derive from light chain of immunoglobulin (AIO (amyloid of immune origin): 5-18 kD glycoprotein, product of a single clone of plasma cells, the N terminal part of lambda or kappa light chain) or, in what used to be referred to as AUO, amyloid of unknown origin, from serum amyloid A (SAA), one of the acute phase proteins that increases many fold in inflammation. The polypeptides are organised as a _ pleated sheet making the material rather inert and insoluble. Minor protein components are also found. Should be distinguished from _ amyloid deposited in the brain and that is derived from amyloid precursor protein (see amyloidogenic glycoprotein.
(18 Nov 1997)
amyloid A-degrading serine protease <enzyme> Reduced in amyloidosis associated with rheumatoid arthritis
Registry number: EC 3.4.21.-
Synonym: amyloid a-degrading activity, aad-protease
(26 Jun 1999)
amyloid angiopathy Deposition of acellular hyaline material in small arteries and arterioles of the leptomeninges and cerebral cortex in the elderly with resulting predilection for recurrent lobar intraparenchymal haematomas.
(05 Mar 2000)
amyloid beta-protein A 4 kD protein, 39-43 amino acids long, expressed by a gene located on chromosome 21. It is the major protein subunit of the vascular and plaque amyloid filaments in individuals with alzheimer's disease and in aged individuals with trisomy 21 (down syndrome). The protein is found predominantly in the nervous system, but there have been reports of its presence in non-neural tissue.
(12 Dec 1998)
amyloid beta-protein precursor A precursor to the amyloid-beta protein (beta/a4). Alterations in the expression of the amyloid beta-protein precursor (abpp) gene, located on chromosome 21, plays a role in the development of the neuropathology common to both alzheimer disease and down syndrome. Abpp is associated with the extensive extracellular matrix secreted by neuronal cells. Upon cleavage, this precursor produces three proteins of varying amino acid lengths: 695, 751, and 770. The beta/a4 (695 amino acids) or beta-amyloid protein is the principal component of the extracellular amyloid in senile plaques found in alzheimer disease, down syndrome and, to a limited extent, in normal aging.
(12 Dec 1998)
amyloid bodies of the prostate An obsolete term for small masses of colloid material often present in the tubules of the gland.
See: corpus amylaceum.
(05 Mar 2000)
amyloid corpuscle One of a number of small ovoid or rounded, sometimes laminated, bodies resembling a grain of starch and found in nervous tissue, in the prostate, and in pulmonary alveoli; of little pathological significance, and apparently derived from degenerated cells or proteinaceous secretions.
Synonym: amniotic corpuscle, amylaceous corpuscle, amyloid corpuscle, colloid corpuscle.
(05 Mar 2000)
amyloid degeneration Infiltration of amyloid between cells and fibres of tissues and organs.
Synonym: waxy degeneration.
(05 Mar 2000)
amyloid kidney A kidney in which amyloidosis has occurred, usually in association with some chronic illness such as multiple myeloma, tuberculosis, osteomyelitis, or other chronic suppurative inflammation; such kidney's are moderately enlarged and grossly manifest a waxy appearance, with amyloid deposited beneath the endothelium in the glomerular loops and in the arterioles, apparently beginning as foci of thickening of the basement membranes.
Synonym: waxy kidney.
(05 Mar 2000)
amyloid nephrosis The nephrotic syndrome due to deposition of amyloid in the kidney.
See: renal amyloidosis.
(05 Mar 2000)
amyloid neuropathies Disorders of the peripheral nervous system associated with deposition of amyloid. Amyloid neuropathies may result from non-hereditary or hereditary amyloidosis. Several different forms of familial amyloid neuropathies have been described, most of which have specific mutations in the prealbumin gene.
(12 Dec 1998)
amyloid p component Amyloid p component is a small, non-fibrillar glycoprotein found in normal serum and in all amyloid deposits. It has a pentagonal (pentaxin) structure. It acts as an acute phase protein in the mouse, modulates immunologic responses in man, inhibits elastase, and has been suggested as an indicator of liver disease.
(12 Dec 1998)
amyloid precursor protein <protein> Individuals with Alzheimer's disease are characterised by extensive accumulation of amyloid in the brain, referred to as senile plaques. These consist of a core of amyloid fibrils surrounded by dystrophic neurites. The principal component of the amyloid fibrils is B/A4, a peptide derived from the larger APP. The specific role of amyloid protein is unclear but it is thought that amyloid deposits may cause neurons to degenerate. Amyloid deposits also occur in brains of older Down's Syndrome patients.
(04 May 1997)
amyloid protein Glycoprotein deposited extracellularly in tissues in amyloidosis. The glycoprotein may either derive from light chain of immunoglobulin (AIO (amyloid of immune origin): 5-18 kD glycoprotein, product of a single clone of plasma cells, the N terminal part of lambda or kappa light chain) or, in what used to be referred to as AUO, amyloid of unknown origin, from serum amyloid A (SAA), one of the acute phase proteins that increases many fold in inflammation. The polypeptides are organised as a _ pleated sheet making the material rather inert and insoluble. Minor protein components are also found. Should be distinguished from _ amyloid deposited in the brain and that is derived from amyloid precursor protein (see amyloidogenic glycoprotein.
(18 Nov 1997)
amyloid protein aa A nonimmunoglobulin amyloid isolated from amyloid fibrils deposited in amyloidosis secondary to chronic inflammatory diseases such as rheumatoid arthritis. Antisera to amyloid protein aa have been used to detect a related serum protein saa.
(12 Dec 1998)
ÇÑ¿µ/¿µÇÑ »çÀü À¯»ç °Ë»ö °á°ú : 3 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • amyloid
    ¾Æ¹Ð·ÎÀ̵å;À¯ÀüºÐü
  • familial
    °¡Á·ÀÇ;°¡Á·¼ºÀÇ;°¡Á· ƯÀ¯ÀÇ
  • neuropathy
    ½Å°æº´;½Å°æÀå¾Ö
ÀÌ ¾Æ·¡ ºÎÅÍ´Â °á°ú°¡ ¾ø½À´Ï´Ù.
KMLE ¾àǰ/ÀǾàǰ ¸ÂÃã °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • Á¦Ç°¸í
    ¼ººÐ/ÇÔ·®
    ±¸ºÐ/º¸Çè±Þ¿©
KMLE ¾àǰ/ÀǾàǰ À¯»ç °Ë»ö °á°ú : 0 ÆäÀÌÁö: 1
  • Á¦Ç°¸í
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