| 영문 | breast cancer | 한글 | 유방암 |
|---|---|---|---|
| 설명 | 유방은 지방조직과 본래의 목적인 젓을 생산하는 샘조직, 그리고 이 샘조직에서 생산한 젓을 밖에까지 내보내는 관조직, 그리고 이 샘조직과 관조직의 주위를 둘러싸고 있으면서 이것들의 모양을 유지해주는 간질로 이루어진다. 유방암이란 샘조직과 관조직에서 기원하는 종양을 말한다. 유방암을 발생시킬 수 있는 위험인자는 여러 가지가 밝혀져 있다. 그중에서 대표적인 것은 다음과 같다. 1.유전적 요인－가족 중에서 유방암에 걸린 사람이 있으면 유방암의 발생확률이 아주 높아진다. 2.에스트로겐에 장기적으로 많이 폭로되는 경우－에스트로겐은 여성호르몬으로 여성에서 정상적으로 생리의 주기에 따라 증감을 되풀이한다. 이 에스트로겐은 유방의 젖샘조직과 관조직의 증식과 성장에 관계한다. 그리고 이 에스트로겐에 많이 폭로되는 사람은 유방암의 확률이 높다. 외부에서 에스트로겐의 제제를 치료목적으로 투여할 경우에 유방암의 발생확률은 정상인에서 보다 현저하게 높아진다. 그리고 아이를 낳지 않고 평생을 사는 여자의 경우에 임신을 하고 아이를 낳은 여자에 비해서 에스트로겐에 많이 폭로가 되므로(임신시에는 비임신시보다 에스트로겐에 적게 폭로가 된다)유방암의 확률이 높다. 3.환경적 요인－지방질 섭취의 증가, 술의 섭취 등은 유방암의 확률을 높인다. 유방암의 증상으로 가장 많은 것은 아프지 않은 유방의 덩어리이다. 그리고 유두에서 분비물이 나오거나, 유방의 피부변화 등이 유방암의 흔한 증상이다. 치료는 유방의 절제술이 가장 효과적인 치료법으로 되어있다. 그리고 이것에 더하여 방사선 치료나 항암제요법도 효과가 있는 것으로 되어있다. 또 에스트로겐의 과잉과 관계가 있는 종양일 경우에는 에스트로겐의 효과를 차단하는 약물도 효과가 있다. |
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| 영문 | incubation period, latent stage | 한글 | 잠복기, 배양기간 |
|---|---|---|---|
| 설명 | 1. 병원체가 체내에 침입(감염)한 후 그로 인한 감염증이 발병할 때까지의 기간. 병원체의 종류에 따라 그 내용이 다르다. 예컨대 말라리아는 침입한 병원체가 인체 내에서 일정한 단계까지 발육한 후 발병하고, 장티푸스는 침입한 병원균이 일정한 수까지 번식을 해야 발병하며, 파상풍은 침입한 병원균이 생성한 독소가 뇌신경에 달한 후에 발병한다. 같은 병이라도 침입한 병원체의 양이나 독력, 감염된 사람의 감수성 등에 따라서도 달라진다. 보통 잠복기에는 병적 징후가 나타나지 않지만 민감한 사람의 경우는 두통-식욕부진-현기증-불쾌감과 같은 전구증상을 나타내는 수도 있다. 2. 보온기간, 배양기간, 발육에 필요한 기간. |
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| 영문 | funnel breast | 한글 | 오목가슴, 누두흉 |
|---|---|---|---|
| 설명 | 앞가슴의 중앙부가 깔대기 모양으로 함몰되어 있는 것. 대부분 유전성으로 심한 경우에는 심장이나 허파에 장애를 일으킨다. 인구 300~400명의 신생아 중 1명이 이 질환에 걸릴 수 있는 것으로 알려져 있다. 보통 이 증상을 갖고 있는 사람은 마른체형이고 척추후만 등 자세의 이상을 동반하는 경우가 많다. 보통 신생아기, 영아기 등 어려서부터 알게 되며, 기형적인 모습 때문에 학교생활이나 사회생활을 할 때 지장을 받을 수도 있다. 남자에게 많이 발생하고, 상기도폐색-기관지연화증과 관련되어 생길 수도 있다. 보통의 경우 증상이 없지만, 심한 경우 심폐기능의 장애로 인해 운동할 때 숨이 차고, 반복되는 감기증상이 있을 수 있다. 승모판 탈출, 조기흥분증후군이 나타나는 경우도 있다. |
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| 영문 | fibrocystic disease of breast | 한글 | 유방 섬유낭병 |
|---|---|---|---|
| 설명 | 젖을 생산하는 젖샘내에 완두콩 또는 큰콩 크기의 결절이 발생하는 증세를 특징으로 하는 병. 30~50대의 부인에게 흔히 발생하며, 그 대부분은 양쪽 유방에 동시에 발생한다. 이러한 결절은 두 손가락 사이에 끼워 촉진할 때는 분명하지만, 흉벽을 손바닥으로 누르면 명료하지 않을 정도로 부드러운 것이 많다. 그 발생 원인에는 여러 가지 설이 많은데, 젖샘조직에 대한 만성적인 자극이 주원인이라 생각되고 있으며, 극히 서서히 진행하는 경과를 밟는다. 진단은 촉진, 초음파진단 등으로 하며, 암과 감별이 곤란할 때는 조직의 일부를 채취하여 검사하는 생검이 이용되기도 한다. |
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| 영문 | ovarian cancer | 한글 | 난소암 |
|---|---|---|---|
| 설명 | 여성의 난소에 발생하는 암. 부인과종양으로서 50세 이상 여성악성종양의 약 18%를 차지한다. 종양은 대개 복부 깊숙히 위치하므로 종양이 많이 진행된 상태에서 발견되는 수가 많으며, 또한 종양의 초기에는 증상이 거의 없는 경우가 많아 더욱 조기발견이 어렵다. 아주 다양한 종류의 암이 발생하며, 예후도 각기 그 종양의 종류에 따라 다르다. 대표적인 암으로 장성낭샘암종(serous cystadenocarcinoma), 점액낭샘암종(mucinous cystadenocarcinoma), 종자세포종(germinoma 등이 있다. 치료는 수술적 치료가 선행되어야 하지만, 많이 진행되어 이미 다른 조직으로 전이가 이루어진 상태에서는 화학요법이 선택적으로 사용된다. |
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| GTN | - Stages of GTN(FIGO, WHO) 1. Stage O; Molar Pregnancy(H-Mole... |
|---|---|
| LCP Disease | Legg-Calve-Perthes Disease ? Stages of LCP Disease(= Juvenile Idiopathic AVN) &nb... |
| MBC | male breast cancer; maximal bladder capacity; maximal breathing capacity; metastatic breast cancer; ... |
| CEA | Carcino-Embryonic Antigen [HP 1825-6] ; Oncofetal Antigens ; Glycopro... |
| AFP | Alpha(α) Feto-Protein [HP 1826, 1858, 1859, 2265] ; Oncofetal Antigens &nbs... |
| BC | Breast Cancer |
|---|---|
| BCDDP | Breast Cancer Detection Demonstration Project |
| BCPT | Breast Cancer PRevention TRial |
| BCRP | Breast Cancer Resistance Protein |
| BCC | Breast cancer cells |
| early stage breast cancer | <oncology> Cancer is confined to the breast and has not spread to other sites in the body. (16 Dec 1997) |
|---|
| BRCA1 breast cancer susceptibility gene | This mutated (changed) version of the BRCA1 gene makes a person susceptible to developing breast cancer. (12 Dec 1998) |
|---|---|
| breast cancer | <oncology> The uncontrolled growth of malignant breast tissue. Breast cancer is currently the most common cancer in women and the leading cause of cancer-related death in the 15-54 age group. Strong risk factors include a prior history for breast cancer or a positive family history for breast cancer. Early detection is possible through the use of monthly breast self-examination, annual clinical exams and mammography. WWW: cancerNET document for patients WWW: cancerNET document for clinicians (05 Jan 1998) |
| breast cancer susceptibility genes | Inherited factors that predispose to breast cancer. Put otherwise, these genes make one more susceptible to the disease and so increase the risk of developing breast cancer. Two of these genes, BRCA1 and BRCA2, have been identified (and prominently publicised). Several other genes (those for the Li-Fraumeni syndrome, Cowden disease, Muir-Torre syndrome, and ataxia-telangiectasia) are also known to predispose to breast cancer. However, since all of these known breast cancer susceptibility genes together do not account for more than a minor fraction (1/5th at most) of breast cancer that clusters in families, it is clear that more breast cancer genes remain to be discovered. (12 Dec 1998) |
| cancer, breast | Cancer of the tissue containing or involving the milk glands (mammary tissue). Breast cancer is diagnosed with self- and physician- examination of the breasts, mammography, ultrasound testing, and biopsy. There are many types of breast cancer that differ in their capability of spreading to other body tissues (metastasis). Treatment of breast cancer depends on the type and location of the breast cancer, as well as the age and health of the patient. The American Cancer Society recommends that a woman should have a baseline mammogram between the ages of 35 and 40 years. Between 40 and 50 years of age mammograms are recommended every other year. After age 50 years, yearly mammograms are recommended. (12 Dec 1998) |
| cancer, breast, familial | A number of factors have been identified that increase the risk of breast cancer. One of the strongest of these risk factors is the history of breast cancer in a relative. About 15-20% of women with breast cancer have such a family history of the disease, clearly reflecting the participation of inherited (genetic) components in the development of some breast cancers. Dominant breast cancer suceptibility genes, including BRCA1 and BRCA2, appear responsible for about 5% of all breast cancer. (12 Dec 1998) |
| cancer, breast, susceptibility genes | Inherited factors that predispose to breast cancer. Put otherwise, these genes make one more susceptible to the disease and so increase the risk of developing breast cancer. Two of these genes, BRCA1 and BRCA2, have been identified (and prominently publicised). Several other genes (those for the Li-Fraumeni syndrome, Cowden disease, Muir-Torre syndrome, and ataxia-telangiectasia) are also known to predispose to breast cancer. However, since all of these known breast cancer susceptibility genes together do not account for more than a minor fraction (1/5th at most) of breast cancer that clusters in families, it is clear that more breast cancer genes remain to be discovered. (12 Dec 1998) |
| genes, breast cancer susceptibility | Inherited factors that predispose to breast cancer. Put otherwise, these genes make one more susceptible to the disease and so increase the risk of developing breast cancer. Two of these genes, BRCA1 and BRCA2, have been identified (and prominently publicised). Several other genes (those for the Li-Fraumeni syndrome, Cowden disease, Muir-Torre syndrome, and ataxia-telangiectasia) are also known to predispose to breast cancer. Howeverm, since all of these known breast cancer susceptibility genes together do not account for more than a minor fraction (1/5th at most) of breast cancer that clusters in families, it is clear that more breast cancer genes remain to be discovered. See related entries to: BRCA1; BRCA2; Breast cancer, familial. (12 Dec 1998) |
| metastatic breast cancer | Breast cancer that has spread to other parts of the body. (16 Dec 1997) |
| familial breast cancer | <oncology> A number of factors have been identified that increase the risk of breast cancer. One of the strongest of these risk factors is the history of breast cancer in a relative. About15-20% of women with breast cancer have such a family history of the disease, clearly reflecting the participation of inherited (genetic) components in the development of some breast cancers. Dominant breast cancer suceptibility genes, including BRCA1 and BRCA2, appear responsible for about 5% of all breast cancer. See related entries to: Breast cancer susceptibility genes; BRCA1; BRCA2. There are 2 genes; BRCA1 and BRCA2 which are susceptibility genes for breast cancer. They are inherited factors that predispose to breast cancer. Put otherwise, these genes make one more susceptible to the disease and so increase the risk of developing breast cancer. Two of these genes, BRCA1 and BRCA2, have been identified (and prominently publicised). Several other genes (those for the li-fraumeni syndrome, cowden disease, muir-torre syndrome, and ataxia-telangiectasia) are also known to predispose to breast cancer. However, since all of these known breast cancer susceptibility genes together do not account for more than a minor fraction (1/5th at most) of breast cancer that clusters in families, it is clear that more breast cancer genes remain to be discovered. (12 Dec 1998) |
| adenovirus early proteins | <molecular biology, protein, virology> Proteins encoded by adenoviruses that are synthesised prior to, and in the absence of, viral DNA replication. The proteins are involved in both positive and negative regulation of expression in viral and cellular genes, and also affect the stability of viral mRNA. Some are also involved in oncogenic transformation. (12 Dec 1998) |
| genes, immediate-early | Genes that show rapid and transient expression in the absence of de novo protein synthesis. The term was originally used exclusively for viral genes where immediate-early referred to transcription immediately following virus integration into the host cell. It is also used to describe cellular genes which are expressed immediately after resting cells are stimulated by extracellular signals such as growth factors and neurotransmitters. (12 Dec 1998) |
| history of medicine, early modern | The period of the history of medicine from 1451 through 1600 a.d. History of medicine, 15th cent. And history of medicine, 16th cent. Are also available. (12 Dec 1998) |
| immediate early gene | Class of genes whose expression is low or undetectable in quiescent cells, but whose transcription is activated within minutes after extracellular stimulation such as addition of a growth factor. C fos and c myc proto-oncogenes were among the first IEG's to be identified. Many IEG's encode transcription factors and therefore have a regulatory function. (18 Nov 1997) |
| immediate-early proteins | Proteins that are coded by immediate-early genes, in the absence of de novo protein synthesis. The term was originally used exclusively for viral regulatory proteins that were synthesised just after viral integration into the host cell. It is also used to describe cellular proteins which are synthesised immediately after the resting cell is stimulated by extracellular signals. (12 Dec 1998) |
| early | 1. In advance of the usual or appointed time; in good season; prior in time; among or near the first; opposed to late; as, the early bird; an early spring; early fruit. "Early and provident fear is the mother of safety." (Burke) "The doorsteps and threshold with the early grass springing up about them." (Hawthorne) 2. Coming in the first part of a period of time, or among the first of successive acts, events, etc. "Seen in life's early morning sky." (Keble) "The forms of its earlier manhood." (Longfellow) "The earliest poem he composed was in his seventeenth summer." (J. C. <philosophy> Shairp) Early English See the Note under English. Early English architecture, the first of the pointed or Gothic styles used in England, succeeding the Norman style in the 12th and 13th centuries. Synonym: Forward, timely, not late, seasonable. Origin: OE. Earlich. See Early. Source: Websters Dictionary (01 Mar 1998) |
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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