| 영문 | Crohn's disease | 한글 | 크론병 |
|---|---|---|---|
| 설명 | 만성적이고 재발을 잘하는 창자의 염증을 특징으로 하는 병. 장의 벽은 안쪽에서부터 점막, 점막하조직, 근육층, 장막의 4개의 층으로 이루어져 있는데, 크론병은 이 모든 층의 염증을 동반한다. 장의 모든 부분에서 생길 수가 있지만 주로 막창자와 연결되는 큰창자의 말단부에 가장 많이 생긴다. 창자의 전층의 염증으로 인해서 장의 폐쇄나 괴양을 만들며 종종 천공된다. |
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| 영문 | infectious disease | 한글 | 감염병 |
|---|---|---|---|
| 설명 | 바이러스로부터 기생충 크기까지의 생물을 원인으로 하는 병. 원인은 접촉전염성이며, 병원에서 감염되는 경우도 있다. 감염을 원인균에 따라 분류하면 바이러스, 세균, 클라미디아, 리켓차, 미코박테리움, 곰팡이, 원충, 윤충, 외부기생충 감염으로 나눌 수 있다. |
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| 영문 | hypertensive heart disease | 한글 | 고혈압심장병 |
|---|---|---|---|
| 설명 | 고혈압에 의해서 생기는 심장병. 고혈압심장병이라는 진단을 붙이기 위해서는 최소한 다음과 같은 조건이 부합되어야 하는데, 첫째 심장혈관계에 심장병을 유발할 수 있을 만한 다른 병변이 없이 좌심실 비대가 있어야 하며, 둘째 고혈압을 앓았다는 병력이 있어야 한다. 주로 고혈압에 의한 심장병은 초기에는 좌심실이 비후라는 것으로 특징되어진다. 즉 혈압이 높으므로 혈액을 순환시키기 위해서는 그만큼 심장의 혈액을 보내는 힘이 좋아야 한다. 그 힘을 얻기위해서는 심근의 비후가 필요로 하여 좌심실 근육의 비후가 생긴다. 그리고 고혈압이 지속이 될 경우에는 결국 심장이 제 구실을 하지 못하고 펌프로서의 기능을 잃어버리게 되어 심장기능상실에 빠지게 된다. |
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| 영문 | pelvic inflammatory disease | 한글 | 골반염질환 |
|---|---|---|---|
| 설명 | 골반주위의 장기에 발생하는 염증을 말함. 주로 여성에서 발생하며 원인은 임균(gonococcus)과 비임균에 의한 감염(non-gonorrheal infection)에 의한다. 증상은 초기에는 질분비물, 하복부동통, 여성의 생식기부위에 발생하는 압통, 월경통, 월경량의 증가 등이다. 일찍 치료해야 하며, 계속적으로 병이 지속시 여성의 불임의 원인이 된다. 후진국에서는 가장 많은 여성불임의 원인이기도 함. 치료는 항생제의 투여이다. |
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| 영문 | Graves' disease | 한글 | 그레이브스병 |
|---|---|---|---|
| 설명 | 갑상샘의 비대와 갑상샘호르몬의 과다분비가 특징인 병이다. 이 병은 주로 25~50세에 호발하고 주로 여자에게서 많이 생긴다. 갑상샘 호르몬은 저장되어 있는 에너지를 소모하여 신체의 대사율을 높이는 호르몬이므로 입맛이 좋은 데도 불구하고 계속적인 체중의 감소, 그리고 축적된 에너지를 소모하여 열생많이 하여서 더위를 참기 힘들어하고 만성 쇠약감이나 근력의 약화를 보일 수가 있다. 그리고 눈에 특징적인 증상이 나타나는데 눈꺼풀이 비정상적으로 위로 올라가 있고, 눈이 아래의 물건을 주시할 경우에 눈꺼풀이 정상적으로는 처져야 하지만 갑상샘 호르몬이 과도하게 나올 경우에는 눈꺼풀이 처지지 않는다. 또 눈알이 앞쪽으로 돌출하는 안구돌출을 볼 수가 있다. 또 피부가 아주 부드럽고 물기가 많아서 축축하다. 그리고 특징적으로 하지의 앞쪽에 피부가 두꺼워져 국소적 융기를 이루는 것이 있는데 이것은 이 병의 특징적인 병터이다. |
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| CD | cadaver donor; canine distemper; canine dose; carbohydrate dehydratase; carbon dioxide; cardiac dise... |
|---|---|
| HD | Haab-Dimmer [syndrome]; Hajna-Damon [broth]; Hansen disease; hearing distance; heart disease; helix ... |
| MD | Doctor of Medicine [Lat. Medicinae Doctor]; magnesium deficiency; main duct; maintenance dose; major... |
| CDAI | Crohn disease activity index |
| AD | accident dispensary; acetate dialysis; active disease; acute dermatomyositis; addict, addiction; ade... |
| CD | Crohn Disease |
|---|---|
| CDAI | Crohn Disease Activity Index |
| AEBS | Antiestrogen binding sites |
| DHS | DNAse I hypersensitive sites |
| EBS | ETS binding sites |
Kugelberg-Welander disease 근 위축증의 유전성 연소형으로서 보통 상염색체성 열성 형질로 유전된다. 척수 전각의 병변이 그 원인이다.
kukuruku 원인 불명이며, 나이지리아에서 볼 수 있는 질환으로, 열
| crohn disease: sites | <radiology> Oesophagus: rare, stomach (2-20%): granulomatous gastritis, pseudo-post Bilroth-I appearance, ramshorn sign, antral-duodenal fistula, duodenum (4-10%): almost always associated with gastric involvement, bulb and proximal half of duodenum, small bowel (80%): regional enteritis, terminal ileum (alone/in combination): 95%, jejunum/ileum: 15%, commonly associated with medial caecal defect, colon (22-55%): granulomatous colitis, particularly on the right side, transverse stripe sign: contrast within coarse mucosal folds, rectum (35-50%) see: Crohn disease (12 Dec 1998) |
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| crohn disease | <radiology> CT: double halo (50%): edematous mucosa/thickened soft tissue, creeping fat (40%): mesenteric infiltration, ultrasound: thickened bowel wall (65%): about 8mm, inflammatory mass (14%), abscess (4%), distended fluid filled loops (12%), Complications: fistula (33%), intramural sinus tracts, abscess, perforations (rare), toxic megacolon, SBO (15%), hydronephrosis (usually right sided), adenocarcinoma in ileum/colon, Differential diagnosis: Yersinia: in TI, resolution in 3-4 months, TB (more severe involvement of caecum), segmental infarction (acute onset, elderly patient), radiation ileitis, lymphoma, carcinoid, eosinophilic gastoenteritis, potassium stricture see: sites, phases, extraintestinal manifestations Cf: ulcerative colitis More info: Crohn disease (12 Dec 1998) |
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| crohn disease: extraintestinal manifestations | <radiology> Fatty liver, gallstones (28-34%), risk 3-5X higher than expected, secondary to malabsorption of bile salts in terminal ileum, correlation with length of diseased ileum and duration of disease, sclerosing cholangitis, bile duct carcinoma, amyloidosis, urolithiasis: oxalate/uric acid stones, migratory arthritis (5-20%), sacroilitis, ankylosing spondylitis, erythema nodosum, uveitis see: Crohn disease (12 Dec 1998) |
| crohn disease: phases | <radiology> Nonstenotic phase: blunting, flattening, distortion, straightening, and thickening of mucosal folds (early event from obstructive lymphadema), aphthous ulcers: nodules with shallow central ulcerations, cobblestoning: serpiginous ulcers separated by areas of oedema, pseudopolyps: hyperplastic mucosa between denuded mucosa, postinflammatory polyps, skip lesions (90%), pseudodiverticula: bulging area of normal wall opposite affected scarred wall, mostly on the antimesenteric side, separation and displacement of small bowel loops (from increase in mesenteric fat, enlarged nodes, or perforation with abscess formation), stenotic phase: string sign: strictures (most in TI) in rigid loops, normal proximal loops may be dilated with stasis ulcers and fecoliths see: Crohn disease (12 Dec 1998) |
| crohn disease vs ulcerative colitis | <radiology> Crohn disease ulcerative colitis location right side left side ulcers deep shallow contraction no yes ileocaecal valve thickened gaping fistulae yes no eccentricity yes no rate of carcinoma slight increase marked increase megacolon unusual yes (12 Dec 1998) |
| Crohn's disease | <disease, gastroenterology> An inflammatory disease of the gastrointestinal tract that seems to have both genetic and environmental causes, not well understood. The peak incidence of onset of this disease is between 15 and 25 years of age. Crohn's also occurs in later years between the ages of 55 and 60. Common symptoms include recurrent abdominal pains, fever, nausea, vomiting, weight loss and diarrhoea which is occasionally bloody. Complications include gastrointestinal bleeding, fistulas and anal fissures. Treatment includes anti-inflammatory drugs and corticosteroids. Surgery is successful in a select few. (27 Sep 1997) |
| disease, crohn's | A chronic inflammatory disease of the intestine. Named after Burrill Crohn who described the disease in 1932. It usually affects persons in their teens or early twenties. It tends to be a chronic, recurrent condition withperiods of remission and exacerbation. In the early stages, there are small scattered shallow crater-like areas (erosions) called apthous ulcers in the inner surface of the bowel. With time, deeper and larger ulcers develop, causing scarring and stiffness of the bowel. The bowel becomes increasingly narrowed, leading to obstruction. Deep ulcers can puncture holes in the bowel wall, leading to infection in the abdominal cavity (peritonitis)and in adjacent organs. When only the large intestine (colon) is involved, the condition is called Crohn's colitis. When only the small intestine is involved, the condition is called Crohn's enteritis. When only the end of the small intestine (the terminal ileum) is involved, it is termed terminal ileitis.When both the small intestine and the large intestine are involved, the condition is called Crohn's enterocolitis (or ileocolitis). Abdominal pain, diarrhoea, vomiting, fever, and weight loss can be symptoms. Crohn's disease can be associated with reddish tender skin nodules, and inflammation of the joints, spine, eyes, and liver. Diagnosis is by barium enema, barium X-ray of the small bowel, and colonoscopy. Treatment includes medications for inflammation, immune suppression, antibiotics, orsurgery. (The disease is also called granulomatous enteritis or regionalenteritis). (12 Dec 1998) |
| attachment sites | <microbiology, molecular biology> Particular loci in both bacterial and phage DNA molecules at which phage DNA is integrated into the bacterial DNA by recombination between these sites. (12 Dec 1998) |
| binding sites | The reactive parts of a macromolecule that directly participate in its specific combination with another molecule. (12 Dec 1998) |
| binding sites, antibody | Local surface sites on antibodies which react with antigen determinant sites on antigens. They are formed from parts of the variable regions of the fab fragment of the immunoglobulin. (12 Dec 1998) |
| chromosome fragile sites | Heritable sensitive regions of chromosomes which show up in vitro as non-staining bands. They are associated with chromosome breakage and other aberrations, and, when located on sex chromosomes, they produce phenotypic abnormalities. No abnormal phenotype has been definitely identified with autosomal fragile sites, but some rare autosomal recessive disorders may be due to homozygosity for fragile sites. Fragile sites are designated by the letters "fra" followed by the designation for the specific chromosome and locus. (12 Dec 1998) |
| contact sites A | Developmentally regulated adhesion sites that appear on the ends of aggregation competent Dictyostelium discoideum at the stage when the starved cells begin to come together to form the grex. Originally detected by the use of Fab fragments of polyclonal antibodies, raised against aggregation competent cells and adsorbed against vegetative cells, to block adhesion in EDTA containing medium. (Cell cell adhesion mediated by contact sites A, unlike that mediated by contact sites B, is not divalent cation sensitive). The fact that a mutant deficient in csA behaves perfectly normally in culture is puzzling. (18 Nov 1997) |
| contact sites B | Developmentally regulated adhesion sites that appear on the ends of aggregation competent Dictyostelium discoideum at the stage when the starved cells begin to come together to form the grex. Originally detected by the use of Fab fragments of polyclonal antibodies, raised against aggregation competent cells and adsorbed against vegetative cells, to block adhesion in EDTA containing medium. (Cell cell adhesion mediated by contact sites A, unlike that mediated by contact sites B, is not divalent cation sensitive). The fact that a mutant deficient in csA behaves perfectly normally in culture is puzzling. (18 Nov 1997) |
| sequence tagged sites | Short, tagged tracts of DNA sequence that are used as landmarks in genome mapping. In most instances, 200 to 500 base pairs of sequence define a sequence tagged site (sts) that is operationally unique in the human genome (i.e., can be specifically detected by the polymerase chain reaction in the presence of all other genomic sequences). The overwhelming advantage of stss over mapping landmarks defined in other ways is that the means of testing for the presence of a particular sts can be completely described as information in a database. (12 Dec 1998) |
| sequence-tagged sites | Short stretches of DNA sequences that can be detected by use of the polymerase chain reaction. (05 Mar 2000) |
| immunologically privileged sites | Sites where allografts are not readily rejected, probably because these particular areas have poor lymphatic drainage. (05 Mar 2000) |
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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