| ¿µ¹® | autoimmune dieases | ÇÑ±Û | ÀÚ°¡¸é¿ªÁúȯ |
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| ¼³¸í | ¸é¿ªÀ̶õ ¿Ü°è¿¡¼ µé¾î¿Â ¹°ÁúÀ» ÆÄ±«Çϰųª ¹«±â·ÂÈ ½ÃÄѼ ÀÚ½ÅÀ» º¸È£ÇÏ´Â ÀÏ·ÃÀÇ ÀÛ¿ëÀ» ¸»ÇÑ´Ù. ¸é¿ª¿¡¼ °¡Àå Áß¿äÇÑ ´Ü°è´Â ÀڽŰú ¿ÜºÎ¿¡¼ µé¾î¿Â ¹°ÁúÀ» ±¸ºÐÇÏ´Â °ÍÀÌ´Ù. ÀÚ°¡ ¸é¿ªÁúȯÀ̶õ ÀÌ·± ÀڽŰú ¿ÜºÎ¿¡¼ µé¾î¿Â ¹°ÁúÀÇ ±¸ºÐÀÌ È®½ÇÇÏÁö ¸øÇÏ¿© ÀÚ½ÅÀÇ ¹°Áú·Î À߸ø ÀνÄÇÏ¿© ½º½º·Î ÀÚ½ÅÀÇ ¹°ÁúÀ» ÆÄ±«ÇÏ´Â º´À» ¸»ÇÑ´Ù. |
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| ¿µ¹® | progesterone | ÇÑ±Û | ÇÁ·Î°Ô½ºÅ×·Ð, Ȳüȣ¸£¸ó |
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| ¼³¸í | 21°³ÀÇ Åº¼Ò¸¦ °¡Áö´Â ½ºÅ×·ÎÀ̵å·Î¼ ÁÖ·Î ³¼ÒÀÇ È²Ã¼ ¹× Źݿ¡¼ ¸¸µé¾îÁö³ª, ±× ¿Ü °íȯÀ̳ª ºÎ½Å°ÑÁú¿¡¼µµ ¹Ì·® ¸¸µé¾îÁø´Ù. ÀÌ È£¸£¸óÀº Àڱÿ¡ ÀÛ¿ëÇÏ¿© ¼öÁ¤¶õ(Á¤ÀÚ¿Í ³ÀÚ°¡ ¸¸³ª¼ »ý±ä žÆÀÇ Àü±¸Ã¼)À» Âø»ó½Ã۱â À§ÇÑ Áغñ·Î Àڱ󻸷À» ¼ºÀå½ÃŰ°í ¹ß´Þ½Ã۴µ¥ °ü¿©Çϰí ÀÓ½ÅÀÌ µÇ¸é, Ȳü´Â ´ë°³ 1°³¿ù°£ ÇÁ·Î°Ô½ºÅ×·ÐÀ» ºÐºñÇÏ¿© ÀÓ½ÅÀ» À¯Áö½Ã۸ç, ±× ÀÌÈÄ¿¡´Â Źݿ¡¼ ºÐºñÇÑ´Ù. ¼öÁ¤ÀÌ ÀϾÁö ¾ÊÀ¸¸é, Ȳü´Â ´õÀÌ»ó ÇÁ·Î°Ô½ºÅ×·ÐÀ» ºÐºñÇÏÁö ¾ÊÀ¸¹Ç·Î, Àڱ󻸷ÀÌ Å»¶ôµÇ¾î ¿ù°æÀÌ ½ÃÀ۵ȴÙ. ±× ¿Ü À¯¹æ¿¡ ÀÛ¿ëÇÏ¿© Á¥ÀÇ »ý»êÀº Áõ°¡½Ã۰í, ºÐºñ¸¦ ¾ïÁ¦ÇÏ´Â ÀÛ¿ëµµ ÀÖ´Ù. |
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| ¿µ¹® | seborrheic dermatitis | ÇÑ±Û | Áö·çÇǺο° |
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| ¼³¸í | ÇǺÎÀÇ ¸¸¼º¿°ÁõÀ¸·Î ¸Ó¸®µ¤°³, ¾ó±¼(´«½ç, ÀÔ¼ú, ´«²¨Ç°, ±Í µî), º¹Àå»ÀºÎÀ§, °Üµå¶ûÀÌ µî¿¡ È£¹ßÇÏ´Â ÇǺκ´ÀÌ´Ù. ÀÌ º´Àº ¸¶¸£°Å³ª ½À±â°¡ Àְųª, ±â¸§±â°¡ ÀÖ´Â ºñ´ÃÀÌ Æ¯Â¡À̰í, ´Ù¾çÇÑ ¸ð¾ç°ú Å©±âÀÇ È²»ö¹ÝÀ» Çü¼ºÇϸç È£Àü°ú ¾Çȸ¦ µÇÇ®ÀÌ Çϰí, ´Ù¼Ò °¡·Á¿î °¨°¢À» µ¿¹ÝÇÑ´Ù. |
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| ¿µ¹® | dermatitis | ÇÑ±Û | ÇǺο° |
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| ¼³¸í | ÇǺο°À̶ó°í Çϸé À̰ÍÀº ½ÀÁøÇǺκ´(eczematous dermatoses)À» À̸£´Â ¸»ÀÌ´Ù. ½ÀÁøÀ̶õ ±Þ¼º±â¿¡´Â °¡·Á¿òÁõÀ» µ¿¹ÝÇÏ´Â ¼öÆ÷³ª, È«¹Ý, ºÎÁ¾ µîÀÌ ³ªÅ¸³ª°í ¸¸¼º±â¿¡´Â ºÎÁ¾, ¼öÆ÷´Â °¨¼ÒµÇ´Â ¹Ý¸é¿¡ ż±È, ºñ´Ã, ÇǺλöÀÇ º¯È µîÀÌ Çü¼ºµÇ´Â ¸ðµç ÇǺκ´µéÀ» À̸£´Â ¸»ÀÌ´Ù. ±× ¹ß»ýºóµµ´Â Áö¿ª¿¡ µû¶ó ´Ù¼Ò Â÷À̰¡ ÀÖÀ¸³ª Àüü ÇǺÎÁúȯÀÇ ¾à 1/3~1/4À» Â÷ÁöÇϰí ÀÖ´Ù. 1.Á¢ÃËÇǺο°(contact dermatitis)-¿ÜºÎ¹°ÁúÀÇ Á¢ÃË¿¡ ÀÇÇØ¼ ¹ß»ýÇÏ´Â ÇǺο°À» ¸»ÇÑ´Ù. ¹°À̳ª ¼¼Á¦¿¡ Àå±âÀû Á¢ÃË¿¡ ÀÇÇØ¼ ¼Õ³¡ºÎÀ§¿¡ »ý±â´Â ÁֺνÀÁø(housewife's eczema)À̳ª °øÀå¿¡¼ À¯µ¶ÇÑ ¹°ÁúÀÇ Á¢ÃË¿¡ ÀÇÇØ¼ »ý±â´Â ÇǺο° µîÀÌ À̰ÍÀÇ ¿¹ÀÌ´Ù. 2.¾ÆÅäÇÇÇǺο°(atopic dermatitis)-ÇǺΰ¡ °¡·Á¿ò¿¡ ´ëÇÑ ¿ªÄ¡°¡ ³·¾Æ¼ ¾ÆÁÖ ½ÉÇÑ °¡·Á¿òÀ» ´À³¢°í ÀÌ·Î ÀÎÇØ¼ 2Â÷ÀûÀ¸·Î »ý±â´Â ½ÀÁøÀ¸·Î À¯ÀüÀûÀÎ °æÇâÀÌ ¾ÆÁÖ °ÇÏ´Ù. ¾ÆÁÖ ½ÉÇÑ °¡·Á¿òÁõ°ú ƯÁ¤ÇÑ ¹ßº´ºÎÀ§·Î ´ë°³ Áø´ÜÀÌ µÇ´Âµ¥ 2¼¼ÀÌÇÏÀÇ À¯¾Æ¿¡°Ô »ý°åÀ» °æ¿ì¿¡´Â ÁÖ·Î »´, À̸¶, µÎÇÇ µîÀÇ ¾ó±¼¿¡ ¸¹ÀÌ »ý±â°í, ¼Ò¾Æ±âÀÇ °æ¿ì¿¡´Â ÁÖ·Î ¹«¸À̳ª ÆÈ²ÞÄ¡ÀÇ »ìÀÌ Á¢È÷´Â ºÎÀ§¿¡ ¸¹ÀÌ »ý±ä´Ù. 1¼¼ ¹Ì¸¸ÀÇ À¯¾Æ¿¡¼ ¸¹ÀÌ ¹ß»ýÇϸç, ³ªÀ̰¡ µé¼ö·Ï È£ÀüµÇ´Â °æ°ú¸¦ º¸ÀδÙ. 3.Áö·ç ÇǺο°(seborrheic dermatitis)-¸¸¼ºÀÇ °æ°ú¸¦ ÃëÇÏ´Â ½ÀÁøÀÇ ÀÏÁ¾À¸·Î ÁÖ·Î ÇÇÁöÀÇ ºÐºñ°¡ ¿Õ¼ºÇÑ ºÎÀ§(¸Ó¸®µ¤°³, ¾ó±¼, °¡½¿ µîÀÇ Áß°£ ºÎÀ§)¿¡ ³ë¶õ»ö ³»Áö ºÓÀº»öÀÇ ¸¸¼º ½ÀÁøÀÇ ¾ç»óÀ» ³ªÅ¸³»´Â º´º¯, ƯÈ÷ ¹øµé¹øµéÇÑ ºñ´ÃÀÌ Æ¯Â¡ÀûÀ¸·Î ÇÏ´Â º´º¯À» ÁÖ Æ¯Â¡À¸·Î ÇÏ´Â ÁúȯÀÌ´Ù. ¿øÀÎÀº È®½ÇÇÏÁö ¾ÊÀ¸³ª Pityrosporum ovaleÀ̶ó´Â ±Õ°ú °¡Á··Â, ½Å°æº´°ú ¿¬°üÀÌ ÀÖ´Ù°í ÇÑ´Ù. |
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| APD | action potential duration; acute polycystic disease; advanced physical diagnosis; anteroposterior di... |
|---|---|
| HCG, hCG | Human Chorionic Gonadotropin; »ç¶÷À¶¸ð¼º¼º¼±ÀÚ±ØÈ£¸£¸ó 1. Placental Glycoprotein Hormone &nbs... |
| AID | acquired immunodeficiency disease; acute infectious disease; acute ionization detector; Agency for I... |
| EAT | Eating Attitudes Test; Ehrlich ascites tumor; electro-aerosol therapy; epidermolysis acuta toxica; e... |
| MPA | 1) Mean Pulmonary Artery 2) Medroxy-Progesterone Acetate(Provera¨Þ) &nb... |
| ACD | Allergic contact dermatitis |
|---|---|
| AD | atophic dermatitis |
| CAD | Chronic actinic dermatitis |
| CD | Contact dermatitis |
| DH | Dermatitis Herpetiformis |
| anaemia, haemolytic, autoimmune | Acquired haemolytic anaemia due to the presence of autoantibodies which agglutinate or lyse the patient's own red cells. (12 Dec 1998) |
|---|---|
| autoimmune | <immunology> Pertaining to autoimmunity. (02 Jan 1998) |
| autoimmune disease | <disease> A disease process that involves the production of host antibodies to host tissue. (27 Sep 1997) |
| autoimmune diseases | Are illnesses which occur when the body tissues are attacked by its own immune system. The immune system is a complex organisation within the body that is designed normally to seek and destroy invaders of the body, particularly infections. Patients with these diseases have unusual antibodies in their blood that target their own body tissues. (12 Dec 1998) |
| autoimmune haemolytic anaemia | <haematology> A condition that results from the cellular destruction (haemolysis) of red blood cells due to antibodies formed to components on the surface of the red blood cells. Origin: Gr. Haima = blood (02 Jan 1998) |
| autoimmune hepatitis | <pathology> A type of chronic active hepatitis that results from circulating auto-antibodies and chronic inflammation of the liver. Symptoms are those of chronic active hepatitis. (27 Sep 1997) |
| autoimmune thrombocytopenia purpura | <haematology> A rare autoimmune disorder characterised by an acute shortage of platelets with resultant bruising and spontaneous bleeding. The platelet count becomes exceedingly low and spontaneous bleeding from the gums, gastrointestinal tract and nose can be seen. Physical examination may demonstrate enlargement of the spleen. A typical rash occurs to do microscopic haemorrhage of small blood vessels in the skin. Platelet counts under 10,000 can lead to spontaneous haemorrhage into the brain causing death. Treatment with corticosteroids is generally effective. Surgical removal of the spleen (splenectomy) is reserved for some patients. Anti-platelet antibodies are detectable in some cases. It may present in either an acute or a chronic form. Acronym: ITP (20 Sep 2002) |
| autoimmune thyroiditis | <endocrinology> Inflammation of the thyroid gland without the formation of pus. Noninfectious nonbacterial thyroid inflammation. (27 Sep 1997) |
| polyendocrinopathies, autoimmune | Autoimmune disease affecting multiple endocrine organs. Type I is characterised by childhood onset and mucocutaneous candidiasis, while type II exhibits any combination of adrenal insufficiency (addison's disease), lymphocytic thyroiditis, hypoparathyroidism, and gonadal failure. In both types organ-specific antibodies against a variety of endocrine glands have been detected. The type II syndrome differs from type I in that it is associated with HLA-a1 and b8 haplotypes, onset is usually in adulthood, and candidiasis is not present. (12 Dec 1998) |
| hepatitis, autoimmune | An unresolving, predominately periportal, hepatitis, usually with hypergammaglobulinaemia and serum autoantibodies. The existence of subgroups (types 1, 2, and 3) based on serological findings are controversial. Additionally, some patients have variant forms, where there are features associated with both autoimmune hepatitis and another type of chronic liver disease (overlap syndromes) or where there are findings incompatible with autoimmune hepatitis (outlier syndromes). (12 Dec 1998) |
| systemic autoimmune diseases | A group of connective tissue disease's characterised by the presence of autoantibodies responsible for immunopathologically mediated tissue lesions; systemic lupus erythematosus is the prototype. (05 Mar 2000) |
| thyroiditis, autoimmune | A progressive disease of the thyroid gland with antibodies in the blood stream directed against the thyroid and infiltration of the gland by lymphoctes (a key type of white blood cells involved in the immune response). This immune response is against one's own thyroid. (it is autoimmune.) predominantly affects women. Can be familial. Also called hashimoto's disease or hashimoto's thyroiditis. (12 Dec 1998) |
| receptors, progesterone | Specific proteins found in or on cells of progesterone target tissues that specifically combine with progesterone. The cytosol progesterone-receptor complex then associates with the nucleic acids to initiate protein synthesis. There are two kinds of progesterone receptors, a and b. Both are induced by oestrogen and have short half-lives. (12 Dec 1998) |
| progesterone | <biochemistry, gynaecology> Produced in the corpus luteum, as an antagonist of oestrogens. Promotes proliferation of uterine mucosa and the implantation of the blastocyst, prevents further follicular development. (18 Nov 1997) |
| progesterone-11HS-horseradish peroxidase | <chemical> Used as a tracer in an immunoassay for progesterone Synonym: p-11hs-hrp, progesterone-11hs-hrp (26 Jun 1999) |
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