| ¿µ¹® | helminth | ÇÑ±Û | ¿¬Ãæ |
|---|---|---|---|
| ¼³¸í | ¸öÀÌ °¡´Ã°í ±æ¸ç ²ÞƲ°Å·Á ±â¾î´Ù´Ï´Â ¹«Ã´Ãßµ¿¹°À» À̸£´Â ¸». ±×¸®½º¾îÀÇ ¡®helmins(¹ú·¹¶ó´Â ¶æ)¡¯¿¡¼ À¯·¡ÇÏ´Â ¿ë¾î·Î, ¿ø·¡´Â ³»ÀåÃæÀ» ¶æÇÏ¿´´Ù. µ¿¹°ºÐ·ùÇлó ¸íĪÀÌ ¾Æ´Ï¶ó ÆíÇüµ¿¹°-¼±Çüµ¿¹°-ȯÇüµ¿¹° µî¿¡ ¼ÓÇÏ´Â µ¿¹°±ºÀ» ¸»Çϸç, ±â»ý»ýȰÀ» ÇÏ´Â Á¾ ¿Ü¿¡ ÀÚÀ¯»ýȰÀ» ÇÏ´Â Á¾µµ Æ÷ÇÔÇÑ´Ù. ¿¬Ãæ·ù´Â °ÇÁ¶ÇÑ »óÅ·¹¼ ¸öÀ» ÁöÄÑÁÙ ¸¸ÅÀÇ ½À±â¸¸ ÀÖ´Â °÷ÀÌ¸é °ÅÀÇ ¸ðµç »ýÅÂÀû ÁöÀ§¸¦ Â÷ÁöÇÒ ¼ö ÀÖµµ·Ï ÀûÀÀÇϰí ÀÖ´Ù. ¹ú·¹°¡ »ì ¼ö ¾ø´Â °÷Àº ¸Å¿ì °ÇÁ¶ÇÑ Àå¼Ò»ÓÀÌ´Ù. µ¿½Ä¹°ÀÇ ±â»ýÃæÀ̱⵵ Çϱ⠶§¹®¿¡ ÀÇÇÐÀûÀ¸·Î Áß¿äÇÏ´Ù. ÀÌ °¡¿îµ¥ ÆíÇüµ¿¹°Àº µî°ú ¹è°¡ ÆíÆòÇÏ°í ¸ö¿¡ µÕ±Ù ¸¶µð°¡ ¾øÀ¸¸ç, ¼ÒȰüÀÌ ¹ß´ÞÇÏÁö ¸øÇϰí Ç×¹®ÀÌ ¾ø´Â ÈíÃæ-Á¶Ãæ µî°ú °°Àº ¹ú·¹·Î ¾Ï¼öÇѸöÀ̸ç, ±â»ýÀ» Çϰųª °æ¿ì¿¡ µû¶ó µ¶¸³»ýȰÀ» ÇÑ´Ù. ¼±Çüµ¿¹°Àº ¸öÀÇ ¸ð¾çÀÌ ±æ°í °¡´Â ¼±ÀÇ ÇüÅ·Π¸¶µð°¡ ¾øÀ¸¸ç, Ç÷°üÀÌ È£Èí±â°¡ ¾ø´Â È¸ÃæÀ̳ª ¿äÃæ-½ÊÀÌÁöÀåÃæ-ÆíÃæ µîÀÇ ±â»ýÃæÀ¸·Î ¾Ï¼öµý¸öÀ¸·Î µÇ¾î ÀÖ´Ù. |
||
| AMA | 1) Anti-Mitochondrial Antibodies 2) American Medical Association |
|---|---|
| Anti-LKM | Antibodies to Liver-Kidney Microsome |
| FA | 1) Fatty Acid 2) Fluorescent Antibodies; Çü±¤ Ç×ü |
| MAbs | Monoclonal Antibodies |
| MHA-TP | Micro-Hemagglutination Assay for antibodies to Treponema Pallidum |
| AEA | A-anti-endomysium antibodies |
|---|---|
| ANCA | Anti-Neutrophil Cytoplasmic Antibodies |
| ACA | Anti-cardiolipin antibodies |
| ACL | Anti-cardiolipin antibodies |
| ACLA | Anti-cardiolipin antibodies |
| antibodies, helminth | Antibodies produced by human or animal cells following clinical or experimental exposure to parasitic helminth antigens. The IgE class of immunoglobulins is usually formed and released, but IgG, IgM, and IgA may also occur. (12 Dec 1998) |
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| antigens, helminth | Any part or derivative of a helminth that elicits an immune reaction. The most commonly seen helminth antigens are those of the schistosomes. (12 Dec 1998) |
|---|---|
| genes, helminth | The hereditary material of helminths. (12 Dec 1998) |
| genes, structural, helminth | DNA sequences that code for RNA and for the proteins required for the enzymatic and structural function of helminthic cells. (12 Dec 1998) |
| RNA, helminth | Ribonucleic acid in helminths having regulatory and catalytic roles as well as involvement in protein synthesis. (12 Dec 1998) |
| helminth | Any parasitic worm, though it usually refers to one living in the intestines of a vertebrate animal. (09 Oct 1997) |
| helminth proteins | Proteins found in any species of helminth. (12 Dec 1998) |
| DNA, helminth | Deoxyribonucleic acid that makes up the genetic material of helminths. (12 Dec 1998) |
| acetylcholine receptor antibodies | <neurology, investigation> A test used to measure the amount of antibodies to acetylcholine receptors on nerve endings. This is a diagnostic test for myasthenia gravis. A normal value is no antibodies in the bloodstream. Acetylcholine receptor (AChR) binding autoantibodies (i.e. Antibodies reactive with several epitopes other than the binding site for acetylcholine or alpha-bungarotoxin) are present in approximately 88% of patients with generalised myasthenia gravis, 70% of ocular myasthenia and in approximately 80% of myasthenia gravis in remission. Although serum concentrations of AChR binding autoantibodies do not in general correlate well with severity of weakness, there is typical decrease in concentration as weakness improves with immunosuppressive therapy. AChR blocking autoantibodies (i.e., antibodies reactive with the AChR binding site) are present in about 50% of patients with myasthenia gravis, 30% with ocular myasthenia gravis and 20% of myasthenia gravis in remission, AChR blocking autoantibodies are the only AChR autoantibodies present in about 1% of myasthenia gravis. AChR modulating autoantibodies (i.e., autoantibodies which cross-link AChRs and cause their removal from muscle membrane surfaces) are present in more than 90% of myasthenia gravis and occasionally are the only AchR autoantibodies detectable in mild, recent onset or ocular-restricted myasthenia gravis. Results for AChR modulating autoantibodies can be transiently false-positive due to curare-like drugs used during general anesthesia. AChR autoantibodies of one or more types are found in at least 80% of ocular myasthenia gravis. Although generally absent in neurological conditions other than myasthenia gravis(and consequently unlikely to cause confusion in neurodiagnosis), false-positive results for AChR autoantibodies occasionally occur in primary biliary cirrhosis, tardive dyskinesia, autoimmune thyroiditis, the elderly, amyotrophic lateral sclerosis patients treated with cobra venom and patients with thymoma in the absence of myasthenia gravis. Approximately 1% of patients with rheumatoid arthritis treated with D-penicillamine develop AChR autoantibodies and myasthenia gravis, both of which disappear when the drug is discontinued. Babies born to ~10% of myasthenia gravis mothers have a transient neonatal form of myasthenia gravis that responds well to anticholinesterase therapy and usually remits within 1 month as maternal IgG disappears. (29 Dec 1997) |
| antibodies | Any of numerous protein molecules produced by the B-cells as a primary immune defense. (16 Dec 1997) |
| antibodies, anticardiolipin | Antiphospholipid antibodies found in association with systemic lupus erythematosus (lupus erythematosus, systemic), antiphospholipid syndrome, and in a variety of other diseases as well as in healthy individuals. The antibodies are detected by solid-phase immunoassay employing the purified phospholipid antigen cardiolipin. (12 Dec 1998) |
| antibodies, anti-idiotypic | Antibodies which react with the individual structural determinants (idiotopes) on the variable region of other antibodies. (12 Dec 1998) |
| antibodies, antineutrophil cytoplasmic | Autoantibodies directed against cytoplasmic constituents of polymorphonuclear leukocytes and/or monocytes. They are used as specific markers for wegener's granulomatosis and other diseases, though their pathophysiological role is not clear. Anca are routinely detected by indirect immunofluorescence with three different patterns: c-anca (cytoplasmic), p-anca (perinuclear), and atypical anca. (12 Dec 1998) |
| antibodies, antinuclear | See: Antinuclear antibodies. (12 Dec 1998) |
| antibodies, antiphospholipid | Autoantibodies directed against phospholipids. These antibodies are characteristically found in patients with systemic lupus erythematosus (lupus erythematosus, systemic), antiphospholipid syndrome, related autoimmune diseases, some non-autoimmune diseases, and also in healthy individuals. (12 Dec 1998) |
| antibodies, archaeal | Immunoglobulins induced by substances elaborated by archaea that have an antigenic activity. (12 Dec 1998) |
Synonyms :
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