| ¿µ¹® | liver cirrhosis | ÇÑ±Û | °£°æÈ(Áõ) |
|---|---|---|---|
| ¼³¸í | Á¤»óÀûÀÎ °£¼¼Æ÷ÀÇ ¸¹Àº ºÎºÐÀÌ ¼Ò½ÇÀÌ µÇ°í ´ë½Å¿¡ ¼¶À¯Á¶Á÷À¸·Î ´ëÄ¡µÇ¾î ÀÖ´Â °£ÀÇ º´Àû »óŸ¦ ¸»ÇÑ´Ù. °£¼¼Æ÷ÀÇ ¸¹Àº ¼Õ»óÀ» °¡Á®¿À´Â ¸ðµç º´¿¡¼ °£°æÈ°¡ ÀϾÙ. ±×·¯³ª ´ëºÎºÐÀÇ °£°æÈÀÇ ¿øÀÎÀº °£¿°°ú ¼ú¿¡ ÀÇÇÑ °£¼Õ»óÀÌ´Ù. °£°æÈÀÇ Áõ»óÀº ¿øÀο¡ µû¶ó¼ ´ÙÀ½°ú °°Àº µÎ °¡Áö·Î ³ª´ ¼ö°¡ ÀÖ´Ù. ù°´Â ¿ì¼± °£ÀÇ ±â´ÉÀÇ Àå¾Ö¿¡ ÀÇÇÑ Áõ»óÀÌ´Ù. °£¼¼Æ÷ÀÇ »ó´ç¼ö°¡ ¼¶À¯Á¶Á÷À¸·Î ´ëüµÇ¾î ÀÖ´Â »óÅÂÀ̹ǷΠ°£ÀÇ ±â´ÉÀÇ Àå¾Ö°¡ »ý±â´Â °ÍÀº ´ç¿¬ÇÏ´Ù. Ȳ´Þ µîÀÌ ´ëÇ¥Àû ¿¹¶ó ÇϰڴÙ. µÎ¹øÂ°´Â ¹®¸Æ¾ÐÇ×Áø(portal hypertension)¿¡ ÀÇÇÑ Áõ»óµéÀÌ´Ù. À§, ÀÛÀºÃ¢ÀÚ³ª ūâÀÚ¿¡¼ ¿µ¾çºÐÀ» Èí¼öÇϱâÀ§ÇÑ ¸ð¼¼Ç÷°üÁ¶Á÷Àº ¸ðµÎ °£À¸·Î ¿¬°áÀÌ µÈ´Ù. Áï ¼Òȱ⿡¼ Èí¼öÇÑ ¿µ¾çºÐÀÌ °¡µæÇÑ ÇÇ´Â ¸ðµÎ °£À¸·Î ¿¬°áµÇ´Âµ¥ À̰ÍÀ» ¹®¸Æ°è(portal system)¶ó°í ÇÑ´Ù. °£°æÈÀÇ °æ¿ì¿¡´Â ¼¶À¯¼ºÁ¶Á÷ÀÌ °£Á¶Á÷À» °ÅÀÇ ´ëÄ¡ÇÔÀ¸·Î Á¤»ó °£¼¼Æ÷³»¿¡¼± ³ÐÀº °ø°£À» Â÷ÁöÇÏ´ø °£³»ÀÇ Ç÷°üµéÀÌ ¼¶À¯Á¶Á÷¿¡ ´¸®°Ô µÈ´Ù. ±×·¯¸é À̰Ͱú ¿¬°áµÈ ¹®¸Æ°èÀÇ ¾Ð·Âµµ ³ô¾ÆÁö°Ô µÈ´Ù. ¹®¸Æ¾ÐÀÇ »ó½ÂÀÌ ÀÖ´Â °æ¿ì¿¡´Â ¹®¸Æ°è¿¡ ¿¬°áÀÌ µÇ¾î ÀÖ´Â ¸ðµç ºÎºÐÀÇ Á¤¸ÆÀÇ ¾Ð·ÂÀÌ ³ô¾ÆÁö°í Á¤¸ÆÀÇ ¼øÈ¯ÀÌ Á¤ÁöµÈ »óŰ¡ µÈ´Ù. Áö¶óÀÇ °æ¿ìµµ ¹®¸Æ°è¿¡ ¿¬°áµÈ Àå±âÀ̹ǷΠ¹®¸Æ¾Ð »ó½Â½Ã¿¡´Â Á¤¸ÆÀÇ ¼øÈ¯ÀÌ ¾ø¾îÁö°í, µ¿¸ÆÀ¸·Î À¯ÀÔÀÌ µÇ´Â Ç÷¾×Àº °è¼Ó µé¾î¿À¹Ç·Î Áö¶óÀÌ Ä¿Áö°Ô µÈ´Ù. ¶Ç ¼ÒȱâÀÇ ¸ð¼¼Ç÷°ü³»¿¡¼ÀÇ ¾Ð·Âµµ ³ô¾ÆÁö°Ô µÇ°í ±×·¯¸é ±× ¾Ð·Â¿¡ ÀÇÇØ¼ ¸¹Àº ¾çÀÇ ¼öºÐÀÌ ¸ð¼¼Ç÷°ü¹ÛÀ¸·Î ºüÁ®³ª¿À°Ô µÈ´Ù. ÀÌ ¼öºÐÀÌ ¸ð¿© º¹¼ö°¡ µÈ´Ù. |
||
| ¿µ¹® | liver function tests | ÇÑ±Û | °£±â´É°Ë»ç |
|---|---|---|---|
| ¼³¸í | Ç÷¾×°Ë»çÁß °¡Àå ¸¹ÀÌ ¾²ÀÌ´Â °Ë»ç¹ýÀ¸·Î ´ÙÀ½ 7°¡Áö¸¦ °Ë»çÇÏ°Ô µÈ´Ù. Ç÷ûÄÝ·¹½ºÅ×·Ñ, ÃѴܹéÁú, ¾ËºÎ¹Î, ºô¸®·çºó, GOT/GPT È¿¼Ò, ¾ËÄ®¸®ÀλêºÐÇØÈ¿¼Ò(alkaline phophatase) µîÀ» °Ë»çÇÏ°Ô µÇ´Â µ¥ °¢ °Ë»çÄ¡¿¡´Â ¸ðµÎ Àǹ̰¡ ÀÖÀ¸¸ç, ÀÌ °Ë»ç Çϳª·Î °£±â´ÉÀÇ Àü¹ÝÀûÀÎ »óÅ¿¡ ´ëÇØ¼ ¾Ë¾Æº¼ ¼ö ÀÖ´Ù. |
||
| ¿µ¹® | liver biopsy | ÇÑ±Û | °£»ý°Ë |
|---|---|---|---|
| ¼³¸í | »ç¶÷ÀÌ »ì¾ÆÀÖ´Â »óÅ¿¡¼ º´Å͸¦ Àß¶ó³»¾î Á÷Á¢ Çö¹Ì°æ µîÀ¸·Î º¸¾Æ Áø´ÜÀ» ³»¸®´Â Áø´Ü¹ýÀÌ´Ù. °£»ý°ËÀº ÁÖ·Î °£¿°À̳ª °£¾ÏÀÇ Áø´ÜÀ̳ª, Èñ±ÍÇÑ À¯Àüº´, ¼±Ãµº´ µîÀÇ È®Áø¿¡ ÀÌ¿ëµÈ´Ù. °£¿°¿¡¼´Â ÇöÀçÀÇ °£¿°ÀÌ ÁøÇ༺ÀÎÁö ȤÀº ºñÁøÇ༺ÀÎÁö ¶Ç´Â ÀÌ¹Ì °£°æÈ»óÅ·Π³Ñ¾î°¬´ÂÁö µîÀÇ ¿©ºÎ¸¦ ¾Ë¾Æº¸°Ô µÈ´Ù. |
||
| ¿µ¹® | infectious disease | ÇÑ±Û | °¨¿°º´ |
|---|---|---|---|
| ¼³¸í | ¹ÙÀÌ·¯½º·ÎºÎÅÍ ±â»ýÃæ Å©±â±îÁöÀÇ »ý¹°À» ¿øÀÎÀ¸·Î ÇÏ´Â º´. ¿øÀÎÀº Á¢ÃËÀü¿°¼ºÀ̸ç, º´¿ø¿¡¼ °¨¿°µÇ´Â °æ¿ìµµ ÀÖ´Ù. °¨¿°À» ¿øÀαտ¡ µû¶ó ºÐ·ùÇÏ¸é ¹ÙÀÌ·¯½º, ¼¼±Õ, Ŭ¶ó¹Ìµð¾Æ, ¸®ÄÏÂ÷, ¹ÌÄÚ¹ÚÅ׸®¿ò, °õÆÎÀÌ, ¿øÃæ, À±Ãæ, ¿ÜºÎ±â»ýÃæ °¨¿°À¸·Î ³ª´ ¼ö ÀÖ´Ù. |
||
| AFP | Alpha(¥á) Feto-Protein [HP 1826, 1858, 1859, 2265] ; Oncofetal Antigens &nbs... |
|---|---|
| CD | cadaver donor; canine distemper; canine dose; carbohydrate dehydratase; carbon dioxide; cardiac dise... |
| HD | Haab-Dimmer [syndrome]; Hajna-Damon [broth]; Hansen disease; hearing distance; heart disease; helix ... |
| MD | Doctor of Medicine [Lat. Medicinae Doctor]; magnesium deficiency; main duct; maintenance dose; major... |
| AD | accident dispensary; acetate dialysis; active disease; acute dermatomyositis; addict, addiction; ade... |
| BRIC | Benign recurrent intrahepatic cholestasis |
|---|---|
| ICP | Intra-hepatic cholestasis of pregnancy |
| PFIC | Progressive Familial Intrahepatic Cholestasis |
| ATSDR | Agency for Toxic Substances and Disease Registry |
| ALD | Alcoholic Liver Disease |
| cholestasis | <gastroenterology> An arrest of the normal flow of bile. This may occur due to a blockage of the bile ducts resulting in an elevation of bilirubin in the bloodstream (jaundice). Origin: Gr. Stasis = stoppage (15 Jan 1998) |
|---|---|
| cholestasis, intrahepatic | Intrahepatic impairment of bile flow. It is usually due to liver cell damage, but may be due to obstruction of intrahepatic bile ducts. It is also called hepatocellular jaundice and medical jaundice. (12 Dec 1998) |
| drug-induced cholestasis | <hepatology> A condition where a drug is interfering with the normal flow of bile from the liver to the gut via the biliary tract. The end result is jaundice. Origin: Gr. Stasis = stoppage (27 Sep 1997) |
| alcoholic liver disease | <gastroenterology> Alcoholic cirrhosis is a condition of irreversible liver disease due to the chronic inflammatory and toxic effects of ethanol on the liver. In cirrhosis, the liver cells are replaced by fibrous scar tissue. Fibrosis leads to the development of portal hypertension. The development of cirrhosis is directly related to the duration and quantity of alcohol consumption. The manifestations of cirrhosis are related to the liver's inability to not adequately remove waste products from the bloodstream and the effects of portal hypertension. (15 Nov 1997) |
| veno-occlusive disease of the liver | Obliterating endophlebitis of small hepatic vein radicles, described in Jamaican children, associated with ingestion of toxic plant substances in bush tea; causes ascites, which may progress to cirrhosis. (05 Mar 2000) |
| chronic active liver disease | Any of several types of hepatitis persisting for more than six months, often progressing to cirrhosis. Synonym: chronic active liver disease. (05 Mar 2000) |
| polycystic liver disease | Gradual cystic dilation of intralobular bile ducts (Meyenburg's complexes) that fail to involute in embryologic development of the liver; frequently associated with bilateral congenital polycystic kidneys and occasionally with cystic involvement of the pancreas, lungs, and other organs. Synonym: polycystic liver disease. (05 Mar 2000) |
| megacolon, toxic | Acute dilatation of the colon associated with amebic or ulcerative colitis. The dilatation may precede perforation of the colon. (12 Dec 1998) |
| goiter, diffuse toxic | Graves' disease, the most common cause of hyperthroidism, too much thyroid hormone. (12 Dec 1998) |
| goiter, toxic multinodular | Condition in which the thyroid gland contains multiple lumps (nodules) that are overactive and produce excess thyroid hormones. This condition is also known as Parry's disease or Plummer's disease. (12 Dec 1998) |
| plants, toxic | Plants or plant parts which are harmful to man or other animals. (12 Dec 1998) |
| hepatitis, toxic | Acute hepatitis caused by true hepatotoxins such as amanita phaloides toxin, carbon tetrachloride, yellow phosphorus, and a variety of drugs. (12 Dec 1998) |
| shock, toxic | See Syndrome, toxic shock. (12 Dec 1998) |
| syndrome, toxic shock | A grave condition occurring predominantly in menstruating women using tampons, toxic shock is characterised by a highly toxic state (with sudden high fever, vomiting, diarrhoea, muscle aching) followed by low blood pressure (hypotension) which can lead to shock (and death). There may be a rash resembling sunburn with peeling of skin. The channing laboratory in boston under dr. Edw. Kass discovered that toxic shock was due to a toxin produced by staph (staphylococcus) aureus bacteria growing under conditions with little or no oxygen. The syndrome occurs rarely in women not using tampons and in men. (12 Dec 1998) |
| drugs toxic to lung | <radiology> Bleomycin, methotrexate, cytoxan, amiodarone (12 Dec 1998) |
Á¦Ç°¸í |
ÆÇ¸Å»ç |
º¸ÇèÄÚµå | ¼ººÐ/ÇÔ·® | ±¸ºÐ/º¸Çè±Þ¿© |
|---|
Á¦Ç°¸í |
ÆÇ¸Å»ç |
º¸ÇèÄÚµå | ¼ººÐ/ÇÔ·® | ±¸ºÐ/º¸Çè±Þ¿© |
|---|