| ¿µ¹® | chronic lymphocytic leukemia | ÇÑ±Û | ¸¸¼º¸²ÇÁ¼º ¹éÇ÷º´ |
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| ¿µ¹® | chronic obstructive pulmonary disease | ÇÑ±Û | ¸¸¼ºÆó¼âÆóº´ |
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| ¿µ¹® | chronic active hepatitis | ÇÑ±Û | ¸¸¼ºÈ°µ¿°£¿° |
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| ¼³¸í | BÇü °£¿°À̳ª ºñAÇü£ºñBÇü °£¿°ÀÇ ¼Ó¹ßÁõÀ¸·Î ³ªÅ¸³ª´Â °£ÀÇ ¸¸¼º¿°ÁõÀÌ´Ù. °°Àº ÇüÅÂÀÇ º´ÀÌ ¼±Ãµ¼º ¶Ç´Â ÈÄõ°¨¸¶±Û·ÎºÒ¸°°áÇÌÁõÀ̳ª ¾î¶² Á¾·ùÀÇ ¾à¹° Åõ¿©¿¡ ¼ö¹ÝÇØ¼ ³ªÅ¸³¯ ¼öµµ ÀÖ´Ù. Ư¡ÀûÀ¸·Î ¹®¸ÆºÎ¿¡ ÇüÁú¼¼Æ÷¿Í Å«Æ÷½Ä¼¼Æ÷ÀÇ Ä§À±, Á¶°¢±«»ç(°£¼Ò¿± ÁÖº¯ºÎ °£¼¼Æ÷ÀÇ ÆÄ±«) ¹× ¼¶À¯Áõ µîÀÇ Á¶Á÷¼Ò°ßÀ» ³ªÅ¸³½´Ù. º´ÀÇ °æ°ú´Â ¸Å¿ì ´Ù¾çÇϸç Àå±â°£ÀÇ ¹«Áõ»ó±â¸¦ º¸ÀÏ ¼öµµ ÀÖ°í ±× »çÀÌ »çÀÌ¿¡ Ȳ´Þ, Àü½Å¼è¾à, ½Ä¿åºÎÁø ¹× ¹ß¿ µîÀÇ Áõ»óÀÌ ³ªÅ¸³ª´Â ¼ö°¡ ÀÖÀ¸¸ç, ¶Ç ¹«¿ù°æÁõ, °üÀý¿°, ÇǺιßÁø, Ç÷°ü¿°, °©»ó»ù¿°, ÄáÆÏ»ç±¸Ã¼¿°, ±Ë¾ç¼º´ëÀå¿°, ½¦±×·»ÁõÈıº µî °£ ÀÌ¿ÜÀÇ Áõ»óÀÌ ³ªÅ¸³ª´Â ¼öµµ ÀÖ°í, °£°æÈÁõ°ú °£±â´É»ó½Ç·Î ÁøÇàµÇ´Â ¼öµµ ÀÖ´Ù. ÀÚ°¡¸é¿ª¸ÞÄ¿´ÏÁòÀÌ °ü¿©µÇ´Â °ÍÀ¸·Î ÃßÃøµÇ°í ÀÖ´Ù. |
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| ¿µ¹® | basal layer of skin | ÇÑ±Û | ±âÀúÃþ |
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| ¼³¸í | Ç¥ÇÇÀÇ °¡Àå ¾Æ·¡ ºÎºÐÀÌ¸ç ¼¼Æ÷ÀÇ ¸ð¾çÀÌ ºñ±³Àû Á÷»ç°¢Çü¿¡ °¡±õ°í ÀÏÁ¤ÇÑ ¹è¿À» ÇÑ´Ù. |
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| ¿µ¹® | radiation | ÇÑ±Û | ¹æ»ç¼± |
|---|---|---|---|
| ¼³¸í | ºûÀÇ ÀüÀÚÀÚ±âÆÄ³ª ȤÀº ÀÔÀÚÆÄ(¥á, ¥â, ¥ã¼±)¸¦ ¸»Çϴµ¥, ¾î¶² ±Ù¿ø¹°Áú¿¡¼ »ý°Ü ¹æÃâµÈ´Ù. À̿½ÖÀ» »ý¼ºÇÏ´Â °í¿¡³ÊÁö ¹æ»ç¼±(X-¼±°ú °¨¸¶¼±)À» ¸»ÇÑ´Ù. ¹æ»ç¼±Çϸé, ÁÖ·Î Àü¸®¹æ»ç¼±À» ÀǹÌÇÑ´Ù. ÀÌ·± ¹æ»ç¼±À» ÀÌ¿ëÇÏ¿©, °¡Àå ±âº»ÀûÀÎ ¹æ»ç¼±ÇÐÀû °Ë»ç¸¦ ÇÒ ¼ö ÀÖÀ¸¸ç, ÀÌ¿Ü Ä¡·á¿¡µµ ÀÌ¿ëÇϰí ÀÖ´Ù. °¢ ¹æ»ç¼±ÀÇ Á¾·ù¿¡ µû¶ó Á¶±Ý¾¿ ÀÎü¿¡ ¹ÌÄ¡´Â ¿µÇâÀÌ ´Ù¸£¸ç, À̸¦ ÀÌ¿ëÇÏ¿© °¢±â ´Ù¸£°Ô Áø´Ü ¹× Ä¡·á¿¡ ÀÌ¿ëÇÑ´Ù. |
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| POEMS | polyneuropathy, organomegaly, endocrinopathy, M protein, skin changes [syndrome] |
|---|---|
| CP | candle power; capillary pressure; cardiac pacing; cardiac performance; cardiopulmonary; caudate puta... |
| CRD | carbohydrate-recognition domain; chronic renal disease; chronic respiratory disease; child restraint... |
| ECG | Electro-Cardio-Graphy(-Gram); ½ÉÀüµµ = EKG 1. Conducting System Structu... |
| ALARA | as low as reasonably achievable [radiation exposure] |
| DUE | DNA unwinding element |
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| DUE | Drug usage evaluation |
| 14C | Changes in |
| ACH | air changes per hour |
| EPC | erosive prepyloric changes |
| radiation, nonionizing | Electromagnetic radiation which does not produce ions in matter through which it passes. Its wavelengths are generally greater than those of far ultraviolet radiation and range through the longest radio waves. (12 Dec 1998) |
|---|---|
| body temperature changes | Any deviation from normal body temperature of the human body, about 98.6 degrees f. Or 37 degrees c. When taken orally. (12 Dec 1998) |
| postmortem changes | Changes that occur in bodies after death. (12 Dec 1998) |
| trophic changes | Abnormalities of the skin, hair, nails, subcutaneous tissues and bone, caused by peripheral nerve lesions. Synonym: neuritic atrophy, neurogenic atrophy, neurotrophic atrophy, trophic changes. (05 Mar 2000) |
| due date | The estimated calendar date when a baby will be born, the date the baby is due to be born. It is also called the estimated date of confinement (EDC). (12 Dec 1998) |
| dystonia, focal, due to blepharospasm | The second most common focal dystonia, the involuntary, forcible closure of the eyelids. The first symptoms may be uncontrollable blinking. Only one eye may be affected initially, but eventually both eyes are usually involved. The spasms may leave the eyelids completely closed causing functional blindness even though the eyes and vision are normal. (12 Dec 1998) |
| dystonia, focal, due to torticollis | Spasmodic torticollis, or torticollis, is the most common of the focal dystonias. In torticollis, the muscles in the neck that control the position of the head are affected, causing the head to twist and turn to one side. In addition, the head may be pulled forward or backward. (12 Dec 1998) |
| thrombotic disease due to protein c deficiency | Protein C is a protein in plasma that enters into the cascade of biochemical events leading to the formation of a clot. Deficiency of protein c results in thrombotic (clotting) disease and excess platelets with recurrent thrombophlebitis (inflammation of the vein that occurs when a clot forms). The clot can break loose and travel through the blood stream (thromboembolism) to the lungs causing a pulmonary embolism, brain causing a stroke (cerebrovascular accident), heart causing an early heart attack, skin causing what in the newborn is called neonatal purpura fulminans, the adrenal gland causing haemorrhage with abdominal pain, abnormally low blood pressure (hypotension), and salt loss. Protein c deficiency is due to possession of one gene (heterozygosity) in chromosome band 2q13-14. The possession of two such genes (homozygosity) is usually lethal. (12 Dec 1998) |
| maternal exposure | Exposure of the female parent, human or animal, to potentially harmful chemical, physical, or biological agents in the environment or to environmental factors that may include ionizing radiation, pathogenic organisms, or toxic chemicals that may affect offspring. It includes pre-conception maternal exposure. (12 Dec 1998) |
| maximum permissible exposure level | The highest level of exposure to a substance, usually noxious, in the environment or during diagnostic and therapeutic procedures, that a body can tolerate without injury. (12 Dec 1998) |
| paternal exposure | Exposure of the male parent, human or animal, to potentially harmful chemical, physical, or biological agents in the environment or to environmental factors that may include ionizing radiation, pathogenic organisms, or toxic chemicals that may affect offspring. (12 Dec 1998) |
| permissible exposure limit | An occupational health standard to safeguard workers against dangerous contaminants in the workplace. (05 Mar 2000) |
| prenatal exposure delayed effects | Delayed effects on offspring of maternal or foetal prenatal exposure to drugs, radiation and other physical agents, manipulation, nutrition, stress, etc. (12 Dec 1998) |
| short-term exposure limit | The maximum concentration of a chemical to which workers may be exposed continuously for up to 15 minutes without danger to health or work efficiency and safety. (05 Mar 2000) |
| dental pulp exposure | The result of pathological changes in the hard tissue of a tooth caused by carious lesions, mechanical factors, or trauma, which render the pulp susceptible to bacterial invasion from the external environment. (12 Dec 1998) |
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