| ICO | idiopathic cyclic oedema; impedance cardiac output |
|---|
| CMO | cystoid macular oedema |
|---|
| Quincke's oedema | A vascular reaction involving the deep dermis or subcutaneous or submucal tissues, representing localised oedema caused by dilatation and increased permeability of the capillaries and characterised by development of giant wheals. (18 Nov 1997) |
|---|
| disease, quincke's | This is angioneurotic oedema (or angioedema), a form of localised swelling of the deeper layers of the skin and fatty tissues beneath the skin. Hereditary angioneurotic oedema (or hereditary angioedema) is a genetic form of angioedema. Persons with it are born lacking an inhibitor protein (called C1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of C1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. (12 Dec 1998) |
|---|---|
| Quincke | Heinrich I., German physician, 1842-1922. See: Quincke's disease, Quincke's oedema, Quincke's pulse, Quincke's puncture, Quincke's sign. (05 Mar 2000) |
| quincke's disease | This is angioneurotic oedema (or angioedema), a form of localised swelling of the deeper layers of the skin and fatty tissues beneath the skin. Hereditary angioneurotic oedema (or hereditary angioedema) is a genetic form of angioedema. Persons with it are born lacking an inhibitor protein (called c1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of c1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. (17 Dec 1998) |
| Quincke's pulse | The capillary pulse as appreciated in the finger and toenails during aortic regurgitation; ebb and flow is seen. Synonym: Quincke's sign. (05 Mar 2000) |
| Quincke's puncture | A diagnostic procedure where a sterile needle is introduced into the lower spine (L2) to collect cerebrospinal fluid for diagnostic purposes. Chemical analysis, cellular analysis and CSF pressure can all be measured with this procedure. This test can aid in the diagnosis of meningitis, subarachnoid haemorrhage and multiple sclerosis. (27 Sep 1997) |
| Quincke's sign | The capillary pulse as appreciated in the finger and toenails during aortic regurgitation; ebb and flow is seen. Synonym: Quincke's sign. (05 Mar 2000) |
| ambulant oedema | Oedema forming during periods of walking with the legs dependent. (05 Mar 2000) |
| angioneurotic oedema | <neurology> An acute or recurring attack of transient oedema suddenly appearing in areas of the skin or mucous membranes and occasionally of the viscera, often associated with dermatographism, urticaria, erythema, and purpura. It can produce dramatic swelling of the subcutaneous tissues (welts beneath the skin) that typically appears around the eyes and lips. Welts may also involve the hands, feet and throat (compromise the airway). This condition is associated with allergies (for example foods, pollen), but may also be a side effect of some medications (for example ACE inhibitors, salicylates). Emotional stress, exposure to cold, water, sunlight, heat and insect bites all have been know to cause angioneurotic oedema. Treatment is with adrenaline, antihistamines, cimetidine, and/or corticosteroids. (12 Dec 1998) |
| angioneurotic oedema, hereditary | A genetic form of angioedema. (Angioedema is also referred to as Quinke's disease.) Persons with it are born lacking an inhibitor protein (called C1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of C1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioedema. (12 Dec 1998) |
| Berlin's oedema | Retinal oedema after blunt trauma to the globe. (05 Mar 2000) |
| blue oedema | The swelling and cyanosis of an extremity in hysterical paralysis. (05 Mar 2000) |
| brain oedema | Brain swelling due to increased volume of the extravascular compartment from the uptake of water in the neuropile and white matter. See: brain swelling. Synonym: brain oedema. (05 Mar 2000) |
| brawny oedema | Swelling of subcutaneous tissues which cannot be indented by compression easily. Usually due to metabolic abnormality, such as increased glycosaminoglycan content, like that which occurs in Graves' disease (pretibial myxoedema) or in early phase of scleroderma. Synonym: brawny oedema. (05 Mar 2000) |
| brown oedema | Oedema of the lungs associated with chronic passive congestion. (05 Mar 2000) |
| bullous oedema | A reddened, swollen appearance of the ureteral orifice in the bladder wall, frequently observed with distal ureteral calculi or in tuberculosis of the ureter. (05 Mar 2000) |
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|