| CJA | Creutzfeldt-Jakob agent |
|---|---|
| CJD | Creutzfeldt-Jakob disease |
| CJS | Creutzfeldt-Jakob syndrome |
| JC | Jakob-Creutzfeldt; joint contracture |
| JCD | Jakob-Creutzfeldt disease |
| EMP | Embden-Meyerhof-Parnas |
|---|---|
| CJD | CREUTZFELDT-JAKOB disease |
| nvCJD | New variant Creutzfeldt-Jakob disease |
| vCJD | Variant Creutzfeldt-Jakob Disease |
| Parnas, Jakob Karol | <person> Polish physiologic chemist, 1884-1955. See: Embden-Meyerhof-Parnas pathway. (05 Mar 2000) |
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| Embden-Meyerhof-Parnas pathway | A pathway that degrades glucose to pyruvate, the six-carbon stage converts glucose to fructose-1,6-bisphosphate, and the three-carbon stage produces ATP while changing glyceraldehyde-3-phosphate to pyruvate. Compare: Entner-Doudoroff pathway. (09 Oct 1997) |
|---|---|
| creutzfeldt-jakob disease | <infectious disease> A very rare form of encephalopathy thought to be caused by a virus (slow-virus), termed a prion. There is little known about the mode of transmission. Human to human transmission has occurred through the use of contaminated brain electrodes and transplantation of infected tissues. The agent can be recovered in the CSF of infected individuals. Standard disinfectants such as formalin, heat, exposure to ultraviolet light or X-rays is ineffective to inactivate the virus. Autoclaving to at least 132 degrees C. Or immersion in 4% sodium hydroxide or 10% sodium hypochlorite solution for 1 hour is recommended for sterilisation. The disease occurs primarily in adults, with peak incidence in the late 50's. Infection results in dementia, myoclonus, ataxia and other neurologic symptoms. The disease progresses rapidly to coma and death after a 3 to 12 month illness. There is no known cure. (27 Sep 1997) |
| creutzfeldt-jakob syndrome | <syndrome> A rare, usually fatal, transmissible encephalopathy occurring in middle life, in which there is partial degeneration of the pyramidal and extrapyramidal systems accompanied by progressive dementia and sometimes muscle wasting, tremor, athetosis, and spastic dysarthria. A familial form has been shown to exhibit autosomal dominant inheritance. The more common sporadic form is probably caused by mutations that produce abnormal prion proteins (prions). A variant of cjd has been reported in patients younger than 30 years old, probably caused by exposure to beef harboring the bovine spongiform encephalopathy agent. (12 Dec 1998) |
| Stilling, Jakob | <person> German ophthalmologist, 1842-1915. See: Stilling colour tables. (05 Mar 2000) |
| syndrome, creutzfeldt-jakob | Better known as creutzfeldt-jakob disease (cjd). A dementing disease of the brain. It is believed due to an unconventional (not a bacteria or virus), transmissible agent called a prion. Symptoms of cjd include forgetfulness, nervousness, jerky trembling hand movements, unsteady gait, muscle spasms, chronic dementia, balance disorder, and loss of facial expression. Cjd is classified as a spongiform encephalopathy. most cases occur randomly (sporadically), but inherited forms exist. There is neither treatment nor cure for cjd. Other names for cjd include jakob-creutzfeldt disease and spastic pseuodoparalysis. (12 Dec 1998) |
| disease, creutzfeldt-jakob | A dementing disease of the brain. It is believed due to an unconventional (not a bacteria or virus), transmissible agent called a prion. Symptoms of CJD include forgetfulness, nervousness, jerky trembling hand movements, unsteady gait, muscle spasms, chronic dementia, balance disorder, and loss of facial expression. CJD is classified as a spongiform encephalopathy. most cases occur randomly (sporadically), but inherited forms exist. There is neither treatment nor cure for CJD. Other names for CJD include Creutzfeldt-Jakob syndrome, Jakob-Creutzfeldt disease, and spastic pseuodoparalysis. (12 Dec 1998) |
| disease, jakob-creutzfeldt | A transmissible degenerative brain disorder technically termed spongiform encephalopathy. Eating mad cow meat or squirrel brain can lead to Jakob-Creuzfeldt-like disease. Better known as Creutzfeldt-Jakob disease (CJD). A dementing disease of the brain, believed due to an unconventional, transmissible agent (a prion). Symptoms of CJD include forgetfulness, nervousness, jerky trembling hand movements, unsteady gait, muscle spasms, chronic dementia, balance disorder, and loss of facial expression. CJD is classified as a spongiform encephalopathy. most cases occur randomly (sporadically), but inherited forms exist. There is neither treatment nor cure for CJD. Other names for CJD include Creutzfeldt-Jakob syndrome, Jakob's disease, and spastic pseuodoparalysis. (12 Dec 1998) |
| disease, jakob's | Better known as Creutzfeldt-Jakob disease (CJD), a dementing disease of the brain. It is believed due to an unconventional, transmissible agent (a prion). Symptoms of CJD include forgetfulness, nervousness, jerky trembling hand movements, unsteady gait, muscle spasms, chronic dementia, balance disorder, and loss of facial expression. CJD is classified as a spongiform encephalopathy. most cases occur randomly (sporadically), but inherited forms exist. There is neither treatment nor cure for CJD. Other names for CJD include Creutzfeldt-Jakob syndrome, Jakob-Creutzfeldt disease, and spastic pseuodoparalysis. (12 Dec 1998) |
| Jakob, Alfons | <person> German neuropsychiatrist, 1884-1931. See: Creutzfeldt-Jakob disease, Jakob-Creutzfeldt disease. (05 Mar 2000) |
| Jakob-Creutzfeldt disease | <infectious disease> A very rare form of encephalopathy thought to be caused by a virus (slow-virus), termed a prion. There is little known about the mode of transmission. Human to human transmission has occurred through the use of contaminated brain electrodes and transplantation of infected tissues. The agent can be recovered in the CSF of infected individuals. Standard disinfectants such as formalin, heat, exposure to ultraviolet light or X-rays is ineffective to inactivate the virus. Autoclaving to at least 132 degrees C. Or immersion in 4% sodium hydroxide or 10% sodium hypochlorite solution for 1 hour is recommended for sterilisation. The disease occurs primarily in adults, with peak incidence in the late 50's. Infection results in dementia, myoclonus, ataxia and other neurologic symptoms. The disease progresses rapidly to coma and death after a 3 to 12 month illness. There is no known cure. (27 Sep 1997) |
| jakob's disease | Better known as Creutzfeldt-Jakob disease (cjd), a dementing disease of the brain. It is believed due to an unconventional, transmissible agent (a prion). Symptoms of cjd include forgetfulness, nervousness, jerky trembling hand movements, unsteady gait, muscle spasms, chronic dementia, balance disorder, and loss of facial expression. Cjd is classified as a spongiform encephalopathy. most cases occur randomly (sporadically), but inherited forms exist. There is neither treatment nor cure for cjd. Other names for cjd include creutzfeldt-jakob syndrome, jakob-creutzfeldt disease, and spastic pseuodoparalysis. (12 Dec 1998) |
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
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