| IMDS | Idiopathic Myelo-Dysplastic Syndrome = Preleukemia = Refractory Anemi... |
|---|---|
| MDS | Myelo-Dysplastic Syndrome = Refractory (Dysmyelopoietic) Anemia = (Id... |
| DMN | dimethylnitrosamine; dorsal motor nucleus; dysplastic melanocytic nevus |
| DNS | deviated nasal septum; diaphragmatic nerve stimulation; did not show [for appointment]; Doctor of Nu... |
| MCDK | multicystic dysplastic kidney |
| DNS | Dysplastic Naevus Syndrome |
|---|---|
| DMN | Dysplastic melanocytic nevi |
| DN | Dysplastic naevi |
| MCDK | Multicystic dysplastic kidney |
| DRO | Differential Reinforcement of Other Behaviour |
| other-directed | Pertaining to a person readily influenced by the attitudes of others. (05 Mar 2000) |
|---|---|
| transferases (other substituted phosphate groups) | <enzyme> A class of enzymes that transfers substituted phosphate groups. Registry number: EC 2.7.8 (12 Dec 1998) |
| multicystic dysplastic kidney disease | <radiology> most severe form of renal dysplasia, most common cause of palpaple abdominal mass in infants, unilateral (almost always; if bilateral, death occurs in utero), ureter absent or atretic, not associated with other cysts or with periportal fibrosis, Potter type II (12 Dec 1998) |
| congenital dysplastic angiectasia | <syndrome> A congenital malformation syndrome characterised by the triad of asymmetric limb hypertrophy, haemangiomata, and nevi. Asymmetric limb hypertrophy is enlargement of one limb and not the corresponding limb on the other side, the enlarged limb being 3 times more likely to be a leg than an arm in ktw; and the limb enlargement is of bone as well as soft tissue. The haemangiomas, abnormal nests of blood vessels that proliferate inappropriately and excessively, cover a remarkable range from small innocuous capillary haemangiomas ( strawberry marks ) to huge cavernous haemangiomas. The nevi are pigmented moles on the skin; in ktw there are often also dark linear streaks on the skin, streaks due to too much pigment. There can be other abnormalities but the triad is the consistent clinical centrepiece of the disease. most persons with ktw have an enlarged leg and do relatively well without treatment or, for example, with only compression from an elastic stocking. Skin ulcers and other skin problems can occur over the swollen leg. Usually, the treatment is conservative. Surgery is almost never needed. The only possible exceptions are the very rare situations in which the leg reaches gigantic proportions or secondary clotting difficulties arise (due to trapping and destruction of blood platelets in a huge haemangioma). Then, amputation may become necessary. The cause of ktw syndrome is unknown. (12 Dec 1998) |
| congenital dysplastic angiomatosis | Autosomal dominant angiomatosis in which there is dysplasia of the underlying tissues, sometimes with overgrowth of bone (Klippel-Trenaunay-Weber syndrome), or encephalotrigeminal angiomatosis (Sturge-Weber syndrome) in which there is an angioma in the distribution of one or more branches of the trigeminal nerve, with vascular anomalies and calcification of the cerebral cortex. (05 Mar 2000) |
| dysplastic | Pertaining to or marked by dysplasia. (05 Mar 2000) |
| dysplastic nevi | Atypical moles; moles whose appearance is different from that of common moles. Dysplastic nevi are generally larger than ordinary moles and have irregular borders. Their colour often is not uniform; they usually are flat, but parts may be raised above the skin surface. (12 Dec 1998) |
| dysplastic nevus | <syndrome> Clinically atypical nevi (usually exceeding 5 mm in diameter and having variable pigmentation and ill defined borders) with an increased risk for development of non-familial cutaneous malignant melanoma. Biopsies show melanocytic dysplasia. Nevi are clinically and histologically identical to the precursor lesions for melanoma in the b-k mole syndrome. (12 Dec 1998) |
| dysplastic nevus syndrome | <syndrome> Clinically atypical nevi (usually exceeding 5 mm in diameter and having variable pigmentation and ill defined borders) with an increased risk for development of non-familial cutaneous malignant melanoma. Biopsies show melanocytic dysplasia. Nevi are clinically and histologically identical to the precursor lesions for melanoma in the b-k mole syndrome. (12 Dec 1998) |
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