| ¿µ¹® | mitral stenosis | ÇÑ±Û | ½Â¸ðÆÇ¸· ÇùÂøÁõ |
|---|---|---|---|
| ¼³¸í | ½Â¸ðÆÇ¸·(Á½ɹæ°ú Á½ɽǻçÀÌ¿¡ Á¸ÀçÇÏ´Â ÆÇ¸·)ÀÌ Á¼¾ÆÁ® ÀÖ´Â º´ÀûÀÎ »óŸ¦ ¸»ÇÔ. Á¤»óÀûÀ¸·Î ½Â¸ðÆÇ»çÀÌÀÇ °ø°£¸éÀûÀº 4~6cmÁ¤µµÀ̸ç, ÀÌ ¸éÀûÀÌ 2.5cmÀÌÇϰ¡ µÇ¸é Áõ»óÀÌ ³ªÅ¸³´Ù. ±×¸®°í 1~2cmÀÌÇϰ¡ µÇ¸é ¼ö¼úÀ» °í·ÁÇØ¾ß ÇÑ´Ù. ¿îµ¿¼º È£Èí°ï¶õÀÌ ÁÖÁõ»óÀ̸ç, ½ÉÀåûÁø»ó ½ÉÀâÀ½ÀÌ µé¸°´Ù. Áõ»ó°ú ÇùÂøÀÇ Á¤µµ¿¡ µû¶ó ´Ù¸£³ª, ´ë°³ ¼ö¼úÀ» °í·ÁÇØ¾ß Çϸç, ¼ö¼úÀº ÆÇ¸·´ëÄ¡¼ú, ÆÇ¸·¼ºÇü¼ú µîÀÌ ÀÖ°í, ÆÇ¸·´ëÄ¡¼ú¿¡µµ, Á¶Á÷ÆÇ¸·À» ÀÌ¿ëÇÏ´Â ¹æ¹ý°ú ±â°èÆÇ¸·À» ÀÌ¿ëÇÏ´Â ¹æ¹ý 2°¡Áö·Î ³ª´ ¼ö ÀÖ´Ù. |
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| ¿µ¹® | Dilatation and Curettage(D & C) | ÇÑ±Û | Àڱñܾ¼ú, ÀڱøñÈ®Àå |
|---|---|---|---|
| ¼³¸í | ÀÚ±ÃÀ̶õ žư¡ ¼öÅÂµÇ¾î¼ ºÐ¸¸Àü±îÁö ¹ßÀ°ÇÏ°í ¼ºÀåÇÏ´Â °ø°£ÀÌ´Ù. Àڱüӿ¡ º´º¯ÀÌ ÀÖ¾î ÀÓ½ÅÀÌ °è¼ÓµÉ ¼ö ¾ø°Å³ª ¾Æ´Ï¸é ´Ù¸¥ ÀÌÀ¯·Î ÀӽŵǾî Àִ žƸ¦ Á¦°ÅÇϰíÀÚ ÇÒ °æ¿ì¿¡ »ç¿ëµÇ´Â ¹æ¹ýÀÌ´Ù. ¿©±â¼ ±Ü¾î³»±â À§ÇÏ¿©´Â ¿ì¼± ÀÚ±ÃÀÇ ÀÔ±¸¿¡ ÇØ´çÇÏ´Â ÀڱøñÀ» È®Àå½ÃÄÑ¾ß ÇÑ´Ù. ¿©±â¿¡´Â ±Þ¼ÓÈ÷ È®ÀåÀ» ½ÃµµÇÏ´Â ¹ý°ú ¼¼È÷ È®ÀåÀ» ½ÃµµÇÏ´Â 2°¡Áö ¹æ¹ýÀÌ ÀÖ´Ù. ÀڱøñÀ» ±Þ¼ÓÈ÷ È®ÀåÇÒ ¶§´Â Çì°¡¸£ ¸ñ°üÈ®Àå±â(Hegar's dilatator)¸¦ »ç¿ëÇÑ´Ù. À̰ÍÀº ÀÛÀº ±Ý¼Ó¸·´ë·Î ÀÛÀº Å©±âºÎÅÍ Å« Å©±â±îÁö ´Ù¾çÇÑ Å©±â°¡ ÀÖ¾î¼ ¿ì¼± ÀÛÀº ¸·´ë·Î ½ÃÀÛÇÏ¿© Á¡Á¡ Å« Å©±âÀÇ ¸·´ë¸¦ Àڱøñ¿¡ ³Ö¾î¼ ÀڱøñÀ» È®Àå½ÃŲ´Ù. ¼¼È÷ È®Àå½Ãų ¶§´Â Laminaria tent¸¦ ¸ñ°ü¿¡ »ðÀÔÇÏ´Â ¹æ¹ýÀ» »ç¿ëÇÑ´Ù. Laminaria tent¶õ ÇØÃÊ·Î ¸¸µç ÀÛÀº ¸·´ë·Î ¼öºÐÀ» Èí¼öÇϸé Á¡Á¡ ´Ã¾î³ª´Â ¼ºÁúÀÌ ÀÖ´Ù. À̰ÍÀ» ÀÚ±ÃÀÇ ¸ñ¿¡ ³ÖÀ¸¸é À̰ÍÀÌ ¼öºÐÀ» Èí¼öÇÏ¿© ´Ã¾î³ª¹Ç·Î õõÈ÷ ÀÚ±ÃÀÇ ¸ñÀÌ ´Ã¾î³´Ù. ÀڱøñÀÌ ÃæºÐÈ÷ ´Ã¾î³ª¸é ±× ¼ÓÀ¸·Î ³¡ÀÌ ¼ù°¡¶ôó·³ »ý±ä ±â±¸¸¦ ³Ö¾î¼ ÀڱüÓÀÇ º´º¯À̳ª ÀÓ½ÅµÈ Å¾Ƹ¦ ±Ü¾î³»´Âµ¥ ¿©±â¿¡ »ç¿ëµÇ´Â ¼ù°¡¶ôó·³ »ý±ä ±â±¸¸¦ Å¥·¿À̶ó°í ÇÑ´Ù. Ãʱâ ÀÓ½ÅÁßÀý Áï À¯»ê°ú °°Àº ÀӽŰú °ü·ÃµÈ °æ¿ì»Ó¸¸ ¾Æ´Ï¶ó, ºñÀӽŠÀÚ±ÃÀÇ Àڱ󻸷Á¶Á÷ÀÇ Ã¤Ãë ¹× Á¦°Å¸¦ À§Çؼµµ ÇàÇØÁö´Â ¼ö±âÀÌ´Ù. ÀÌ´Â ¿øÄ¢ÀûÀ¸·Î ¸¶ÃëÇÏ¿¡ ½Ç½ÃµÇ´Â °ÍÀ¸·Î Àڱøñ°üÀ» È®ÀåÇÏ°í ±â±¸·Î Àڱà ³»¿ë¹°À» Á¦°ÅÇϰí Å¥·¿À¸·Î Àڱ󻺮À» ±ú²ýÀÌ ÇÑ´Ù. ÀÚ±Ãõ°øÀ̳ª ÀڱøñÀÇ ÆÄ¿ µîÀÇ À§ÇèÀÌ µû¸£¸ç, ¼ö¼úÈÄ °¨¿° ¶Ç´Â ÃâÇ÷ µî¿¡ ´ëÇÑ ÁÖÀǰ¡ ÇÊ¿äÇÏ´Ù. |
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| ¿µ¹® | occlusion | ÇÑ±Û | Æó»ö |
|---|---|---|---|
| ¼³¸í | Ç÷°üÀ̳ª ȤÀº ³»°À» ÀÌ·ç´Â °üÀÌ ¸·È÷´Â °æ¿ì¸¦ Æó»öÀ̶ó°í ÇÑ´Ù. ÀÌÁß Ç÷°ü¿¡ ÀÖ¾î¼ Æó»öÀÌ ¹ß»ýÇϸé, ±× Ç÷°ü¿¡ ÀÇÇØ Ç÷¾×À» °ø±Þ¹Þ´Â ÀÌÇϺÎÀ§¿¡¼ ÇãÇ÷(Ç÷¾×ÀÌ °ø±ÞµÇÁö ¾Ê¾Æ ±× Á¶Á÷ÀÌ Á×±â Á÷ÀüÀÇ ´Ü°è. ÀÌ ºÎÀ§´Â ´ë»ç°¡ °¨¼ÒÇϰí, ¿Âµµ°¡ ¶³¾îÁö¸ç, ¿À·¡ ¹æÄ¡Çصθé Á×°Ô µÈ´Ù)À̳ª, ȤÀº ±«»ç(¼¼Æ÷°¡ Á×´Â ´Ü°è. ½ÉÀå¿¡¼´Â °æ»öÀ̶ó°í ºÎ¸§)¿¡ ºüÁö°Ô µÈ´Ù. À̶§ Æó»öÀ» ÀÏÀ¸Å°´Â °æ¿ì´Â ´ÙÀ½ 2°¡Áö °æ¿ì°¡ ÀÖ´Ù. ¿©±â¼ Ç÷ÀüÁõÀ̶ó´Â ¸»Àº Çǰ¡ ¸ð¿©¼ À̸¥¹Ù ¡°ÇǶ±¡±À» Çü¼ºÇÑ »óŸ¦ ¸»ÇÑ´Ù. ½±°Ô ¸»Çϸé, ÇǺο¡¼ »óó°¡ »ý±â¸é, »óóÁÖÀ§·Î ÇǶ±ÀÌ »ý°Ü ´õÀÌ»ó Çǰ¡ È帣Áö ¾Êµµ·Ï Çϴµ¥, ÀÌ·± Çö»óÀÌ Çǰ¡ ±»¾î¼´Â ¾ÈµÇ´Â Ç÷°ü³»¿¡¼ ¹ß»ýÇÏ¿©, Ç÷°üÀ» ¸·´Â °æ¿ì¸¦ ¸»ÇÑ´Ù. ÀÌ¿¡ ºñÇØ »öÀüÁõÀ̶ó´Â ¸»Àº ´Ù¸¥ °÷¿¡¼ »ý±ä ¡°Ç÷Àü¡±À̳ª ȤÀº ¡°Áö¹æ¡± ¡°°ø±â¡±µîÀÌ Ç÷¾×³»·Î µé¾î¿Í Ç÷¾×À» ¶°µ¹¾Æ ´Ù´Ï´Ù°¡ Å©±â°¡ ÀÛÀº Ç÷°üÀ» ¸·¾Æ Æó»öÀ» ÀÏÀ¸Å°´Â °æ¿ì¸¦ ¸»ÇÑ´Ù. |
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| ¿µ¹® | artery | ÇÑ±Û | µ¿¸Æ |
|---|---|---|---|
| ¼³¸í | ½ÉÀå¿¡¼ ³ª¿À´Â ÇǸ¦ ¿î¹ÝÇÏ´Â Ç÷°üÀ» ¸»ÇÑ´Ù. |
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| JVP | [POMD P 49 - 52] 1) Jugular Vein Pressure 2) Jugular Venous Pulse ... |
|---|---|
| ECG | Electro-Cardio-Graphy(-Gram); ½ÉÀüµµ = EKG 1. Conducting System Structu... |
| STANDOUT | soft thresholding and depth cueing of unspecified techniques |
| URD | unspecified respiratory disease; upper respiratory disease |
| AS | acetylstrophanthidin; acidified serum; acoustic schwannoma; acoustic stimulation; active sarcoidosis... |
| RAS | Renal Artery Stenosis |
|---|---|
| TRAS | Transplant renal artery stenosis |
| BCO | Bilateral carotid artery occlusion |
| CRAO | Central retinal artery occlusion |
| CAO | Coronary artery occlusion |
| renal artery stenosis | <cardiology, nephrology> A narrowing of the renal artery or one of its main branches accounts for 2 to 5% of cases of hypertension. (27 Sep 1997) |
|---|---|
| central retinal artery occlusion | <ophthalmology> The sudden blockage of the retinal artery with a blood clot that commonly leads to a painless but irreversible blindness in that eye. (12 Jan 1998) |
| retinal artery occlusion | Occlusion or closure of the central retinal artery causing sudden, usually nearly complete, loss of vision in one eye. Occlusion of the branch retinal artery causes sudden visual loss in only a portion of the visual field. (12 Dec 1998) |
| mesenteric artery occlusion | Obstruction of arterial flow in the mesenteric circulation by an embolus or thrombus; usually refers to occlusion of the superior mesenteric artery, although atherosclerotic narrowing may involve all three major splanchnic branches (coeliac, superior, and inferior mesenteric). (05 Mar 2000) |
| aortic stenosis | Progressive narrowing of the aortic valve resulting in the obstructed passage of blood from the left ventricle into the aorta. Causes for aortic stenosis include rheumatic fever, congenital and idiopathic sclerosis. Chronic stenosis can lead to left ventricular enlargement and congestive heart failure. (27 Sep 1997) |
| aortic valve stenosis | Narrowing of the orifice of the aortic valve or of the supravalular or subvalvular regions. (12 Dec 1998) |
| aqueductal stenosis | <radiology> most common cause of congenital hydrocephalus (43%), aqueduct develops about the 6th week of gestation, M:F = 2:1, other congenital anomalies (16%): thumb deformities, prognosis: 11-30% mortality aetiology: infectious (50%): toxoplasmosis, CMV, syphillis, mumps, influenza, developmental: forking, narrowing, transverse septum (X-linked recessive), neoplastic (extremely rare): glioma, pinealoma, meningioma (12 Dec 1998) |
| buttonhole stenosis | Extreme narrowing, usually of the mitral valve. (05 Mar 2000) |
| calcific nodular aortic stenosis | Most common type of aortic stenosis, occurring usually in elderly men, in which the cusps contain calcified fibrous nodules on both surfaces; the causes include rheumatic fever, atherosclerosis, age-related degeneration, and congenitally bicuspid aortic valve. (05 Mar 2000) |
| valvular aortic stenosis | <radiology> Secondary to fusion of commisures between cusps types: bicuspid/unicuspid (95%): in 1-2% of population; M>F; commonly associated with coarctation, tricuspid (5%), dysplastic thickened aortic cusps in infants with crtical aortic stenosis: may stimulate neonatal sepsis, associated with L-R shunts (atrial septal defect, VSD), marked CMG (thickened wall of LV), pulmonary venous hypertension, congestive heart failure child/adult: LV configuration with normal heart size, postenotic dilatation, calcified valve (60% of patients greater than24 y.o.) see: aortic stenosis (12 Dec 1998) |
| carotid stenosis | The narrowing or stricture of a carotid artery that may lead to transient ischemic attacks (see cerebral ischemia, transient) and stroke (see cerebrovascular disorders). (12 Dec 1998) |
| mitral stenosis | <cardiology> A congenital or acquired heart valve abnormality that describes the narrowing and ineffective opening of the mitral valve. (13 Nov 1997) |
| mitral valve stenosis | A rheumatic disease causing diffuse thickening of the mitral valve leaflets by fibrous tissue or calcific deposits. (harrisons' principles of internal medicine, 13th ed, p1052) (12 Dec 1998) |
| congenital pyloric stenosis | <radiology> Not seen until 3 weeks, projectile vomiting, palpable olive in RUQ/epigastrium (12 Dec 1998) |
| muscular subaortic stenosis | <cardiology> A congenital heart disease that results in abnormal thickening of the ventricular septum and left ventricular wall. Enlargement of the ventricular septum can result in ventricular outflow obstruction (subaortic stenosis) and eventual cardiomyopathy. (27 Sep 1997) |
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