| ¿µ¹® | hyperthermia, fever | ÇÑ±Û | °í¿ |
|---|---|---|---|
| ¼³¸í | ü¿ÂÁ¶Àý ÁßÃß°¡ ÀÌ»óÀ» ÃÊ·¡ÇØ Ã¼¿ÂÀÌ °è¼Ó »ó½ÂÇÏ¿© Áö¼ÓµÇ°í ÀÖ´Â »óŸ¦ ¸»ÇÑ´Ù. ¿øÀÎÀº ´Ù¾çÇϸç ü¿ÂÁ¶Àý ÁßÃßÀÇ Àå¾Ö(³úÁúȯ), Á¶Á÷Àå¾Ö¿¡ ÀÇÇÑ °ÍÀÌ ÀÖ´Ù. ü¿ÂÀº ÀϹÝÀûÀ¸·Î °Üµå¶ûÀÌ, ÀÔ¾È, °ðâÀÚ¿¡¼ Àç°í, ½Ã°£Àº 10ºÐÀ» ÇÊ¿ä·Î ÇÑ´Ù. Åë»ó 37¡É ÀÌÇÏ(°Üµå¶ûÀÌ)·Î, ÀÔ¾È ¿Âµµ´Â À̺¸´Ù 0.1~0.2¡É ³ôÀ¸¸ç, °ðâÀڿµµ´Â 0.2~0.5¡É ³ô´Ù. °íü¿ÂÁõ¿¡¼µµ µ¿°á°ú À¯»çÇÑ ¼¼Æ÷ º¯È°¡ ³ªÅ¸³´Ù. Áï Ç÷°ü³»ÇǼ¼Æ÷¿¡ ¼Õ»óÀÌ »ý±â°í Ç÷°üÅõ°ú°¡ Áõ°¡µÇ¾î ºÎÁ¾, ¼öÆ÷ µîÀÌ Çü¼ºµÈ´Ù. |
||
| ¿µ¹® | dengue fever | ÇÑ±Û | µ±â |
|---|---|---|---|
| ¼³¸í | ÁÖ·Î ¿´ëÁö¹æ¿¡¼ ¹ß»ýÇÏ´Â ¹ÙÀÌ·¯½º¼º ¿º´. 4~8ÀÏÀÇ Àẹ±â¸¦ °ÅÃÄ ±Þ°ÝÇÏ°Ô ¹ß¿Çϰí, °á¸·ÃæÇ÷-°üÀýÅë-±ÙÀ°Åë-¹éÇ÷±¸°¨¼Ò µîÀÇ Áõ»óÀ» º¸ÀδÙ. ´ëºÎºÐ 7~10ÀÏ ÈÄ¿¡´Â ȸº¹µÇ¸ç, »ç¸ÁÇÏ´Â ¿¹´Â µå¹°´Ù. ¹Ì±¹ÀÇ ¼¼±ÕÇÐÀÚÀÎ C.F. Å©·¹À̱״ 1907³â Çʸ®ÇÉ¿¡¼ µ±â¿Àº ¸ð±â¿¡ ÀÇÇØ °¨¿°µÈ´Ù´Â °ÍÀ» È®ÀÎ-º¸°íÇß´Ù. Á¦2Â÷ ¼¼°è´ëÀü Á÷ÈÄ ¹Ì±¹ÀÇ ¼¼±ÕÇÐÀÚ A. B. ¼¼À̺óÀº ±×ÀÇ ¿¬±¸¿¡ ÀÇÇØ µ±â¿ ¹ÙÀÌ·¯½º¿¡´Â ¸é¿ªÇÐÀûÀ¸·Î ³×°¡Áö ÇüÀÌ ÀÖ´Ù°í ¹àÇû´Ù. µ±â¿ ¹ÙÀÌ·¯½º¸¦ ¸Å°³ÇÏ´Â ¸ð±â¿¡´Â ÀÌÁýÆ®½£¸ð±â(Aedes aegypti)¿Í ÈòÁÙ½£¸ð±â(Aedes albopictus)°¡ ¾Ë·ÁÁ® ÀÖ´Ù. ÀÌ º´¿¡ °É¸®°í ³ª¸é ¸é¿ªÀÌ »ý±âÁö¸¸ Áö¼Ó±â°£Àº ºñ±³Àû ª´Ù. |
||
| ¿µ¹® | typhus fever | ÇÑ±Û | ¹ßÁøÆ¼Çª½º |
|---|---|---|---|
| ¼³¸í | °í¿°ú ¹ßÁøÀÌ ÁÖÁõ¼¼ÀÎ ¿¼º-±Þ¼ºÀÇ ¹ýÁ¤Àü¿°º´. ¼¼°èº¸°Ç±â±¸(WHO)ÀÇ º¸°Ç±ÔÄ¢¿¡ ÀÇÇÑ ±¹Á¦ °¨½Ã Àü¿°º´ÀÇ ÇϳªÀ̱⵵ ÇÏ´Ù. ¶ÇÇÑ, ÀÌ º´ÀÇ À¯ÇàÀº ÀüÀï°ú °ü°è°¡ ±í¾î ÀüÀïÆ¼Çª½º ¶Ç´Â ±â±Ù¿-±³µµ¼Ò¿ µîÀÇ º°¸íÀÌ ÀÖ´Ù. ±×°ÍÀº ÀÌ º´ÀÇ ¸Å°³°ïÃæÀÎ ¡®¿ÊÀÌ¡¯°¡ ÀÇ·ù³ª ¸öÀÌ ´õ·¯¿ï ¶§ ¹ß»ýÇϱ⠽¬¿ì¹Ç·Î ±º´ë³ª ±³µµ¼Ò, ÀüÀïÅÍ µî ȯ°æÀÌ ³ª»Û °÷¿¡¼ Å©°Ô À¯ÇàÇÏ°Ô µÇ¾ú±â ¶§¹®À̾ú´Ù. Á¦1Â÷ ¼¼°è´ëÀü ´ç½Ã ·¯½Ã¾Æ¿¡¼ ¹ß»ýÇÑ È¯ÀÚÀÇ ¼ö´Â 2,500¸¸ ¸íÀ̳ª µÇ¾ú°í, ¿µ±¹°ú ±âŸ À¯·´¿¡¼µµ ÀüÀï-±â±Ù ¶§¹®¿¡ ÀÌ º´ÀÌ ´ëÀ¯ÇàÇß´ø ±â·ÏÀÌ ÀÖÀ¸¸ç, ÀϺ»¿¡¼µµ Á¦2Â÷ ¼¼°è´ëÀü Á÷ÈÄ¿¡ À¯Çà Çß¾ú´Ù. ¿À´Ã³¯¿¡´Â ¾ÆÇÁ¸®Ä«-À¯·´-¾Æ½Ã¾ÆÀÇ ÀϺÎÁö¿ª¿¡¸¸ Á¸ÀçÇÒ »ÓÀÌ´Ù. º´¿øÃ¼´Â ¸®ÄÉÂ÷ ÇÁ·Î¹ÙÁ¦Å°(Rickettsia prowazeki)·Î ¿ÊÀÌ¿¡ ±â»ýÁõ½ÄÇÏ¿© ¿ÊÀÌÀÇ ºÐº¯°ú ÇÔ²² ¹è¼³µÇ¸é¼ ¿ÊÀ̰¡ ÈíÇ÷Çϱâ À§ÇØ ÇǺο¡ ÁØ »óó³ª »ç¶÷ÀÌ ¼ÕÀ¸·Î ±Ü¾î »ý±ä »óó¸¦ ÅëÇÏ¿© °¨¿°µÈ´Ù. Àẹ±â°£Àº 10~14ÀÏÀÌ¸ç ±ÞÀÛ½º·± ¿ÀÇÑÀ̳ª ÇÔ²² ¹ß¿ÇÏ¿© 3ÀÏ Á¤µµ °æ°úµÇ¸é 40¡É ÀüÈÄÀÇ °í¿ÀÌ ³ª°Ô µÇ°í, µÎÅë-°üÀýÅë-°á¸·ÃæÇ÷ µî°ú Áö¸§ 2mm ¾ÈÆÆÀÇ ºÓ°í ÀÛÀº ÃâÇ÷¼º ¹ßÁøÀÌ ¿Â¸ö¿¡ ¸¹ÀÌ »ý±ä´Ù. Áõ¼¼´Â ÀåÆ¼Çª½º¿Í ºñ½ÁÇϰí, ¹ÙÀÏ-Æç¸¯½º¹ÝÀÀ(Weil-Felix test)À̶ó ÇÏ´Â Ç÷û¹ÝÀÀ¿¡ ÀÇÇØ °¨º°µÈ´Ù. Ŭ·Î¶÷Æä´ÏÄÝÀ̳ª Åׯ®¶ó»çÀÌŬ¸°°èÀÇ Ç×»ý¹°ÁúÀÌ Æ¯È¿¸¦ º¸¿© Ä¡»çÀ²µµ ³·¾ÆÁ³´Ù(20%). ¿¹¹æÀ¸·Î´Â »ìÃæÁ¦·Î ¿ÊÀ̸¦ ±¸Á¦ÇÏ´Â °ÍÀÌ °¡Àå Áß¿äÇϸç, ¹ßÁøÆ¼Çª½º¹é½Å Á¢Á¾µµ À¯È¿ÇÏ´Ù. |
||
| ¿µ¹® | scarlet fever | ÇÑ±Û | ¼ºÈ«¿ |
|---|---|---|---|
| ¼³¸í | AÇü¿ëÇ÷»ç½½¾Ë±Õ¿¡ ÀÇÇÑ ¹ýÁ¤ ±Þ¼ºÀü¿°º´ÀÌ´Ù. 5~10¼¼ÀÇ ¼Ò¾Æ¿¡¼ È£¹ßÇÑ´Ù. ÀÛÀº ħ¹æ¿ïÀ» ÅëÇÏ¿© °¨¿°µÇ¸ç Àẹ±âÀÎ 2~4ÀÏÀ» Áö³ª, ÃʱâÁõ»óÀº °í¿, ÀεÎÁ¡¸·, Æíµµ°¡ ÇöÀúÇÏ°Ô ¹ßÀûÁ¾´ëµÈ´Ù. 1~2ÀÏ ÈÄ ºñÀ¶ÇÕ¼ºÀÇ È«»ö¼Ò±¸ÁøÀÌ Àü½Å¿¡ ÃâÇöÇÑ´Ù. ÀÔ ¾ðÀú¸®¿¡´Â ¹ßÁøÀÌ ¾ø°í ÀÔ¼úÁÖÀ§´Â â¹éÇÏ¸ç µþ±âÇô¸¦ º»´Ù. 2~3ÀÏ ÈÄ ¹ßÁøÀÌ »ç¶óÁø´Ù. º´º¯ºÎÀÇ ´ëºÎºÐÀº Ç¥ÃþÀÌ ¹Ú¸®µÇ´Âµ¥ ƯÈ÷ ¼Õ°ú ¹ßÀÇ À¶ÇÕ¼º Å»Çǰ¡ Ư¡ÀÌ´Ù. ÇöÀç´Â °ú°Å¿¡ ºñÇÏ¿© ÈξÀ º´ÀÌ °¡º¿öÁ®¼ ÁßÀÌ¿°, ¹ÙÀ§, ȳó¸²ÇÁÀý¿°µµ µå¹°´Ù. ÀεÎÁ¡¸·¹è¾çÀ¸·Î ±ÕÀ» Áõ¸íÇÏ¿© Áø´ÜÇÑ´Ù. A±º »ç½½¾Ë±Õ°¨¿°ÀÇ ÇÕº´ÁõÀ¸·Î´Â ¸é¿ªº¹ÇÕü¼ºÄáÆÏ¿°°ú ·ù¸¶Æ¼½º¿ÀÌ µÈ´Ù. |
||
| ¿µ¹® | fever | ÇÑ±Û | ¿ |
|---|---|---|---|
| ¼³¸í | ü¿ÂÁ¶Àý ÁßÃß°¡ ÀÌ»óÀ» ÃÊ·¡ÇØ Ã¼¿ÂÀÌ °è¼Ó »ó½ÂÇÏ¿© Áö¼ÓµÇ°í ÀÖ´Â »óŸ¦ ¸»ÇÑ´Ù. ¿øÀÎÀº ´Ù¾çÇϸç ü¿ÂÁ¶Àý ÁßÃß¿¡ Àå¾Ö(³úÁúȯ), Á¶Á÷ Àå¾Ö¿¡ ÀÇÇÑ °ÍÀÌ ÀÖ´Ù. ü¿ÂÀº ÀϹÝÀûÀ¸·Î °Üµå¶ûÀÌ, ÀÔ¾È, °ðâÀÚ¿¡¼ Àç°í, ½Ã°£Àº 10ºÐÀ» ÇÊ¿ä·Î ÇÑ´Ù. Åë»ó 37¡ÉÀÌÇÏ(°Üµå¶ûÀÌ)·Î, ÀÔ¾È ¿Âµµ´Â À̺¸´Ù 0.1~0.2¡É ³ôÀ¸¸ç, °ðâÀڿµµ´Â 0.2~0.5¡É ³ô´Ù. |
||
| HFRS | Hemorrhagic Fever with Renal Syndrome [HP 849-50] = Korean Hemorrhagic Fever &nbs... |
|---|---|
| FMF | familial Mediterranean fever; fetal movement felt; flow microfluorometry; forced midexpiratory flow |
| MSF | macrophage slowing factor; macrophage spreading factor; Medicins sans Frontieres [Doctors without Bo... |
| RF | 1) Renal Failure 2) Rheumatic Fever ? Rheumatic Fever  ... |
| HF | Hageman factor; haplotype frequency; hard filled [capsule]; hay fever; head of fetus; head forward; ... |
| FMF | Familial Mediterranean Fever |
|---|---|
| MSF | Mediterranean Spotted Fever |
| AGEP | Acute generalized exanthematous pustulosis |
| EMR | Eastern Mediterranean Region |
| ARF | Acute Rheumatic Fever |
| Mediterranean exanthematous fever | An affection occurring sporadically in the Mediterranean littoral marked by a severe chill with abrupt rise of temperature, pains in the joints, tonsillitis, diarrhoea, vomiting, and, on the third to fifth day, a rash of elevated nonconfluent macules beginning on the thighs and spreading to the entire body; lasts from ten days to two weeks and then disappears by rapid lysis without desquamation; probably caused by Rickettsia conorii, like Boutonneuse fever. (05 Mar 2000) |
|---|
| exanthematous fever | Fever associated with an exanthem. (05 Mar 2000) |
|---|---|
| mediterranean fever | See Familial Mediterranean Fever. (12 Dec 1998) |
| familial mediterranean fever | An inherited intestinal disorder that is characterised by recurrent fevers and intestinal inflammation. Usually has onset between the ages of 5 and 15 years and is more common in those of Mediterranean descent. Symptoms include fevers and abdominal pain. Some may also suffer from chest pains, multiple joint pains and red swollen legs. (27 Sep 1997) |
| fever, mediterranean | See Familial Mediterranean Fever. (12 Dec 1998) |
| exanthematous | Relating to an exanthema. (05 Mar 2000) |
| anaemia, mediterranean | Better known today as thalassaemia (or as beta thalassaemia or thalassaemia major).The clinical picture of this important type of anaemia was first described in 1925 by the paediatrician Thomas Benton Cooley. The name thalassaemia was coined by the Nobel Prise winning pathologist George Whipple and the professor of paediatrics Wm Bradford at U. Of Rochester because thalassa in Greek means the sea (like the Mediterrranean Sea) + -aemia means in the blood so thalassaemia means sea in the blood. Thalassaemia is not just one disease. It is a complex contingent of genetic (inherited) disorders all of which involve underproduction of haemoglobin, the indispensable molecule in red blood cells that carries oxygen. The globin part of normal adult haemoglobin is made up of 2 alpha and 2 beta polypeptide chains. In beta thalassaemia, there is a mutation (change) in both beta globin chains leading to underproduction (or absence) of beta chains, underproduction of haemoglobin, and profound anaemia. The gene for beta thalassaemia is relatively frequent in people of Mediterranean origin (for example, from Italy and Greece). Children with this disease inherit one gene for it from each parent. The parents are carriers (heterozygotes) with just one thalassaemia gene, are said to have thalassaemia minor, and are essentially normal. Their children affected with beta thalassaemia seem entirely normal at birth because at birth we still have predominantly foetal haemoglobin which does not contain beta chains. The anaemia surfaces in the first few months after birth and becomes progressively more severe leading to pallor and easy fatiguability, failure to thrive (grow), bouts of fever (due to infections) and diarrhoea. Treatment based on blood transfusions is helpful but not curative. Gene therapy will, it is hoped, be applicable to this disease. (12 Dec 1998) |
| mediterranean anaemia | Better known today as thalassaemia (or as beta thalassaemia or thalassaemia major).the clinical picture of this important type of anaemia was first described in 1925 by the paediatrician thomas benton cooley. The name thalassaemia was coined by the nobel prise winning pathologist george whipple and the professor of paediatrics wm bradford at u. Of rochester because thalassa in greek means the sea (like the mediterrranean sea) + -aemia means in the blood so thalassaemia means sea in the blood. Thalassaemia is not just one disease. It is a complex contingent of genetic (inherited) disorders all of which involve underproduction of haemoglobin, the indispensable molecule in red blood cells that carries oxygen. The globin part of normal adult haemoglobin is made up of 2 alpha and 2 beta polypeptide chains. In beta thalassaemia, there is a mutation (change) in both beta globin chains leading to underproduction (or absence) of beta chains, underproduction of haemoglobin, and profound anaemia. The gene for beta thalassaemia is relatively frequent in people of mediterranean origin (for example, from italy and greece). Children with this disease inherit one gene for it from each parent. The parents are carriers (heterozygotes) with just one thalassaemia gene, are said to have thalassaemia minor, and are essentially normal. Their children affected with beta thalassaemia seem entirely normal at birth because at birth we still have predominantly foetal haemoglobin which does not contain beta chains. The anaemia surfaces in the first few months after birth and becomes progressively more severe leading to pallor and easy fatiguability, failure to thrive (grow), bouts of fever (due to infections) and diarrhoea. Treatment based on blood transfusions is helpful but not curative. Gene therapy will, it is hoped, be applicable to this disease. (12 Dec 1998) |
| mediterranean islands | Scattered islands in the mediterranean sea. The chief islands are the balearic islands (belong to spain; majorca and minorca are among these), corsica (belongs to france), crete (belongs to greece), cyprus (a republic), the cyclades, dodecanese and ionian islands (belong to greece), malta (a republic), sardinia and sicily (belong to italy). (12 Dec 1998) |
| Mediterranean lymphoma | A spectrum of conditions ranging from a benign plasma cell hyperplasia to a highly malignant lymphoma of the small intestine. (12 Dec 1998) |
| mediterranean region | The mediterranean sea, the mediterranean islands, and the countries bordering on the sea collectively. (12 Dec 1998) |
| Mediterranean theileriosis | tropical theileriosis |
| absorption fever | An elevation of temperature often occurring, without other untoward symptoms, shortly after childbirth, assumed to be due to absorption of uterine discharges through abrasions of the vaginal wall. (05 Mar 2000) |
| acclimating fever | Elevated temperature with malaise that occurs upon working in a very hot environment. (05 Mar 2000) |
| aden fever | <virology> A tropical disease caused by dengue virus (Arbovirus), that is transmitted by the bite of an infected mosquito of the genus Aedes). Four severity grades of the illness are seen: Grade I: fever and constitutional symptoms. Grade II: grade I plus spontaneous bleeding of skin, gums or gastrointestinal tract. Grade III: grade II plus agitation and circulatory failure. Grade IV: profound shock. Grade I infection is seen most frequently in world travelers, where it is usually self-limited and rarely fatal. The other grades are referred to as dengue haemorrhagic fever and are often fatal. Dengue haemorrhagic fever appears to be an infection by one of the other dengue viruses. Prior immunity to a different dengue virus type appears to be important in the development of the more serious haemorrhagic form. Vaccines are available. Protection from mosquitoes is an important preventive measure. (15 Jan 1998) |
| aestivoautumnal fever | <infectious disease> A tropical parasitic disease caused by one of the genus Plasmodium and carried by infected mosquitoes of the genus Anopheles. This parasite uses red blood cells to complete its reproductive cycle. Common symptoms of an attack include high fever, chills, sweats and body aches. (27 Sep 1997) |
Á¦Ç°¸í |
ÆÇ¸Å»ç |
º¸ÇèÄÚµå | ¼ººÐ/ÇÔ·® | ±¸ºÐ/º¸Çè±Þ¿© |
|---|
Á¦Ç°¸í |
ÆÇ¸Å»ç |
º¸ÇèÄÚµå | ¼ººÐ/ÇÔ·® | ±¸ºÐ/º¸Çè±Þ¿© |
|---|