| ¿µ¹® | infection | ÇÑ±Û | °¨¿° |
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| ¼³¸í | º´¿ø¹Ì»ý¹°ÀÌ »ç¶÷À̳ª µ¿¹° ¶Ç´Â ½Ä¹°ÀÇ Á¶Á÷. ü¾×-Ç¥¸é¿¡ Á¤ÂøÇÏ¿© Áõ½ÄÇÏ´Â »óÅÂ. ÀÌ °æ¿ì µ¿¹° ¶Ç´Â ÀÎü¿¡ ¿©·¯ °¡Áö Áõ»ó, Áï Áúº´À» ÀÏÀ¸Å°´Â °æ¿ì¿Í ÀÏÀ¸Å°Áö ¾Ê´Â °æ¿ì°¡ ÀÖ´Ù. ¿¹¸¦ µé¸é, ÀϺ»³ú¿°¹ÙÀÌ·¯½º°¡ ÀÎü¿¡ ħÀÔÇÏ¿© ü³»¿¡ Áõ½ÄÇÏ¸é ¾î¶² »ç¶÷¿¡°Ô´Â °í¿-µÎÅë-ÀǽÄÀå¾Ö-°æ·Ã µîÀÇ Áõ»óÀÌ ÀϾ ¹ßº´À» ¾ËÁö¸¸, ´ë´Ù¼öÀÇ »ç¶÷Àº ü³»¿¡¼ ¹ÙÀÌ·¯½º°¡ Áõ½ÄÇÏ´õ¶óµµ Áõ¼¼ÀÇ Á¤µµ°¡ ³·°í ¹ß¿À̳ª ±× ¹ÛÀÇ Áõ¼¼µµ ¾ø¾î °¨¿°À» ¸ð¸¥´Ù. ÀÌ¿Í °°ÀÌ º´¿ø¹Ì»ý¹°Àº ÀÎü¿¡ °¨¿°µÇ´õ¶óµµ ¹ßº´ÇÏ´Â °æ¿ì¿Í ÇÏÁö ¾Ê´Â °æ¿ì°¡ ÀÖ´Ù. ÀüÀÚ¸¦ Áõ»ó°¨¿°, ÈÄÀÚ¸¦ ¹«Áõ»ó°¨¿°À̶ó ÇÑ´Ù. °¨¿°ÀÇ ±Ù¿øÀÌ µÇ´Â ȯÀÚ-º¸±ÕÀÚ-°¨¿°µ¿¹°-¸Å°³µ¿¹°-º´¿øÃ¼¸¦ Æ÷ÇÔÇÑ ¹è¼³¹° ¹× ±×¿¡ ÀÇÇØ °¨¿°µÈ °ÍÀ» °¨¿°¿øÀ̶ó Çϰí, ÀÌ·¯ÇÑ °¨¿°¿ø¿¡¼ Á÷Á¢ ¶Ç´Â °£Á¢À¸·Î »ýü¿¡ º´¿øÃ¼°¡ ħÀÔÇÏ´Â °æ·Î¸¦ °¨¿°°æ·Î¶ó ÇÑ´Ù. °¨¿°°æ·Î¿¡´Â °ø±â°¨¿°-Á¢Ã˰¨¿°-°æ±¸°¨¿°-°æÇǰ¨¿° µîÀÌ ÀÖ´Ù. ¶ÇÇÑ °¨¿°ÁõÀº Àü¿°¼º°ú ºñÀü¿°¼ºÀÇ µÎ °¡Áö·Î ³ª´ ¼ö ÀÖ´Ù. ÀüÀÚ´Â Áúº´ÀÇ °æ°ú Áß¿¡(¶§·Î´Â Àẹ±â³ª ȸº¹±â¿¡) °¨¿°ÇÑ »ýüÀÇ ºÐºñ¹° ¶Ç´Â ¹è¼³¹°°ú ÇÔ²² º´¿øÃ¼°¡ ³ª¿Í¼ Á¢ÃË ¶Ç´Â ¸Å°³¿¡ ÀÇÇØ ´Ù¸¥ °³Ã¼¸¦ °¨¿°½ÃŰ´Â °æ¿ì¸¦ ¸»ÇÑ´Ù. ¸¶¸¶-µðÇÁÅ׸®¾Æ-¼ºÈ«¿-Æä½ºÆ®-ÄÝ·¹¶ó-ÀÌÁú µîÀÌ ÀÌ¿¡ ¼ÓÇÑ´Ù. ÈÄÀÚ´Â º´¿øÃ¼°¡ °¨¿°ÇÑ »ýü¿¡¼ ¹è¼³µÇÁö ¾Ê°Å³ª ¹è¼³µÇ´õ¶óµµ ´Ù¸¥ °³Ã¼¿¡´Â °¨¿°À» ÀÏÀ¸Å°Áö ¾Ê´Â °ÍÀ¸·Î ¿©±â¿¡´Â ÆÄ»ódz-¸»¶ó¸®¾Æ-¹ßÁøÆ¼Çª½º-»êÈÄ¿ µîÀÌ ÀÖ´Ù. |
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| ¿µ¹® | droplet infection | ÇÑ±Û | ºñ¸»°¨¿°, ÀÛÀº¹æ¿ï°¨¿° |
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| ¼³¸í | º¸±ÕÀÚ³ª Áõ»óÀÌ Àִ ȯÀÚ È¤Àº ÀÌ¹Ì °¨¿°µÇ¾î ÀÖ´Â »ç¶÷ÀÇ È£Èí¿¡¼ ³ª¿Â Á÷°æ 10¸¶ÀÌÅ©·Ð ¶Ç´Â ±× ÀÌÇÏÀÇ ¾×üÀÔÀÚ¿¡ ºÎÀ¯Çϰí ÀÖ´Â º´¿øÃ¼ÀÇ ÈíÀÔ¿¡ ÀÇÇÑ È£Èí±â°¨¿°À» À̸¥´Ù. ÀÎÇ÷翣ÀÚ³ª Æíµµ¿°°ú °°ÀÌ È¯ÀÚ°¡ ±âħÀ» Çϰųª ´ëÈ µµÁß¿¡ ÀÚÀßÇÑ ºñ¸»°ú ÇÔ²² º´¿ø±ÕÀÌ °ø±â¿Í ÇÔ²² º´¿ø±ÕÀÌ ¹æÃâµÇ¾î °ø±â¿Í ÇÔ²² È£Èí±â·Î ÈíÀÔµÊÀ¸·Î½á °¨¿°µÇ´Â °ÍÀ» ¸»ÇÑ´Ù. °áÇÙ-À¯Ç༺°¨±â-¹éÀÏÇØ-µðÇÁÅ׸®¾Æ-Æó·Å µîÀÌ ÀÌ¿¡ ÀÇÇÏ¿© ÀüÆÄµÈ´Ù. |
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| ¿µ¹® | wound infection | ÇÑ±Û | »ó󰨿° |
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| ¼³¸í | Àý¼Õ µîÀÇ ±â°èÀû »óÇØ, ÀÎÀ§Àû ºÎ»ó ¶Ç´Â Ÿ±ÕÀÇ Ä§ÀÔ¿¡ ÀÇÇØ »óó³ Á¶Á÷¿¡¼ ħÀÔÇÏ¿© °¨¿°½ÃŰ´Â °Í. |
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| ¿µ¹® | secondary infection | ÇÑ±Û | ÀÌÂ÷°¨¿° |
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| ¼³¸í | ¾î¶² º´¿øÃ¼ÀÇ °¨¿°¿¡ ÀÇÇÏ¿© º»ÀÎÀÇ ÀúÇ×·ÂÀÌ ¾àÇØÁ³À» ¶§ ¸öÀÇ ´Ù¸¥ ºÎÀ§·Î ÀüÀÌÇÏ¿© ´Ù½Ã °¨¿°À» ÀÏÀ¸Å°´Â °Í. º´¿øÃ¼°¡ ÀÎü¿¡ ħÀÔÇÏ¿© ƯÁ¤ÇÑ ±â°üÀ̳ª Á¶Á÷¿¡¼ º´¿øÃ¼°¡ Áõ½ÄÇϰí, ±×°÷¿¡ ƯÀ¯ÀÇ º´Å͸¦ ÀÏÀ¸Å°´Â °ÍÀÌ 1Â÷°¨¿° ¶Ç´Â Ãʰ¨¿°ÀÌ´Ù. ÀÌ 1Â÷°¨¿°ÀÇ º´ÅÍÀÇ º´¿øÃ¼°¡ Ç÷°ü-¸²ÇÁ°ü-±â°ü-¼ÒȰü-¿ä°ü µîÀÇ ±æÀ» µû¶ó °°Àº ±â°üÀÇ ´Ù¸¥ ºÎÀ§³ª ´Ù¸¥ ±â°üÀ¸·Î ¿î¹ÝµÇ¾î °¨¿°À» ÀÏÀ¸Å²´Ù. µû¶ó¼ 1Â÷°¨¿°¿¡ ÀÇÇÏ¿© ÃæºÐÇÑ ¸é¿ªÀÌ µÉ °æ¿ì¿¡´Â 2Â÷°¨¿°ÀÌ ÀϾÁö ¾Ê´Â´Ù. ¿¹¸¦ µé¾î, À¯Ç༺ °¨±â¿¡ °É·ÈÀ» ¶§ ¼¼±Õ¿¡ ÀÇÇÑ Æó·ÅÀÌ µÚµû¸£´Â °æ¿ì¸¦ À̸¥´Ù. Æó·Å±Õ, ȳó¾Ë±Õ, ´ëÀå±Õ µûÀ§°¡ ÀÖ´Ù. |
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| MOTT | mycobacteria other than tuberculosis |
|---|---|
| NTM | Non-Tuberculosis Mycobacteria |
| RGM | Rapidly Growing Mycobacteria |
| NTM | nontuberculous mycobacteria |
| d/t | due to |
| MOTT | Mycobacteria Other Than Tuberculosis |
|---|---|
| DUE | DNA unwinding element |
| DUE | Drug usage evaluation |
| MGIT | Mycobacteria Growth Indicator Tube |
| NTM | Non-Tuberculous Mycobacteria |
| other-directed | Pertaining to a person readily influenced by the attitudes of others. (05 Mar 2000) |
|---|---|
| transferases (other substituted phosphate groups) | <enzyme> A class of enzymes that transfers substituted phosphate groups. Registry number: EC 2.7.8 (12 Dec 1998) |
| due date | The estimated calendar date when a baby will be born, the date the baby is due to be born. It is also called the estimated date of confinement (EDC). (12 Dec 1998) |
| dystonia, focal, due to blepharospasm | The second most common focal dystonia, the involuntary, forcible closure of the eyelids. The first symptoms may be uncontrollable blinking. Only one eye may be affected initially, but eventually both eyes are usually involved. The spasms may leave the eyelids completely closed causing functional blindness even though the eyes and vision are normal. (12 Dec 1998) |
| dystonia, focal, due to torticollis | Spasmodic torticollis, or torticollis, is the most common of the focal dystonias. In torticollis, the muscles in the neck that control the position of the head are affected, causing the head to twist and turn to one side. In addition, the head may be pulled forward or backward. (12 Dec 1998) |
| thrombotic disease due to protein c deficiency | Protein C is a protein in plasma that enters into the cascade of biochemical events leading to the formation of a clot. Deficiency of protein c results in thrombotic (clotting) disease and excess platelets with recurrent thrombophlebitis (inflammation of the vein that occurs when a clot forms). The clot can break loose and travel through the blood stream (thromboembolism) to the lungs causing a pulmonary embolism, brain causing a stroke (cerebrovascular accident), heart causing an early heart attack, skin causing what in the newborn is called neonatal purpura fulminans, the adrenal gland causing haemorrhage with abdominal pain, abnormally low blood pressure (hypotension), and salt loss. Protein c deficiency is due to possession of one gene (heterozygosity) in chromosome band 2q13-14. The possession of two such genes (homozygosity) is usually lethal. (12 Dec 1998) |
| atypical mycobacteria | Species of mycobacteria other than M. Tuberculosis or M. Bovis that can cause disease in immunocompromised humans. (05 Mar 2000) |
| group III mycobacteria | Mycobacteria that are either colourless or that slowly produce a light yellow pigment when grown in the presence of light. Organisms placed in this group belong to the species Mycobacterium intracellulare. Synonym: nonchromogens. (05 Mar 2000) |
| group II mycobacteria | Mycobacteria that produce a yellow pigment even when grown in the dark; when grown in the light, the pigment is orange. These organisms behave as do saprophytes in humans and are nonpathogenic to laboratory animals. Synonym: scotochromogens. (05 Mar 2000) |
| group I mycobacteria | Mycobacteria that produce a bright yellow colour when grown in the presence of light. Organisms placed in this group appear to belong to the species Mycobacterium kansasii. Synonym: photochromogens. (05 Mar 2000) |
| group IV mycobacteria | Mycobacteria that grow rapidly and that do not produce pigment. Organisms placed in this group belong to such species as Mycobacterium ulcerans and M. Marinum. (05 Mar 2000) |
| mycobacteria | <microbiology> Bacteria with unusual cell walls that are resistant to digestion, being waxy, very hydrophobic and rich in lipid, especially esterified mycolic acids. Staining properties differ from those of gram-negative and gram-positive organisms, being acid-fast. Many are intracellular parasites, causing serious diseases such as leprosy and tuberculosis. Cell wall has strong immunostimulating (adjuvant) properties due to muramyl dipeptide (MDP). Mycobacterium bovis causes tuberculosis in cattle, attenuated strain is Bacille Calmette-Guerin (BCG), used for immunisation. Mycobacterium leprae is the causative agent of leprosy. Mycobacterium microti is a mycobacterium that causes tuberculosis like disease in small rodents (Microtus microtus is the vole), will infect mice but not humans and is therefore much used as a laboratory model. Releases large amounts of cAMP which may inhibit lysosome phagosome fusion. Mycobacterium tuberculosis Is an obligate anaerobic nonmotile bacterium, causative agent of tuberculosis in humans. Lives intracellularly in macrophages. (18 Nov 1997) |
| agonal infection | An acute infection, commonly pneumonic or septic, occurring toward the end of any disease and often the cause of death. Synonym: agonal infection. (05 Mar 2000) |
| airborne infection | A mechanism of transmission of an infectious agent by particles, dust, or droplet nuclei suspended in the air. (05 Mar 2000) |
| apical infection | Implantation of microorganisms at the apex of a tooth, usually the result of the migration of microorganisms from the pulp canal through the apical foramen. (05 Mar 2000) |
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