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"Indiana type familial amyloid polyneuropathy"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial amyloid neuropathy
    °¡Á·¼º¾Æ¹Ð·ÎÀ̵å½Å°æº´(Áõ)
  • polyneuropathy
    ¿©·¯½Å°æº´(Áõ), ´Ù¹ß½Å°æº´(Áõ)
  • paraneoplastic polyneuropathy
    ½Å»ý¹°µþ¸²¿©·¯½Å°æº´(Áõ), ½Å»ý¹°µþ¸²´Ù¹ß½Å°æº´(Áõ)
  • amyloid angiopathy
    ¾Æ¹Ð·ÎÀ̵åÇ÷°üº´(Áõ)
  • amyloid kidney
    ¾Æ¹Ð·ÎÀ̵åÄáÆÏ, ¾Æ¹Ð·ÎÀ̵å½ÅÀå
  • amyloid nephrosis
    ¾Æ¹Ð·ÎÀ̵åÄáÆÏÁõ, ¾Æ¹Ð·ÎÀ̵å½ÅÀåÁõ
  • amyloid plaque
    ¾Æ¹Ð·ÎÀ̵åÆÇ
  • acholuric familial jaundice
    ¹«´ãÁó´¢°¡Á·¼ºÈ²´Þ
  • familial
    °¡Á·(¼º)-
  • familial adenomatous polyposis
    °¡Á·¼º»ùÁ¾Æú¸³Áõ
  • familial aminoglycoside ototoxicity
    °¡Á·¼º¾Æ¹Ì³ë±Û¸®Äڽõå±Íµ¶¼º
  • familial annulare erythema
    °¡Á·¼ºÀ±»óÈ«¹Ý, °¡Á·¼º°í¸®È«¹Ý
  • familial cardiomyopathy
    °¡Á·¼º½ÉÀå±ÙÀ°º´(Áõ), °¡Á·¼º½É±Ùº´(Áõ)
  • familial dysautonomia
    °¡Á·¼ºÀÚÀ²½Å°æÀÌ»ó(Áõ)
  • familial goiter
    °¡Á·¼º°©»ó»ùÁ¾
´ëÇÑÀÇÇù Çʼö ÀÇÇпë¾îÁý »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 12 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial adenomatous polyposis
    °¡Á·¼ºÅ«Ã¢ÀÚÆú¸³Áõ, °¡Á·¼º´ëÀåÆú¸³Áõ
  • polyneuropathy neuropathy
    ½Å»ý¹°µþ¸²¿©·¯½Å°æº´Áõ, ½Å»ý¹°µþ¸²´Ù¹ß½Å°æº´Áõ
  • polyneuropathy
    ¿©·¯½Å°æº´Áõ, ´Ù¹ß½Å°æº´Áõ
  • paraneoplastic polyneuropathy
    ½Å»ý¹°µþ¸²¿©·¯½Å°æº´Áõ, Á¾¾çµþ¸²´Ù¹ß½Å°æº´Áõ
  • tension type headache
    ±äÀåÇüµÎÅë
  • storage-type
    ÃàÀûÇü
  • type
    Çü, À¯Çü
  • blood type
    Ç÷¾×Çü
  • Borrmann type
    º¸¸£¸¸Çü
  • cellular type
    ¼¼Æ÷Çü
  • lepromatous type
    ³ªÁ¾Çü
  • scirrhous type
    °æÈ­Çü
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • polyneuropathy
    ´Ù¹ß½Å°æº´Áõ, ¿©·¯½Å°æº´Áõ
  • amyloid kidney
    ¾Æ¹Ð·ÎÀ̵åÄáÆÏ, ¾Æ¹Ð·ÎÀ̵å½ÅÀå
  • amyloid nephrosis
    ¾Æ¹Ð·ÎÀ̵åÄáÆÏÁõ
  • amyloid plaque
    ¾Æ¹Ð·ÎÀ̵åÆÇ
  • amyloid angiopathy
    ¾Æ¹Ð·ÎÀ̵åÇ÷°üº´Áõ
  • acholuric familial jaundice
    (¢¡hereditary spherocytosis) À¯ÀüµÕ±ÙÀûÇ÷±¸Áõ, À¯Àü±¸ÇüÀûÇ÷±¸
  • familial cardiomyopathy
    °¡Á·½ÉÀå±ÙÀ°º´Áõ
  • familial dyskeratotic comedo
    °¡Á·°¢È­ÀÌ»ó¸éÆ÷
  • familial dysautonomia
    °¡Á·ÀÚÀ²½Å°æ±â´ÉÀÌ»ó
  • endogenous familial hypertriglyceridemia
    ³»Àΰ¡Á·°íÁß¼ºÁö¹æÇ÷Áõ
  • familial eosinophilia
    °¡Á·È£»ê±¸Áõ°¡Áõ
  • familial annulare erythema
    °¡Á·À±»óÈ«¹Ý, °¡Á·°í¸®È«¹Ý
  • familial pigmented purpuric eruption
    °¡Á·»ö¼ÒÀڹݹßÁø
  • familial
    °¡Á·-
  • familial hyperlipoproteinemia
    °¡Á·°íÁö¹æ´Ü¹éÇ÷Áõ
¿¾ ´ëÇÑÀÇÇù 2 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • arsenical polyneuropathy
    ºñ¼Ò(¼º) ´Ù¹ß¼º ½Å°æº´Áõ.
  • polyneuropathy
    ´Ù¹ß½Å°æº´Áõ.
  • polyneuropathy
    ´Ù¹ß½Å°æº´Áõ(ÒýÛ¡ãêÌèÜ»ñø)
  • porphyric polyneuropathy
    Æ÷¸£ÇǸ°¼º ´Ù¹ß½Å°æº´Áõ.
  • porphyric polyneuropathy
    Æ÷¸£ÇǸ°¼º ´Ù¹ß½Å°æº´Áõ(¡­àõ ÒýÛ¡ãêÌèÜ»ñø)
  • relapsing polyneuropathy
    Àç¹ß¼º ´Ù¹ß½Å°æº´Áõ(¡­ÒýÛ¡ãêÌèÜ»ñø).
  • relapsing polyneuropathy
    Àç¹ß¼º ´Ù¹ß½Å°æº´Áõ(¡­ÒýÛ¡ãêÌèÜ»ñø)
  • amyloid
    ¾Æ¹Ð·ÎÀ̵å.
  • amyloid
    ¾Æ¹Ð·ÎÀ̵å
  • amyloid angiopathy
    À¯ÀüºÐÇ÷°üº´Áõ(ëºîþÝÏ úìηܻñø)
  • amyloid bodies<³ª> corpora amylacea
    ¾Æ¹Ð·ÎÀ̵å¼Òü(¡­á³ô÷).
  • amyloid degeneration
    ¾Æ¹Ð·ÎÀ̵庯¼º(¡­Ü¨àõ).
  • amyloid disease
    ¾Æ¹Ð·ÎÀ̵庴(¡­Ü»).
  • amyloid kidney
    ¾Æ¹Ð·ÎÀÌµå ½Å
  • amyloid nephrosis
    ¾Æ¹Ð·ÎÀ̵å½ÅÁõ(¡­ãìñø).
¿¾ ´ëÇÑÀÇÇù 3 ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial amyloid polyneuropathy
    °¡Á·¼º ¾Æ¹Ð·ÎÀÌµå ´Ù¹ß½Å°æº´Áõ
  • familial amyloid elastosis
    °¡Á·¼º À¯ÀüºÐ ź·Â¼¶À¯Áõ
  • familial amyloidotic polyneuropathy
    °¡Á·Àû ¾Æ¹Ð·ÎÀÌµå ´Ù¹ß½Å°æº´Áõ.
  • familial amyloidotic polyneuropathy
    °¡Á·Àû ¾Æ¹Ð·ÎÀÌµå ´Ù¹ß½Å°æº´Áõ
  • amyloid
    ¾Æ¹Ð·ÎÀ̵å.
  • amyloid
    ¾Æ¹Ð·ÎÀ̵å
  • amyloid angiopathy
    À¯ÀüºÐÇ÷°üº´Áõ(ëºîþÝÏ úìηܻñø)
  • amyloid bodies<³ª> corpora amylacea
    ¾Æ¹Ð·ÎÀ̵å¼Òü(¡­á³ô÷).
  • amyloid degeneration
    ¾Æ¹Ð·ÎÀ̵庯¼º(¡­Ü¨àõ).
  • amyloid disease
    ¾Æ¹Ð·ÎÀ̵庴(¡­Ü»).
  • amyloid kidney
    ¾Æ¹Ð·ÎÀÌµå ½Å
  • amyloid nephrosis
    ¾Æ¹Ð·ÎÀ̵å½ÅÁõ(¡­ãìñø).
  • amyloid precurssor protein
    ¾Æ¹Ð·ÎÀ̵å Àü±¸ ´Ü¹éÁú
  • amyloid tumor
    ¾Æ¹Ð·ÎÀ̵åÁ¾¾ç.
  • laryngeal amyloid
    Èĵξƹ̷ÎÀ̵å
´ëÇÑÇØºÎÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • Chief cell [Type I glomus cell]
    °ú¸³¼¼Æ÷
    [¿¾ ¿ë¾î] ÁÖ¼¼Æ÷(Á¦1Çü»ç±¸¼¼Æ÷)
  • Regular type
    ±ÔÄ¢Çü
    [¿¾ ¿ë¾î] ±ÔĢġ¹Ð°áÇÕÁ¶Á÷
  • Muscular type of artery
    ±ÙÀ°Çüµ¿¸Æ
    [¿¾ ¿ë¾î] ±ÙÇüµ¿¸Æ
  • Muscular type of lymphatic vessel
    ±ÙÀ°Çü¸²ÇÁ°ü
    [¿¾ ¿ë¾î] ±ÙÇüÀӯİü
  • Muscular type of vein
    ±ÙÀ°ÇüÁ¤¸Æ
    [¿¾ ¿ë¾î] ±ÙÇüÁ¤¸Æ
  • Type B spermatogonium
    ´ÊÁ¤Á¶¼¼Æ÷
    [¿¾ ¿ë¾î] BÁ¤Á¶¼¼Æ÷
  • Anovulatory type
    ¹«¹è¶õÇü
    [¿¾ ¿ë¾î] ¹«¹è¶õÇü
  • Ovulatory type
    ¹è¶õÇü
    [¿¾ ¿ë¾î] ¹è¶õÇü
  • Irregular type
    ºÒ±ÔÄ¢Çü
    [¿¾ ¿ë¾î] ºÒ±ÔĢġ¹Ð°áÇÕÁ¶Á÷
  • Calcified hypertrophic type
    ¼®È¸È­ºñ´ëÇü
    [¿¾ ¿ë¾î] ¼®È¸È­ºñ´ëÇü
  • Fibrous type of lymphatic vessel
    ¼¶À¯Çü¸²ÇÁ°ü
    [¿¾ ¿ë¾î] ¼¶À¯ÇüÀӯİü
  • Type II hair cell
    ¿øÁÖÅм¼Æ÷
    [¿¾ ¿ë¾î] ÀüÆÄ¿¬Á¢¼¼Æ÷
  • Type A spermatogonium
    À¸¶äÁ¤Á¶¼¼Æ÷
    [¿¾ ¿ë¾î] AÁ¤Á¶¼¼Æ÷
  • Type I hair cell
    Á¶·Õ¹ÚÅм¼Æ÷
    [¿¾ ¿ë¾î] ¹è»ó¿¬Á¢¼¼Æ÷
  • Elastic type of artery
    ź·ÂÇüµ¿¸Æ
    [¿¾ ¿ë¾î] ź·ÂÇüµ¿¸Æ
´ëÇѱâ»ýÃæÇÐȸ ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 3 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • familial aggregation
    °¡Á·ÁýÀû¼º
  • polymyarian type
    ´Ù±ÙÀ°Çü
  • type specimen
    ±âÁØÇ¥º»
´ëÇÑ»ýÈ­ÇкÐÀÚ»ý¹°ÇÐȸ ¿ë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • amyloid
    ¾Æ¹Ð·ÎÀ̵å
  • familial high-density lipoprotein deficiency °¡Á·¼º °í¹Ðµµ ÁöÁú´Ü¹éÁú °áÇÌ (Ê«ðéàõÍÔÚËÓøò·òõ
    Ó±ÛÜòõÌÀù¹)
  • familial hypercholesterolemia
    °¡Á·¼º(Ê«ðéàõ) °ú(Φ)ÄÝ·¹½ºÅ×·ÑÇ÷Áõ(úìñø)
  • familial lysosomal lipase deficiency
    °¡Á·¼º(Ê«ðéàõ) ¶óÀÌ¼Ò¼Ø ¶óÀÌÆäÀ̽º °áÇÌ(ÌÀù¹)
  • familial methemoglobinemia
    °¡Á·¼º(Ê«ðéàõ) ¸ÞÅ׸ð±Û·ÎºóÇ÷Áõ(úìñø)
  • ABO blood group (type) system
    ABOÇ÷¾×Çü (úìäûúþ) ½Ã½ºÅÛ
  • C-type particles
    C-Çü(û¡) ÀÔÀÚ(Ø£í­)
  • C-type virus
    "C-Çü(û¡) ¹ÙÀÌ·¯½º, (ÔÒ) C-type particles"
  • dehydrogenase-type mechanism
    µðÇÏÀ̵å·ÎÀú³×À̽ºÇü(úþ) ±âÀü(Ѧï®)
  • delayed-type hypersensitivity
    Áö¿¬Çü °ú¹ÎÁõ(òÀæÅû¡Î¦ÚÂñø)
  • immediate-type hypersensitivity
    Áï½ÃÇü °ú¹ÎÁõ(ñíãÁúþΦÚÂñø)
  • L-type structure
    L-Çü(úþ)±¸Á¶(ϰðã)
  • mixed-type inhibitor
    È¥ÇÕÇü ÀúÇØÁ¦(ûèùêúþîÁúªð¥)
  • plant-type ferredoxin
    ½Ä¹°Çü(ãÕÚªû¡) Æä·¹µ¶½Å
  • plaque-type mutant
    ÇöóÅ©Çü(û¡) º¯ÀÌü(ܨì¶ô÷)
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 10 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • amyloid infiltration
    ¾Æ¹Ð·ÎÀ̵åħÀ±
  • familial
    °¡Á·¼ºÀÇ
  • familial polyposis
    °¡Á·¼ºÆú¸³Áõ
  • bell type
    Á¾¸ð¾ç, Á¾Çü
  • intracanalicular type
    ¼Ò°ü³»Çü
  • mobile type diagnostic X ray apparatus
    À̵¿Çü Áø´ÜX¼±ÀåÄ¡
  • onion-skin type
    ¾çÆÄ²®Áú¸ð¾ç
  • phased linear array type
    À§»óÂ÷¼±Çü¹è¿­½Ä
  • RF coil type
    °íÁÖÆÄÄÚÀÏÀ¯Çü
  • split electrode type probe
    ºÐÇÒÀü±ØÅ½ÃËÀÚ
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
ECG Electro-Cardio-Graphy(-Gram); ½ÉÀüµµ
   = EKG
  1. Conducting System Structu...
FAP familial adenomatous polyposis; familial amyloid polyneuropathy; fatty acid polyunsaturated; fatty a...
PMD Progressive Muscular Dystrophy; ÁøÇ༺ ±ÙÀÌ¿µ¾çÁõ
  Types of PMD(Progressive Muscular Dystroph...
FAP Familial Amyloid Polyneuropathy
TAPVR Total Anomalous Pulmonary Venous Return
  = TAPVC
  4 Types of TAPVR
&...
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
FAP Familial Amyloid Polyneuropathy
FAP Familial Amyloidotic Polyneuropathy
AIDP Acute inflammatory demyelinating polyneuropathy
CIDP Chronic Inflammatory Demyelinating Polyneuropathy
CIP Critical illness polyneuropathy
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • familial amyloid polyneuropathy
    °¡Á·¼º ¾Æ¹Ð·ÎÀÌµå ´Ù¹ß ½Å°æº´Áõ
  • Indiana
    ÀδپƳª
  • polyneuropathy
    ´Ù¹ß¼º ½Å°æ º´º¯, ´Ù¹ß¼º ½Å°æº´Áõ, ´Ù¹ß ½Å°æº´Áõ
    ¿©·¯ °³ÀÇ ½Å°æÀ» ÀÌȯÇÏ´Â ÁúȯÀ¸·Î¼­ º¸Åë ¾çÃø¼ºÀÌ°í ¹Ì¸¸¼ºÀÌ´Ù.
  • amyloid degeneration
    ¾Æ¹Ð·ÎÀÌµå º¯¼º
  • amyloid infiltration
    ¾Æ¹Ð·ÎÀ̵å ħÀ±
  • serum amyloid protein A
    Ç÷û ¾Æ¹Ð·ÎÀ̵å ÇÁ·Îƾ A
  • amaurotic familial idiocy
    °¡Á·¼º Èæ³»Àå ¹éÄ¡
    µ¿ÀǾî=Tay-Sach's disease.
  • asymptomatric familial hyperbilirubinemia
    ¹«ÁõÈÄ °¡Á·¼º °úºô¸®·çºóÇ÷Áõ
  • benign familial pemphigus
    ¾ç¼º °¡Á·¼º õÆ÷â
    µå¹°°Ô ¹ß»ýÇÏ´Â, À¯Àü¼ºÀ̸ç Áö¼ÓÀûÀ¸·Î Àç¹ßÀ» ¹Ýº¹ÇÏ´Â ¼ÒÆ÷¼º ¹× ÀÛÀº ¼öÆ÷¼º ÇǺο°À¸·Î, ¾×¿Í, ¼­ÇýºÎ ¹× ¸ñ ºÎÀ§¸¦ °¡Àå Àß Ä§¹üÇÏÁö¸¸, ¶§·Î´Â ±¤¹üÇÑ ºÎÀ§¸¦ ħ¹üÇÑ´Ù. º´º¯Àº ´Ù¹ß¼ºÀ̸ç, ¼öÁÖÀÏ ³»Áö ¼ö°³¿ù ÈÄ¿¡ ¼èÅðÇÑ´Ù. ÀÌ ÁúȯÀº »ó¿°»öü ¿ì¼º À¯ÀüÀ» ÇÑ´Ù.
  • familial adenomatous polyposis
    °¡Á·¼º ¼±Á¾¼º ¿ëÁ¾Áõ
  • familial benign chronic pemphigus
    °¡Á·¼º ¾ç¼º ¸¸¼º õÆ÷â
  • familial cold urticaria
    °¡Á·¼º Çѳà µÎµå·¯±â
  • familial cutaneous collagenosis
    °¡Á·¼º ÇǺΠ±³¿øÁõ
  • familial dysbetalipoproteinemia
    °¡Á·¼º ÀÌ»ó º£Å¸ ¸®Æ÷ ÇÁ·ÎÅ×ÀÎ Ç÷Áõ
  • familial fibrous dysplasia
    °¡Á·¼º ¼¶À¯ ÀÌÇü¼º
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
vesicular stomatitis-indiana virus The type species of vesiculovirus causing a disease symptomatically similar to foot-and-mouth disease in cattle, horses, and pigs. It may be transmitted to other species including humans, where it causes influenza-like symptoms.
(12 Dec 1998)
familial amyloid neuropathy <neurology> A disorder in which various peripheral nerves are infiltrated with amyloid and their functions disturbed, an abnormal prealbumin is also formed and is present in the blood; characteristically, it begins during mid-life and is found largely in persons of Portuguese descent. Other rare clinical types occur.
Inheritance: autosomal dominant.
Synonym: familial amyloidosis, hereditary amyloidosis.
(05 Mar 2000)
type I familial hyperlipoproteinaemia Hyperlipoproteinaemia characterised by the presence of large amounts of chylomicrons and triglycerides in the plasma when the patient has a normal diet, and their disappearance on a fat-free diet; low alpha-and beta-lipoproteins on a normal diet, with increase on fat-free diet; decreased plasma postheparin lipolytic activity; and low tissue lipoprotein lipase activity. It is accompanied by bouts of abdominal pain, hepatosplenomegaly, pancreatitis, and eruptive xanthomas; autosomal recessive inheritance.
See: familial lipoprotein lipase inhibitor.
Synonym: Burger-Grutz syndrome, familial fat-induced hyperlipaemia, familial hyperchylomicronaemia, familial hypertriglyceridemia, idiopathic hyperlipaemia.
(05 Mar 2000)
type II familial hyperlipoproteinaemia Hyperlipoproteinaemia characterised by increased plasma levels of beta-lipoproteins, cholesterol, and phospholipids, but normal triglycerides; heterozygotes have mild lipid changes and are susceptible to atherosclerosis in middle age, but homozygotes have severe changes often with generalised xanthomatosis and xanthelasma, and frank clinical atherosclerosis as young adults. The primary defect is a deficiency of apoprotein of VLDL, and the disorder is divided into two classes: 1) type IIA, which has elevated LDL due to a deficiency of the receptor or a modified apolipoprotein B-100; 2) type IIB, which has elevated LDL and triglycerides; autosomal dominant inheritance.
Synonym: familial hyperbetalipoproteinaemia, familial hypercholesteraemic xanthomatosis, familial hypercholesterolaemia.
(05 Mar 2000)
type III familial hyperlipoproteinaemia Hyperlipoproteinaemia characterised by increased plasma levels of LDL, beta-lipoproteins, pre-beta-lipoproteins, cholesterol, phospholipids, and triglycerides; hypertriglyceridemia induced by a high carbohydrate diet, and glucose tolerance is abnormal; frequent eruptive xanthomas and atheromatosis, particularly coronary artery disease; biochemical defect lies in apolipoproteins; there are many varieties.
Synonym: carbohydrate-induced hyperlipaemia, dysbetalipoproteinaemia, familial hyperbetalipoproteinaemia and hyperprebetalipoproteinaemia, familial hypercholesterolaemia with hyperlipaemia.
(05 Mar 2000)
type IV familial hyperlipoproteinaemia Plasma levels of VLDL, pre-beta-lipoproteins and triglycerides are increased on a normal diet, but beta-lipoproteins, cholesterol, and phospholipids are normal; hypertriglyceridemia is induced by a high carbohydrate diet; may be accompanied by abnormal glucose tolerance and susceptibility to ischemic heart disease; probably autosomal recessive inheritance.
Synonym: carbohydrate-induced hyperlipaemia, familial hyperprebetalipoproteinaemia, familial hypertriglyceridemia.
(05 Mar 2000)
type V familial hyperlipoproteinaemia Hyperlipoproteinaemia characterised by increased plasma levels of chylomicrons, VLDL, pre-beta-lipoproteins, and triglycerides, and slight rise of cholesterol on a normal diet, with beta-lipoproteins normal; may be accompanied by bouts of abdominal pain, hepatosplenomegaly, susceptibility to atherosclerosis, and abnormal glucose tolerance; probably autosomal recessive inheritance.
Synonym: combined fat-and carbohydrate-induced hyperlipaemia, familial hyperchylomicronaemia with hyperprebetalipoproteinaemia, mixed hyperlipaemia.
(05 Mar 2000)
acute demyelinating polyneuropathy <neurology> A neurologic condition.
Synonym: Guillain-Barre syndrome.
Origin: Gr. Pathos = disease
(27 Sep 1997)
acute inflammatory polyneuropathy <neurology, syndrome> Acute infective polyneuritis that results in a form of peripheral neuropathy with temporary loss of movement and sensation due to inflammation of multiple nerves and loss of myelin.
The exact cause is unknown but has been associated with an abnormal immune response to viral infection, particularly cytomegalovirus infection, in which there is cell-mediated immunity to a component of myelin. The disease may be autoimmune in origin and complete recovery can take up to six months.
Synonym: Guillain-Barre syndrome
(12 Jul 2000)
alcoholic polyneuropathy <neurology> A disorder that occurs secondary to the damage to nerve cells from habitual alcohol abuse.
The effects of alcoholic polyneuropathy may be due in part to the direct toxic effect alcohol has on nervous tissue in combination with specific nutritional deficiencies (for example B1 or thiamin deficiency and B12 deficiency).
Common symptoms include sensory changes, numbness, tingling, muscle weakness, muscle cramps, heat intolerance, impotence, difficulty urinating, swallowing difficulty, speech impairment, diarrhoea and constipation.
Origin: Gr. Pathos = disease
(27 Sep 1997)
arsenical polyneuropathy An axon loss polyneuropathy that results from subacute or chronic arsenic poisoning; almost always preceded by gastrointestinal symptoms; one of the heavy metal neuropathies.
(05 Mar 2000)
axonal polyneuropathy A type of polyneuropathy in which axon degeneration is the sole/predominant feature; many aetiologies, particularly toxic and metabolic; on nerve conduction studies, affects amplitudes of the responses, but does not cause conduction slowing or block.
Synonym: axonal polyneuropathy.
(05 Mar 2000)
axon loss polyneuropathy A type of polyneuropathy in which axon degeneration is the sole/predominant feature; many aetiologies, particularly toxic and metabolic; on nerve conduction studies, affects amplitudes of the responses, but does not cause conduction slowing or block.
Synonym: axonal polyneuropathy.
(05 Mar 2000)
buckthorn polyneuropathy Ascending polyneuropathy resulting from ingestion of the fruit of Karwinskia humboldtiana.
(05 Mar 2000)
chronic inflammatory demyelinating polyneuropathy An uncommon, acquired, demyelinating sensorimotor polyneuropathy, clinically characterised by insidious onset, and slow evolution, (either steady progression or stepwise), and chronic course; symmetrical weakness is a predominant symptom, often involving proximal leg muscles, accompanied by paresthesias, but not pain; CSF examination shows elevated protein, while electrodiagnostic studies reveal evidence of a demyelinating process, primarily conduction slowing rather than block; sometimes responds to prednisone.
(05 Mar 2000)
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