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¿µ¹® poststreptococcal glomerulonephritis ÇÑ±Û »ç½½¾Ë±Õ°¨¿°ÈÄÅ丮ÄáÆÏ¿°, ¿¬¼â±¸±Õ°¨¿°ÈĻ籸ü½Å¿°
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¿µ¹® glomerulonephritis ÇÑ±Û Å丮ÄáÆÏ¿°, »ç±¸Ã¼½Å¿°
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  2.¸¸¼ºÅ丮ÄáÆÏ¿°-Áö¼ÓÀûÀΠ¿ä°Ë»ç»óÀÇ ÀÌ»ó, Áܹ鴢, Ç÷´¢¿Í ½ÅÀåÀÇ ±â´ÉÀÌ ¼­¼­È÷ ¾ø¾îÁö´Â Áúȯ±ºÀÌ´Ù. ÈçÈ÷ ¸»±âÄáÆÏÀ̶ó ºÎ¸£¸ç °ÅÀÇ ¸ðµç ÄáÆÏÀÇ º´ÀÌ ÀÌ ¸¸¼º Å丮ÄáÆÏ¿°À¸·Î ÁøÇàµÈ´Ù. 
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  • ¿µ¹®
    ÇѱÛ
  • IgA nephropathy
    IgAÄáÆÏº´(Áõ), IgA½ÅÀ庴(Áõ)
  • crescentic glomerulonephritis
    Ãʽ´ÞÅ丮ÄáÆÏ¿°, Ãʽ´޻籸ü½Å¿°
  • chronic glomerulonephritis
    ¸¸¼ºÅ丮ÄáÆÏ¿°, ¸¸¼º»ç±¸Ã¼½Å¿°
  • diffuse proliferative glomerulonephritis
    ±¤¹üÀ§Áõ½ÄÅ丮ÄáÆÏ¿°, ±¤¹üÀ§Áõ½Ä»ç±¸Ã¼½Å¿°
  • exudative proliferative glomerulonephritis
    »ïÃâÁõ½ÄÅ丮ÄáÆÏ¿°, »ïÃâÁõ½Ä»ç±¸Ã¼½Å¿°
  • focal embolic glomerulonephritis
    ÃÊÁ¡»öÀüÅ丮ÄáÆÏ¿°, ÃÊÁ¡»öÀü»ç±¸Ã¼½Å¿°
  • focal glomerulonephritis
    ÃÊÁ¡Å丮ÄáÆÏ¿°, ÃÊÁ¡»ç±¸Ã¼½Å¿°
  • focal proliferative glomerulonephritis
    ÃÊÁ¡Áõ½ÄÅ丮ÄáÆÏ¿°, ÃÊÁ¡Áõ½Ä»ç±¸Ã¼½Å¿°
  • focal segmental glomerulonephritis
    ÃÊÁ¡ºÐÀýÅ丮ÄáÆÏ¿°, ÃÊÁ¡ºÐÀý»ç±¸Ã¼½Å¿°
  • glomerulonephritis
    Å丮ÄáÆÏ¿°, »ç±¸Ã¼½Å¿°
  • idiopathic crescentic glomerulonephritis
    Ư¹ßÃʽ´ÞÅ丮ÄáÆÏ¿°, Ư¹ßÃʽ´޻籸ü½Å¿°
  • intercapillary glomerulonephritis
    ¸ð¼¼°ü»çÀÌÅ丮ÄáÆÏ¿°, ¸ð¼¼°ü°£»ç±¸Ã¼½Å¿°
  • membranoproliferative glomerulonephritis
    ¸·Áõ½ÄÅ丮ÄáÆÏ¿°, ¸·Áõ½Ä»ç±¸Ã¼½Å¿°
  • membranous glomerulonephritis
    ¸·Å丮ÄáÆÏ¿°, ¸·»ç±¸Ã¼½Å¿°
  • mesangial proliferative glomerulonephritis
    Ç÷°ü»çÀÌÁúÁõ½ÄÅ丮ÄáÆÏ¿°, Ç÷°ü°£¼¼Æ÷Áõ½Ä»ç±¸Ã¼½Å¿°
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  • ¿µ¹®
    ÇѱÛ
  • IgA nephropathy
    ¸é¿ª±Û·Îºí¸°¿¡ÀÌÄáÆÏº´Áõ, ¸é¿ª±Û·Îºí¸°¿¡À̽ÅÀ庴Áõ
  • glomerulonephritis
    Å丮ÄáÆÏ¿°, »ç±¸Ã¼½Å¿°
  • crescentic glomerulonephritis
    Ãʽ´ÞÅ丮ÄáÆÏ¿°, Ãʽ´޻籸ü½Å¿°
  • idiopathic crescentic glomerulonephritis
    Ư¹ßÃʽ´ÞÅ丮ÄáÆÏ¿°, Ư¹ßÃʽ´޻籸ü½Å¿°
  • membranoproliferative glomerulonephritis
    ¸·Áõ½ÄÅ丮ÄáÆÏ¿°, ¸·Áõ½Ä»ç±¸Ã¼½Å¿°
  • membranous glomerulonephritis
    ¸·Å丮ÄáÆÏ¿°, ¸·»ç±¸Ã¼½Å¿°
  • poststreptococcal glomerulonephritis
    »ç½½¾Ë±Õ°¨¿°ÈÄÅ丮ÄáÆÏ¿°, ¿¬¼â±¸±Õ°¨¿°ÈĻ籸ü½Å¿°
  • rapidly progressive glomerulonephritis
    ±Þ¼ÓÁøÇàÅ丮ÄáÆÏ¿°, ±Þ¼ÓÁøÇà»ç±¸Ã¼½Å¿°
  • mesangial proliferative glomerulonephritis
    Ç÷°ü»çÀÌÁúÁõ½ÄÅ丮ÄáÆÏ¿°, Ç÷°ü°£¼¼Æ÷Áõ½Ä»ç±¸Ã¼½Å¿°
¿¾ ´ëÇÑÀÇÇù ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • crescentric glomerulonephritis
    Ãʽ´ÞÅ丮ÄáÆÏ¿°, Ãʽ´޻籸ü½Å¿°
  • diffuse proliferative glomerulonephritis
    »êÀçÁõ½ÄÅ丮ÄáÆÏ¿°, ¹Ì¸¸Áõ½Ä»ç±¸Ã¼½Å¿°
  • exudative proliferative glomerulonephritis
    »ïÃâÁõ½ÄÅ丮ÄáÆÏ¿°, »ïÃâÁõ½Ä»ç±¸Ã¼½Å¿°
  • focal glomerulonephritis
    ÃÊÁ¡Å丮ÄáÆÏ¿°, ÃÊÁ¡»ç±¸Ã¼½Å¿°
  • focal embolic glomerulonephritis
    ÃÊÁ¡»öÀüÅ丮ÄáÆÏ¿°, ÃÊÁ¡»öÀü»ç±¸Ã¼½Å¿°
  • focal proliferative glomerulonephritis
    ÃÊÁ¡Áõ½ÄÅ丮ÄáÆÏ¿°, ÃÊÁ¡Áõ½Ä»ç±¸Ã¼½Å¿°
  • focal segmental glomerulonephritis
    ÃÊÁ¡ºÐÀýÅ丮ÄáÆÏ¿°, ÃÊÁ¡ºÐÀý»ç±¸Ã¼½Å¿°
  • glomerulonephritis
    Å丮ÄáÆÏ¿°, »ç±¸Ã¼½Å¿°
  • intercapillary glomerulonephritis
    ¸ð¼¼°üÅ丮ÄáÆÏ¿°, ¸ð¼¼°ü°£»ç±¸Ã¼½Å¿°
  • membranoproliferative glomerulonephritis
    ¸·Áõ½ÄÅ丮ÄáÆÏ¿°, ¸·Áõ½Ä»ç±¸Ã¼½Å¿°
  • membranous glomerulonephritis
    ¸·Å丮ÄáÆÏ¿°, ¸·»ç±¸Ã¼½Å¿°
  • mesangiocapillary glomerulonephritis
    »çÀÌÁú¸ð¼¼°üÅ丮ÄáÆÏ¿°, »ç±¸Ã¼°£Áú¸ð¼¼°ü»ç±¸Ã¼½Å¿°
  • poststreptococcal glomerulonephritis
    »ç½½¾Ë±Õ°¨¿°ÈÄÅ丮ÄáÆÏ¿°, ¿¬¼â±¸±Õ°¨¿°ÈĻ籸ü½Å¿°
  • rapidly progressive glomerulonephritis
    ±Þ¼ÓÁøÇàÅ丮ÄáÆÏ¿°, ±Þ¼ÓÁøÇà»ç±¸Ã¼½Å¿°
  • subacute diffuse glomerulonephritis
    ¾Æ±Þ¼º»êÀçÅ丮ÄáÆÏ¿°, ¾Æ±Þ¼º¹ü¹ß»ç±¸Ã¼½Å¿°
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  • ¿µ¹®
    ÇѱÛ
  • IgA, secretory
    ºÐºñÇü IgA
  • anti-IgA
    Ç× IgA Ç×ü
  • iga deficiency
    ¸é¿ª±Û·ÎºÒ¸° A °áÇÌ
  • iga nephroathy
    ¸é¿ª±Û·ÎºÒ¸° A ½Åº´Áõ
  • immunoglobulin a(iga)
    ¸é¿ª±Û·ÎºÒ¸° A(Øó湡­)
  • acute glomerulonephritis
    ±Þ¼º»ç±¸Ã¼½Å¿°
  • acute glomerulonephritis
    ±Þ¼º »ç±¸Ã¼½Å¿°(¡­ÞêϹô÷ãìæú).
  • glomerulonephritis
    »ç±¸Ã¼¼º ½Å¿°
  • glomerulonephritis
    »ç±¸Ã¼½Å¿°.
  • glomerulonephritis
    »ç±¸Ã¼½Å¿°.(ÞêϹô÷ãìæú)
  • glomerulonephritis
    »ç±¸Ã¼½Å¿°
  • glomerulonephritis,diffuse proliferative
    ¹Ì¸¸¼º Áõ½Ä¼º(ڵؼàõ ñòãÖàõ)
  • glomerulonephritis,focal embolic
    ÃÊÁ¡¼º »öÀü¼º
  • glomerulonephritis,focal proliferative
    ÃÊÁ¡¼º Áõ½Ä¼º
  • glomerulonephritis,membranoproliferative
    ¸·Áõ½Ä¼º
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  • ¿µ¹®
    ÇѱÛ
  • anti-IgA
    Ç× IgA Ç×ü
  • bullous dermatosis,linear iga
    ¼±»ó IgA (àÊß¾¡­)
  • iga deficiency
    ¸é¿ª±Û·ÎºÒ¸° A °áÇÌ
  • iga nephroathy
    ¸é¿ª±Û·ÎºÒ¸° A ½Åº´Áõ
  • immunoglobulin a(iga)
    ¸é¿ª±Û·ÎºÒ¸° A(Øó湡­)
  • intra-epidermal neutrophilic IgA dermatosis
    Ç¥Çdz»È£Áß±¸¼º ¸é¿ª±Û·ÎºÒ¸°A ÇǺκ´
  • isolated iga deficiency
    ´Üµ¶¼º¸é¿ª±Û·ÎºÒ¸°A°áÇÌ(Ó¤Ô¼àõ Øóæ¹ ¡­ ÌÀù¹)
  • linear IgA bullous dematosis
    ¼±»ó ¸é¿ª±Û·ÎºÒ¸°A ¼öÆ÷¼ºÇǺκ´
  • linear IgA dermatitis
    ¼±»ó ¸é¿ª±Û·ÎºÒ¸°A ÇǺο°
  • linear IgA dermatosis
    ¼±»ó ¸é¿ª±Û·ÎºÒ¸°A ÇǺκ´(Áõ)
  • secretory IgA
    ºÐºñÇü IgA.
  • acute glomerulonephritis
    ±Þ¼º »ç±¸Ã¼½Å¿°(¡­ÞêϹô÷ãìæú).
  • acute glomerulonephritis
    ±Þ¼º»ç±¸Ã¼½Å¿°
  • crescentric glomerulonephritis
  • focal glomerulonephritis
    ÃÊÁ¡¼º »ç±¸Ã¼ ½Å¿°.
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  • ¿µ¹®
    ÇѱÛ
  • IgA
    (å²) immunoglobulin A
KI ÀÇÇпë¾î »çÀü °Ë»ö À¯»ç °Ë»ö °á°ú : 1 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
  • focal glomerulonephritis
    ÃÊÁ¡¼º»ç±¸Ã¼½Å¿°
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
MCGN mesangiocapillary glomerulonephritis; minimal change glomerulonephritis; mixed cryoglobulinemia with...
Ig Immuno-globulin
  IgA; Immuno-globulin A; (27)(30)60(100) - (170)(80)(250)380 mg/dL
IGA infantile genetic agranulocytosis
IgA immunoglobulin A
S-IgA secretory immunoglobulin A
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 1
IgA GN IgA glomerulonephritis
IgA-D IgA deficiency
IgA-IC IgA immune complex
IgA NP IgA nephropathy
IgA RF IgA rheumatoid factor
°æºÏ´ë Ä¡°ú´ëÇÐ ±¸°­³»°ú ±³½Ç »çÀü À¯»ç °Ë»ö °á°ú : 8 ÆäÀÌÁö: 1
  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • iga nephroathy
    ¸é¿ª ±Û·ÎºÒ¸° A ½Åº´Áõ
  • linear IgA disease
    ¼±»ó ¸é¿ª ±Û·ÎºÒ¸° A Áúȯ
  • secretory IgA
    ºÐºñÇü ¸é¿ª ±Û·ÎºÎ¸° A
    Ç÷û, Ÿ¾×, ´«¹°, È£Èí±â, ¼ÒÈ­°ü µîÀÇ ºÐºñ¾× Áß ÇÔÀ¯µÇ¾î ÀÖ´Ù.
  • acute glomerulonephritis
    ±Þ¼º »ç±¸Ã¼ ½Å¿°
    ÇüÅÂÇÐÀûÀ¸·Î´Â ±Þ¼º ¹Ì¸¸¼º Áõ½Ä¼º »ç±¸Ã¼½Å¿°À» ÀÏÀ¸Å°°í ÀÓ»óÀûÀ¸·Î´Â Ç÷´¢, °íÁú¼ÒÇ÷Áõ, ÇÌ´¢, °íÇ÷¾Ð µîÀÇ ±Þ¼º ½Å¿° ÁõÈıºÀ» ÁÖ¼Ò·Î ÇÏ´Â ½Å¿°À¸·Î ´Ü¹é´¢ ¹× ºÎÁ¾ µîµµ µ¿¹ÝµÉ ¼ö ÀÖÀ¸³ª ½ÅÁõÈıºº¸´Ù Á¤µµ°¡ ½ÉÇÏÁö ¾ÊÀº °æ¿ì°¡ ´ëºÎºÐÀÌ´Ù.
  • acute poststreptococcal glomerulonephritis
    ±Þ¼º ¿¬¼â»ó ±¸±Õ¼º »ç±¸Ã¼ ½Å¿°
    A±º ¿¬¼â»ó ±¸±Õ °¨¿°ÀÌ ¿øÀÎÀÌ µÇ¾î ¹ß»ýÇÏ´Â ±Þ¼º ½Å¿°ÀÌ´Ù. ¿¬¼â»ó ±¸±Õ¿¡ ÀÇÇÑ Æíµµ¿° µîÀÌ Ä¡À¯µÇ¾î¼­ 1-2ÁÖ ÈÄ¿¡ µ¹¿¬ ºÎÁ¾, ÇÌ´¢, °íÇ÷¾Ð µîÀ» µ¿¹ÝÇØ¼­ ¹ßº´ÇÑ´Ù. ¼Ò¾Æ¿¡ ¸¹ÀÌ ¹ßº´Çϳª ¼ºÀο¡°Ôµµ µå¹°Áö´Â ¾ÊÀ¸¸ç, ¾à 90%´Â 1-2°³¿ù ÈÄ¿¡ ¿ÏÀüÈ÷ Ä¡À¯µÈ´Ù. Ç÷û ASLO °ªÀÇ »ó½Â°ú Ç÷û º¸Ã¼°¡ÀÇ °¨¼Ò°¡ Áø´Ü¿¡ µµ¿òÀÌ µÈ´Ù. ÀÌ Áõ»óÀº ¿¬¼â»ó ±¸±ÕÀÇ Ç׿ø°ú ±× Ç×ü¿¡ ÀÇÇÑ ¸é¿ª º¹ÇÕü ½Å¿°ÀÌ´Ù. ƯÈ÷ ¼Ò¾Æ¿¡¼­´Â ±Þ¼º±â¿¡ °íÇ÷¾Ð¿¡ ÀÇÇÑ ½ÉºÎÀü, °æ·ÃÀ» ÀÏÀ¸Å°±â ½±±â ¶§¹®¿¡ ÁÖÀǸ¦ ¿äÇÑ´Ù.
  • focal glomerulonephritis
    ÃÊÁ¡¼º »ç±¸Ã¼ ½Å¿°
  • focal proliferative glomerulonephritis
    ÃÊÁ¡¼º Áõ½Ä¼º »ç±¸Ã¼ ½Å¿°
  • membranoproliferative glomerulonephritis
    ¸· Áõ½Ä¼º »ç±¸Ã¼ ½Å¿°
    ¸¸¼º »ç±¸Ã¼ ½Å¿°ÀÇ ÇÑ À¯ÇüÀ¸·Î, Ç÷°ü°£ ¼¼Æ÷ Áõ½Ä°ú »ç±¸Ã¼ ¸ð¼¼Ç÷°üÀÇ ºÒ±ÔÄ¢ÀûÀÎ ºñÈĸ¦ Ư¡À¸·Î ÇÑ´Ù. ÀÌ¿¡´Â µÎ °³ÀÇ ¾ÆÇüÀÌ ÀÖ´Ù. Á¦ 1ÇüÀº ³»ÇǼ¼·ÎÇÏ ÀüÀÚ ¹Ðµµ Ä§Âø°ú ÀüÇüÀûÀÎ º¸Ã¼ °æ·Î Ȱ¼ºÈ­°¡ ³ªÅ¸³ª¸ç, Á¦2ÇüÀº ½ÉÇÑ ÀüÀÚ ¹Ðµµ Ä§ÂøÀÌ »ç±¸Ã¼ ±âÀú¸·¿¡ »ý±â¸ç C3 ½Å¿° ÀÎÀÚ¸¦ Æ÷ÇÔÇÑ ±³´ëÀû º¸Ã¼ °æ·Î Ȱ¼ºÈ­°¡ ÀϾ´Ù. À̴ û¼Ò³âÀ̳ª ÀþÀº ¼ºÀÎÃþ¿¡°Ô ¹ßº´Çϸç, ºÒ±ÔÄ¢ÀûÀΠȸº¹À» µ¿¹ÝÇϸ鼭 õõÈ÷ ÁøÇàÇÏ¿© ±Ã±ØÀûÀ¸·Î´Â ½ÅºÎÀü¿¡ ºüÁø´Ù.
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 1
glomerulonephritis, IgA Chronic form of glomerulonephritis characterised by recurring haematuria with only slight proteinuria and by deposits of IgA immunoglobulin in the mesangial areas of the renal glomeruli. It usually occurs in young males.
(12 Dec 1998)
secretory component of IgA <immunology, protein> A polypeptide chain of about 60 kD that aids secretion of the IgA, a portion of the IgA receptor on the plasmalemma of the inner side of the epithelial cells lining the gut, which is proteolysed when the IgA receptor complex has travelled through the cell after receptor mediated endocytosis at the inner face, to the outer (luminal) face.
(18 Nov 1997)
IgA <immunology> Major class of immunoglobulin of external secretions in mammals, also found in serum and body fluids such as tears and saliva and in the respiratory, reproductive, urinary and gastrointestinal tracts.
Immunoglobulin A protects the bodys mucosal surfaces from infection and in secretions, it is found as a dimer (400 kD) joined by a short J chain and linked to a secretory portion or transport piece. In serum found as a monomer (170 kD).
Immunoglobulin A is the main mechanism for providing local immunity against infections in the gut or respiratory tract and may act by reducing the binding between an immunoglobulin A coated micro organism and a host epithelial cell. Present in human colostrum but not transferred across the placenta. Have heavy chains.
(30 Mar 1998)
IgA deficiency A dysgammaglobulinaemia characterised by a deficiency of IgA.
(12 Dec 1998)
IgA nephropathy <nephrology, pathology> This is a form of glomerulonephritis that results from the deposition of circulating IgA antibody in the kidney tissues.
Inflammation of the glomerulus (glomerulonephritis) is the result. This condition may present as acute glomerulonephritis, chronic glomerulonephritis or rapidly progressive glomerulonephritis.
Berger's is usually detected in an individual with one or two bouts of bloody urine (usually begins during or soon after a respiratory infection) and no other symptoms of renal disease. Only rarely, will Berger's disease permanently affect kidney function and progress to chronic renal failure. This renal disorder more commonly affects males in the 16-40 age group.
Origin: Gr. Pathos = disease
(27 Sep 1997)
IgA, secretory The principle ig found in exocrine secretions such as milk, respiratory and intestinal mucin, saliva and tears. It is composed of two to four molecules of 7s IgA and one molecule each of j chain and secretory component (sc), also called secretory piece. Molecular weight appr. 400,000.
(12 Dec 1998)
IgA-specific serine endopeptidase <enzyme> Extracellular microbial enzymes whose only substrate is human IgA of the iga1 subclass; cleave the immunoglobulin at a specific internal prolyl-threonyl peptide bond in the heavy chain to yield intact faba and fca fragments; consider also EC 3.4.24.13
Registry number: EC 3.4.21.72
Synonym: immunoglobulin a(1) protease, iga1 protease, immunoglobulin a1 protease, IgA protease
(26 Jun 1999)
linear IgA bullous disease in children A rare self-limiting bullous disease, chiefly of the trunk, perioral, and pelvic areas, with onset in the first decade, successively less severe recurrences, and total remission at adolescence; linear epidermal basement membrane zone deposit of IgA is found in involved and in normal skin.
Synonym: linear IgA bullous disease in children.
(05 Mar 2000)
acute crescentic glomerulonephritis <nephrology> A relatively uncommon (affecting 1 out of 10,000 people) form of acute glomerulonephritis that results in damage within the glomerulus of the kidney. There is rapid loss of kidney function with the formation of crescents on microscopic analysis (kidney biopsy). This disorder may result in acute glomerulonephritis or nephrotic syndrome, but ultimately results in renal failure and end-stage renal disease.
Symptoms include smoky coloured urine (pyuria), decreased urine output, swelling and hypertension. Any conditions which can cause a vasculitis increase the risk of this disorder. Some examples include lupus, Goodpasture's syndrome, Henoch-Schonlein purpura, IgA nephropathy, membranoproliferative glomerulonephritis, anti-glomerular basement membrane antibody disease, history for malignant tumours and exposure to hydrocarbon solvents.
(27 Sep 1997)
acute glomerulonephritis <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood).
Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis).
(27 Sep 1997)
acute haemorrhagic glomerulonephritis <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood).
Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis).
(27 Sep 1997)
acute post-streptococcal glomerulonephritis <nephrology> A disease of the kidneys that results in inflammation of the glomerulus (the portion of the kidney that filters the blood).
Conditions which may cause glomerulonephritis include post-streptococcal disease (strep throat), lupus, syphilis, bacterial endocarditis, membranoproliferative glomerulonephritis, sepsis, vasculitis, Goodpasture's syndrome, typhoid fever, Henoch-Schonlein purpura, hepatitis or a viral infection (for example mumps, measles, mononucleosis).
(27 Sep 1997)
anti-basement membrane glomerulonephritis Glomerulonephritis resulting from anti-basement membrane antibodies, characterised by smooth linear deposits of IgG and C3 along glomerular capillary walls; includes rapidly progressive glomerulonephritis and glomerulonephritis in Goodpasture's syndrome.
(05 Mar 2000)
Berger's focal glomerulonephritis Glomerulonephritis affecting a small proportion of renal glomeruli which commonly presents with haematuria and may be associated with acute upper respiratory infection in young males, not usually due to streptococci; associated with IgA deposits in the glomerular mesangium and may also be associated with systemic disease, as in Henoch-Schonlein purpura.
Synonym: Berger's disease, Berger's focal glomerulonephritis, focal nephritis, IgA nephropathy.
(05 Mar 2000)
rapidly progressive glomerulonephritis <nephrology> A relatively uncommon (affecting 1 out of 10,000 people) form of acute glomerulonephritis that results in damage within the glomerulus of the kidney. There is rapid loss of kidney function with the formation of crescents on microscopic analysis (kidney biopsy). This disorder may result in acute glomerulonephritis or nephrotic syndrome, but ultimately results in renal failure and end-stage renal disease.
Symptoms include smoky coloured urine (pyuria), decreased urine output, swelling and hypertension. Any conditions which can cause a vasculitis increase the risk of this disorder. Some examples include lupus, Goodpasture's syndrome, Henoch-Schonlein purpura, IgA nephropathy, membranoproliferative glomerulonephritis, anti-glomerular basement membrane antibody disease, history for malignant tumours and exposure to hydrocarbon solvents.
(27 Sep 1997)
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