| misc | miscarriage; miscellaneous |
|---|---|
| DASD | Direct Access Storage Device = RAM |
| SPD | Storage Pool Disease |
| CESD | cholesterol ester storage disease |
| CSC | blow on blow (administration of small amounts of drugs at short intervals) [Fr. coup sur coup]; coll... |
| CESD | Cholesterol ester storage disease |
|---|---|
| GSD | Glycogen Storage Disease |
| GSDII | Glycogen Storage Disease type II |
| GSD 1a | Glycogen storage disease type 1a |
| GSD I | Glycogen storage diseases type I |
| cleaning | In dentistry, a procedure whereby accretions are removed from the teeth or from a dental prosthesis. See: dental prophylaxis. Ultrasonic cleaning, in dentistry, the use of a high-frequency vibrating point to remove deposits from tooth structure; also the process of cleaning dentures by placing them in a special liquid in a container that generates high-frequency vibrations. (05 Mar 2000) |
|---|---|
| cleaning-in-place | <procedure> Cleaning and sterilising a bioreactor system without dismantling it. (14 Nov 1997) |
| ear canal, self-cleaning | most of the time the ear canals are self-cleaning, that is, there is a slow and orderly migration of ear canal skin from the eardrum to the outer opening. Old earwax is constantly being transported from the deeper areas of the ear canal to the opening where it usually dries, flakes, and falls out. (12 Dec 1998) |
| ear cleaning | When so much wax accumulates that it blocks the ear canal (and hearing), your physician may have to wash it out, vacuum it, or remove it with special instruments. Alternatively, your physician may prescribe ear drops what are designed to soften the wax (such as Cerumenex). Never put anything smaller than your elbow in your ear! Wax is not formed in the deep part of the ear canal near the eardrum, but only in the outer part of the canal. So when a patient has wax pushed up against the eardrum, it is often because he has been probing his ear with such things as cotton-tipped swabs (such as Q-Tips), bobby pins, or twisted napkin corners. Such objects only serve as ramrods to push the wax in deeper. Also, the skin of the ear canal and the eardrum is very thin, fragile and easily injured. The ear canal is more prone to infection after it has been whipped clean of the good coating type wax. In addition, we have seen many perforated eardrums as a result of these efforts. (12 Dec 1998) |
| electron cyclotron discharge cleaning | Using relatively low power microwaves (at the electron cyclotron frequency) to create a weakly ionised, essentially unconfined hydrogen plasma in the vacuum chamber. The ions react with impurities on the walls of the tokamak and help remove them from the chamber. For instance, Alcator C-mod typically applies electron cyclotron discharge cleaning for a few days prior to beginning a campaign, and a few hours before each day's run. (09 Oct 1997) |
| brancher glycogen storage disease | Type of glycogen storage disease, due to deficiency of amylo-1,4-1,6-transglucosidase (brancher enzyme). Synonym: brancher deficiency glycogenosis, debrancher deficiency. (05 Mar 2000) |
| glycogen storage disease | <hepatology> A group of inherited metabolic disorders involving the enzymes responsible for the synthesis and degradation of glycogen. In some patients, prominent liver involvement is presented. In others, more generalised storage of glycogen occurs, sometimes with prominent cardiac involvement. Synonym: glycogenosis (12 Sep 2002) |
| glycogen storage disease type I | <disease> An autosomal recessive disease in which gene expression of glucose-6-phosphatase is absent, resulting in hypoglycaemia due to lack of glucose production. Accumulation of glycogen in liver and kidney leads to organomegaly, particularly massive hepatomegaly. Increased concentrations of lactic acid and hyperlipidemia appear in the plasma. Clinical gout often appears in early childhood. Inheritance: autosomal recessive. (12 Dec 1998) |
| glycogen storage disease type II | <disease> Glycogenosis due to alpha-1,4-glucosidase (acid maltase) deficiency. It affects muscle, heart, and other organs. (12 Dec 1998) |
| glycogen storage disease type III | <disease> An autosomal recessive metabolic disorder due to deficient expression of amylo-1,6-glucosidase (one part of the glycogen debranching enzyme system). The clinical course of the disease is similar to that of glycogen storage disease type I, but milder. Massive hepatomegaly, which is present in young children, diminishes and occasionally disappears with age. Levels of glycogen with short outer branches are elevated in muscle, liver, and erythrocytes. Six subgroups have been identified, with subgroups type IIIa and type IIIb being the most prevalent. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type IV | <disease> An autosomal recessive metabolic disorder due to a deficiency in expression of branching enzyme (alpha-1,4-glucan-6-alpha-glucosyltransferase), resulting in an accumulation of abnormal glycogen with long outer branches. Clinical features are muscle hypotonia and cirrhosis. Death from liver disease usually occurs before age 2. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type V | <disease> Glycogenosis due to muscle phosphorylase deficiency. Characterised by painful cramps following sustained exercise. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type VI | <disease> A hepatic glycogen storage disease in which there is an apparent deficiency of hepatic phosphorylase activity. However, studies have not been able to distinguish between phosphorylase deficiency and phosphorylase kinase deficiency in patients with hepatic glycogenosis. (12 Dec 1998) |
| glycogen storage disease type VII | <disease> An autosomal recessive muscle glycogen storage disease in which there is deficient expression of muscle phosphofructokinase activity, resulting in increased concentrations of glucose-6-phosphate and fructose-6-phosphate and low concentrations of fructose-1,6-diphosphate in muscle tissue. Glycogen storage in muscle is increased, perhaps due to activation of glycogen synthase by accumulated glucose-6-phosphate. It has been proposed that shunting of glucose-6-phosphate and fructose-6-phosphate into the pentose phosphate pathway may result in increased synthesis of purines and pyrimidines, causing hyperuricaemia and gout. Erythrocytes from patients may show decreased phosphofructokinase activity and 2,3-diphosphoglycerate deficiency. Exercise intolerance is present and severe congenital muscular dystrophy has been reported. Inheritance: autosomal recessive (12 Dec 1998) |
| glycogen storage disease type VIII | <disease> An x-linked recessive hepatic glycogen storage disease resulting from lack of expression of phosphorylase-b-kinase activity. Symptoms are relatively mild; hepatomegaly, increased liver glycogen, and decreased leukocyte phosphorylase are present. Liver shrinkage occurs in response to glucagon. Inheritance: X-linked recessive (12 Dec 1998) |
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|