| ¿µ¹® | edema | ÇÑ±Û | ºÎÁ¾ |
|---|---|---|---|
| ¼³¸í | Á¶Á÷³»¿¡ ¸²ÇÁ¾×À̳ª Á¶Á÷ÀÇ »ïÃâ¹° µîÀÇ ¾×ü°¡ Àú·ùµÇ¾î¼ °úÀ× Á¸ÀçÇÏ´Â »óŸ¦ ¸»ÇÑ´Ù. ÀÌ·± Çö»óÀº »ý±â´Â ºÎÀ§¿¡ µû¶ó ÇÇÇϺÎÁ¾, ÆóºÎÁ¾, º¹¼ö¶ó Çϸç, ¹ß»ý ±âÀü¿¡ µû¶ó ºÐ·ùÇÏ¸é ¿ïÇ÷¼º ºÎÁ¾, ¿°Áõ¼º ºÎÁ¾, Ç÷°ü¿îµ¿¼º ºÎÁ¾, ±â¾Æ¼º ºÎÁ¾, ÄáÆÏÅ¿ ºÎÁ¾ µîÀ¸·Î ºÐ·ùµÈ´Ù. ¿ïÇ÷¼º ºÎÁ¾Àº ±â´É¼º ºÎÁ¾À̶ó°íµµ Çϸç, Á¤¸ÆÀ̳ª ¸²ÇÁ°üÀÇ ÇùÂø, Æó¼â·Î ÀÎÇÏ¿© Á¤»óÀûÀÎ È帧ÀÌ ¹æÇع޾ÒÀ» ¶§¿¡ ¹ß»ýÇÏ´Â °ÍÀÌ´Ù. ¿°Áõ¼º ºÎÁ¾Àº ¿°Áõ¼º º¯È·Î ÀÎÇØ¼ Ç÷°üÀÇ Åõ°ú¼ºÀÌ Ç×ÁøµÇ¾î Ç÷°ü¼Ó¿¡ ÀÖ´Â ¼öºÐÀÌ ¸¹ÀÌ Á¶Á÷À¸·Î ºüÁ® ³ª°¡¼ »ý±â´Â ºÎÁ¾À̰í, Ç÷°ü¿îµ¿¼º ºÎÁ¾Àº Ç÷°ü¿îµ¿½Å°æ ¸¶ºñ·Î À¯¹ßµÇ¸ç, ±â¾Æ¼º ºÎÁ¾Àº ¸¸¼ºÀûÀ¸·Î ¿µ¾çÀÌ ºÎÁ·ÇÑ °æ¿ì¿¡ »ý±â´Â ºÎÁ¾ÀÌ´Ù. ÄáÆÏÅ¿ ºÎÁ¾Àº ÄáÆÏº´ÀÌ ÀÖ´Â °æ¿ì¿¡ ¼Òº¯À¸·Î ¿µ¾çºÐÀÌ ºüÁ®³ª°¡´Â °æ¿ì¿¡ À¯¹ßµÈ´Ù. ºÎÁ¾Àº »ý±â´Â ºÎÀ§¿¡ µû¶ó ´Ù¼Ò Â÷À̰¡ ÀÖÀ»Áö¶óµµ ¹ß»ý±âÀü¿¡¼´Â ¼·Î ¹ÐÁ¢ÇÑ °ü·ÃÀ» ¸Î°í ÀÖÀ¸¹Ç·Î ±Ùº»ÀûÀÎ ¿øÀÎÀ» ã¾Æ³»¾î Ä¡·á¸¦ ½ÃÀÛÇÏ´Â °ÍÀÌ ¹Ù¶÷Á÷ÇÏ´Ù. |
||
| HAE | health appraisal examination; hearing aid evaluation; hepatic artery embolism; hereditary angioneuro... |
|---|---|
| HANE | hereditary angioneurotic edema |
| HPE | hepatic portoenterostomy; high-permeability edema; history and physical examination; holoprosencepha... |
| EF | Edema Factor |
| HACE | High Altitude Cerebral Edema |
| HAE | Hereditary angioneurotic edema |
|---|---|
| HANE | Hereditary Angio Neurotic Edema |
| HAE | Hereditary Angio-Edema |
| BE | Brain edema |
| CSME | Clinically significant macular edema |
| angioneurotic oedema, hereditary | A genetic form of angioedema. (Angioedema is also referred to as Quinke's disease.) Persons with it are born lacking an inhibitor protein (called C1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of C1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioedema. (12 Dec 1998) |
|---|---|
| hereditary angioneurotic oedema | A genetic form of angioedema. (angioedema is also referred to as quinke's disease.) persons with it are born lacking an inhibitor protein (called c1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of c1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioedema. (12 Dec 1998) |
| oedema, hereditary angioneurotic | A genetic form of angioedema. (Angioedema is also referred to as Quinke's disease.) Persons with it are born lacking an inhibitor protein (called C1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of C1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioedema. (12 Dec 1998) |
| edema | <clinical sign> The presence of abnormally large amounts of fluid in the intercellular tissue spaces of the body, usually applied to demonstrable accumulation of excessive fluid in the subcutaneous tissues. Oedema may be localised, due to venous or lymphatic obstruction or to increased vascular permeability or it may be systemic due to heart failure or renal disease. Collections of oedema fluid are designated according to the site, for example ascites (peritoneal cavity), hydrothorax (pleural cavity) and hydropericardium (pericardial sac). Massive generalised oedema is called anasarca. Origin: Gr. Oide ma = swelling (18 Nov 1997) |
| edema, cardiac | A manifestation of congestive heart failure caused by increased venous and capillary pressures and often associated with the retention of sodium by the kidneys. (12 Dec 1998) |
| angioneurotic | <neurology> Denoting a neuropathy affecting the vascular system. See: angioedema. (18 Nov 1997) |
| angioneurotic oedema | <neurology> An acute or recurring attack of transient oedema suddenly appearing in areas of the skin or mucous membranes and occasionally of the viscera, often associated with dermatographism, urticaria, erythema, and purpura. It can produce dramatic swelling of the subcutaneous tissues (welts beneath the skin) that typically appears around the eyes and lips. Welts may also involve the hands, feet and throat (compromise the airway). This condition is associated with allergies (for example foods, pollen), but may also be a side effect of some medications (for example ACE inhibitors, salicylates). Emotional stress, exposure to cold, water, sunlight, heat and insect bites all have been know to cause angioneurotic oedema. Treatment is with adrenaline, antihistamines, cimetidine, and/or corticosteroids. (12 Dec 1998) |
| Albright's hereditary osteodystrophy | An inherited form of hyperparathyroidism associated with ectopic calcification and ossification and skeletal defects, notably the small fourth metacarpals, but intelligence is normal. There are dominant, recessive and X-linked forms. See: pseudohypoparathyroidism. Synonym: Albright's syndrome. (05 Mar 2000) |
| angioedema, hereditary | A genetic form of angioedema. (Angioedema is also referred to as Quinke's disease.) Persons with it are born lacking an inhibitor protein (called C1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of C1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioneurotic oedema. (12 Dec 1998) |
| canine hereditary blindness | An autosomal dominant condition seen in dogs of the collie and several other breeds. (05 Mar 2000) |
| colourectal neoplasms, hereditary nonpolyposis | A syndrome characterised by autosomal dominant inheritance, a low mean age (41 years) for occurrence of colon cancer, and a marked increase in the proportion of tumours in the proximal colon. (12 Dec 1998) |
| corneal dystrophies, hereditary | Bilateral hereditary disorders of the cornea, usually autosomal dominant, which may be present at birth but more frequently develop during adolescence and progress slowly throughout life. Central macular dystrophy is transmitted as an autosomal recessive defect. (12 Dec 1998) |
| hereditary | <genetics> Transferred via genes from parent to child. (16 Dec 1997) |
| hereditary amyloidosis | <neurology> A disorder in which various peripheral nerves are infiltrated with amyloid and their functions disturbed, an abnormal prealbumin is also formed and is present in the blood; characteristically, it begins during mid-life and is found largely in persons of Portuguese descent. Other rare clinical types occur. Inheritance: autosomal dominant. Synonym: familial amyloidosis, hereditary amyloidosis. (05 Mar 2000) |
| hereditary angioedema | A genetic form of angioedema. (angioedema is also referred to as quinke's disease.) persons with it are born lacking an inhibitor protein (called c1 esterase inhibitor) that normally prevents activation of a cascade of proteins leading to the swelling of angioedema. Patients can develop recurrent attacks of swollen tissues, pain in the abdomen, and swelling of the voice box (larynx) which can compromise breathing. The diagnosis is suspected with a history of recurrent angioedema. It is confirmed by finding abnormally low levels of c1 esterase inhibitor in the blood. Treatment options include antihistamines and male steroids (androgens) that can also prevent the recurrent attacks. Also called hereditary angioneurotic oedema. (12 Dec 1998) |
Á¦Ç°¸í |
ÆÇ¸Å»ç |
º¸ÇèÄÚµå | ¼ººÐ/ÇÔ·® | ±¸ºÐ/º¸Çè±Þ¿© |
|---|
Á¦Ç°¸í |
ÆÇ¸Å»ç |
º¸ÇèÄÚµå | ¼ººÐ/ÇÔ·® | ±¸ºÐ/º¸Çè±Þ¿© |
|---|