| EHL | Electro-Hydrauric Lithotripsy |
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| EHL | effective half-life; electrohydraulic lipotripsy; endogenous hyperlipidemia; Environmental Health Laboratory; essential hyperlipemia; extensor hallucis longus |
| EHL | Electrohydraulic lithotripsy |
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| EHL | extensor hallucis longus |
| Ehlers, Edward | <person> A Danish dermatologist, 1863-1937. See: Ehlers-Danlos syndrome. (05 Mar 2000) |
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| Ehlers-Danlos syndrome | <dermatology, rheumatology> A connective-tissue disorder characterised by hyperelasticity of skin, poor wound healing, hyperextensibility of joints, soft-tissue calcifications (spheroids), vascular lesions: aortic dissection, aneurysms, rarely: tortuous arch, ectatic pulmonary artery, tissue fragility most likely to be haematomas, aortic rupture after angiography, clinical types: gravis, mitis, benign hypermobile, ecchymotic, X-linked, associated with medullary sponge kidney Inheritance: autosomal dominant, X-linked. Synonym: dermatosparaxis. (12 Dec 1998) |
| ehlite | <chemical> A mineral of a green colour and pearly luster; a hydrous phosphate of copper. Origin: From Ehl near Linz, where it occurs. Source: Websters Dictionary (01 Mar 1998) |
Synonyms : Ehlers-Danlos Disease, Disease, Ehlers-Danlos, Ehlers Danlos Disease, Ehlers Danlos Syndrome, Syndrome, Ehlers-Danlos
| Ehlers-Danlos syndrome |
Ehlers-Danlos syndrome is a group of inherited disorders that affect connective tissue, the tissue that binds and supports the body's muscles, ligaments, organs, and skin. The dermatosparaxis type is characterized by extremely fragile and sagging skin. This form of Ehlers-Danlos syndrome was formerly called type VIIC. Ehlers-Danlos syndrome, dermatosparaxis type is a subtype of Ehlers-Danlos syndrome. ...
Ãâó: goldbamboo.com/glossary-1e.html
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| Ehlers-Danlos s. |
1. a group of inherited disorders of the connective tissue, occurring in at least ten types, I to X, based on clinical, genetic, and biochemical evidence, varying in severity from mild to lethal, and transmitted genetically as autosomal recessive, autosomal dominant, or X-linked recessive traits. The major manifestations include hyperextensible skin and joints, easy bruisability, friability of tissues with bleeding and poor wound healing, calcified subcutaneous spheroids, and pseudotumors; variably present in some types are cardiovascular, gastrointestinal, orthopedic, and ocular defects. The biochemical defects are known for several types. Type IV, with prominent vascular manifestations, is caused by defects in the structure, synthesis, or secretion of one type of procollagen; type VI, with prominent ocular manifestations, is caused by a deficiency of lysyl hydroxylase; type VII, with multiple joint dislocations, is called also arthrochalasis multiplex congenita and is caused by mutations involving the normal cleavage sites of some procollagen chains; type IX is X-linked cutis laxa; and type X is due to a defect in fibronectin that interferes with normal platelet aggregation. See accompanying illustration. 2. cutaneous asthenia (def. 1).
Ãâó: www.mercksource.com/pp/us/cns/cns_health_library.j...
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