| 영문 | dental caries | 한글 | 충치, 치아우식증 |
|---|---|---|---|
| 설명 | 치아의 조직이 세균에 의해 용해내지 파괴되어 점차 통증을 나타내다가 치아를 잃게 되는 하나의 세균성 병이라고 볼 수 있다. 우리나라에서는 영구치의 충치 이환율이 약 80%로, 한 사람이 2~3개의 충치를 갖고 있는 셈이며, 구강 보건상태는 도시, 농촌지역, 해안지역 순으로 좋다. 원인은 치태(dental plaque)로서, 이것은 점액이나 탈락한 잇몸의 세포들, 그리고 세균 등으로 이루어져 있으며, 점착성이 강하여 치아 표면에 잘 부착되어 양치질이나 칫솔질로는 쉽게 제거되지 않는다. 치태에는 그물모양의 구조내에 막대균과 알균이 섞여 있는데, 특히 내산성 사슬알균과 젖산균이 많다. 현재 가장 유력한 충치의 원인균으로 밝혀지고 있는 이 사슬알균은 설탕을 합성하는 능력을 가지고 있으며, 덱스트란(dextran)을 형성한다고 생각되는데, 이 덱스트란은 물에 녹지 않으며 끈적거리는 점착성이 있어 치태가 치아표면에 잘 붙어 있게 한다. 이 사슬알균은 설탕으로부터 유기산과 치태를 형성한다. 치태 중에는 사슬알균이 생산하는 산이 정체되어 있기 때문에, 이 산에 의해 치아 표면의 단단한 석회질을 녹인다. 대부분이 인산칼슘으로 구성되어 있는 치아의 표면은 산에 의해 칼슘이 분해되어 치아 표면이 거칠어지면서 충치가 진행된다. 이와 같이 세균과 이 세균이 생산하는 화학적 물질에 의해서 충치가 생긴다는 가설을 세균-화학설이라고 한다. 충치의 발생원인에는 이 세균-화학설 이외에도 미생물이 치아표면의 유기질 부위에 먼저 부착되어 산을 만들어 파괴시킨다는 단백용해설도 있으며, 또한 산에 의하지 않는 치아의 파괴, 음식물이 원인이 된다고 주장하는 학설도 있다. 설탕은 충치를 가장 잘 유발시키는 물질로, 섭취 빈도에 따라 충치가 증가한다는 것이 증명되고 있다. 비타민은 충치 요인에 그리 중요하지 않은 것으로 보이며 음식물 내에 존재하는 칼슘과 인 또한 중요하지 않다고 한다. 유전적 소인은 중요한 역할을 하리라 생각되지만 증명하기가 어렵다. |
||
| Dent, dent | dentistry, dentist, dental, dentition |
|---|---|
| FDP(s) | 1) Fibrinolytic split Products(= FSP) 2) Fibrinogen Degradation Products |
| FSP | familial spastic paraplegia; fibrin split products; fibrinogen split products; fine suspended partic... |
| DENT | Dental Exposure Normalization Technique |
| DDS | damaged disc syndrome; dendrodendritic synaptosome; dental distress syndrome; depressed DNA synthesi... |
| GBI | Gingival Bleeding Index |
|---|---|
| GCF | Gingival Crevicular Fluid |
| GF | Gingival fibroblasts |
| GO | Gingival overgrowth |
| GR | Gingival recession |
dentagra
| dent | 1. <prefix> Relating to the teeth or dentition, dental. See: odonto-. Origin: L. Dens, tooth 2. A slight depression, or small notch or hollow, made by a blow or by pressure; an indentation. "A blow that would have made a dent in a pound of butter." (De Quincey) Origin: A variant of Dint. Source: Websters Dictionary (21 Jun 2000) |
|---|---|
| superior gingival branches of superior dental plexus | Branches of superior dental plexus to gingiva of upper jaw. Synonym: rami gingivales superiores plexus dentalis superioris. (05 Mar 2000) |
| inferior gingival branches of inferior dental plexus | Branches of inferior dental plexus to the gingiva of the lower jaw. Synonym: rami gingivales inferiores plexus dentalis inferioris. (05 Mar 2000) |
| superior dental branches of superior dental plexus | Branches passing from the superior dental plexus to the roots of the teeth of the upper jaw. Synonym: rami dentales superiores plexus dentalis superioris. (05 Mar 2000) |
| inferior dental branches of inferior dental plexus | Branches passing from the inferior dental plexus to the roots of the teeth of the lower jaw. Synonym: rami dentales inferiores plexus dentalis inferioris. (05 Mar 2000) |
| biological products | Complex pharmaceutical substances, preparations, or agents of organic origin, usually obtained by biological methods or assay, that depend for their action on the processes affecting immunity. They are used especially in diagnosis and treatment of disease (as vaccines or pollen extracts). Biological products are differentiated from biological factors in that the latter are compounds with biological or physiological activity made by living organisms. (12 Dec 1998) |
| blood products | Biopharmaceutical products purified from human blood, such as the blood clotting factor VIII used to treat haemophiliacs. (Recombinant factor VIII is also on the market.) The term also refers to biopharmaceuticals that act on blood or the cells that make blood. These products are often produced by the cells themselves, but in such tiny amounts that extracting them from blood is impractical, and so they are genetically engineered. (14 Nov 1997) |
| gene products, env | Retroviral proteins, often glycosylated, coded by the envelope (env) gene. They are usually synthesised as protein precursors (polyproteins) and later cleaved into the final products by a viral protease. (12 Dec 1998) |
| gene products, gag | Proteins coded by the retroviral gag gene. The products are usually synthesised as protein precursors or polyproteins, which are then cleaved by viral proteases to yield the final products. Many of the final products are associated with the nucleoprotein core of the virion. Gag is short for group-specific antigen. (12 Dec 1998) |
| gene products, nef | Products of the HIV nef gene (formerly 3'-orf gene). The products trans-suppress viral replication and function as negative regulators of transcription. Nef stands for negative factor. (12 Dec 1998) |
| gene products, pol | Retroviral proteins coded by the pol gene. Often synthesised as a gag-pol fusion protein (fusion proteins, gag-pol) and later cleaved into final products that include reverse transcriptase, endonuclease/integrase, and viral protease. Pol is short for polymerase, the enzyme class of reverse transcriptase. (12 Dec 1998) |
| gene products, rev | Trans-acting nuclear proteins whose functional expression are required for HIV viral replication. Specifically, the rev gene products are required for processing and translation of the HIV gag and env mRNAs, and thus rev regulates the expression of the viral structural proteins. Rev can also regulate viral regulatory proteins. A cis-acting antirepression sequence (car) in env, also known as the rev-responsive element (rre), is responsive to the rev gene product. Rev is short for regulator of virion. (12 Dec 1998) |
| gene products, rex | Post-transcriptional regulatory proteins required for the accumulation of mRNAs that encode the gag and env gene products in HTLV-I and HTLV-II. The rex (regulator x; x is undefined) products act by binding to elements in the ltr. (12 Dec 1998) |
| gene products, tat | Trans-acting transcription factors. Nuclear proteins whose expression is required for HIV viral replication. The tat protein stimulates HIV-ltr-driven RNA synthesis for both viral regulatory and viral structural proteins. Tat stands for trans-activation of transcription. (12 Dec 1998) |
| gene products, tax | Transcriptional trans-acting proteins of the promoter elements found in the long-terminal repeats (ltr) of HTLV-I and HTLV-II. The tax (trans-activator x; x is undefined) proteins act by binding to enhancer elements in the ltr. (12 Dec 1998) |
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|