| ¿µ¹® | senile dementia | ÇÑ±Û | ³ëÀÎÄ¡¸Å |
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| ¿µ¹® | dementia | ÇÑ±Û | Ä¡¸Å |
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| ¼³¸í | ³úÀÇ ±âÁúÀû Àå¾Ö¿¡ ÀÇÇÏ¿© ÈÄõÀûÀ¸·Î ÀϾ´Â ȸº¹ºÒ´ÉÀÇ Áö´ÉÀå¾Ö·Î¼, ÀǽÄÀå¾Ö¿¡ ÀÇÇÑ °ÍÀÌ ¾Æ´Ñ °ÍÀ» ¸»ÇÑ´Ù. Áö´É, ÀÇÁö, ±â¾ï µî Á¤½ÅÀûÀÎ ´É·ÂÀÌ µÎµå·¯Áö°Ô °¨ÅðÇÑ »óÅ·μ Á¤½Å¹Ú¾à°ú °°Àº Áö´É Àå¾ÖÀε¥, Á¤½Å¹Ú¾àÀÌ ÁÖ·Î Áö´É¹ßÀ°ÀÌ ÁöüµÇ°Å³ª Á¤ÁöµÈ °ÍÀÎ µ¥ ºñÇÏ¿© Ä¡¸Å´Â ÀÏ´Ü ¹ß´ÞµÈ Áö´ÉÀÌ ´ë³úÀÇ ÁúȯÀ¸·Î ÀÎÇØ Áö¼ÓÀûÀ¸·Î ÀúÇÏµÈ »óŶó´Â Á¡¿¡¼ ±¸º°µÈ´Ù. °ú°ÅÀÇ Ã¼Çè¿¡ ÀÇÇÑ ±â¾ïÀ̳ª Áö½ÄÀÌ ´ÜÆíÀûÀ¸·Î ³²¾Æ ÀÖ°í, ÀüüÀûÀ¸·Î ÀÏÁ¤ÇÏÁö ¾ÊÀº Áö´ÉÀå¾Ö°¡ ³ªÅ¸³´Ù. |
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| ¿µ¹® | infectious disease | ÇÑ±Û | °¨¿°º´ |
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| ¿µ¹® | hypertensive heart disease | ÇÑ±Û | °íÇ÷¾Ð½ÉÀ庴 |
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| ¿µ¹® | pelvic inflammatory disease | ÇÑ±Û | °ñ¹Ý¿°Áúȯ |
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| HD | Haab-Dimmer [syndrome]; Hajna-Damon [broth]; Hansen disease; hearing distance; heart disease; helix ... |
|---|---|
| AD | accident dispensary; acetate dialysis; active disease; acute dermatomyositis; addict, addiction; ade... |
| PD | Doctor of Pharmacy; Dublin Pharmacopoeia; interpupillary distance; Paget disease; pancreatic duct; p... |
| CD | cadaver donor; canine distemper; canine dose; carbohydrate dehydratase; carbon dioxide; cardiac dise... |
| MD | Doctor of Medicine [Lat. Medicinae Doctor]; magnesium deficiency; main duct; maintenance dose; major... |
| UHDRS | Unified Huntington Disease Rating Scale |
|---|---|
| HC | Huntington chorea |
| MID | 6-multiinfarct dementia |
| ADC | AIDS dementia complex |
| AD | Alzheimer Dementia |
Kugelberg-Welander disease ±Ù À§ÃàÁõÀÇ À¯Àü¼º ¿¬¼ÒÇüÀ¸·Î¼ º¸Åë »ó¿°»öü¼º ¿¼º ÇüÁú·Î À¯ÀüµÈ´Ù. ô¼ö Àü°¢ÀÇ º´º¯ÀÌ ±× ¿øÀÎÀÌ´Ù.
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| Huntington disease | <neurology> An inherited adult-onset disease of the central nervous system. It is characterised by dementia and bizarre involuntary movements. The disease is progressive and there is currently no known cure. The identification of the gene (huntingtin) on chromosome 4p now allows for direct mutation analysis. The gene contains a trinucleotide repeat (CAG) that is found to be expanded in length in affected patients. The normal allele size ranges from 11 to 34 triplet repeat units, while 42 repeats or greater is considered diagnostic of Huntington disease. As in other trinucleotide repeat disorders, the phenomenom of anticipation has been observed, in this case expressed as earlier age of onset in offspring, particularly with paternal transmission. A further complication is the presence of two neighboring trinucleotide repeats (both CCG) which can expand independently without causing the disease. Early PCR primer sets encompassed these adjacent repeats, potentially yielding false positive test results, newer primers hone in more closely on the CAG repeat sequence. Careful attention must be paid to the psychosocial support structure of prospective test subjects in Huntington disease genetic counseling. Established protocols require systematic neuropsychiatric assessment and informed consent prior to DNA testing. Inheritance: autosomal dominant. (29 Dec 1997) |
|---|---|
| huntington's disease | An hereditary disorder with mental and physical deterioration leading to death. Although characterised as an adult-onset disease (as is usually the case), we have seen children with full-blown huntington's disease. (12 Dec 1998) |
| disease, huntington's | An hereditary disorder with mental and physical deterioration leading to death. Although characterised as an adult-onset disease (as is usually the case), we have seen children with full-blown Huntington's disease. (12 Dec 1998) |
| Huntington | George, U.S. Physician, 1850-1916. See: Huntington's chorea, Huntington's disease. (05 Mar 2000) |
| Huntington chorea | <neurology> An inherited adult-onset disease of the central nervous system. It is characterised by dementia and bizarre involuntary movements. The disease is progressive and there is currently no known cure. The identification of the gene (huntingtin) on chromosome 4p now allows for direct mutation analysis. The gene contains a trinucleotide repeat (CAG) that is found to be expanded in length in affected patients. The normal allele size ranges from 11 to 34 triplet repeat units, while 42 repeats or greater is considered diagnostic of Huntington disease. As in other trinucleotide repeat disorders, the phenomenom of anticipation has been observed, in this case expressed as earlier age of onset in offspring, particularly with paternal transmission. A further complication is the presence of two neighboring trinucleotide repeats (both CCG) which can expand independently without causing the disease. Early PCR primer sets encompassed these adjacent repeats, potentially yielding false positive test results, newer primers hone in more closely on the CAG repeat sequence. Careful attention must be paid to the psychosocial support structure of prospective test subjects in Huntington disease genetic counseling. Established protocols require systematic neuropsychiatric assessment and informed consent prior to DNA testing. Inheritance: autosomal dominant. (29 Dec 1997) |
| Huntington's chorea | A progressive disorder usually beginning in young to middle age, consisting of a triad of choreoathetosis, dementia, and autosomal dominant inheritance with complete penetrance. Bilateral marked wasting of the putamen and the head of the caudate nucleus is characteristic. Synonym: chronic progressive chorea, degenerative chorea, hereditary chorea, Huntington's disease. (05 Mar 2000) |
| AIDS dementia | <immunology> A frequent cerebral condition in people with AIDS that results in the loss of cognitive capacity, affecting the ability to function in a social or occupational setting. Its cause has not been determined exactly, but may result from HIV infection of cells in the brain or an inflammatory reaction to such infection. (09 Oct 1997) |
| AIDS dementia complex | <immunology> A frequent cerebral condition in people with AIDS that results in the loss of cognitive capacity, affecting the ability to function in a social or occupational setting. Its cause has not been determined exactly, but may result from HIV infection of cells in the brain or an inflammatory reaction to such infection. (09 Oct 1997) |
| Alzheimer's dementia | <disease> A progressive, neurodegenerative disease characterised by loss of function and death of nerve cells in several areas of the brain leading to loss of cognitive function such as memory and language. The cause of nerve cell death is unknown but the cells are recognised by the appearance of unusual helical protein filaments in the nerve cells (neurofibrillary tangles) and by degeneration in cortical regions of brain, especially frontal and temporal lobes. Alzheimer's disease is the most common cause of dementia. (22 May 1997) |
| vascular dementia | A state of diminished cognition that is the results from repeated cerebral strokes with a step-like deterioration in intellectual functions with focal neurological signs, as the result of multiple infarctions of the cerebral hemispheres. Synonym: multi-infarct dementia. (07 Mar 2000) |
| paralytic dementia | Dementia and paralysis resulting from a chronic syphilitic meningoencephalitis. Synonym: dementia paralytica. (05 Mar 2000) |
| catatonic dementia | Dementia with catatonic symptoms. (05 Mar 2000) |
| multi-infarct dementia | <neurology> This form of dementia is caused by a number of strokes in the brain. These strokes can cause specific symptoms, depending on their severity and location and can cause general symptoms of dementia. MID cannot be treated, once the nerve cells die, they cannot be replaced. However, the underlying condition leading to strokes (for example, high blood pressure, diabetes) can be treated, which may help prevent further damage. Synonym: vascular dementia. (22 May 1997) |
| presenile dementia | Dementia of Alzheimer's disease developing before age 65. Synonym: Alzheimer's disease. Primary dementia, dementia occurring independently as a mental disorder. (05 Mar 2000) |
| primary senile dementia | <disease> A progressive, neurodegenerative disease characterised by loss of function and death of nerve cells in several areas of the brain leading to loss of cognitive function such as memory and language. The cause of nerve cell death is unknown but the cells are recognised by the appearance of unusual helical protein filaments in the nerve cells (neurofibrillary tangles) and by degeneration in cortical regions of brain, especially frontal and temporal lobes. Alzheimer's disease is the most common cause of dementia. (22 May 1997) |
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