| 영문 | endocrine gland | 한글 | 내분비샘 |
|---|---|---|---|
| 설명 | 신체의 내부로 분비가 되어서 다른 세포의 기능이나 활성의 변화를 일으키는 물질인 호르몬을 분비하는 곳을 이르는 말이다. 대표적인 내분비샘은 갑상샘, 부갑상샘, 부신, 이자, 뇌하수체, 난소, 고환, 솔방울샘, 가슴샘 등이다. 마지막에 나오는 2개의 기관은 내분비 기능을 하는 것으로 생각이 되지만 확실히 그것이 무슨 기능을 하는 지에 대해선 아직까지 정확하게 밝혀지지 않고 있다.  |
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| 영문 | congenital syphilis | 한글 | 선천매독 |
|---|---|---|---|
| 설명 | 임부가 매독에 감염되어 있으면 임신 후기에 매독균이 태반을 통해 혈행성으로 태아에 감염(수직감염)된 것을 말하다. 대부분은 유산, 사산이 되지만 출생하면 제2기 이후의 발진을 보인다. 발현시기에 따라서 ① 태아매독, ② 유아매독, ③ 만발성 선천매독으로 분류된다. ①에서는 뼈연골염, 간-지라 비대와 매독성 천포창, ②에서는 파로가성마비와 매독성 코염, ③에서는 허친슨 세징후(허친슨 치아, 속귀성 난청, 실질성 각막염)에 따라 특징이 있다. 기타 수두증, 지능발육 불량 등을 자주 볼 수 있다. 매독 혈청반응은 대부분의 경우 양성으로 나온다. 매우 드물게 간세포내에서 매독균을 무수히 볼 수 있다. 간세포 주변의 섬유화와 함께 불규칙한 흉터(hepar lobatum)를 만들 수 있다. |
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| 영문 | congenital rubella syndrome | 한글 | 선천풍진증후군 |
|---|---|---|---|
| 설명 | 임신기간 중에 산모가 풍진에 걸리면 이 풍진 바이러스는 태반을 통해서 태아에게 전달되어서 태아의 풍진감염을 일으킨다. 임신 첫 3개월 동안, 특히 임신 첫달에 태아가 풍진의 감염을 받으면, 신생아에서 선천기형, 즉 눈에서 촛점을 정확히 맞추어주는 렌즈의 역할을 하는 수정체의 혼탁(백내장), 심장기형, 귀머거리 및 심한 지능박약을 동반하는 소두증 등이 발생하는 수가 많다. |
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| 영문 | congenital heart disease | 한글 | 선천심장병 |
|---|---|---|---|
| 설명 | 선천적으로 심장의 구조에 이상이 있는 병. |
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| 영문 | lacrimal glands | 한글 | 눈물샘 |
|---|---|---|---|
| 설명 | 눈물을 만드는 곳이다. 눈물은 눈의 건조를 막고 이물질을 씻어내리는 역할을 한다. 눈물은 대개 눈물샘에서 만들어져 눈을 적신 뒤 눈물관을 거친뒤에 눈물주머니에 모여서 코눈물관으로 흘러 코 안쪽으로 내려가게 된다. |
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| CHA | Canadian Hospital Association; Catholic Health Association; Chinese hamster; chronic hemolytic anemi... |
|---|---|
| MEN | Multiple Endocrine Neoplasia ; AD Trait 1. MEN Type I(= Wermer Syndro... |
| CDH | 1) Chronic Daily Headache = CTH = ... |
| CDH | ceramide dihexoside; congenital diaphragmatic hernia; congenital dislocation of hip; congenital dysp... |
| POSSUM | Pictures of Standard Syndromes and Undiagnosed Malformations |
| C.M. | Congenital Malformations |
|---|---|
| APs | Anterior pituitary glands |
| MSG | Minor salivary glands |
| SMSG | Submandibular Salivary Glands |
| DRO | Differential Reinforcement of Other Behaviour |
| cerebral arteriovenous malformations | Vascular anomalies in which tangles of arteries are connected directly to veins without intervening capillaries. The resulting vessels are thin-walled owing to poorly developed elastic and muscle tissue within the media. They can be located anywhere in the brain and can produce headaches, seizures, focal neurologic deficits, or intracranial haemorrhage. Familial cases are rare, indicating that the problem reflects sporadic abnormalities in embryologic development. (12 Dec 1998) |
|---|---|
| other-directed | Pertaining to a person readily influenced by the attitudes of others. (05 Mar 2000) |
| transferases (other substituted phosphate groups) | <enzyme> A class of enzymes that transfers substituted phosphate groups. Registry number: EC 2.7.8 (12 Dec 1998) |
| endocrine glands | Ductless glands that secrete substances which are released directly into the circulation and which influence metabolism and other body functions. (12 Dec 1998) |
| bone diseases, endocrine | Diseases of the bones related to hyperfunction or hypofunction of the endocrine glands. (12 Dec 1998) |
| multiple endocrine adenomatosis | The presence of functioning tumours in more than one endocrine gland, commonly the pancreatic islets and parathyroid glands, which may be associated with Zollinger-Ellison syndrome; dominant inheritance. Synonym: multiple endocrine adenomatosis. (05 Mar 2000) |
| multiple endocrine deficiency syndrome | <syndrome> Acquired deficiency of the function of several endocrine glands, usually on an auto-immune basis. Synonym: multiple glandular deficiency syndrome. (05 Mar 2000) |
| multiple endocrine neoplasia | (type I) This is a hereditary disorder in which two or more of the following glands: parathyroid, pancreas, pituitary, adrenals or thyroid develop hyperplasia or a tumour. (type II) This is a hereditary disorder in which two or more of the following glands: thyroid, adrenal or parathyroid, develop overgrowth (hyperplasia) or malignant cells (cancer). The underlying cause is genetic and a positive family history for this illness is a risk factor. Incidence: approximately 3 in 100,000 people in the general population. Origin: Gr. Plassein = to form (27 Sep 1997) |
| multiple endocrine neoplasia 1 | <radiology> Multiple endrocrine neoplasia syndrome three P's. Pituitary adenoma, 65% can develop Cushing's, acromegaly, prolactinoma, parathyroid hyperplasia / adenoma, 88% can develop hyper-PTH pancreatic isleT-cell tumour, gastrinoma (Z-E) most common, 50% of Z-E can develop MEN-1, inconstant features: bronchial/intestinal carcinoid, thyroid adenoma, adrenal cortical tumour, lipoma, thymoma tissue expression Primary hyperparathyroidism (90%), Gastrinoma (30%), Prolactinoma (15%), Other (10%). Synonym: Wermer syndrome (12 Dec 1998) |
| multiple endocrine neoplasia 2 | <radiology> Multiple endocrine neoplasia syndrome, medullary thyroid carcinoma, usually multifocal; metastasis to local nodes, lung, liver, usually calcify in liver, pheochromocytoma, almost always bilateral, parathyroid hyperplasia, may be secondary to calcitonin secreted by medullary thyroid carcinoma inconstant feature: adrenal cortical hyperplasia Synonym: Sipple syndrome (12 Dec 1998) |
| multiple endocrine neoplasia 3 | <radiology> Multiple endocrine neoplasia syndrome (type 2B, type 3), medullary thyroid carcinoma, pheochromocytoma, marfanoid habitus (Cf: Marfan syndrome), mucosal neuromas, neurofibromas, ganglioneuromatosis coli More info: MEN syndrome 2B Synonym: Schimke, marfanoid syndrome (12 Dec 1998) |
| multiple endocrine neoplasia type 1 | A rare syndrome characterised by hyperplasia and/or neoplasms of the pituitary, parathyroid glands, and pancreatic islets. Hyperparathyroidism occurs in 90% of the cases and is usually the first manifestation of the syndrome. The most frequent pancreatic manifestation is gastrinoma typically leading to zollinger-ellison syndrome. The appearance of this condition has been limited to the loss of allelic heterozygosity at the 11q13 locus on the long arm of chromosome 11. Patients overall exhibit long survival times. Chemotherapy is rare and surgical management is generally dependent on the genetic expression in individual patients. (12 Dec 1998) |
| multiple endocrine neoplasia type 2 | <syndrome> This is a hereditary disorder in which two or more of the following glands: thyroid, adrenal or parathyroid, develop overgrowth (hyperplasia) or malignant cells (cancer). The underlying cause is genetic and a positive family history for this illness is a risk factor. Incidence: approximately 3 in 100,000 people in the general population. (27 Sep 1997) |
| multiple endocrine neoplasia type 2a | A type of multiple endocrine neoplasia characterised by a virtually 100% incidence of medullary thyroid carcinoma, a 50% incidence of pheochromocytoma, and a lesser incidence of parathyroid adenomas associated with hyperparathyroidism. The condition is always transmitted through autosomal dominant inheritance. Genetic testing can identify individuals with the trait in early infancy. Treatment is usually excision of the enlarged parathyroid glands. (12 Dec 1998) |
| multiple endocrine neoplasia type 2b | A type of multiple endocrine neoplasia occurring as an isolated congenital presentation or as a distinct autosomal dominant disease. It is characterised by the 100% incidence of medullary thyroid carcinoma and frequent pheochromocytomas; patients seldom exhibit hyperparathyroidism. It is distinguished from men 2a by its characteristic physical appearance resulting from numerous neural defects including mucosal neuromas of the eyelids, lips, and tongue. The neural abnormalities also include widespread neurogangliomatosis of the gastrointestinal tract leading to abnormal gut motility. Treatment usually requires total thyroidectomy following evaluation for the presence of pheochromocytomas. (12 Dec 1998) |
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|
제품명 |
판매사 |
보험코드 | 성분/함량 | 구분/보험급여 |
|---|