| ¿µ¹® | atherosclerosis | ÇÑ±Û | Á׻󵿸ưæÈÁõ |
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| ¼³¸í | Á׻󵿸ưæÈÁõÀ̶õ ´ëµ¿¸Æ°°Àº ź·Â¼º µ¿¸ÆÀÇ ³»Ãþ¿¡ Áö¹æ°ú ÄÝ·¹½ºÅ×·Ñ·Î ÀÌ·ç¾îÁø Á×Á¾ÀÌ »ý±â´Â º´ÅÂÀÌ´Ù. Á×Á¾ÀÌ »ý±ä µ¿¸ÆÀº Á×Á¾ÀÌ Ç÷°üÀÇ ³»ºÎ·Î µ¹ÃâÀ» ÇÏ°Ô µÇ¾î¼ Ç÷°üÀÇ ³»°æÀÌ ÀÛ¾ÆÁö°Ô µÈ´Ù. ÀÌ·Î ÀÎÇØ¼ Ç÷·ù°¡ °¨¼ÒÇÏ°Ô µÇ°í °è¼Ó ÁøÇàÀÌ µÉ °æ¿ì¿¡´Â ¿ÏÀüÈ÷ µ¿¸ÆÀ» ¸·À» ¼öµµ ÀÖ´Ù. ÁÖ·Î Áß°£Å©±âÀÇ µ¿¸Æ¿¡ ¸¹ÀÌ »ý±â¸ç ´ë°³ ½ÄÀÌ¿Í Ç÷Áß¿¡ ÀÖ´Â Áö¹æÀÇ ³óµµ, °íÇ÷¾Ð, ±×¸®°í À¯Àü°ú °ü·ÃÀÌ ÀÖ´Ù°í ÇÑ´Ù. |
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| ¿µ¹® | renal biopsy | ÇÑ±Û | ÄáÆÏ»ý°Ë |
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| ¼³¸í | ÄáÆÏÀÇ º´º¯ÀÌ ÀÇ½ÉµÉ ¶§ È®ÁøÀ» À§ÇØ ÁÖ»ç¹Ù´Ã µîÀ» ÀÌ¿ëÇÏ¿© ÄáÆÏÁ¶Á÷À» ÀϺΠ¶¼¾î³»¼ Çö¹Ì°æÀ¸·Î °Ë°æÇÏ´Â °Í. |
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| ¿µ¹® | renal hypertension | ÇÑ±Û | ÄáÆÏ¼º°íÇ÷¾Ð |
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| ¿µ¹® | renal cell carcinoma | ÇÑ±Û | ÄáÆÏ¼¼Æ÷¾ÏÁ¾ |
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| ¼³¸í | ÄáÆÏ¿¡ »ý±ä ¿ø½ÃÄáÆÏÁ¶Á÷¿¡¼ ¹ß»ýÇÑ ¾Ï. ÁÖ·Î ¿ø½Ã¼¼´¢°üÁ¶Á÷¿¡¼ ¹ß»ýÇÑ´Ù. ´ëÇ¥ÀûÀÎ ¼¼Æ÷Á¶Á÷ÇüÀº ¿°»ö½Ã ¼¼Æ÷ÁúÀÌ ¸¼°Ô ºñ¾îº¸ÀÌ´Â ¸¼Àº¼¼Æ÷¾ÏÁ¾ÀÌ´Ù. Ä¡·á´Â ¼ö¼ú°ú Ç×¾ÏÈÇпä¹ýÀÌ¸ç ¾ÆÁÖ µå¹°Áö¸¸ ÀúÀý·Î ³´´Â °æ¿ìµµ ÀÖ´Â °ÍÀ¸·Î º¸°íµÇ¾î ÀÖ´Ù. |
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| ¿µ¹® | renal transplantation | ÇÑ±Û | ÄáÆÏÀÌ½Ä |
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| ¼³¸í | ÄáÆÏº´À» °¡Áö°í ÀÖÀ¸³ª Ä¡·á°¡ ºÒ°¡´ÉÇÑ ¸¸¼ºÄáÆÏ±â´É»ó½Ç µîÀÇ Áúº´À» °¡Áø ȯÀÚÀÇ ½ÅÀåÀ» ¶¼¾î³»°í ȯÀÚ¿Í Ç׿ø¼ºÀÌ À¯»çÇÑ »ç¶÷ÀÇ ÄáÆÏÀ» À̽ÄÇØÁÖ´Â °Í. ÀÌ ¶§ ¼·Î°£ÀÇ Ç׿ø¼ºÀÇ À¯»çÁ¡ÀÌ ¸¹¾Æ¾ß °ÅºÎ¹ÝÀÀÀÌ ÀϾÁö ¾Ê´Â´Ù. ±×¸®°í ÀÏ´Ü ÄáÆÏÀ̽ÄÀ» ¹ÞÀº »ç¶÷Àº ¿À·£±â°£ µ¿¾È ¸é¿ª¾ïÁ¦Á¦¸¦ Åõ¿©ÇÏ¿© °ÅºÎ¹ÝÀÀÀ» ÁÙ¿©¾ß ÇÑ´Ù. ´ë°³ ÀÌ½ÄµÈ ÄáÆÏÀº ¾ûµ¢»À¿À¸ñ¿¡ À§Ä¡ÇÏ°Ô µÈ´Ù. |
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| ECG | Electro-Cardio-Graphy(-Gram); ½ÉÀüµµ = EKG 1. Conducting System Structu... |
|---|---|
| CAS | calcarine sulcus; calcific aortic stenosis; Cancer Attitude Survey; carbohydrate-active steroid; car... |
| Athsc | atherosclerosis |
| VHAS | Verapamil in Hypertension Atherosclerosis Study |
| RCA | red cell agglutination; relative chemotactic activity; renal cell carcinoma; right carotid artery; r... |
| ACAS | Asymptomatic Carotid Atherosclerosis Study |
|---|---|
| AS | Atherosclerosis |
| ARIC | Atherosclerosis Risk In Communities |
| CLAS | Cholesterol Lowering Atherosclerosis Study |
| ELSA | EUROPEAN LACIDIPINE STUDY ON ATHEROSCLEROSIS |
| atherosclerosis | <cardiology, pathology> The progressive narrowing and hardening of the arteries over time. This is known to occur to some degree with aging, but other risk factors that accelerate this process have been identified. These factors include: high cholesterol, high blood pressure, smoking, diabetes and family history for atherosclerotic disease. (27 Sep 1997) |
|---|---|
| capsular branches of renal artery | <anatomy, artery> Branches arising from the renal artery outside of the kidney that are distributed to the renal capsule. Synonym: rami capsulares arteriae renalis. (05 Mar 2000) |
| renal artery | <anatomy, artery> A branch of the abdominal aorta which supplies the kidneys, adrenal glands and ureters. (12 Dec 1998) |
| renal artery obstruction | Narrowing or occlusion of the renal arteries. It is due usually to atherosclerosis, fibromuscular dysplasia, thrombosis, embolism, or external pressure. It may result in renovascular hypertension. (12 Dec 1998) |
| renal artery stenosis | <cardiology, nephrology> A narrowing of the renal artery or one of its main branches accounts for 2 to 5% of cases of hypertension. (27 Sep 1997) |
| posterior branch of renal artery | <anatomy, artery> Terminal branch of renal artery (with anterior branch) becoming the posterior segmental artery of kidney. Synonym: ramus posterior arteriae renalis. (05 Mar 2000) |
| ureteric branches of the renal artery | <anatomy, artery> Supplies ureteric (renal) pelvis and superior portion of ureter. Synonym: rami ureterici arteriae renalis. (05 Mar 2000) |
| anastomotic branch of middle meningeal artery to lacrimal artery | <anatomy, artery> A branch of the middle meningeal artery arising in the cranial cavity which runs anteriorly through the superior orbital fissure to anastomose with the lacrimal artery. See: orbital branch of middle meningeal artery. Synonym: ramus anastomoticus arteriae meningeae mediae cum lacrimali. (05 Mar 2000) |
| acute renal failure | <nephrology> A sudden decline in renal function may be triggered by a number of acute disease processes. Examples include sepsis (infection), shock, trauma, kidney stones, kidney infection, drug toxicity (aspirin or lithium), poisons or toxins (drug abuse) or after injection with an iodinated contrast dye (adverse effect). Chronic renal failure represents a slow decline in kidney function over time. Chronic renal failure may be caused by a number of disorders which include long-standing hypertension, diabetes, congestive heart failure, lupus or sickle cell anaemia. Both forms of renal failure result in a life-threatening metabolic derangement. (27 Sep 1997) |
| aminoaciduria, renal | Impairment of renal tubular transport of amino acids. (12 Dec 1998) |
| back-pressure renal atrophy | <radiology> Caliectasis without obstruction, due to repeated episodes of obstruction, gradual loss of renal pyramids (12 Dec 1998) |
| base of renal pyramid | The outer broad part of a renal pyramid that lies next to the cortex. Synonym: basis pyramidis renis. (05 Mar 2000) |
| branchio-oto-renal syndrome | <syndrome> An autosomal dominant disorder manifested by various combinations of preauricular pits, branchial fistulae or cysts, lacrimal duct stenosis, hearing loss, structural defects of the outer, middle, or inner ear, and renal dysplasia. Associated defects include asthenic habitus, long narrow facies, constricted palate, deep overbite, and myopia. Hearing loss may be due to mondini type cochlear defect and stapes fixation. (12 Dec 1998) |
| captopril renal scan | <radiology> In a kidney with a lesion in the afferent arteriole (e.g. Atherosclerotic plaque), reflex constriction of the efferent arteriole occurs through angiotensin system thus maintaining renal perfusion. ACE inhibition prevents constriction of efferent arteriole. Therefore, perfusion is decreased to a kidney with afferent lesions and the renal scan to looks WORSE. Bottom line: renal scans appear WORSE with captopril administration if there is a lesion in the afferent arteriole. See: renal artery stenosis (12 Dec 1998) |
| carcinoma, renal cell | Carcinoma of the renal parenchyma usually occurring in middle age or later and composed of tubular cells in varying arrangements. It was first described in 1826. Possible causal factors are environmental, hormonal, cellular, and genetic. Smoking is a definite risk factor and obesity is associated with increased risk. Renal cell carcinoma accounts for approximately 3% of adult cancer; the male-female ratio is 2:1. It is more common among urban residents than rural. (12 Dec 1998) |
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