| ¿µ¹® | heart failure | ÇÑ±Û | ½ÉÀå±â´É»ó½Ç |
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| ¿µ¹® | congestive heart failure | ÇÑ±Û | ¿ïÇ÷¼º½ÉÀå±â´É»ó½Ç |
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| ¼³¸í | ½ÉÀåÀÇ ¿ø·¡ ±â´É, Áï Ç÷¾×À» ¸»ÃÊ·Î º¸³»´Â ÆßÇÁ¿Í °°Àº ±â´ÉÀÌ °ÅÀÇ ¾ø¾îÁø »óŸ¦ ¸»ÇÑ´Ù. ÇǸ¦ º¸³»´Â ÆßÇÁÀÇ ¿ªÇÒÀÌ ¸ØÃß¾úÀ¸¹Ç·Î ½ÉÀåÀ¸·Î µé¾î¿À´Â ÇÇ´Â ½ÉÀåÀ¸·Î µé¾î¿ÀÁö ¸øÇϰí Á¤¸Æ¼Ó¿¡¼ Á¤Ã¼ÇÏ°Ô µÈ´Ù. ±×¸®ÇÏ¿© Ç÷¾×ÀÇ ´ëºÎºÐÀÌ ¸»ÃÊÀÇ Á¤¸Æ¿¡ ¸Ó¹°°Ô µÇ¾î ¿©·¯ °¡Áö Áõ»óÀÌ ³ªÅ¸³´Ù. ƯÈ÷ ÇãÆÄ¸¦ µ¹¾Æ Á½ɹæÀ¸·Î µé¾î°¡¾ßÇÏ´Â Çǰ¡ ÆßÇÁ ±â´ÉÀÌ ¾ø¾îÁ®¼ ¿Þ½É¹æ¿¡ ¸¹Àº Çǰ¡ Á¤Ã¼Çϰí ÀÖ¾î¼ µé¾î°¡Áö ¸øÇؼ ÇãÆÄ¿¡ °íÀÌ°Ô µÈ´Ù. ±×·¯¸é ÇãÆÄ¿¡ »ê¼Ò¿Í ÀÌ»êÈź¼Ò¸¦ ±³È¯ÇÏ´Â ±â´ÉÀÌ ¾ø¾îÁ®¼ ¼ûÀ» ½¬Áö ¸øÇÏ´Â °á°ú¸¦ ³½´Ù. Áï ¼ûÀÌ Â÷°í ¼û½¬±â Èûµç Áõ¼¼°¡ ³ªÅ¸³´Ù. ¶Ç ¿Â¸öÀ» ¼øÈ¯ÇÑ ´ÙÀ½¿¡ ¿ì½É¹æÀ¸·Î µé¾î¿Í¾ßÇÏ´Â Çǵµ ¿ì½É¹æÀ¸·Î µé¾î¿ÀÁö ¸øÇؼ ¸»ÃÊ¿¡ Á¤Ã¼ÇϹǷΠ¿Â¸öÀÌ º×°Ô µÈ´Ù. |
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| ¿µ¹® | necrosis | ÇÑ±Û | ±«»ç |
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| ¼³¸í | ¼¼Æ÷°¡ Á×´Â °ÍÀ» ¸»ÇÑ´Ù. ¿øÀÎÀº ¾î¶² ¿Ü»óÀÏ ¼öµµ ÀÖ°í, ȤÀº µ¿¸ÆÀÌ Á¼¾ÆÁ®¼ Çǰ¡ ÅëÇÏÁö ¾Ê¾Æ Á×°Ô µÇ´Â °ÍÀÏ ¼öµµ ÀÖ´Ù. ¾î¶² °æ¿ìÀ̵ç, ±«»çµÈ Á¶Á÷ÀÌ Ã¼³»¿¡ ÀÖÀ¸¸é, °á±¹Àº ¿©·¯ ÇÕº´ÁõÀ» ºÒ·¯ ÀÏÀ¸Å°¹Ç·Î Á¦°ÅµÇ¾î¾ß ÇÑ´Ù. ±«Àú±«»ç: gangrenous necrosis ±«Àú±«»ç¶ó´Â ¿ë¾î´Â º¸Åë ±«»ç¶ó´Â °Í°ú Å©°Ô ´Ù¸¥ °ÍÀº ¾øÀ¸³ª ÀÓ»ó, ƯÈ÷ ¿Ü°ú¿¡¼ ÈçÈ÷ ¾²°í ÀÖ´Â ¸»ÀÌ´Ù. ÀÌ´Â ÆÈ´Ù¸®, ƯÈ÷ ´Ù¸®¿¡¼ ¸¹ÀÌ »ý±â´Âµ¥ Ç÷¾×°ø±ÞÀÌ ¼Ò½ÇµÇ°í ±× ÈÄ¿¡ ¼¼±Õ°¨¿°À» ¹Þ¾Æ¼ »ý±â´Â º´ÅÍÀÌ´Ù. ¸¸¾à Ç÷·ù°ø±ÞÀÇ Â÷´Ü¿¡ ÀÇÇÑ Á¶Á÷ÀÇ Á×À½ÀÌ ÁÖº´º¯À̰í ÀÌÂ÷Àû ¼¼±ÕÀÇ °¨¿°ÀÌ Àû°í, ÀÖ´õ¶óµµ ¾ÆÁÖ ¹Ì¾àÇØ¼ º´ÅͰ¡ ¸¶¸£°í ¾×ü¼ººÐÀÌ ÀûÀ» °æ¿ì¿¡ À̰ÍÀ» °Ç¼º±«Àú(dry gangrene)À̶ó°í Çϰí, ¼¼±ÕÀÇ °¨¿°ÀÌ ¾ÆÁÖ ¸¹¾Æ¼ Á×Àº Á¶Á÷ÀÌ ºÐÇØ°¡ µÇ¾î¼ ¾×ü¼ººÐÀÌ ±«ÀúÀÇ Á¶Á÷¿¡ ¸¹ÀÌ ÀÖ¾î¼ º¸±â¿¡ ½ÀÇØ º¸À̸é À̰ÍÀ» ½À¼º±«Àú(wet gangrene)À̶ó°í ÇÑ´Ù. |
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| ¿µ¹® | fibrinoid necrosis | ÇÑ±Û | ¼¶À¯¼Ò¼º±«»ç |
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| ¼³¸í | ÃʱâÀÇ ·ù¸¶Æ¼½º ȤÀº ¾Ç¼ºÁ¾¾ç µî¿¡¼ º¼ ¼ö ÀÖ´Â Ç÷°ü-°áÇÕÁ¶Á÷ÀÇ Æ¯À¯ÇÑ º¯È·Î Ç÷°üº®À̳ª ÁÖÀ§ÀÇ °áÇÕÁ¶Á÷ÀÌ ¼¶À¯¼Ò¸ð¾çÀ¸·Î ±ÕÁúÇÑ ¹°Áú·Î µÇ°í, È£»ê¼ºÀ¸·Î ¿°»öµÈ´Ù. À̰ÍÀº ¼¶À¯¼Ò¿Í °ÅÀÇ ¶È°°Àº ¿°»ö¼ºÀ» ³ªÅ¸³»±â ¶§¹®¿¡ ¿°Áõ¿¡¼ Ç÷°üÀ¸·ÎºÎÅÍ ¼¶À¯¼Ò ¶Ç´Â ¼¶À¯¼Ò¸ð¾çÀÇ ¹°ÁúÀÌ »ïÃâÇÏ¿© °áÇÕÁ¶Á÷ ¼¶À¯°£¿¡ Ä§ÂøÇÏ¿© ±¸Á¶°¡ ºÒ¸í·áÇÑ °ÍÀ¸·Î »ý°¢µÇ¾úÀ¸³ª, ÃÖ±Ù Á¶Á÷ÈÇÐ, ÀüÀÚÇö¹Ì°æ µî¿¡ ÀÇÇÏ¿© °áÇÕ¼¶À¯ÀÚü¿¡µµ º¯È°¡ ÀÖ´Â °ÍÀ¸·Î ¸é¿ª±Û·ÎºÒ¸°ÀÇ Ä§Âøµµ Áõ¸íµÇ°í ÀÖ´Ù. |
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| ¿µ¹® | coagulation necrosis | ÇÑ±Û | ÀÀ°í±«»ç |
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| ¼³¸í | ¼¼Æ÷ÀÇ Á×À½À» ³ªÅ¸³»´Â ÇüÅÂÇÐÀû º¯È·Î¼ ±«»çºÎÀ§°¡ ÀÀ°íÇÑ »óÅ·ΠÀÖ´Â °Í. ±«»ç¼¼Æ÷´Â È£»ê¼º µ¢¾î¸®·Î º¸À̸ç Àû¾îµµ ¼öÀϰ£ ¼¼Æ÷ÀÇ ±âº» À±°ûÀÌ º¸Á¸µÇ±â ¶§¹®¿¡ Á¶Á÷±¸Á¶¸¦ ½Äº°ÇÒ ¼ö ÀÖ´Ù. ´ëÇ¥ÀûÀ¸·Î ÇãÇ÷±«»ç¸¦ µé ¼ö ÀÖ´Ù. °æ»ö°ú ÇãÇ÷¿¡ ÀÇÇÑ Àú»ê¼ÒÁõ½Ã ½ÉÀå±ÙÀ̳ª ÄáÆÏ¿¡¼ °üÂûµÇ°Å³ª Àü±â ¹× ¼öÀºÁßµ¶½Ã ÄáÆÏÀÇ ¿ä¼¼°ü¿¡¼ °üÂûµÇ´Â °ÍÀ¸·Î ´Ü¹éÀÇ º¯¼º ÈÄ ³²¾ÆÀֱ⠶§¹®ÀÌ´Ù. ¼¼Æ÷ÀÇ À±°ûÀÌ ³²¾Æ ÀÖ¾î ¿ø·¡ Á¶Á÷ÀÇ ±¸Á¶¸¦ ÇüÅÂÇÐÀûÀ¸·Î ÀνÄÇÒ ¼ö ÀÖ´Â °ÍÀÌ Æ¯Â¡ÀÌ´Ù. |
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| ARF | acute renal failure; acute respiratory failure; acute rheumatic fever; Addiction Research Foundation... |
|---|---|
| ARN | acute renal necrosis; acute retinal necrosis; arcuate nucleus; Association of Rehabilitation Nurses |
| POF | pattern of failure; position of function; premature ovarian failure; primary ovarian failure; pyruva... |
| CRF | case report form; chronic renal failure; chronic respiratory failure; coagulase-reacting factor; con... |
| RF | radial fiber; radio frequency; receptive field; regurgitant fraction; Reitland-Franklin [unit]; rela... |
| ARF | Acute Renal Failure |
|---|---|
| RCBF | Renal cortical blood flow |
| CRF | CHRONIC RENAL FAILURE |
| ESRF | End Stage Renal Failure |
| ESRD | End-stage renal failure |
cortical evoked potential (ÇÇÁú À¯¹ß ÀüÀ§
| acute renal failure | <nephrology> A sudden decline in renal function may be triggered by a number of acute disease processes. Examples include sepsis (infection), shock, trauma, kidney stones, kidney infection, drug toxicity (aspirin or lithium), poisons or toxins (drug abuse) or after injection with an iodinated contrast dye (adverse effect). Chronic renal failure represents a slow decline in kidney function over time. Chronic renal failure may be caused by a number of disorders which include long-standing hypertension, diabetes, congestive heart failure, lupus or sickle cell anaemia. Both forms of renal failure result in a life-threatening metabolic derangement. (27 Sep 1997) |
|---|---|
| laminar cortical necrosis | The breaking down of a definite cell layer in the cerebral cortex, encountered typically after temporary cardiac arrest or perinatal hypoxia. (05 Mar 2000) |
| renal cortical adenoma | <tumour> One of the usually small adenoma's sometimes found in the renal cortex and derived from renal tubular tissue. (05 Mar 2000) |
| renal cortical lobule | One of the subdivisions of the kidney, consisting of a medullary ray and that portion of the convoluted port (renal corpuscles and convoluted tubules) associated with its collecting duct. Synonym: lobulus corticalis renalis, renal cortical lobule, renculus, reniculus, renunculus. (05 Mar 2000) |
| renal failure | Chronic renal failure represents a slow decline in kidney function over time. Chronic renal failure may be caused by a number of disorders which include long-standing hypertension, diabetes, congestive heart failure, lupus or sickle cell anaemia. If renal function declines to a low enough level (end-stage renal disease) kidney dialysis may be necessary. A sudden decline in renal function may be triggered by a number of acute disease processes. Examples include sepsis (infection), shock, trauma, kidney stones, kidney infection, drug toxicity (aspirin or lithium), poisons or toxins (drug abuse) or after injection with an iodinated contrast dye (adverse effect). Both forms of renal failure result in a life-threatening metabolic derangement. (27 Sep 1997) |
| chronic renal failure | <nephrology> Chronic renal failure represents a slow decline in kidney function over time. Chronic renal failure may be caused by a number of disorders which include long-standing hypertension, diabetes, congestive heart failure, lupus or sickle cell anaemia. If renal function declines to a low enough level (end-stage renal disease) kidney dialysis may be necessary. A sudden decline in renal function may be triggered by a number of acute disease processes. Examples include sepsis (infection), shock, trauma, kidney stones, kidney infection, drug toxicity (aspirin or lithium), poisons or toxins (drug abuse) or after injection with an iodinated contrast dye (adverse effect). Both forms of renal failure result in a life-threatening metabolic derangement. (27 Sep 1997) |
| renal papillary necrosis | <nephrology> Necrosis of renal papillae, occurring in acute pyelonephritis, especially in diabetics, or in analgesic nephropathy; renal failure may result. Synonym: necrotizing papillitis. (05 Mar 2000) |
| acute kidney failure | <nephrology> A sudden decline in renal function may be triggered by a number of acute disease processes. Examples include sepsis (infection), shock, trauma, kidney stones, kidney infection, drug toxicity (aspirin or lithium), poisons or toxins (drug abuse) or after injection with an iodinated contrast dye (adverse effect). Chronic renal failure represents a slow decline in kidney function over time. Chronic renal failure may be caused by a number of disorders which include long-standing hypertension, diabetes, congestive heart failure, lupus or sickle cell anaemia. Both forms of renal failure result in a life-threatening metabolic derangement. (27 Sep 1997) |
| acute respiratory failure | Loss of pulmonary function either acute or chronic that results in hypoxaemia or hypercarbia. (05 Mar 2000) |
| kidney failure, acute | A clinical syndrome characterised by a sudden decrease in glomerular filtration rate, often to values of less than 1 to 2 ml per minute. It is usually associated with oliguria (urine volumes of less than 400 ml per day) and is always associated with biochemical consequences of the reduction in glomerular filtration rate such as a rise in blood urea nitrogen (bun) and serum creatinine concentrations. (12 Dec 1998) |
| liver failure, acute | A form of liver failure with rapid onset. It is often induced by the toxic effect of drugs and various toxic substances in experimental studies in animals and in clinical states in humans. If coma ensues, the constellation of neurological symptoms is referred to as hepatic encephalopathy. (12 Dec 1998) |
| acute tubular necrosis | <nephrology> A kidney disorder that results in damage to the renal tubule cells leading to acute renal failure. Acute tubular necrosis can result from any condition which deprives the kidney of oxygen (ischaemia). Acute tubular necrosis may occur as a complication of shock, trauma or sepsis. Conditions such as diabetes or liver disease can predispose people to the development of acute tubular necrosis. Certain medications (for example aminoglycosides, amphotericin B, cyclosporine) are known to cause acute tubular necrosis as a toxic side effect. Radiopaque contrast dyes, used in some radiologic procedures, may also result in acute tubular necrosis as a rare complication from contrast dye use. Acronym: ATN (13 Nov 1997) |
| retinal necrosis syndrome, acute | <ophthalmology> Mild to fulminant necrotizing vaso-occlusive retinitis associated with a high incidence of retinal detachment and poor vision outcome. (12 Dec 1998) |
| kidney tubular necrosis, acute | Acute kidney failure resulting from destruction of tubular epithelial cells. It is commonly attributed to exposure to toxic agents or renal ischemia following severe trauma. (12 Dec 1998) |
| adenoma, adrenal cortical | A benign neoplasm of adrenal cortical cells resembling normal adrenal cells histologically but possessing functional autonomy. In general it does not exceed 5 cm in its largest dimension, although benign tumours exceeding 20 cm have been reported. Adrenal cortical adenomas produce hypercortisolism and hyperaldosteronism, but seldom produce adrenogenital syndromes. For the most part the prognosis after surgery is reasonably favourable. (12 Dec 1998) |
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