| renal tubular transport, inborn errors | Genetically determined disorders of the reabsorptive functions of the kidney with regard to specific nephron segments responsible for specific transport functions, classifiable by proximal nephron function, loop of henle function, and distal nephron function. The transport defects can be selective or nonselective. (12 Dec 1998) |
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| renal tubule | <anatomy> Small structures in the kidney that filter the blood and produce the urine. (12 Dec 1998) |
| renal vein | <anatomy, vein> The short thick veins which return blood from the kidneys to the vena cava. (25 Jun 1999) |
| renal veins | Large veins formed at the renal hilus by the merger of the segmental veins anterior to the corresponding arteries; they open at right angles into the inferior vena cava at the level of the second lumbar vertebra. The left renal vein receives the left suprarenal vein and the left gonadal vein, and passes through the angle between the abdominal aorta and superior mesenteric artery where it may be compressed. Synonym: venae renales. (05 Mar 2000) |
| glycosuria, renal | Glycosuria occurring when there is only the normal amount of sugar in the blood, due to inherited inability of the renal tubules to reabsorb glucose completely. (12 Dec 1998) |
| microcystic disease of renal medulla | Presence of small cysts in the renal medulla associated with anaemia, sodium depletion, and chronic renal failure. It is of two types: 1) fatal autosomal recessive or juvenile type (also called familial juvenile nephrophthisis), beginning at about age 10 with an average duration of 6 to 8 years; 2) autosomal dominant or adult type, beginning at about age 30 but with a more fulminant course. Synonym: microcystic disease of renal medulla. (05 Mar 2000) |
| chronic renal failure | <nephrology> Chronic renal failure represents a slow decline in kidney function over time. Chronic renal failure may be caused by a number of disorders which include long-standing hypertension, diabetes, congestive heart failure, lupus or sickle cell anaemia. If renal function declines to a low enough level (end-stage renal disease) kidney dialysis may be necessary. A sudden decline in renal function may be triggered by a number of acute disease processes. Examples include sepsis (infection), shock, trauma, kidney stones, kidney infection, drug toxicity (aspirin or lithium), poisons or toxins (drug abuse) or after injection with an iodinated contrast dye (adverse effect). Both forms of renal failure result in a life-threatening metabolic derangement. (27 Sep 1997) |
| congenital renal cysts | <radiology> Congenital solitary cyst, multilocular cyst, multicystic disease (renal dysplasia), polycystic disease, autosomal-recessive (childhood) form, autosomal-dominant (adult) form, medullary sponge kidney, medulary cystic disease see also: Potter syndrome (12 Dec 1998) |
| congenital renal osteodystrophy | <radiology> Tubular form of renal osteodystrophy, vitamin D-resistant rickets, Fanconi syndrome, renal tubular acidosis (12 Dec 1998) |
| multiple renal cysts | <radiology> Adult (autosomal-dominant) polycystic kidney disease, multiple simple cysts, tuberous sclerosis, von Hippel-Lindau syndrome, Meckel-Gruber syndrome (12 Dec 1998) |
| cribriform area of the renal papilla | The apex of a renal papilla pierced by 10 to 22 openings of the papillary ducts, the foramina papillaria. Synonym: area cribrosa papillae renalis. (05 Mar 2000) |
| posterior branch of renal artery | <anatomy, artery> Terminal branch of renal artery (with anterior branch) becoming the posterior segmental artery of kidney. Synonym: ramus posterior arteriae renalis. (05 Mar 2000) |
| crossed renal ectopia | <radiology> M more than F, right (67%), may fuse: crossed-fused renal ectopia (12 Dec 1998) |
| haematologic, gastrointestinal, and renal agents | A collective grouping for agents that act on the haematopoietic, gastrointestinal, and renal systems. (12 Dec 1998) |
| haemorrhagic fever with renal syndrome | <syndrome> An acute febrile disease occurring predominately in asia. It is characterised by fever, prostration, vomiting, haemorrhagic phenonema, shock, and renal failure. It is caused by any one of several closely related species of the genus hantavirus. The most severe form is caused by hantaan virus whose natural host is the rodent apodemus agrarius. A milder form is caused by seoul virus and related species and transmitted by the rodents rattus rattus and r. Norvegicus. (12 Dec 1998) |