| PHC | personal health costs; posthospital care; premolar hypodontia, hyperhidrosis, [premature] canities [... |
|---|---|
| SCCB | small-cell carcinoma of the bronchus |
| SCCC | squamous cell cervical carcinoma |
| SCCHN | squamous cell carcinoma of the head and neck |
| SCCL | small cell carcinoma of the lung |
| renal-splenic venous shunt | Anastomosis of the splenic vein to the left renal vein, usually end-to-side, for control of portal hypertension. Synonym: renal-splenic venous shunt. (05 Mar 2000) |
|---|---|
| renal surface of spleen | The surface of the spleen in contact with the left kidney. Synonym: facies renalis lienis, facies renalis splenis. (05 Mar 2000) |
| renal surface of suprarenal gland | Surface of suprarenal gland in contact with the kidney. (05 Mar 2000) |
| renal surface of the suprarenal gland | The surface of the suprarenal gland in contact with the kidney. Synonym: renal surface of the suprarenal gland. (05 Mar 2000) |
| renal tb | <radiology> Putty kidney: caseous necrosis with calcium deposition, Cf: medullary nephrocalcinosis, renal calcification, haematogenous origin, clinically unilateral in 75%, findings: papillary necrosis, infundibular stenosis, autonephrectomy, CXR abnormal in 50% (12 Dec 1998) |
| renal threshold | Concentration of plasma substance above which the substance appears in the urine. (05 Mar 2000) |
| renal transplantation | Transplantation of a kidney from a compatible donor to restore kidney function in a recipient suffering from renal failure. (05 Mar 2000) |
| renal tubular acidosis | <nephrology> A rare sometimes familial disorder of the renal tubule characterised by the inability to excrete urine of normal acidity. This leads to a hyperchloraemic acidosis which is often associated with one or more secondary complications such as hypercalcinuria with nephrolithiasis and nephrocalcinosis, rickets, or osteomalacia and severe potassium depletion. (25 Jun 1999) |
| renal tubular transport, inborn errors | Genetically determined disorders of the reabsorptive functions of the kidney with regard to specific nephron segments responsible for specific transport functions, classifiable by proximal nephron function, loop of henle function, and distal nephron function. The transport defects can be selective or nonselective. (12 Dec 1998) |
| renal tubule | <anatomy> Small structures in the kidney that filter the blood and produce the urine. (12 Dec 1998) |
| renal vein | <anatomy, vein> The short thick veins which return blood from the kidneys to the vena cava. (25 Jun 1999) |
| renal veins | Large veins formed at the renal hilus by the merger of the segmental veins anterior to the corresponding arteries; they open at right angles into the inferior vena cava at the level of the second lumbar vertebra. The left renal vein receives the left suprarenal vein and the left gonadal vein, and passes through the angle between the abdominal aorta and superior mesenteric artery where it may be compressed. Synonym: venae renales. (05 Mar 2000) |
| glycosuria, renal | Glycosuria occurring when there is only the normal amount of sugar in the blood, due to inherited inability of the renal tubules to reabsorb glucose completely. (12 Dec 1998) |
| microcystic disease of renal medulla | Presence of small cysts in the renal medulla associated with anaemia, sodium depletion, and chronic renal failure. It is of two types: 1) fatal autosomal recessive or juvenile type (also called familial juvenile nephrophthisis), beginning at about age 10 with an average duration of 6 to 8 years; 2) autosomal dominant or adult type, beginning at about age 30 but with a more fulminant course. Synonym: microcystic disease of renal medulla. (05 Mar 2000) |
| chronic renal failure | <nephrology> Chronic renal failure represents a slow decline in kidney function over time. Chronic renal failure may be caused by a number of disorders which include long-standing hypertension, diabetes, congestive heart failure, lupus or sickle cell anaemia. If renal function declines to a low enough level (end-stage renal disease) kidney dialysis may be necessary. A sudden decline in renal function may be triggered by a number of acute disease processes. Examples include sepsis (infection), shock, trauma, kidney stones, kidney infection, drug toxicity (aspirin or lithium), poisons or toxins (drug abuse) or after injection with an iodinated contrast dye (adverse effect). Both forms of renal failure result in a life-threatening metabolic derangement. (27 Sep 1997) |