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"reaction deficiency"¿¡ ´ëÇÑ °Ë»ö °á°úÀÔ´Ï´Ù. °Ë»ö °á°ú º¸´Â µµÁß¿¡ Tab ۸¦ ´©¸£½Ã¸é °Ë»ö âÀÌ ¼±Åõ˴ϴÙ.
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  • ¿µ¹®
    ÇѱÛ
  • downgrading reaction
    ÇÏÇâ¹ÝÀÀ
  • exergonic reaction
    ¿¡³ÊÁö¹æÃâ¹ÝÀÀ
  • exoergic reaction
    ¿¡³ÊÁö¹æÃâ¹ÝÀÀ
  • exothermic reaction
    ¹ß¿­¹ÝÀÀ
  • explosive reaction
    Æø¹ßÀû¹ÝÀÀ
  • endergonic reaction
    1. ¿¡³ÊÁöÁõ°­¹ÝÀÀ 2. ¿¡³ÊÁöÈí¼ö¹ÝÀÀ
  • endothermic reaction
    Èí¿­¹ÝÀÀ
  • enzyme-antienzyme reaction
    È¿¼ÒÇ×È¿¼Ò¹ÝÀÀ
  • eosinophilic leukemoid reaction
    È£»ê±¸¹éÇ÷º´¸ð¾ç¹ÝÀÀ
  • equilibrium reaction
    ÆòÇü¹ÝÀÀ
  • erythrocyte sedimentation reaction
    ÀûÇ÷±¸Ä§°­¹ÝÀÀ
  • escape reaction
    µµÇǹÝÀÀ
  • febrile transfusion reaction
    ¹ß¿­¼º¼öÇ÷¹ÝÀÀ
  • Fernandez reaction
    Æä¸£³­µ¥½º¹ÝÀÀ
  • Feulgen¡¯s reaction
    Æ÷ÀϰչÝÀÀ
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  • ¿µ¹®
    ÇѱÛ
  • consensual pupillary reaction
    °ø°¨µ¿°ø¹ÝÀÀ
  • conversion reaction
    Àüȯ¹ÝÀÀ
  • counter reaction
    ¿ª¹ÝÀÀ
  • coupled reaction
    °ø¿ª¹ÝÀÀ, ¦Áø¹ÝÀÀ, ¿¬°á¹ÝÀÀ
  • cross reaction
    ±³Â÷¹ÝÀÀ
  • cutaneous reaction
    ÇǺιÝÀÀ
  • cutaneous drug reaction
    ÇǺξ๰¹ÝÀÀ
  • cytotoxic reaction
    µ¶¼º¹ÝÀÀ, ¼¼Æ÷µ¶¼º¹ÝÀÀ
  • reaction center
    (¢¡germinal center) Á¾ÀÚÁß½É, ¹èÁß½É
  • reaction time crossover
    ¹ÝÀÀ½Ã°£±³Â÷
  • defensive reaction
    ¹æ¾îÀÛ¿ë
  • deficient release reaction
    ¹æÃâ¹ÝÀÀ°áÇÌ
  • delayed reaction
    Áö¿¬¹ÝÀÀ
  • delayed allergic reaction
    Áö¿¬¾Ë·¹¸£±â¹ÝÀÀ
  • delayed cell-mediated reaction
    Áö¿¬¼¼Æ÷¸Å°³¹ÝÀÀ
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  • ¿µ¹®
    ÇѱÛ
  • Weil-Felix reaction
    ¿ÍÀÏ-Æç¸¯½º ¹ÝÀÀ
  • Widal reaction
    ºñ´Þ[ÀåÆ¼Çª½ºÇ÷û]¹ÝÀÀ
  • Zimmerman reaction
    Áü¸Þ¸£¸¸¹ÝÀÀ
  • acrosome reaction
    ֟ ¹ÝÀÀ
  • activator reaction
    Ȱ¼ºÈ­¹ÝÀÀ.
  • acute hemolytic transfusion reaction
    ±Þ¼º¿ëÇ÷¼º¼öÇ÷¹ÝÀÀ
  • acute phase reaction
    ±Þ¼º±â¹ÝÀÀ(¡­Ñ¢Úãëë)
  • addition reaction
    ºÎ°¡¹ÝÀÀ.
  • adverse reaction
    À¯ÇØ(êóúª)¹ÝÀÀ, ¿ª(æ½)¹ÝÀÀ.
  • adverse reaction
    ºÎÀÛ¿ë
  • aerobic reaction
    È£±â¼º ¹ÝÀÀ
  • affective reaction
    Á¤µ¿¹ÝÀÀ, Á¤°¨¹ÝÀÀ
  • agglutination reaction
    ÀÀÁý¹ÝÀÀ(¡­Úãëë).
  • allergic reaction
    ¾Ë·¹¸£±â(¼º) ¹ÝÀÀ.
  • allergic reaction
    ¾Ë·¹¸£±â(¼º) ¹ÝÀÀ.
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  • ¿µ¹®
    ÇѱÛ
  • hexokinase deficiency
    Çí¼ÒŰ³ªÁ¦°áÇÌ.
  • hexokinase deficiency
    Çí¼ÒÄ«À̳×À̽º°áÇÌ.
  • hexosaminidase a deficiency
    Çí¼Ò»ç¹Ì´Ïµ¥À̽º A °áÇÌ(Áõ)
  • histogenetic deficiency
    Á¶Á÷¹ß»ý°áÇÌ
  • hormone deficiency
    È£¸£¸ó°áÇÌ
  • iduronate sulfatase deficiency
    Iduronate sulfatase deficiency
  • iduronosulfate sulfatase deficiency
  • iga deficiency
    ¸é¿ª±Û·ÎºÒ¸° A °áÇÌ
  • immune deficiency disease
    ¸é¿ª°áÇÌÁúȯ<º´>.
  • immunity deficiency =immunodeficiency
    ¸é¿ª°áÇÌ(¡­ÌÀù¹).
  • immunoglobulin A deficiency
    ¸é¿ª±Û·ÎºÒ¸° A °áÇÌ(Áõ)
  • immunologic deficiency state
    ¸é¿ª°áÇÌ »óÅÂ.
  • immunologic deficiency syndrome
    ¸é¿ª°áÇÌ ÁõÈıº(¡­ÌÀù¹ ñøý¦ÏØ)
  • immunological deficiency state
    ¸é¿ª°áÇÌ »óÅÂ.
  • inosine phosphorylase deficiency
    À̳ë½ÅÆ÷½ºÆ÷¸±¶óÁ¦°áÇÌ(Áõ)
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  • ¿µ¹®
    ÇѱÛ
  • oxidation-reduction reaction
    »êȭȯ¿ø ¹ÝÀÀ(ß«ûùü½êªÚãëë)
  • oxidoreduction reaction
    »êȭȯ¿ø¹ÝÀÀ(ß«ûùü½êªÚãëë)
  • partial reaction
    ºÎºÐ ¹ÝÀÀ(Ý»ÝÂÚãëë)
  • Pauly reaction
    ÆÄ¿ï¸® ¹ÝÀÀ(Úãëë)
  • pericyclic reaction
    °í¸®Çü Çùµ¿¹ÝÀÀ(úðÔÒÚãëë)
  • phosphoroclastic reaction
    ÀλêÀý´Ü ¹ÝÀÀ(×òß«ï·Ó¨Úãëë)
  • photochemical reaction
    ±¤È­ÇйÝÀÀ(ÎÃûùùÊÚãëë)
  • photochemical reaction center
    ±¤È­ÇÐ ¹ÝÀÀÁß½É(ÎÃûùùÊÚãëëñéãý)
  • photonuclear reaction
    ±¤ºØ±«¹ÝÀÀ(ÎÃÝÚÎÕÚãëë)
  • P-K reaction
    P-K ¹ÝÀÀ(Úãëë)
  • polymerase chain reaction
    ÁßÇÕÈ¿¼Ò ¿¬¼â¹ÝÀÀ(ñìùêý£áÈÖ§áðÚãëë)
  • Prausnitz-Kuestner reaction
    ÇÁ¶ó¿ì½º´ÏÃ÷-Å¥¿¡½ºÆ®³Ê ¹ÝÀÀ(Úãëë)
  • precipitin reaction
    ħ°­¼Ò(öØË½áÈ) ¹ÝÀÀ(Úãëë)
  • pseudounimolecular reaction
    À§´ÜºÐÀÚ¹ÝÀÀ(êÊÓ¤ÝÂí­Úãëë)
  • Quellung reaction
KMLE ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 7
ADS acute death syndrome; acute diarrheal syndrome; Alcohol Dependence Scale; alternative delivery syste...
AID acquired immunodeficiency disease; acute infectious disease; acute ionization detector; Agency for I...
AIDS acquired immune deficiency syndrome
AIDS-KS acquired immune deficiency syndrome with Kaposi's sarcoma
AMD acid maltase deficiency; acromandibular dysplasia; actinomycin D; adrenomyelodystrophy; age-related ...
KMLE ÀÚµ¿ÃßÃâ ÀÇÇоà¾î »çÀü À¯»ç °Ë»ö °á°ú : 5 ÆäÀÌÁö: 7
SCID Severe Combined Immune Deficiency
TD Thiamine deficiency
VAD Vitamin A deficiency
cmd cartilage matrix deficiency
CED chronic energy deficiency
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  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • erythema wheal reaction
    È«¹Ý ±¸Áø ¹ÝÀÀ
    ¾Ë·¹¸£±â¿øÀÇ °¨ÀÛ »óŸ¦ °Ë»öÇÒ ¸ñÀûÀ¸·Î ¾Ë·¹¸£±â¿ø ÃßÃâ¹°À» ÇǺΠǥ¸é¿¡ ÇÑ ¹æ¿ï ¶³¾î¶ß¸®°í ±× ¾×À» ÅëÇØ¼­ ÇǺΠǥ¸éÀ» ¹Ù´Ã·Î ±ÜÀ¸¸é À¯¸®µÇ´Â È­ÇÐ Àü´Þ ¹°Áú ¶§¹®¿¡ ÆØÁø ¹× È«¹ÝÀÌ ÃâÇöÇÑ´Ù. ¾Ë·¹¸£±â¿ø °Ë»çÀÎ °æ¿ì´Â ÆØÁø°æ ¶Ç´Â È«¹Ý°æÀÌ ´ëÁ¶ÀÇ 2¹è ÀÌ»ó, ȤÀº ±¸ÁøÀÇ Á÷°æÀÌ 5mm ÀÌ»óÀ» ¾ç¼ºÀ¸·Î ÇÑ´Ù.
  • escape reaction
    µµÇÇ ¹ÝÀÀ
  • eutectic reaction
    °øÁ¤ ¹ÝÀÀ
    ÇÕ±ÝÀ» ¿ëÀ¶ »óÅ¿¡¼­ ³Ã°¢ÇÒ ¶§ ÇϳªÀÇ ¾×»ó¿¡¼­ µÎ °³ÀÇ °í»óÀÌ µ¿½Ã¿¡ Çü¼ºµÇ´Â ¹ÝÀÀ.
  • exergonic reaction
    ¹ß¿­ ¹ÝÀÀ, ¿¡³ÊÁö ¹ß»ý ¹ÝÀÀ, ¿¡³ÊÁö ¹æÃâ ¹ÝÀÀ
  • exothermal reaction
    ¹ß¿­ ¹ÝÀÀ
  • explosive reaction
    Æø¹ß ¹ÝÀÀ
  • false positive reaction
    °¡¾ç¼º ¹ÝÀÀ
  • first order reaction
    ÀÏÂ÷ ¹ÝÀÀ
    ¹ÝÀÀ ¼Óµµ°¡ ¹ÝÀÀ¿¡ °ü¿©ÇÏ´Â ¹°Áú Áß ¾î´À ÇϳªÀÇ ³óµµ¿¡ ºñ·ÊÇÏ´Â ¹ÝÀÀ.
  • flora's myasthenic reaction
    Ç÷ζó ±Ù¹«·Â ¹ÝÀÀ
    ¿Ü»ó¼º ½Å°æ ¼è¾àÀÇ °æ¿ì, ±ÙÀ°¿¡ Àü±â ÀÚ±ØÀ» Àå½Ã°£¿¡ °ÉÃÄ °¡ÇÒ ¶§ º¼ ¼ö ÀÖ´Â Åן´Ï¼º ¹ÝÀÀ.
  • fright reaction
    ³î¶÷ ¹ÝÀÀ
  • heat of reaction
    ¹ÝÀÀ ¿­
  • hyperkinetic reaction
    °ú´Ù ¿îµ¿ ¹ÝÀÀ, °ú´Ù ¿îµ¿¼º ¹ÝÀÀ
  • immune reaction
    ¸é¿ª ¹ÝÀÀ
    Ç׿ø°ú Ç×ü »çÀÌÀÇ ¹ÝÀÀ.
  • inflammatory reaction
    ¿°Áõ ¹ÝÀÀ
    ¿°Áõ ÀÎÀÚ°¡ »ýü¿¡ ÀÛ¿ëÇÏ¸é »ýÃ¼Ãø¿¡´Â ¹æ¾î±â´ÉÀÌ »ý±ä´Ù. ±× ¿°Áõ¼Ò¿¡¼­ÀÇ Ç×ü³ª È÷½ºÅ¸¹Î, ¼¼·ÎÅä´Ñ µîÀÇ È­Çй°ÁúÀ» ÇÔÀ¯ÇÑ Ç÷Àå ¼ººÐÀ̳ª Á¶Á÷¾×ÀÇ ±¹¼ÒÀû »ïÃâ, ¹éÇ÷±¸ÀÇ Ä§À±, ȸº¹À» À§ÇÑ ¼¶À¯ Áõ»ý µîÀÇ »ýÃ¼Ãø¿¡ »ý±â´Â ¹ÝÀÀ Çö»óÀ» ¸»ÇÑ´Ù. ¿°Áõ ÀÎÀÚÀÇ Á¾·ù, ¾çÀ̳ª »ýÃ¼ÃøÀÇ »óÅ¿¡ µû¶ó¼­ ¿°Áõ ¹ÝÀÀÀÌ »ý±â´Â ¾ç»óÀº ´Ù¸£´Ù.
  • inhibition reaction
    ¾ïÁ¦, ¾ïÁ¦ ¹ÝÀÀ, ÀúÁö, ÀúÁö ¹ÝÀÀ
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IgA deficiency A dysgammaglobulinaemia characterised by a deficiency of IgA.
(12 Dec 1998)
IgG deficiency A dysgammaglobulinaemia characterised by a deficiency of IgG.
(12 Dec 1998)
immune deficiency Impaired ability of the bodys defence mechanisms to combat infections by bacteria, viruses and fungi.
(13 Nov 1997)
immune deficiency diseases Those diseases in which immune reactions are suppressed or reduced. Reasons may include congenital absence of B and/or T lymphocytes or viral killing of helper lymphocytes (see HIV).
(18 Nov 1997)
immunity deficiency Inabillity to mount a normal immune response. Immunodeficiency can be due to a genetic disease or acquired as in aids due to HIV.
(12 Dec 1998)
immunoglobulin G subclass deficiency A rare inherited disorder in which there are reduced levels of one or more IgG subclasses resulting from defective heavy chain genes or an abnormality in the regulation of immunoglobulin isotype switching.
(05 Mar 2000)
immunological deficiency Inabillity to mount a normal immune response. Immunodeficiency can be due to a genetic disease or acquired as in aids due to HIV.
(12 Dec 1998)
immunologic deficiency syndromes Syndromes in which there is a deficiency or defect in the mechanisms of immunity, either cellular or humoral.
(12 Dec 1998)
iodine deficiency Iodine is a natural requirement of our diets. Iodine deficiency can lead to inadequate production of thyroid hormone from the thyroid gland (hypothyroidism). For example, in some parts of zaire, ecuador, india, and chile, remote, mountainous areas, such as in the alps (in the past), andes and the himalayas have a particular predisposition to severe iodine deficiency, goiter, and hypothyroidism. Since the addition of iodine to table salt, iodine deficiency is rarely seen in the united states.
(12 Dec 1998)
iron deficiency Deficiency of iron results in anaemia because iron is necessary to make haemoglobin, the key molecule in red blood cells responsible for the transport of oxygen. In iron deficiency anaemia, the red cells are unusally small (microcytic) and pale (hypochromic).
Characteristic features of iron deficiency anaemia in children include failure to thrive (grow) and increased infections.
The treatment of iron deficiency anaemia , whether it be in children or adults, is with iron and iron-containing foods. Food sources of iron include meat, poultry, eggs, vegetables and cereals (especially those fortified with iron). According to the National Academy of Sciences, the recommended dietary allowances of iron are 15 milligrams per day for women and 10 milligrams per day for men.
(12 Dec 1998)
iron deficiency anaemia Hypochromic microcytic anaemia characterised by low serum iron, increased serum iron-binding capacity, decreased serum ferritin, and decreased marrow iron stores.
Synonym: hypoferric anaemia.
(05 Mar 2000)
taste deficiency Reduced or absent ability to detect a bitter taste in a group of compounds of which phenylthiocarbamide is the prototype, due to the homozygous state of a common allele.
See: phenylthiourea.
(05 Mar 2000)
thiamin deficiency An endemic form of polyneuritis (nerve inflammation), due to an unbalanced diet, with a deficiency of vitamin B1(thiamin). Common in those who chronically abuse alcohol.
Synonym: beriberi.
(27 Sep 1997)
thiamine deficiency A nutritional condition produced by a deficiency of thiamine in the diet, characterised by anorexia, irritability, and weight loss. Later, patients experience weakness, peripheral neuropathy, headache, and tachycardia. In addition to being caused by a poor diet, thiamine deficiency in the united states most commonly occurs as a result of alcoholism, since ethanol interferes with thiamine absorption. In countries relying on polished rice as a dietary staple, beriberi prevalence is very high.
(12 Dec 1998)
thrombotic disease due to protein c deficiency Protein C is a protein in plasma that enters into the cascade of biochemical events leading to the formation of a clot. Deficiency of protein c results in thrombotic (clotting) disease and excess platelets with recurrent thrombophlebitis (inflammation of the vein that occurs when a clot forms). The clot can break loose and travel through the blood stream (thromboembolism) to the lungs causing a pulmonary embolism, brain causing a stroke (cerebrovascular accident), heart causing an early heart attack, skin causing what in the newborn is called neonatal purpura fulminans, the adrenal gland causing haemorrhage with abdominal pain, abnormally low blood pressure (hypotension), and salt loss. Protein c deficiency is due to possession of one gene (heterozygosity) in chromosome band 2q13-14. The possession of two such genes (homozygosity) is usually lethal.
(12 Dec 1998)
ÀÌ ¾Æ·¡ ºÎÅÍ´Â °á°ú°¡ ¾ø½À´Ï´Ù.
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