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  • ¿µ¹®
    ÇѱÛ
  • lymphocytosis stimulating factor
    ¸²ÇÁ±¸Áõ°¡ÀÚ±ØÀÎÀÚ
  • migration inhibition factor
    À̵¿ÀúÁöÀÎÀÚ
  • mitogenic factor
    ºÐ¿­ÃËÁøÀÎÀÚ
  • myocardial depressant factor
    ½É(Àå)±Ù(À°)¾ïÁ¦ÀÎÀÚ
  • macrophage aggregating factor
    Å«Æ÷½Ä¼¼Æ÷ÀÀÁýÀÎÀÚ, ´ë½Ä¼¼Æ÷ÀÀÁýÀÎÀÚ
  • macrophage arming factor
    Å«Æ÷½Ä¼¼Æ÷¹«ÀåÀÎÀÚ, ´ë½Ä¼¼Æ÷¹«ÀåÀÎÀÚ
  • macrophage chemotactic factor
    Å«Æ÷½Ä¼¼Æ÷È­Çнò¸²ÀÎÀÚ, ´ë½Ä¼¼Æ÷È­Çнò¸²ÀÎÀÚ
  • macrophage colony-stimulating factor
    Å«Æ÷½Ä¼¼Æ÷Áý¶ôÀÚ±ØÀÎÀÚ, ´ë½Ä¼¼Æ÷Áý¶ôÀÚ±ØÀÎÀÚ
  • macrophage migration inhibitory factor
    Å«Æ÷½Ä¼¼Æ÷À̵¿ÀúÁöÀÎÀÚ, ´ë½Ä¼¼Æ÷À̵¿ÀúÁöÀÎÀÚ
  • macrophage-activating factor
    Å«Æ÷½Ä¼¼Æ÷Ȱ¼ºÀÎÀÚ, ´ë½Ä¼¼Æ÷Ȱ¼ºÀÎÀÚ
  • macrophage-derived growth factor
    Å«Æ÷½Ä¼¼Æ÷À¯·¡¼ºÀåÀÎÀÚ, ´ë½Ä¼¼Æ÷À¯·¡¼ºÀåÀÎÀÚ
  • nerve growth factor
    ½Å°æ¼ºÀåÀÎÀÚ
  • neutron kerma factor
    Áß¼ºÀÚÄ¿¸¶°è¼ö
  • neutrophil chemotactic factor
    Áß¼º±¸È­ÇÐÁÖ¼ºÀÎÀÚ, È£Áß±¸½ò¸²ÀÎÀÚ
  • occupancy factor
    °ÅÁÖ°è¼ö
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  • ¿µ¹®
    ÇѱÛ
  • lymphocyte inhibitory factor
    ¸²ÇÁ±¸¾ïÁ¦ÀÎÀÚ
  • lymphocytosis stimulating factor
    ¸²ÇÁ±¸Áõ°¡ÀÚ±ØÀÎÀÚ
  • macrophage aggregating factor
    Å«Æ÷½Ä¼¼Æ÷ÀÀÁýÀÎÀÚ
  • macrophage arming factor
    Å«Æ÷½Ä¼¼Æ÷¹«ÀåÀÎÀÚ
  • macrophage chemotactic factor
    Å«Æ÷½Ä¼¼Æ÷È­ÇÐÁÖ¼ºÀÎÀÚ, Å«Æ÷½Ä¼¼Æ÷È­Çнò¸²ÀÎÀÚ
  • macrophage colony-stimulating factor
    Å«Æ÷½Ä¼¼Æ÷Áý¶ôÀÚ±ØÀÎÀÚ
  • macrophage migration inhibitory factor
    Å«Æ÷½Ä¼¼Æ÷À̵¿ÀúÁöÀÎÀÚ
  • macrophage-activating factor
    Å«Æ÷½Ä¼¼Æ÷Ȱ¼ºÀÎÀÚ
  • macrophage-derived growth factor
    Å«Æ÷½Ä¼¼Æ÷À¯·¡¼ºÀåÀÎÀÚ
  • migration inhibition factor
    Æ÷½Ä¼¼Æ÷À̵¿ÀúÇØÀÎÀÚ
  • mitogenic factor
    ºÐ¿­ÃËÁøÀÎÀÚ
  • myocardial depressant factor
    ½ÉÀå±Ù¾ïÁ¦ÀÎÀÚ
  • nerve growth factor
    ½Å°æ¼ºÀåÀÎÀÚ
  • neutron kerma factor
    Áß¼ºÀÚÄ¿¸¶°è¼ö
  • neutrophil chemotactic factor
    È£Áß±¸ÁÖ¼ºÀÎÀÚ, È£Áß±¸½ò¸²ÀÎÀÚ
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  • ¿µ¹®
    ÇѱÛ
  • NGF=>nerve growth factor
    ½Å°æ¼ºÀåÀÎÀÚ
  • PAF =platelet activating factor
    Ç÷¼ÒÆÇȰ¼ºÀÎÀÚ.
  • PAF= platelet activating factor
    Ç÷¼ÒÆÇ Ȱ¼ºÀÎÀÚ.
  • Q factor
    Å¥ ÀÎÀÚ
  • Q-factor
    Å¥-ÀÎÀÚ (ì×í­)
  • R factor
    ³»¼ºÀÎÀÚ.
  • R factor
    ³»¼ºÀÎÀÚ.
  • Rh factor
    RhÀÎÀÚ.
  • Stuart-Prower factor
    ½ºÆ©¾îÆ®-ÇÁ¶ó¿ö ÀÎÀÚ
  • T cell activating factor
    T¼¼Æ÷Ȱ¼ºÀÎÀÚ
  • T cell factor (TCF)
    T¼¼Æ÷
  • T cell growth factor (TCGF, IL-2)
    T¼¼Æ÷ Áõ½ÄÀÎÀÚ
  • T cell replacing factor
    T¼¼Æ÷ ´ëüÀÎÀÚ
  • T-cell growth factor
    T-¼¼Æ÷¼ºÀåÀÎÀÚ
  • TGF => transforming growth factor
    Àüȯ¼ºÀåÀÎÀÚ
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  • ¿µ¹®
    ÇѱÛ
  • hexokinase deficiency
    Çí¼ÒÄ«À̳×À̽º°áÇÌ.
  • hexosaminidase a deficiency
    Çí¼Ò»ç¹Ì´Ïµ¥À̽º A °áÇÌ(Áõ)
  • histogenetic deficiency
    Á¶Á÷¹ß»ý°áÇÌ
  • hormone deficiency
    È£¸£¸ó°áÇÌ
  • iduronate sulfatase deficiency
    Iduronate sulfatase deficiency
  • iduronosulfate sulfatase deficiency
  • iga deficiency
    ¸é¿ª±Û·ÎºÒ¸° A °áÇÌ
  • immune deficiency disease
    ¸é¿ª°áÇÌÁúȯ<º´>.
  • immunity deficiency =immunodeficiency
    ¸é¿ª°áÇÌ(¡­ÌÀù¹).
  • immunoglobulin A deficiency
    ¸é¿ª±Û·ÎºÒ¸° A °áÇÌ(Áõ)
  • immunologic deficiency state
    ¸é¿ª°áÇÌ »óÅÂ.
  • immunologic deficiency syndrome
    ¸é¿ª°áÇÌ ÁõÈıº(¡­ÌÀù¹ ñøý¦ÏØ)
  • immunological deficiency state
    ¸é¿ª°áÇÌ »óÅÂ.
  • inosine phosphorylase deficiency
    À̳ë½ÅÆ÷½ºÆ÷¸±¶óÁ¦°áÇÌ(Áõ)
  • insulin deficiency diabetes
    Àν¶¸°°áÇ̼º ´ç´¢º´.
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    ÇѱÛ
  • F' factor
    F' ÀÎÀÚ(ì×í­)
  • fibrin-stabilizing factor
    ¼¶À¯¼Ò ¾ÈÁ¤È­ÀÎÀÚ(àéë«áÈäÌïÒûùì×í­)
  • Fitzgerald factor
    ÇÍÁ¦¶öµå ÀÎÀÚ(ì×í­)
  • g factor
    g ÀÎÀÚ(ì×í­)
  • G factor
    G ÀÎÀÚ(ì×í­)
  • glucose tolerance factor
    ±Û·çÄÚ½º ³»¼º ÀÎÀÚ(Ò±àõì×í­)
  • growth factor
    ¼ºÀåÀÎÀÚ (à÷íþì×í­)
  • Hageman factor
    ÇØ±×¸Õ ÀÎÀÚ (ì×í­)
  • heat labile citrovorum factor
    ¿­ºÒ¾ÈÁ¤(æðÝÕäÌïÒ) ½ÃÆ®·Î¹ö·³ ÀÎÀÚ(ì×í­)
  • helper factor
    µµ¿òÀÌ ÀÎÀÚ(ì×í­)
  • hydration factor
    ¼öÈ­ ÀÎÀÚ(â©ûùì×í­)
  • hypercalcemic factor
    °ú(Φ)Ä®½·Ç÷Áõ(úìñø) ÀÎÀÚ(ì×í­)
  • hyperglycemic factor
    °úÇ÷´ç ÀÎÀÚ(ΦúìÓØì×í­)
  • hypocalcemic factor
    Àú(î¸)Ä®½·Ç÷Áõ(úìñø) ÀÎÀÚ(ì×í­)
  • hypoglycemic factor
    ÀúÇ÷´ç ÀÎÀÚ(î¸úìÓØì×í­)
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TGF T-cell growth factor; transforming growth factor; tuboglomerular feedback; tumor growth factor
AAMD American Association on Mental Deficiency; ¹Ì±¹ Á¤½Å ¹Ú¾à Çùȸ
AAMD-ABS American Association on Mental Deficiency-Adaptive Behavior Scale; ¹Ì±¹ Á¤½Å ¹Ú¾à Çùȸ ÀûÀÀ Çൿ ôµµ...
AIDS Acquired Immuno(Immune)-Deficiency Syndrome; ÈÄõ¼º ¸é¿ª °áÇÌ ÁõÈıº
IDA   1) Imino-Diacetic Acid
  2) Iron Deficiency Anemia
   &nb...
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SCID Severe Combined Immune Deficiency
TD Thiamine deficiency
VAD Vitamin A deficiency
cmd cartilage matrix deficiency
CED chronic energy deficiency
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  • ¿µ¹®
    ÇѱÛ
    ¼³¸í
  • covering factor
    ÇǺ¹ ÀÎÀÚ
  • cultural and ethnic factor
    ¹®È­ ¹ÎÁ·Àû ¿äÀÎ
  • cytotoxic factor
    ¼¼Æ÷ µ¶¼º ÀÎÀÚ
  • D and C ÀÚ±ÃÀÇ °æºÎ È®Àå°ú ³»¸· ¼ÒÆÄ.

    D factor

    D-ÀÎÀÚ
  • Decay accelerating factor
    ºØ±« °¡¼Ó ¿ä¼Ò
  • diabetogenic factor
    ´ç´¢ À¯¹ß ÀÎÀÚ
  • differentiation factor
    °¨º° ¿äÀÎ, °¨º° ¿ä¼Ò, °¨º° ÀÎÀÚ
  • diffusion factor
    È®»ê ÀÎÀÚ
  • dilution factor
    Èñ¼® ÀÎÀÚ
  • dose modifying factor
    ¼±·® ¼ö½Ä °è¼ö
  • drug resistance factor
    ¾àÁ¦ ³»¼º ÀÎÀÚ
  • drug resistance transfer factor
    ¾àÁ¦ ³»¼º Àü´Þ ÀÎÀÚ
  • EDA : electronic dental anesthesiaÀÇ ¾àÀÚ.

    edaphic factor

    ÅäÁö ÀÎÀÚ
  • effector-inhibitory factor
    È¿°ú±â ¾ïÁ¦ ÀÎÀÚ
  • emotional factor
    Á¤¼­ ¿äÀÎ
CancerWEB ¿µ¿µ ÀÇÇлçÀü À¯»ç °Ë»ö °á°ú : 15 ÆäÀÌÁö: 7
deficiency, glucose-6-phosphate dehydrogenase Deficiency of G6PD is the commonest disease-causing enzyme defect in humans affecting an estimated 400 million people. The G6PD gene is on the X chromosome. Males with the enzyme deficiency develop anaemia due to breakup of their red blood cells when they are exposed to oxidant drugs such as the antimalarial primaquine, the sulfonamide antibiotics or sulfones, naphthalene moth balls, or fava beans.
(12 Dec 1998)
deficiency, iron Deficiency of iron results in anaemia because iron is necessary to make haemoglobin, the key molecule in red blood cells responsible for the transport of oxygen. In iron deficiency anaemia, the red cells are unusally small (microcytic) and pale (hypochromic). Characteristic features of iron deficiency anaemia in children include failure to thrive (grow) and increased infections. The treatment of iron deficiency anaemia , whether it be in children or adults, is with iron and iron-containing foods. Food sources of iron include meat, poultry, eggs, vegetables and cereals (especially those fortified with iron). According to the National Academy of Sciences, the Recommended Dietary Allowances of iron are 15 milligrams per day for women and 10 milligrams per day for men.
(12 Dec 1998)
deficiency, lactase Lack of the enzyme lactase resulting in failure to digest lactose in milk (lactose intolerance).
(12 Dec 1998)
deficiency, magnesium Can occur due to inadequate intake or impaired intestinal absorption of magnesium. Low magnesium (hypomagnesaemia) is often associated with low calcium (hypocalcaemia) and potassium (hypokalaemia) levels. Deficiency of magnesium causes increased irritability of the nervous system with tetany (spasms of the hands and feet, muscular twitching and cramps, spasm of the larynx, etc.). According to the National Academy of Sciences, the Recommended Dietary Allowances of magnesium are 420milligrams per day for men and 320 milligrams per day for women. The upperlimit of magnesium as supplements is 350 milligrams daily, in addition to the magnesium from food and water.
(12 Dec 1998)
deficiency mutant Mutant with a nutritional requirement not present in the wild type organism.
Synonym: defective organism, deficiency mutant.
(05 Mar 2000)
deficiency, protein c Protein C is a protein in plasma that enters into the cascade of biochemical events leading to the formation of a clot. Deficiency of protein C results in thrombotic (clotting) disease.
(12 Dec 1998)
deficiency, selenium Deficiency of the essential mineral selenium causes Keshan disease, a fatal form of cardiomyopathy (disease of the heart muscle) first observed in Keshan province in China and since found elsewhere. According to the National Academy of Sciences, the Recommended Dietary Allowances of selenium are 70 milligrams per day for men and 55 milligrams per day for women. Food sources of selenium include seafoods, some meats such as kidney and liver, and some grains and seeds.
(12 Dec 1998)
deficiency symptom Manifestation of a lack, in varying degrees, of some substance (e.g., hormone, enzyme, vitamin) necessary for normal structure and/or function of an organism.
(05 Mar 2000)
deficiency, zinc Deficiency of zinc is associated with short stature, anaemia, increased pigmentation of skin (hyperpigmentation), enlarged liver and spleen (hepatosplenomegaly), impaired gonadal function (hypogonadism), impaired wound healing, and immune deficiency. (For a genetic disorder that impairs zinc uptake, please see Acrodermatitis enteropathica). According to the National Academy of Sciences, the Recommended Dietary Allowances of zinc are 12 milligrams per day for women and 10 milligrams per day for men. Food sources of zinc include meat including liver, eggs, seafood, nuts and cereal.
(12 Dec 1998)
disaccharidase deficiency A digestive disorder caused by the body being unable to produce enough disaccharidase, an enzyme responsible for breaking down carbohydrates in the small intestine. This may cause symptoms such as intestinal gas and diarrhoea.
(09 Oct 1997)
IgA deficiency A dysgammaglobulinaemia characterised by a deficiency of IgA.
(12 Dec 1998)
IgG deficiency A dysgammaglobulinaemia characterised by a deficiency of IgG.
(12 Dec 1998)
immune deficiency Impaired ability of the bodys defence mechanisms to combat infections by bacteria, viruses and fungi.
(13 Nov 1997)
immune deficiency diseases Those diseases in which immune reactions are suppressed or reduced. Reasons may include congenital absence of B and/or T lymphocytes or viral killing of helper lymphocytes (see HIV).
(18 Nov 1997)
immunity deficiency Inabillity to mount a normal immune response. Immunodeficiency can be due to a genetic disease or acquired as in aids due to HIV.
(12 Dec 1998)
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