| NF | nafcillin; National Formulary; nephritic factor; neurofibromatosis; neurofilament; neutral fraction;... |
|---|---|
| RF | radial fiber; radio frequency; receptive field; regurgitant fraction; Reitland-Franklin [unit]; rela... |
| NF | Neuro-Fibromatosis = Von Recklinghausen's Disease NF 1; Neuro-Fibroma... |
| VHL | Von Hippel-Lindau Syndrome = Cerebelloretinal Hemangioblastomatosis |
| VEG | von Egner gland [protein] |
| GM-CSF | Granulocyte colony-stimulating factor , granulocyte-macrophage colony-stimulating factor |
|---|---|
| G-CSF | Granulocyte-Macrophage Colony-Stimulating Factor , Granulocyte Colony-Stimulating Factor |
| HB-EGF | Heparin binding epidermal growth factor-like growth factor |
| HB-EGF | Heparin-binding epidermal growth factor (EGF)-like growth factor |
| HGF | Hepatocyte Growth Factor-Scatter Factor |
edaphic factor
| Hess, Carl von | <person> German ophthalmologist, 1863-1923. See: Hess screen. (05 Mar 2000) |
|---|---|
| Prowazek, Stanislas von | <person> German protozoologist, 1876-1915. See: Prowazekia, Prowazek bodies, Prowazek-Greeff bodies, Halberstaedter-Prowazek bodies. (05 Mar 2000) |
| Hofmann, Georg von | <person> Austrian bacteriologist, 1843-1890. See: Hofmann's bacillus. (05 Mar 2000) |
| Hofmeister, Franz von | <person> German surgeon, 1867-1926. See: Hofmeister's operation, Hofmeister-Polyaanastomosis. (05 Mar 2000) |
| Purkinje, Johannes von | <person> Bohemian anatomist and physiologist, 1787-1869. See: Purkinje conduction, Purkinje images, Purkinje shift, Purkinje system, Purkinje cell, Purkinje's corpuscle, Purkinje's fibres, Purkinje's figures, Purkinje's layer, Purkinje's network, Purkinje's phenomenon, Purkinje-Sanson images. (05 Mar 2000) |
| Hufner, Carl Gustav von | <person> German physician, 1840-1908. See: Hufner's equation. (05 Mar 2000) |
| Soemmerring, Samuel Thomas von | <person> German anatomist, 1755-1830. See: Soemmerring's ganglion, Soemmerring's ligament, Soemmerring's muscle, Soemmerring's spot, ring of Soemmerring. (05 Mar 2000) |
| Spee, Ferdinand Graf von | <person> German embryologist, 1855-1937. See: curve of Spee. (05 Mar 2000) |
| Stein, Stanislav von | <person> Russian otologist, *1855. See: Stein's test. (05 Mar 2000) |
| Stellwag, Carl von | <person> Austrian ophthalmologist, 1823-1904. See: Stellwag's sign. (05 Mar 2000) |
| Neumann, Isidor Edler von Heilwart | <person> Austrian dermatologist, 1832-1906. See: Neumann's disease. (05 Mar 2000) |
| Strumpel, Ernst Adolf von | <person> German physician, 1853-1925. See: Strumpell's disease, Strumpell's phenomenon, Strumpell's reflex, Fleischer-Strumpell ring, Strumpell-Marie disease, Marie-Strumpell disease, Strumpell-Westphal disease, Westphal-Strumpell pseudosclerosis. (05 Mar 2000) |
| Davidoff, M. von | <person> German histologist, +1904. See: Davidoff's cells. (05 Mar 2000) |
| Nussbaum, Johann von | <person> German surgeon, 1829-1890. See: Nussbaum's bracelet. (05 Mar 2000) |
| syndrome, von hippel-lindau | The cardinal features of von hippel-lindau (vhl) syndrome are benign blood-vessel tumours that most typically affect the eye and the brain. The eye tumours are termed angiomata and are in the retina. The brain tumours are termed haemangioblastoma and are in the cerebellum. Vhl is complex. There can also be blood-vessel tumours (haemangiomata) in the spinal cord, adrenal glands, liver, and lungs. Pheochromocytoma (a benign tumour of adrenal-like tissue) occurs in some patients. The combination of high blood pressure (hypertension) with angioma may cause bleeding under the skull (subarachnoid haemorrhage). Kidney tumours (like hypernephromas) may be malignant and metastasize. An abnormal elevation of red blood cells (polycythemia) can be due to the haemangioblastoma of the cerebellum or the hypernephroma. Multiple cysts can occur in the pancreas and kidneys. Patients with kidney problems or pancreatic cysts do not have pheochromocytoma, and visa versa. Lab findings in vhl may include high calcium (hypercalcaemia) and low potassium (hypokalaemia) occurring with the pheochromocytoma. Vhl is inherited as an autosomal dominant trait. The gene on one of the non-sex chromosomes is dominant over the normal gene with which it is paired so that one vhl gene is sufficient to cause the vhl syndrome. If a person has vhl, the chance for each of their children to receive the vhl gene is one-half (50%). The vhl gene has been mapped to chromosome 3 (the 3rd volume in the book of life) in region 3p26-p25. The vhl gene has the characteristics of a tumour-suppressor gene. The person with vhl inherits one inactive copy of the vhl gene (a germline mutation) from one of their parents. But the normal gene with which it is paired is still enough to suppress the formation of a tumour. Then, in one cell in the vhl patient's body, another mutation (a somatic mutation) occurs, inactivating the vhl gene. Thus, both copies of the vhl gene are inactivated and a tumour arises in the vhl patient. The syndrome is named for the german ophthalmologist eugen von hippel who described the charcteristic eye blood-vessel tumours in 1904 and the swedish pathologist arvid lindau who recognised the association between the eye tumours and the blood-vessel tumours of the cerebellum and other parts of the central nervous system in 1926-7. (12 Dec 1998) |