| TSS | Toxic Shock Syndrome |
|---|---|
| TSST-1 | Toxic Shock Syndrome Toxin-1 = Enterotoxin type F |
| CGS | cardiogenic shock; catgut suture |
| CS | calf serum; campomelic syndrome; carcinoid syndrome; cardiogenic shock; caries-susceptible; carotid ... |
| CST | cardiac stress test; cavernous sinus thrombosis; certified surgical technologist; chemostatin; Chris... |
| insulin shock | A severe condition that occurs when the level of blood glucose (sugar) drops quickly. The signs are shaking, sweating, dizziness, double vision, convulsions, and collapse. Insulin shock may occur when an insulin reaction is not treated quickly enough. See: hypoglycaemia. (09 Oct 1997) |
|---|---|
| insulin shock treatment | Formerly used treatment for serious mental disorders in which the patient was given insulin to induce a seizure; supplanted by electroshock therapy. (05 Mar 2000) |
| irreversible shock | Shock that has progressed beyond the stage when it will respond to transfusion or other form of treatment, and recovery is impossible. (05 Mar 2000) |
| oligaemic shock | Shock associated with pronounced fall in blood volume, sometimes resulting from increased permeability of blood vessels. (05 Mar 2000) |
| osmotic shock | Passage of solvent into a membrane bound structure due to osmosis, causing rupture of the membrane. A method of lysing cells or organelles. (18 Nov 1997) |
| electric shock | A sudden violent impression caused by the passage of a current of electricity through any portion of the body. (05 Mar 2000) |
| electrohydraulic shock wave lithotripsy | Destruction of calculi (urinary tract or other) by fragmentation using shock waves sent transcutaneously. (05 Mar 2000) |
| toxic shock | See toxic shock syndrome. (12 Dec 1998) |
| toxic shock syndrome | <syndrome> A blood-borne bacterial infection caused by the genus Staphylococcus. Usually effects menstruating females under the age of thirty and was associated in the past with the use of a particular type of tampon (no longer used). Common symptoms include: fever, chills, vomiting, sore throat, headache and decreased urine output. Progression of disease can result in lowering of blood pressure (shock) and renal failure. Treatment includes hospitalisation and intravenous antibiotics emergently. (27 Sep 1997) |
| endotoxin shock | Shock induced by release of endotoxin from Gram-negative bacteria, especially by Escherichia coli. (05 Mar 2000) |
| erethistic shock | Traumatic or toxic delirium following shock. Synonym: delirious shock. (05 Mar 2000) |
| extracorporeal shock wave lithotripsy | <procedure> This procedure uses sound waves delivered inside a water bath to pulverise kidney stones painlessly inside the body. (11 Nov 1997) |
| acetoacetyl-acyl carrier protein synthase | <enzyme> E coli enzyme, that catalyses condensation of malonyl-acyl carrier protein plus acetyl-acyl carrier protein; not inhibited by cerulenin Registry number: EC 2.3.1.- Synonym: acetoacetyl-acp synthase (26 Jun 1999) |
| acid soluble spore protein | <molecular biology> A DNA binding protein in the spores of some bacteria, thought to stabilise the DNA in an A configuration, so protecting it from cleavage by enzymes or UV light. (18 Nov 1997) |
| acute-phase protein | <haematology> These plasma proteins (in addition to fibrinogen) increase 25% or more in response to inflammation and injury are under direct control of interleukin-6 (IL-6) (hepatocyte-stimulating factor). Other proteins which increase are ceruloplasmin, C3 and C4 which increase 50% or more; alpha-1 acid glycoprotein, alpha-1 antitrypsin, haptoglobin and fibrinogen (the major determinant of viscosity 1 ) which increase two- to fourfold; C-reactive protein (CRP) and serum amyloid A which increase several hundred-fold. Despite long-held clinical opinion to the contrary, available data indicate that neither ESR nor measurement of specific acute-phase reactants are useful in excluding underlying infection or inflammation regardless of the pretest probability. These proteins are secreted into the blood in increased or decreased quantities by hepatocytes in response to trauma, inflammation, or disease. They can serve as inhibitors or mediators of the inflammatory processes. Certain acute-phase proteins have been used to diagnose and follow the course of diseases or as tumour markers. See also: amyloid, c-reactive protein, erythrocyte sedimentation rate, viscosity. (25 Jun 1999) |